المؤلفون: Mezydlo, Aleksandra, Treiber, Nils, Kerschensteiner, Martin, Ullrich Gavilanes, Emily, Eichenseer, Katharina, Ancău, Mihai, Wens, Adinda, Ares Carral, Carla, Schifferer, Martina, Snaidero, Nicolas, Misgeld, Thomas

المصدر: Neuron 111(11), 1748-1759.e8 (2023). doi:10.1016/j.neuron.2023.03.031

مصطلحات موضوعية: Mammals, Multiple Sclerosis, pathology [Axons], General Neuroscience, oligodendrocytes, CLEM, oligodendrocyte precursor cell, Mice, myelin, remyelination, pathology [Oligodendroglia], pathology [Myelin Sheath], in vivo microscopy, Animals, ddc:610, demyelination

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::997f7b882b4cb803e4acdf7841faf1a4Test
https://doi.org/10.1016/j.neuron.2023.03.031Test

المؤلفون: Marina Herwerth, Selin Kenet, Martina Schifferer, Anne Winkler, Melanie Weber, Nicolas Snaidero, Mengzhe Wang, Melanie Lohrberg, Jeffrey L. Bennett, Christine Stadelmann, Bernhard Hemmer, Thomas Misgeld

المصدر: Brain 145(5), 1726-1742 (2022). doi:10.1093/brain/awac079

مصطلحات موضوعية: Aquaporin 4, metabolism [Astrocytes], pathology [Axons], Neuromyelitis Optica, aquaporin-4, neurodegeneration, neuromyelitis optica, astrocytopathy, Axons, neuroinflammation, Mice, nervous system, Astrocytes, Immunoglobulin G, Animals, Humans, metabolism [Neuromyelitis Optica], metabolism [Autoantibodies], metabolism [Immunoglobulin G], Neurology (clinical), ddc:610, Autoantibodies

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba1f0677eaa15885525cadb37c8d4f20Test
https://pub.dzne.de/record/164078Test

المؤلفون: Hannes Glaβ, Peter Heutink, Marcel Naumann, Arun Pal, Nicole Kreiter, Benedikt Kretner, René Günther, Masin Abo-Rady, Andreas Hermann, Ashutosh Dhingra, Tobias M. Böckers, Jared Sterneckert, Banaja P. Dash, Julia Japtok

المصدر: Life Science Alliance
Life science alliance 4(4), e202000764-(2021). doi:10.26508/lsa.202000764

مصطلحات موضوعية: 0301 basic medicine, metabolism [Cytoskeleton], Health, Toxicology and Mutagenesis, Apoptosis, Plant Science, metabolism [Axons], 0302 clinical medicine, C9orf72, Loss of Function Mutation, DNA Breaks, Double-Stranded, Axon, Amyotrophic lateral sclerosis, Induced pluripotent stem cell, Cells, Cultured, Cellular Senescence, Cytoskeleton, Research Articles, Motor Neurons, Ecology, pathology [Axons], Neurodegeneration, physiopathology [Amyotrophic Lateral Sclerosis], Cell biology, DNA-Binding Proteins, genetics [Amyotrophic Lateral Sclerosis], medicine.anatomical_structure, Gain of Function Mutation, metabolism [Organelles], Research Article, Programmed cell death, TARDBP protein, human, genetics [DNA-Binding Proteins], Biology, Biochemistry, Genetics and Molecular Biology (miscellaneous), 03 medical and health sciences, ddc:570, medicine, Humans, pathology [Amyotrophic Lateral Sclerosis], genetics [C9orf72 Protein], Loss function, Repetitive Sequences, Nucleic Acid, Phenocopy, Organelles, C9orf72 Protein, Amyotrophic Lateral Sclerosis, metabolism [Motor Neurons], FUS protein, human, medicine.disease, Axons, 030104 developmental biology, nervous system, Microscopy, Fluorescence, RNA-Binding Protein FUS, Energy Metabolism, genetics [RNA-Binding Protein FUS], 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1c8e9fe2a2303f8e4b32f90e68075bfTest
https://pubmed.ncbi.nlm.nih.gov/33619157Test

المؤلفون: Arun Pal, Peter Reinhardt, Jared Sterneckert, Alexander Storch, Patrick Neumann, Andreas Hermann, Hannes Glaß

المصدر: International Journal of Molecular Sciences, Vol 21, Iss 5, p 1797 (2020)
International Journal of Molecular Sciences
Volume 21
Issue 5
International journal of molecular sciences 21(5), 1797-(2020). doi:10.3390/ijms21051797

مصطلحات موضوعية: Vesicular Transport Proteins, metabolism [Axons], etiology [Motor Neuron Disease], pathology [Mitochondria], metabolism [Lysosomes], lcsh:Chemistry, metabolism [Vesicular Transport Proteins], pathology [Neurons], lcsh:QH301-705.5, Spectroscopy, Cells, Cultured, Chorea acanthocytosis, Neurons, pathology [Axons], Parkinsonism, pathology [Dendrites], human induced pluripotent stem cells (ipsc), General Medicine, Middle Aged, pathology [Induced Pluripotent Stem Cells], Computer Science Applications, Dendritic microtubule, metabolism [Motor Neuron Disease], metabolism [Induced Pluripotent Stem Cells], mitochondria, chorea acanthocytosis (chac), physiopathology [Neuroacanthocytosis], metabolism [Neurons], ddc:540, Female, medicine.symptom, Neuroacanthocytosis, Adult, Cell type, microfluidic chambers (mfcs), pathology [Motor Neuron Disease], organelle trafficking, Induced Pluripotent Stem Cells, motoneurons (mn), Models, Biological, Article, Catalysis, Inorganic Chemistry, lysosomes, genetics [Vesicular Transport Proteins], medicine, Humans, metabolism [Dendrites], Motor Neuron Disease, Physical and Theoretical Chemistry, Molecular Biology, business.industry, Organic Chemistry, Chorea, Dendrites, medicine.disease, metabolism [Mitochondria], Axons, Peripheral neuropathy, Dyskinesia, lcsh:Biology (General), lcsh:QD1-999, Mutation, Axoplasmic transport, business, Neuroscience

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cdf7ab5af61fb2ad4cf9e368c019d96bTest
https://www.mdpi.com/1422-0067/21/5/1797Test

المؤلفون: Stephan A. Müller, Maria Inês Cunha, Stefan F. Lichtenthaler, Anne Winkler, Bettina Schmid, Franziska van der Meer, Tjakko J. van Ham, Ioannis Alexopoulos, Mikael Simons, Minhui Su, Minou Djannatian, Christine Stadelmann, Martina Schifferer, Ludovico Cantuti-Castelvetri

المساهمون: Clinical Genetics

المصدر: Journal of Experimental Medicine, 217(5):e20191390. Rockefeller University Press
The Journal of Experimental Medicine
Journal of experimental medicine 217(5), e20191390 (2020). doi:10.1084/jem.20191390
Journal of Experimental Medicine

مصطلحات موضوعية: 0301 basic medicine, Proteome, metabolism [Myelin Sheath], drug effects [Oligodendroglia], metabolism [Microglia], metabolism [Lysophosphatidylcholines], drug effects [Microglia], pathology [Phagocytes], Myelin, Mice, 0302 clinical medicine, Phagosomes, Immunology and Allergy, Macrophage, drug effects [Axons], Zebrafish, Cells, Cultured, Myelin Sheath, Phagocytes, pathology [Axons], drug effects [Phagocytes], Microglia, biology, Chemistry, pharmacology [Tumor Necrosis Factor-alpha], metabolism [Myeloid Differentiation Factor 88], 3. Good health, Cell biology, Oligodendroglia, medicine.anatomical_structure, Spinal Cord, Larva, drug effects [Myelin Sheath], Immunology, genetics [Mutation], pathology [Spinal Cord], Article, metabolism [Oligodendroglia], 03 medical and health sciences, Inflammation resolution, drug effects [Phagosomes], pathology [Myelin Sheath], medicine, Animals, ddc:610, drug effects [Remyelination], Remyelination, pathology [Inflammation], Inflammation, Innate immune system, Tumor Necrosis Factor-alpha, Macrophages, drug effects [Larva], Lysophosphatidylcholines, metabolism [Proteome], biology.organism_classification, Spinal cord, Axons, Disease Models, Animal, 030104 developmental biology, pathology [Oligodendroglia], Mutation, Myeloid Differentiation Factor 88, metabolism [Phagosomes], pathology [Demyelinating Diseases], 030217 neurology & neurosurgery, Neuroscience, Demyelinating Diseases

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8afc169c9da206914a69fcfd5c9cbd3Test
https://pure.eur.nl/en/publications/92b7bc9e-deb4-4a4e-9a67-9b4171d74756Test

المؤلفون: Hilton, Brett J, Hilton, Brett Jason, Bradke, Frank

المصدر: Development 144(19), 3417-3429 (2017). doi:10.1242/dev.148312

مصطلحات موضوعية: Central Nervous System, 0301 basic medicine, Aging, physiopathology [Central Nervous System], growth & development [Mammals], Central nervous system, Biology, Models, Biological, 03 medical and health sciences, Myelin, pathology [Aging], ddc:570, Extracellular, medicine, Animals, Axon, Growth cone, Molecular Biology, Mammals, pathology [Axons], Regeneration (biology), Anatomy, Axons, Nerve Regeneration, 030104 developmental biology, medicine.anatomical_structure, nervous system, Axoplasmic transport, pathology [Central Nervous System], Neuroscience, Intracellular, Developmental Biology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67ca0f854e04e73a26866bd1aafb90b0Test
https://doi.org/10.1242/dev.148312Test

المؤلفون: Petra Huehnchen, Matthias Endres, Dieter Beule, Yangfan Peng, Andranik Ivanov, Christian Schinke, Valeria Fernandez Vallone, Peter Körtvelyessy, Luca Nolte, Harald Stachelscheid, Wolfgang Boehmerle

المصدر: Neurobiology of Disease, Vol 155, Iss, Pp 105391-(2021)
Neurobiology of disease 155, 105391 (2021). doi:10.1016/j.nbd.2021.105391

مصطلحات موضوعية: 0301 basic medicine, Vincristine, Sensory Receptor Cells, Cell Survival, drug effects [Cell Survival], physiology [Cell Survival], Replacement, Induced Pluripotent Stem Cells, Neurosciences. Biological psychiatry. Neuropsychiatry, Antineoplastic Agents, Lithium, Pharmacology, 3R, Time-Lapse Imaging, Neuroprotection, Cell Line, toxicity [Antineoplastic Agents], Induced pluripotent stem cell derived sensory neurons (iPSC-DSN), 03 medical and health sciences, 0302 clinical medicine, ddc:570, medicine, Humans, Doxorubicin, Viability assay, drug effects [Axons], Induced pluripotent stem cell, Cisplatin, pathology [Axons], Dose-Response Relationship, Drug, drug effects [Induced Pluripotent Stem Cells], pathology [Sensory Receptor Cells], Bortezomib, business.industry, methods [Time-Lapse Imaging], Neurotoxicity, pathology [Induced Pluripotent Stem Cells], medicine.disease, Axons, 030104 developmental biology, Neurology, Chemotherapy induced neuropathy, drug effects [Sensory Receptor Cells], Transcriptome, business, 030217 neurology & neurosurgery, RC321-571, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9571353875d9bd1977ab98d74656b62cTest
https://doi.org/10.1016/j.nbd.2021.105391Test

المؤلفون: Adrian Minh Schumacher, Martin Kerschensteiner, Paula Sánchez, Christoph Mahler, Thomas Misgeld, Philip R. Williams, Jan Philipp Bewersdorf, Oliver Griesbeck, Maarten E. Witte, Jonas Lehmitz, Ronald Naumann, Alexander Scheiter

المصدر: Neuron
Neuron 101(4), 615-624.e5 (2019). doi:10.1016/j.neuron.2018.12.023

مصطلحات موضوعية: 0301 basic medicine, Male, Multiple Sclerosis, experimental autoimmune encephalomyelitis, Excitotoxicity, chemistry.chemical_element, metabolism [Axons], Calcium, plasma membrane, medicine.disease_cause, Article, metabolism [Cell Membrane], 03 medical and health sciences, Mice, 0302 clinical medicine, Calcium imaging, in vivo microscopy, medicine, Humans, Animals, metabolism [Calcium], ddc:610, Rupture, Ion Transport, pathology [Axons], General Neuroscience, Multiple sclerosis, Endoplasmic reticulum, Experimental autoimmune encephalomyelitis, Cell Membrane, Glutamate receptor, medicine.disease, axon degeneration, pathology [Cell Membrane], Axons, ddc, Cell biology, calcium imaging, metabolism [Multiple Sclerosis], 030104 developmental biology, Membrane, chemistry, nervous system, Female, etiology [Multiple Sclerosis], 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d1a6fd34dfe311c366d7144618c73e9Test
https://hdl.handle.net/21.11116/0000-0003-68A5-E21.11116/0000-0003-68A7-C21.11116/0000-0003-68A8-B21.11116/0000-0003-68A9-ATest

المؤلفون: Sina Stern, Kevin C. Flynn, Michele Curcio, Walter Witke, Brett J. Hilton, Christine B. Gurniak, Frank Bradke, Barbara Schaffran, Molly J.E. Larson, Claudia J. Laskowski, Sebastian Dupraz, Andrea Tedeschi, Telma E. Santos

المصدر: Neuron 103(6), 1073-1085.e6 (2019). doi:10.1016/j.neuron.2019.07.007

مصطلحات موضوعية: 0301 basic medicine, Intravital Microscopy, Regulator, genetics [Cofilin 2], metabolism [Axons], metabolism [Spinal Cord Injuries], metabolism [Cofilin 2], genetics [Nerve Regeneration], Mice, 0302 clinical medicine, pathology [Neurons], Axon, Neurons, Microscopy, Confocal, pathology [Axons], genetics [Spinal Cord Injuries], General Neuroscience, Axon extension, Cofilin, Cell biology, metabolism [Cofilin 1], medicine.anatomical_structure, metabolism [Neurons], Actin depolymerizing factor, pathology [Spinal Cord Injuries], Cofilin 1, Cofilin 2, metabolism [Actins], metabolism [Growth Cones], Growth Cones, macromolecular substances, Biology, Time-Lapse Imaging, 03 medical and health sciences, medicine, Animals, ddc:610, Actin, Spinal Cord Injuries, Regeneration (biology), genetics [Cofilin 1], Embryogenesis, pathology [Growth Cones], Actins, Axons, Nerve Regeneration, Rats, 030104 developmental biology, Destrin, metabolism [Destrin], nervous system, genetics [Destrin], 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b07de3102c075dac51ac6dc9b6a076d7Test
https://pubmed.ncbi.nlm.nih.gov/31557454Test

المؤلفون: Jason R. Plemel, Aaron J. Moulson, Sohrab B. Manesh, Brett J. Hilton, Jie Liu, Peggy Assinck, Wolfram Tetzlaff, Greg J. Duncan

المصدر: Nature Communications
Nature Communications, Vol 9, Iss 1, Pp 1-16 (2018)
Duncan, G J, Manesh, S B, Hilton, B J, Assinck, P, Liu, J, Moulson, A, Plemel, J R & Tetzlaff, W 2018, ' Locomotor recovery following contusive spinal cord injury does not require oligodendrocyte remyelination ', Nature Communications, vol. 9, 3066 . https://doi.org/10.1038/s41467-018-05473-1Test
Nature Communications 9(1), 3066 (2018). doi:10.1038/s41467-018-05473-1

مصطلحات موضوعية: Male, 0301 basic medicine, Receptor, Platelet-Derived Growth Factor alpha, metabolism [Myelin Sheath], General Physics and Astronomy, pathology [Neural Stem Cells], metabolism [Axons], metabolism [Spinal Cord Injuries], myelin gene regulatory factor, mouse, Mice, Myelin, 0302 clinical medicine, Neural Stem Cells, Spinal Cord/metabolism, metabolism [Transcription Factors], lcsh:Science, Spinal cord injury, Myelin Sheath, Myelin Sheath/metabolism, Mice, Knockout, Multidisciplinary, pathology [Axons], Behavior, Animal, Cell Differentiation, genetics [Transcription Factors], Neural Stem Cells/pathology, Neural stem cell, Oligodendroglia, medicine.anatomical_structure, Spinal Cord, Female, ddc:500, pathology [Spinal Cord Injuries], metabolism [Spinal Cord], Science, Spinal Cord Injuries/metabolism, Transcription Factors/genetics, pathology [Spinal Cord], Axons/metabolism, Biology, Nerve Regeneration/physiology, Article, General Biochemistry, Genetics and Molecular Biology, metabolism [Oligodendroglia], 03 medical and health sciences, medicine, Animals, Remyelination, Spinal Cord Injuries, Cell Proliferation, Regeneration (biology), Myelin regulatory factor, General Chemistry, medicine.disease, Spinal cord, Axons, Oligodendrocyte, Nerve Regeneration, Disease Models, Animal, Oligodendroglia/metabolism, 030104 developmental biology, physiology [Remyelination], nervous system, Remyelination/physiology, physiology [Nerve Regeneration], pathology [Oligodendroglia], lcsh:Q, Neuroscience, Gene Deletion, 030217 neurology & neurosurgery, Transcription Factors

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5933f6aa6c3d7fc8203d8644e2007c29Test
https://doi.org/10.1038/s41467-018-05473-1Test