التفاصيل البيبلوغرافية
| العنوان: |
Association of prenatal exposure to fine particulate matter pollution with childhood eczema. |
| المؤلفون: |
Yao, Tsung‐Chieh, Huang, Hsin‐Yi, Pan, Wen‐Chi, Wu, Chao‐Yi, Tsai, Shun‐Yu, Hung, Chi‐Yen, Lu, Kun‐Lin, Chang‐Chien, Ju, Tseng, Chih‐Lin, Wu, Chih‐Da, Chen, Yu‐Chen, Huang, Yvonne J., Tsai, Hui‐Ju |
| المصدر: |
Allergy; Jul2021, Vol. 76 Issue 7, p2241-2245, 5p |
| مصطلحات موضوعية: |
ECZEMA, PARTICULATE matter, TOBACCO smoke pollution, POLLUTION, PRENATAL exposure |
| مستخلص: |
Eczema was defined as having physician-diagnosed eczema and presence of eczema in the last 12 months through a modified International Study of Asthma and Allergies in Childhood (ISAAC) questionnaire provided by parents/guardians of the study participants.7 We applied a distributed lag nonlinear model to examine the exposure-lag-response association of eczema with mean weekly PM SB 2.5 sb estimates using a highly spatial-temporal resolution hybrid kriging/land-use regression model. This study has identified, for the first time, a sensitive window of exposure to PM SB 2.5 sb at gestational weeks 7 to 17 on the risk of developing childhood eczema, which may provide insight into underlying mechanisms. In gestational week 12, the concentration-response relationship indicated that the AOR of childhood eczema was significantly higher than 1.0 at PM SB 2.5 sb concentrations greater than 21.2 g/m SP 3 sp (Figure S3). Atopic eczema (or atopic dermatitis) is a chronic relapsing inflammatory skin disease affecting 15-30% of children worldwide.1 Although previous studies have attempted to link higher prenatal exposure to particulate matter with childhood eczema,2-6 most studies examined particulate matter with an aerodynamic diameter of 10 m or less (PM SB 10 sb ), but not particulate matter with an aerodynamic diameter of 2.5 m or less (PM SB 2.5 sb ), and yielded inconsistent results. [Extracted from the article] |
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| قاعدة البيانات: |
Complementary Index |
