المؤلفون: Sofia Meyer zu Reckendorf, Diana Moser, Anna Blechschmidt, Venkata Neeha Joga, Daniela Sinske, Jutta Hegler, Stefanie Deininger, Alberto Catanese, Sabine Vettorazzi, Gregor Antoniadis, Tobias Boeckers, Bernd Knöll

المصدر: The journal of neuroscience 42(12), 2474-2491 (2022). doi:10.1523/JNEUROSCI.1305-21.2022
J Neurosci

مصطلحات موضوعية: genetics [Facial Nerve Injuries], Male, PTEN, metabolism [PTEN Phosphohydrolase], Mice, genetics [Nerve Regeneration], Animals, ddc:610, nerve regeneration, motoneuron, Research Articles, Facial Nerve Injuries, Motor Neurons, physiology [Axons], Hyperplasia, pathology [Hyperplasia], CREB, Akt, General Neuroscience, pathology [Facial Nerve Injuries], PTEN Phosphohydrolase, Hypertrophy, metabolism [Motor Neurons], pathology [Hypertrophy], Axons, Nerve Regeneration, Female, facial nerve, Tumor Suppressor Protein p53

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d7cc4a3aa84840bc1e960c5e0fb96f1Test
https://doi.org/10.1523/jneurosci.1305-21.2022Test

المؤلفون: Banaja P. Dash, Axel Freischmidt, Jochen H. Weishaupt, Andreas Hermann

المصدر: International Journal of Molecular Sciences; Volume 23; Issue 17; Pages: 9652
International journal of molecular sciences 23(17), 9652 (2022). doi:10.3390/ijms23179652 special issue: "Neurological Diseases: A Molecular Genetic Perspective"

مصطلحات موضوعية: TARDBP protein, human, amyotrophic lateral sclerosis (ALS), Gene Expression, motor neurons (MN), genetics [DNA-Binding Proteins], metabolism [Neurodegenerative Diseases], Catalysis, Inorganic Chemistry, Superoxide Dismutase-1, RNA sequencing (RNA-Seq), Humans, Physical and Theoretical Chemistry, differentially expressed genes (DEG), protein-protein interaction (PPI), Molecular Biology, Spectroscopy, Organic Chemistry, SOD1 protein, human, metabolism [Motor Neurons], General Medicine, genetics [Superoxide Dismutase-1], Computer Science Applications, metabolism [Induced Pluripotent Stem Cells], DNA-Binding Proteins, genetics [Amyotrophic Lateral Sclerosis], human induced pluripotent stem cells (iPSC), metabolism [RNA], ddc:540, Mutation, RNA, metabolism [DNA-Binding Proteins]

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::664975ccbe2bbcefa6d819315187f6aaTest

المؤلفون: Genin, Emmanuelle, C, Bannwarth, Sylvie, Ropert, Baptiste, Lespinasse, Françoise, Mauri-Crouzet, Alessandra, Augé, Gaelle, Fragaki, Konstantina, Cochaud, Charlotte, Donnarumma, Erminia, Lacas-Gervais, Sandra, Wai, Timothy, Paquis-Flucklinger, Véronique

المساهمون: Institut de Recherche sur le Cancer et le Vieillissement (IRCAN), Université Nice Sophia Antipolis (1965 - 2019) (UNS)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Université Côte d'Azur (UniCA), G5 Biologie mitochondriale – Mitochondrial biology, Institut Pasteur Paris (IP)-Centre National de la Recherche Scientifique (CNRS)-Université Paris Cité (UPCité), Centre Commun de Microscopie Appliquée (CCMA), Université de Nice Sophia-Antipolis (UNSA), This work was made possible by grants to V.P.-F. from the ANR (Agence Nationale de la Recherche) ANR-16-CE16-0024-01 and from the FRM (Fondation pour la Recherche Médicale) FRM: MND202004011475., We acknowledge Thibault Léger and the Proteomics facility of the Institut Jacques Monod, University of Paris Cité for mass spectrometry analysis. We also thank Christelle Boscagli for technical help. We gratefully acknowledge the IRCAN’s Molecular and Cellular Core Imaging (PICMI) facility, supported financially by FEDER, Conseil régional Provence Alpes-Côte d’Azur, Conseil Départemental 06, Cancéropôle PACA, Gis Ibisa and Inserm, the IRCAN’s Animal core facility, supported by FEDER, Région Provence Alpes-Côte d’Azur, Conseil Départemental 06 and Inserm, the IRCAN’s Histology core facility, supported by Région Provence Alpes-Côte d’Azur, Cancéropôle PACA and Université Côte d’Azur and, the University’s CCMA Electron Microscopy facility supported by Université Côte d’Azur, Région Provence Alpes-Côte d’Azur, Conseil Départemental 06 and Gis Ibisa, ANR-16-CE16-0024,MicroGol,Implication des fonctions sécrétoires de l'appareil de Golgi dans le développement des microcéphalies postnatales avec déficit intellectuel(2016)

المصدر: ISSN: 0006-8950.

مصطلحات موضوعية: amyotrophic lateral sclerosis, frontotemporal dementia, motor neuron disease, mitochondrion, ALS = amyotrophic lateral sclerosis, FTD = frontotemporal dementia, IMM = inner mitochondrial membrane, MICOS = mitochondrial contact site and cristae organizing system, MN = motor neuron, PHB = prohibitin, PLA = proximity ligation assay, RC = respiratory chain, MESH: Amyotrophic Lateral Sclerosis* / genetics Amyotrophic Lateral Sclerosis* / metabolism Animals Frontotemporal Dementia* / genetics Humans Membrane Proteins* / genetics Membrane Proteins* / metabolism Mice Mitochondria / metabolism Mitochondrial Proteins* / genetics Mitochondrial Proteins* / metabolism Motor Neurons / metabolism Prohibitins Transcription Factors / genetics, [SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologie

العلاقة: info:eu-repo/semantics/altIdentifier/pmid/35656794; pasteur-03853884; https://pasteur.hal.science/pasteur-03853884Test; PUBMED: 35656794

الإتاحة: https://doi.org/10.1093/brain/awac197Test
https://pasteur.hal.science/pasteur-03853884Test

المؤلفون: Scekic-Zahirovic, Jelena, Sanjuan-Ruiz, Inmaculada, Kan, Vanessa, Megat, Salim, De Rossi, Pierre, Dieterlé, Stéphane, Cassel, Raphaelle, Jamet, Marguerite, Kessler, Pascal, Wiesner, Diana, Tzeplaeff, Laura, Demais, Valérie, Sahadevan, Sonu, Hembach, Katharina M, Muller, Hans-Peter, Picchiarelli, Gina, Mishra, Nibha, Antonucci, Stefano, Dirrig-Grosch, Sylvie, Kassubek, Jan, Rasche, Volker, Ludolph, Albert, Boutillier, Anne-Laurence, Roselli, Francesco, Polymenidou, Magdalini, Lagier-Tourenne, Clotilde, Liebscher, Sabine, Dupuis, Luc

المساهمون: Mécanismes Centraux et Périphériques de la Neurodégénérescence, Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM), Ludwig-Maximilians University [Munich] (LMU), Universität Zürich [Zürich] = University of Zurich (UZH), Laboratoire de neurosciences cognitives et adaptatives (LNCA), Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS), CRBS - Centre de Recherche en Biomédecine de Strasbourg (Inserm UMS38/UNISTRA), University of Ulm (UUlm), Deutsches Zentrum für Neurodegenerative Erkrankungen [Ulm] (DZNE), German Research Center for Neurodegenerative Diseases - Deutsches Zentrum für Neurodegenerative Erkrankungen (DZNE), Centre de Neurochimie, Harvard Medical School [Boston] (HMS), Harvard University [Cambridge], Munich Cluster for systems neurology [Munich] (SyNergy), Technische Universität Munchen - Université Technique de Munich [Munich, Allemagne] (TUM)-Ludwig-Maximilians-Universität München (LMU), Université de Strasbourg Inserm Faculté de médecine, Klinikum der Universitat Munchen, Ludwig-Maximilians-Universität München (LMU), University hospital of Zurich [Zurich], Universität Ulm - Ulm University [Ulm, Allemagne], Massachusetts Institute of Technology (MIT), Ludwig-Maximilians University Hospital (LMU Munich), ANR-16-CE92-0031,EpiFUS,Rôle de FUS dans la régulation des modifications épigénétiques : conséquences pour la sclérose latérale amyotrophique et la démence frontotemporale(2016), ANR-16-CE16-0015,ToFU,Interactions entre TAU, FUS et TDP-43 dans les maladies neurodégénératives(2016), ANR-19-CE17-0016,SPREADALS,Propagation et toxicité des facteurs pathogéniques de la sclérose latérale amyotrophique(2019), Technische Universität München [München] (TUM)-Ludwig-Maximilians-Universität München (LMU), Anne Laurence, Boutillier, Rôle de FUS dans la régulation des modifications épigénétiques : conséquences pour la sclérose latérale amyotrophique et la démence frontotemporale - - EpiFUS2016 - ANR-16-CE92-0031 - AAPG2016 - VALID, Interactions entre TAU, FUS et TDP-43 dans les maladies neurodégénératives - - ToFU2016 - ANR-16-CE16-0015 - AAPG2016 - VALID, Propagation et toxicité des facteurs pathogéniques de la sclérose latérale amyotrophique - - SPREADALS2019 - ANR-19-CE17-0016 - AAPG2019 - VALID, University of Zurich, Liebscher, Sabine, Dupuis, Luc, Centre de Recherche en Biomédecine de Strasbourg (CRBS), Dieterle, Stéphane

المصدر: Nature Communications
Nature Communications, Nature Publishing Group, 2021, 12 (1), pp.3028. ⟨10.1038/s41467-021-23187-9⟩
Nature Communications, Vol 12, Iss 1, Pp 1-19 (2021)
Nature Communications 12(1), 3028 (2021). doi:10.1038/s41467-021-23187-9
Nature Communications, Nature Publishing Group, 2021, 12 (1), ⟨10.1038/s41467-021-23187-9⟩
Nature Communications, 2021, 12 (1), ⟨10.1038/s41467-021-23187-9⟩
Nature Communications, 2021, 12 (1), pp.3028. ⟨10.1038/s41467-021-23187-9⟩

مصطلحات موضوعية: Male, Cytoplasm, [SDV]Life Sciences [q-bio], General Physics and Astronomy, Gene Expression, Synaptic Transmission, MESH: Synapses, Mice, MESH: Animals, Gene Knock-In Techniques, MESH: Amyotrophic Lateral Sclerosis, MESH: Gene Knock-In Techniques, metabolism [RNA-Binding Protein FUS], Motor Neurons, MESH: RNA-Binding Protein FUS, FUS protein, mouse, 3100 General Physics and Astronomy, [SDV] Life Sciences [q-bio], genetics [Amyotrophic Lateral Sclerosis], Phenotype, [SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC], Female, ddc:500, MESH: Motor Neurons, MESH: Mutation, MESH: Gene Expression, Science, 610 Medicine & health, 1600 General Chemistry, Genetics and Molecular Biology, MESH: Phenotype, Article, 1300 General Biochemistry, Genetics and Molecular Biology, MESH: Mice, Inbred C57BL, MESH: Synaptic Transmission, Animals, [SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC], MESH: Mice, metabolism [Cytoplasm], metabolism [Amyotrophic Lateral Sclerosis], MESH: Cytoplasm, Amyotrophic Lateral Sclerosis, General Chemistry, metabolism [Motor Neurons], metabolism [Synapses], MESH: Male, Mice, Inbred C57BL, physiology [Synaptic Transmission], General Biochemistry, Mutation, Synapses, RNA-Binding Protein FUS, 11493 Department of Quantitative Biomedicine, genetics [RNA-Binding Protein FUS], MESH: Female

وصف الملف: application/pdf; 2021_NatureCummun.pdf - application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::c8536f94d17455134cebd44e2729e13bTest
https://www.hal.inserm.fr/inserm-03376335Test

المؤلفون: Bernd Knöll, Renate Lüllmann-Rauch, Paul Saftig, Christian Haass, Yasuo Uchiyama, Wolfgang Wurst, Renate Wanner, Anja Capell, Rudi D'Hooge, Georg Werner, Stijn Stroobants, Patrick Lüningschrör, Benedikt Wefers, Daniela Sinske, Soichiro Kakuta, Michael Sendtner, Benjamin Dombert, Markus Damme

المصدر: Cell Reports, Vol 30, Iss 10, Pp 3506-3519.e6 (2020)
Cell reports 30(10), 3506-3519.e6 (2020). doi:10.1016/j.celrep.2020.02.060

مصطلحات موضوعية: 0301 basic medicine, MODIFIER, Vacuole, pathology [Facial Nerve], VARIANTS, Axonal Transport, metabolism [Lysosomes], 0302 clinical medicine, innervation [Muscles], Risk Factors, Axon, lcsh:QH301-705.5, genetics [Nerve Tissue Proteins], Mice, Knockout, Motor Neurons, DEMENTIA, Muscles, ultrastructure [Autophagosomes], ultrastructure [Axon Initial Segment], Frontotemporal lobar degeneration, metabolism [Autophagosomes], Cell biology, genetics [Membrane Proteins], Facial Nerve, medicine.anatomical_structure, genetics [Frontotemporal Lobar Degeneration], Life Sciences & Biomedicine, PROTEINS, Nerve Tissue Proteins, ultrastructure [Motor Neurons], Biology, General Biochemistry, Genetics and Molecular Biology, MECHANISMS, Lipofuscin, 03 medical and health sciences, pathology [Brain Stem], MOTILITY, Lysosome, Organelle, medicine, Animals, Genetic Predisposition to Disease, ddc:610, metabolism [Cell Nucleus], PROGRANULIN, deficiency [Membrane Proteins], Axon Initial Segment, Cell Nucleus, FRONTOTEMPORAL LOBAR DEGENERATION, Science & Technology, deficiency [Nerve Tissue Proteins], MUTATIONS, Autophagosomes, metabolism [Axon Initial Segment], Membrane Proteins, ultrastructure [Lysosomes], metabolism [Motor Neurons], Cell Biology, medicine.disease, Axon initial segment, Mice, Inbred C57BL, 030104 developmental biology, nervous system, lcsh:Biology (General), Axoplasmic transport, Frontotemporal Lobar Degeneration, Lysosomes, 030217 neurology & neurosurgery, Brain Stem

وصف الملف: Print

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5eaa4dcd1581611e9ae6d198c56b849fTest
http://www.sciencedirect.com/science/article/pii/S221112472030228XTest

المؤلفون: Florian Wegner, Selma Staege, Andreas Hermann, Stefan Stefanov, Hannes Glass, Susanne Petri, Dieter Edbauer, Rajat Bhatnagar, Antje Janosch, Petra Freitag, Anne-Karin Kahlert, Marc Bickle, Jared Sterneckert, Tanja Richter, Arun Pal, Carina Schludi, Walter Just, Norman Kalmbach, Masin Abo-Rady

المصدر: Stem Cell Reports, Vol 14, Iss 3, Pp 390-405 (2020)
Stem Cell Reports
Stem cell reports 14(3), 390-405 (2020). doi:10.1016/j.stemcr.2020.01.010

مصطلحات موضوعية: 0301 basic medicine, amyotrophic lateral sclerosis, genetics [Transcriptome], Apoptosis, metabolism [Axons], Biochemistry, genetics [Gain of Function Mutation], Pathogenesis, metabolism [HSP40 Heat-Shock Proteins], Gene Knockout Techniques, 0302 clinical medicine, C9orf72, disease modeling, Axon, Amyotrophic lateral sclerosis, drug effects [Axons], drug effects [Motor Neurons], Induced pluripotent stem cell, lcsh:QH301-705.5, HSP70, pharmacology [Pyrrolidinones], Motor Neurons, lcsh:R5-920, DNA Repeat Expansion, drug effects [Induced Pluripotent Stem Cells], gene editing, drug effects [Cell Differentiation], pathology [Nerve Degeneration], Cell Differentiation, Pyrrolidinones, Cell biology, metabolism [Induced Pluripotent Stem Cells], genetics [Amyotrophic Lateral Sclerosis], medicine.anatomical_structure, metabolism [C9orf72 Protein], Gain of Function Mutation, lcsh:Medicine (General), pathology [Motor Neurons], induced pluripotent stem cells, metabolism [HSP70 Heat-Shock Proteins], genetics [DNA Repeat Expansion], C9ORF72, axonal trafficking, drug effects [Apoptosis], Biology, Cytoplasmic Granules, Models, Biological, Article, 03 medical and health sciences, Heat shock protein, Genetics, medicine, Humans, pharmacology [Benzhydryl Compounds], HSP70 Heat-Shock Proteins, ddc:610, Benzhydryl Compounds, pathology [Amyotrophic Lateral Sclerosis], genetics [C9orf72 Protein], C9orf72 Protein, metabolism [Motor Neurons], Cell Biology, HSP40 Heat-Shock Proteins, medicine.disease, metabolism [Cytoplasmic Granules], Axons, Hsp70, 030104 developmental biology, drug effects [Cytoplasmic Granules], HSP40, lcsh:Biology (General), heat shock proteins, Nerve Degeneration, Transcriptome, Trinucleotide repeat expansion, 030217 neurology & neurosurgery, Developmental Biology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e36d802ed14d96d26a9814e300c2698Test
http://www.sciencedirect.com/science/article/pii/S221367112030031XTest

المؤلفون: Emmanuelle C Genin, Sylvie Bannwarth, Baptiste Ropert, Françoise Lespinasse, Alessandra Mauri-Crouzet, Gaelle Augé, Konstantina Fragaki, Charlotte Cochaud, Erminia Donnarumma, Sandra Lacas-Gervais, Timothy Wai, Véronique Paquis-Flucklinger

المساهمون: Institut de Recherche sur le Cancer et le Vieillissement (IRCAN), Université Nice Sophia Antipolis (1965 - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Université Côte d'Azur (UCA), Biologie mitochondriale – Mitochondrial biology, Institut Pasteur [Paris] (IP)-Centre National de la Recherche Scientifique (CNRS)-Université Paris Cité (UPCité), Centre Commun de Microscopie Appliquée (CCMA), Université de Nice Sophia-Antipolis (UNSA), This work was made possible by grants to V.P.-F. from the ANR (Agence Nationale de la Recherche) ANR-16-CE16-0024-01 and from the FRM (Fondation pour la Recherche Médicale) FRM: MND202004011475., We acknowledge Thibault Léger and the Proteomics facility of the Institut Jacques Monod, University of Paris Cité for mass spectrometry analysis. We also thank Christelle Boscagli for technical help. We gratefully acknowledge the IRCAN’s Molecular and Cellular Core Imaging (PICMI) facility, supported financially by FEDER, Conseil régional Provence Alpes-Côte d’Azur, Conseil Départemental 06, Cancéropôle PACA, Gis Ibisa and Inserm, the IRCAN’s Animal core facility, supported by FEDER, Région Provence Alpes-Côte d’Azur, Conseil Départemental 06 and Inserm, the IRCAN’s Histology core facility, supported by Région Provence Alpes-Côte d’Azur, Cancéropôle PACA and Université Côte d’Azur and, the University’s CCMA Electron Microscopy facility supported by Université Côte d’Azur, Région Provence Alpes-Côte d’Azur, Conseil Départemental 06 and Gis Ibisa, ANR-16-CE16-0024,MicroGol,Implication des fonctions sécrétoires de l'appareil de Golgi dans le développement des microcéphalies postnatales avec déficit intellectuel(2016), Lemesle, Marie, Implication des fonctions sécrétoires de l'appareil de Golgi dans le développement des microcéphalies postnatales avec déficit intellectuel - - MicroGol2016 - ANR-16-CE16-0024 - AAPG2016 - VALID

المصدر: Brain-A Journal of Neurology
Brain-A Journal of Neurology, 2022, 145 (10), pp.3415-3430. ⟨10.1093/brain/awac197⟩

مصطلحات موضوعية: Motor Neurons, PHB = prohibitin, Amyotrophic Lateral Sclerosis, Membrane Proteins, PLA = proximity ligation assay, MICOS = mitochondrial contact site and cristae organizing system, MESH: Amyotrophic Lateral Sclerosis* / genetics Amyotrophic Lateral Sclerosis* / metabolism Animals Frontotemporal Dementia* / genetics Humans Membrane Proteins* / genetics Membrane Proteins* / metabolism Mice Mitochondria / metabolism Mitochondrial Proteins* / genetics Mitochondrial Proteins* / metabolism Motor Neurons / metabolism Prohibitins Transcription Factors / genetics, IMM = inner mitochondrial membrane, Mitochondria, Mitochondrial Proteins, Mice, ALS = amyotrophic lateral sclerosis, [SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologie, Frontotemporal Dementia, Prohibitins, motor neuron disease, mitochondrion, Animals, Humans, FTD = frontotemporal dementia, [SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologie, Neurology (clinical), RC = respiratory chain, MN = motor neuron, Transcription Factors

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43e4acee00dfd65b95c24c9f606e3ce2Test
https://pubmed.ncbi.nlm.nih.gov/35656794Test

المؤلفون: Seifert, Anne, Drechsler, Hauke, Japtok, Julia, Korten, Till, Diez, Stefan, Hermann, Andreas

المصدر: International Journal of Molecular Sciences, Vol 22, Iss 2422, p 2422 (2021)
International journal of molecular sciences 22(5), 2422-(2021). doi:10.3390/ijms22052422
International Journal of Molecular Sciences
Volume 22
Issue 5

مصطلحات موضوعية: metabolism [Microtubules], Kinesins, gliding motility assays, Microtubules, Axonal Transport, Article, molecular motors, Cell Line, lcsh:Chemistry, Humans, motor neurons, lcsh:QH301-705.5, metabolism [RNA-Binding Protein FUS], metabolism [Amyotrophic Lateral Sclerosis], Amyotrophic Lateral Sclerosis, physiopathology [Amyotrophic Lateral Sclerosis], Kinesin, metabolism [Motor Neurons], FUS protein, human, KIF5B protein, human, genetics [Amyotrophic Lateral Sclerosis], axonopathy, lcsh:Biology (General), lcsh:QD1-999, ddc:540, Mutation, RNA-Binding Protein FUS, axonal transport, genetics [RNA-Binding Protein FUS], physiology [Motor Neurons]

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::ef91019ea3d39d68003d57630d8e5536Test
https://www.mdpi.com/1422-0067/22/5/2422Test

المؤلفون: Markus H. Schwab, Tatjana Kleele, Stefan Engelhardt, Yan Xiao, Thomas Misgeld, Tim Czopka, Diane L. Sherman, Monika S. Brill, Gabriela Plucińska, Matthias Kneussel, Peter J. Brophy, Mengzhe Wang, Petros Avramopoulos

المصدر: The journal of cell biology 220(4), e201911114 (2021). doi:10.1083/jcb.201911114
Wang, M, Kleele, T, Xiao, Y, Plucinska, G, Avramopoulos, P, Engelhardt, S, Schwab, M H, Kneussel, M, Czopka, T, Sherman, D, Brophy, P J, Misgeld, T & Brill, M S 2021, ' Completion of neuronal remodeling prompts myelination along developing motor axon branches ', Journal of Cell Biology . https://doi.org/10.1083/jcb.201911114Test
The Journal of Cell Biology

مصطلحات موضوعية: animal structures, Delayed myelination, metabolism [Myelin Sheath], viruses, Neuromuscular Junction, Mice, Transgenic, Development, Neurotransmission, Biology, Synaptic Transmission, Article, 03 medical and health sciences, Mice, 0302 clinical medicine, ddc:570, medicine, Animals, Spotlight, Axon, Cytoskeleton, Myelin Sheath, 030304 developmental biology, Motor Neurons, 0303 health sciences, Trafficking, Neuronal Plasticity, Mechanism (biology), Cell Biology, metabolism [Motor Neurons], biochemical phenomena, metabolism, and nutrition, Axons, medicine.anatomical_structure, nervous system, embryonic structures, Neuronal remodeling, Axoplasmic transport, Neuregulin, Neuroscience, 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ecb1d60a36096529a25017c77f4762a1Test

المؤلفون: Óscar López-Pérez, Inês Baldeiras, Isabel Santana, Mònica Povedano, Sergio Martínez-Yélamos, Inga Zerr, Carla Marco, Enrique Santamaría, Isidro Ferrer, Franc Llorens, Pol Andrés-Benito, Joaquín Fernández-Irigoyen, Maria J. Colomina, Raúl Domínguez

المساهمون: Universidad Pública de Navarra. Departamento de Ciencias de la Salud, Nafarroako Unibertsitate Publikoa. Osasun Zientziak Saila

المصدر: International Journal of Molecular Sciences
Volume 21
Issue 22
Dipòsit Digital de la UB
Universidad de Barcelona
Academica-e. Repositorio Institucional de la Universidad Pública de Navarra
instname
International Journal of Molecular Sciences, Vol 21, Iss 8680, p 8680 (2020)
Academica-e: Repositorio Institucional de la Universidad Pública de Navarra
Universidad Pública de Navarra
International journal of molecular sciences 21(22), 8680 (2020). doi:10.3390/ijms21228680

مصطلحات موضوعية: Male, Proteomics, Pathology, cerebrospinal fluid [Chemokine CXCL12], amyotrophic lateral sclerosis, cerebrospinal fluid [Frontotemporal Dementia], lcsh:Chemistry, 0302 clinical medicine, Cerebrospinal fluid, Amyotrophic lateral sclerosis, lcsh:QH301-705.5, Spectroscopy, Aged, 80 and over, Motor Neurons, 0303 health sciences, Biochemical markers, General Medicine, Middle Aged, CXCL12, AAAS, 3. Good health, Computer Science Applications, cerebrospinal fluid [Alzheimer Disease], medicine.anatomical_structure, cerebrospinal fluid [Biomarkers], Frontotemporal Dementia, ddc:540, Marcadors bioquímics, Biomarker (medicine), cerebrospinal fluid [Amyotrophic Lateral Sclerosis], Female, Alzheimer's disease, Neuroglia, Frontotemporal dementia, medicine.medical_specialty, Receptors, CXCR4, Proteòmica, Catalysis, Article, cerebrospinal fluid, Inorganic Chemistry, S1006A, 03 medical and health sciences, proteomics, metabolism [Receptors, CXCR], Alzheimer Disease, parasitic diseases, medicine, Humans, Physical and Theoretical Chemistry, Molecular Biology, metabolism [Neuroglia], 030304 developmental biology, Aged, Receptors, CXCR, CXCR4, Pyramidal tracts, business.industry, Multiple sclerosis, Organic Chemistry, biomarkers, metabolism [Motor Neurons], Spinal cord, medicine.disease, CXCR7, Chemokine CXCL12, lcsh:Biology (General), lcsh:QD1-999, metabolism [Receptors, CXCR4], business, 030217 neurology & neurosurgery, Biomarkers, Esclerosi lateral amiotròfica

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b368c8ac31be532d678237b86fa1cc2Test