دورية أكاديمية
Cyclic arginine-glycine-aspartic acid peptide inhibits macrophage infiltration of the kidney and carotid artery lesions in apo-E-deficient mice.
العنوان: | Cyclic arginine-glycine-aspartic acid peptide inhibits macrophage infiltration of the kidney and carotid artery lesions in apo-E-deficient mice. |
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المؤلفون: | Elitok, Saban, Brodsky, Sergey V., Patschan, Daniel, Orlova, Tatyana, Lerea, Kenneth M., Chander, Praveen, Goligorsky, Michael S. |
المصدر: | American Journal of Physiology: Renal Physiology; Jan2006, Vol. 290, pF159-F166, 8p, 16 Graphs |
مصطلحات موضوعية: | PEPTIDES, MACROPHAGES, KIDNEYS, APOLIPOPROTEIN E, AMINO acids, CAROTID artery |
مستخلص: | Interactions of leukocytes with the vascular endothelium culminating in their diapedesis represent not only a crucial event in immune surveillance and defense but are also critically involved in the pathogenesis of many inflammatory diseases, including atherosclerosis. Our previous in vitro studies using atomic force microscopy measurement of monocyte-endothelial cell interaction have demonstrated that a cyclic arginine-glycine-aspartic acid peptide (cRGD) inhibited their adhesion through very late antigen (α |
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قاعدة البيانات: | Complementary Index |