نتائج البحث - "metabolism [Cytoskeleton]"

1

Biomolecular condensation involving the cytoskeleton

المؤلفون: Satabdee Mohapatra, Susanne Wegmann

المصدر: Brain research bulletin 194, 105-117 (2023). doi:10.1016/j.brainresbull.2023.01.009

مصطلحات موضوعية: metabolism [Cytoskeleton], metabolism [Actins], Condensation, General Neuroscience, metabolism [Microtubules], Microtubules, Liquid-liquid phase separation, Cytoskeletal Proteins, ddc:150, Intermediate filaments, metabolism [Actin Cytoskeleton], metabolism [Cytoplasm], Actin, Cytoskeleton

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::693e19e69139959b760f3b5e1ce45ba7Test
https://pub.dzne.de/record/247519Test

2

Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s disease

المؤلفون: Berta Puig, Jakob Matschke, Mohsin Shafiq, Neelam Younas, Markus Glatzel, Matthias Schmitz, Aneeqa Noor, Isidre Ferrer, Hermann C. Altmeppen, Saima Zafar, Inga Zerr

المصدر: Molecular Neurodegeneration, Vol 16, Iss 1, Pp 1-14 (2021)
Dipòsit Digital de la UB
Universidad de Barcelona
Molecular Neurodegeneration
Molecular neurodegeneration 16(1), 11 (2021). doi:10.1186/s13024-021-00422-x

مصطلحات موضوعية: 0301 basic medicine, Co-immunoprecipitation, metabolism [Cytoskeleton], Rapidly progressive Alzheimer’s disease, Disease, lcsh:Geriatrics, lcsh:RC346-429, Prion protein oligomers, 0302 clinical medicine, Tubulin, Proteïnes citosquelètiques, Cytoskeleton, Neurons, biology, Chemistry, Growth arrest specific 2 like 2, Brain, Alzheimer's disease, Cell biology, metabolism [Neurons], GAS, Disease Progression, metabolism [Alzheimer Disease], Research Article, Immunoprecipitation, metabolism [Amyloid beta-Peptides], G2L2, Prion Proteins, 03 medical and health sciences, Cellular and Molecular Neuroscience, metabolism [Prion Proteins], Alzheimer Disease, ddc:570, Humans, Prion protein, Molecular Biology, Actin, lcsh:Neurology. Diseases of the nervous system, Amyloid beta-Peptides, Binding protein, PrPC, Molecular medicine, rpAD, Growth arrest specific proteins, Cytoskeletal proteins, lcsh:RC952-954.6, 030104 developmental biology, Malaltia d'Alzheimer, metabolism [Brain], biology.protein, Neurology (clinical), pathology [Cytoskeleton], 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8bb5ec7f75f2f6ab829e32422cc6b2eTest
https://doaj.org/article/7c71674b4d0c4550b9d3d91ece6ae9d5Test

3

Molecular underpinnings of cytoskeletal cross-talk

المؤلفون: Erwin Frey, Willi L. Stepp, Angela Oberhofer, Dennis Zimmermann, Zeynep Ökten, Peter Spieler, Emanuel Reithmann, Bettina Schmid

المصدر: Proceedings of the National Academy of Sciences of the United States of America 117(8), 3944-3952 (2020). doi:10.1073/pnas.1917964117
Proc Natl Acad Sci U S A

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::62869088493e03f31f8022a2d3471ef8Test
https://doi.org/10.1073/pnas.1917964117Test

4

Cytoskeleton dynamics in axon regeneration

المؤلفون: Oriane Blanquie, Frank Bradke

المصدر: Current opinion in neurobiology 51, 60-69 (2018). doi:10.1016/j.conb.2018.02.024

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53c351a41b31fc51f223a07ee55882d8Test
https://doi.org/10.1016/j.conb.2018.02.024Test

5

Concomitant gain and loss of function pathomechanisms in C9ORF72 amyotrophic lateral sclerosis

المؤلفون: Hannes Glaβ, Peter Heutink, Marcel Naumann, Arun Pal, Nicole Kreiter, Benedikt Kretner, René Günther, Masin Abo-Rady, Andreas Hermann, Ashutosh Dhingra, Tobias M. Böckers, Jared Sterneckert, Banaja P. Dash, Julia Japtok

المصدر: Life Science Alliance
Life science alliance 4(4), e202000764-(2021). doi:10.26508/lsa.202000764

مصطلحات موضوعية: 0301 basic medicine, metabolism [Cytoskeleton], Health, Toxicology and Mutagenesis, Apoptosis, Plant Science, metabolism [Axons], 0302 clinical medicine, C9orf72, Loss of Function Mutation, DNA Breaks, Double-Stranded, Axon, Amyotrophic lateral sclerosis, Induced pluripotent stem cell, Cells, Cultured, Cellular Senescence, Cytoskeleton, Research Articles, Motor Neurons, Ecology, pathology [Axons], Neurodegeneration, physiopathology [Amyotrophic Lateral Sclerosis], Cell biology, DNA-Binding Proteins, genetics [Amyotrophic Lateral Sclerosis], medicine.anatomical_structure, Gain of Function Mutation, metabolism [Organelles], Research Article, Programmed cell death, TARDBP protein, human, genetics [DNA-Binding Proteins], Biology, Biochemistry, Genetics and Molecular Biology (miscellaneous), 03 medical and health sciences, ddc:570, medicine, Humans, pathology [Amyotrophic Lateral Sclerosis], genetics [C9orf72 Protein], Loss function, Repetitive Sequences, Nucleic Acid, Phenocopy, Organelles, C9orf72 Protein, Amyotrophic Lateral Sclerosis, metabolism [Motor Neurons], FUS protein, human, medicine.disease, Axons, 030104 developmental biology, nervous system, Microscopy, Fluorescence, RNA-Binding Protein FUS, Energy Metabolism, genetics [RNA-Binding Protein FUS], 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1c8e9fe2a2303f8e4b32f90e68075bfTest
https://pubmed.ncbi.nlm.nih.gov/33619157Test

6

Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes

المؤلفون: Michael T. Heneka, Susanne Schmidt, Luc Bousset, Lena Wischhof, Alexandru Odainic, Daniele Bano, Stephanie Schwartz, Cira Dansokho, Jasper Spitzer, Ronald Melki, Hannah Scheiblich, Angelika Griep, Dilek Mercan, Eicke Latz, Frederik Eikens

المساهمون: Universität Bonn = University of Bonn, Dendrite Differenciation Group [DZNE - Bonn], German Research Center for Neurodegenerative Diseases - Deutsches Zentrum für Neurodegenerative Erkrankungen (DZNE), Institute of Innate Immunity, Bonn, Centre National de la Recherche Scientifique (CNRS), Service MIRCEN (MIRCEN), Université Paris-Saclay-Centre National de la Recherche Scientifique (CNRS)-Institut de Biologie François JACOB (JACOB), Direction de Recherche Fondamentale (CEA) (DRF (CEA)), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Direction de Recherche Fondamentale (CEA) (DRF (CEA)), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA), University of Massachusetts Medical School [Worcester] (UMASS), University of Massachusetts System (UMASS), European Project: 821522,PD-MitoQUANT - H2020-EU.3.1.7. - Innovative Medicines Initiative 2 (IMI2), European Project: (grant No 116060),IMPRiND, University of Bonn, Université Paris-Saclay-Institut de Biologie François JACOB (JACOB)

المصدر: Cell 184(20), 5089-5106.e21 (2021). doi:10.1016/j.cell.2021.09.007
Cell
Cell, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩
Cell, Elsevier, 2021, 184, pp.5089-5106. ⟨10.1016/j.cell.2021.09.007⟩

مصطلحات موضوعية: Male, metabolism [Cytoskeleton], alpha-synuclein, animal diseases, metabolism [Leucine-Rich Repeat Serine-Threonine Protein Kinase-2], genetics [Transcriptome], microglia, Apoptosis, metabolism [Microglia], environment and public health, tunneling nanotubes, chemistry.chemical_compound, 0302 clinical medicine, metabolism [Reactive Oxygen Species], heterocyclic compounds, metabolism [alpha-Synuclein], Cytoskeleton, degradation, Aged, 80 and over, 0303 health sciences, Nanotubes, Microglia, Pattern recognition receptor, pathology [Microglia], LRRK2, metabolism [Cell Membrane Structures], Mitochondria, 3. Good health, Cell biology, ultrastructure [Microglia], medicine.anatomical_structure, alpha-Synuclein, [SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC], Female, Intracellular, Cell signaling, metabolism [Actins], Down-Regulation, clearance, Biology, Leucine-Rich Repeat Serine-Threonine Protein Kinase-2, Fibril, Cell Membrane Structures, General Biochemistry, Genetics and Molecular Biology, genetics [Inflammation], Protein Aggregates, 03 medical and health sciences, Immune system, medicine, Animals, Humans, ddc:610, Aged, pathology [Inflammation], 030304 developmental biology, Inflammation, Synucleinopathies, Alpha-synuclein, synucleinopathies, metabolism [Mitochondria], Actins, nervous system diseases, Mice, Inbred C57BL, nervous system, chemistry, Proteolysis, Reactive Oxygen Species, Transcriptome, 030217 neurology & neurosurgery, cell-to-cell transfer

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7433f384592a810ad8cadc6135dda0eeTest
https://doi.org/10.1016/j.cell.2021.09.007Test

7

Neuronal polarization: From spatiotemporal signaling to cytoskeletal dynamics

المؤلفون: Max Schelski, Frank Bradke

المصدر: Molecular and cellular neuroscience 84, 11-28 (2017). doi:10.1016/j.mcn.2017.03.008

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7321e5a26066538a0e572575e9f131aaTest
https://doi.org/10.1016/j.mcn.2017.03.008Test

8

Dual Inhibition of GSK3β and CDK5 Protects the Cytoskeleton of Neurons from Neuroinflammatory-Mediated Degeneration In Vitro and In Vivo

المصدر: Stem Cell Reports
Stem cell reports 12(3), 502-517 (2019). doi:10.1016/j.stemcr.2019.01.015
Stem Cell Reports, Vol 12, Iss 3, Pp 502-517 (2019)

مصطلحات موضوعية: 0301 basic medicine, metabolism [Cytoskeleton], Biochemistry, Microtubules, neuroinflammation, 0302 clinical medicine, drug therapy [Alzheimer Disease], stem cell-based phenotypic screening, metabolism [Zebrafish], Phosphorylation, lcsh:QH301-705.5, Zebrafish, metabolism [Nerve Degeneration], Cytoskeleton, Neurons, lcsh:R5-920, metabolism [Inflammation], Neurodegeneration, Phosphoproteomics, neurodegeneration, metabolism [Neurites], 3. Good health, Cell biology, Neuroprotective Agents, drug effects [Microtubules], metabolism [Neurons], lcsh:Medicine (General), metabolism [Alzheimer Disease], Signal Transduction, drug effects [Signal Transduction], Neurite, induced pluripotent stem cells, Phenotypic screening, metabolism [Glycogen Synthase Kinase 3 beta], metabolism [Microtubules], CDK5, drug effects [Neurites], Biology, Neuroprotection, Article, 03 medical and health sciences, drug effects [Phosphorylation], Alzheimer Disease, Genetics, medicine, Neurites, drug effects [Neurons], Animals, Humans, ddc:610, Neuroinflammation, Inflammation, Glycogen Synthase Kinase 3 beta, pharmacology [Neuroprotective Agents], Cyclin-dependent kinase 5, GSK3β, drug therapy [Inflammation], Cyclin-Dependent Kinase 5, Cell Biology, medicine.disease, biology.organism_classification, 030104 developmental biology, drug effects [Cytoskeleton], lcsh:Biology (General), nervous system, Nerve Degeneration, metabolism [Cyclin-Dependent Kinase 5], drug therapy [Nerve Degeneration], ALS, 030217 neurology & neurosurgery, Developmental Biology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98a62262d3eda3ad763fb4978fc17ddeTest

9

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells

المؤلفون: Shu Liu, Yvonne Duernberger, Katrin Riemschoss, Benedetta Bolognesi, Annika Hornberger, Oleksandra Buravlova, Stephan A. Müller, Lydia Paulsen, Ina Vorberg, Philipp von Eisenhart-Rothe, Stefan F. Lichtenthaler, André Hossinger, Gian Gaetano Tartaglia, Verena Arndt, Sebastian Hogl, Nieves Lorenzo-Gotor

المصدر: Life Science Alliance
Recercat. Dipósit de la Recerca de Catalunya
instname
Life science alliance 2(4), e201800280 (2019). doi:10.26508/lsa.201800280

مصطلحات موضوعية: metabolism [Cytoskeleton], Health, Toxicology and Mutagenesis, Glutamine, animal diseases, Plant Science, Protein aggregation, metabolism [Peptide Termination Factors], Mice, 0302 clinical medicine, Asparagine, Cytoskeleton, Research Articles, 0303 health sciences, Ecology, biology, Chemistry, food and beverages, RNA-Binding Proteins, chemistry [Saccharomyces cerevisiae Proteins], Animals, Cell Line, Tumor, Cytoplasmic Granules, DNA-Binding Proteins, Gene Ontology, Peptide Termination Factors, Prions, Protein Domains, Proteolysis, Saccharomyces cerevisiae Proteins, chemistry [Peptide Termination Factors], Cell biology, ddc, Protein folding, metabolism [DNA-Binding Proteins], Research Article, Saccharomyces cerevisiae, metabolism [RNA-Binding Proteins], Fibril, Biochemistry, Genetics and Molecular Biology (miscellaneous), 03 medical and health sciences, Stress granule, ddc:570, 030304 developmental biology, fungi, Colocalization, chemistry [Prions], metabolism [Saccharomyces cerevisiae Proteins], biology.organism_classification, metabolism [Cytoplasmic Granules], Cytosol, metabolism [Prions], SUP35 protein, S cerevisiae, 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::519833838ae0f8065a48060f79452133Test
http://hdl.handle.net/10230/42509Test

10

Cytoskeleton-associated risk modifiers involved in early and rapid progression of sporadic Creutzfeldt-Jakob disease

المؤلفون: Mohsin Shafiq, Isidre Ferrer, Saima Zafar, Matthias Schmitz, Olivier Andreoletti, Neelam Younas, Waqas Tahir, Nadeem Sheikh, Inga Zerr

المساهمون: Universitat de Barcelona, German Center for Neurodegenerative Diseases, University of the Punjab, Institute of neuropathology, IDIBELL-University Hospital Bellvitge, University of Barcelona, Centro de Investigacion Biomédica en Red sobre Enfermedades Neurodegenerativas (CIBERNED), Instituto de Salud Carlos III [Madrid] (ISC), Interactions hôtes-agents pathogènes [Toulouse] (IHAP), Institut National de la Recherche Agronomique (INRA)-Ecole Nationale Vétérinaire de Toulouse (ENVT), Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées

المصدر: Dipòsit Digital de la UB
Universidad de Barcelona
Recercat. Dipósit de la Recerca de Catalunya
instname
Molecular Neurobiology
Molecular Neurobiology, Humana Press, 2018, 55 (5), pp.4009-4029. ⟨10.1007/s12035-017-0589-0⟩
Molecular neurobiology 55(5), 4009-4029 (2017). doi:10.1007/s12035-017-0589-0

مصطلحات موضوعية: 0301 basic medicine, Male, Pathology, metabolism [Cytoskeleton], [SDV]Life Sciences [q-bio], metabolism [Microglia], MM1, Creutzfeldt-Jakob Syndrome, 0302 clinical medicine, Risk Factors, Cerebellum, Citosquelet, Amyloid precursor protein, Phosphoprotein Phosphatases, ROCK2, Phosphorylation, RNA, Small Interfering, Cells, Cultured, Degeneració (Patologia), Cytoskeleton, metabolism [Phosphoprotein Phosphatases], SSH1 protein, human, Neurons, rho-Associated Kinases, biology, Microglia, metabolism [Calcineurin], Calcineurin, Microfilament Proteins, Lim Kinases, Degeneration (Pathology), SSH1, Cofilin, Middle Aged, Patologia, Metabolisme, 3. Good health, Cell biology, metabolism [Cofilin 1], CJD, medicine.anatomical_structure, Neurology, metabolism [Lim Kinases], metabolism [Neurons], Knockout mouse, Cortex, Disease Progression, Female, Protein Binding, Cofilin 1, medicine.medical_specialty, metabolism [Actins], Cell Survival, Neuroscience (miscellaneous), metabolism [Amyloid beta-Peptides], Mice, Transgenic, macromolecular substances, LIMK1, metabolism [rho-Associated Kinases], Lim kinase, metabolism [PrPC Proteins], 03 medical and health sciences, Cellular and Molecular Neuroscience, LIMK, Downregulation and upregulation, ddc:570, medicine, Animals, Humans, Malaltia de Creutzfeldt-Jakob, PrPC Proteins, metabolism [RNA, Small Interfering], Gene Silencing, Actin, Aged, pathology [Creutzfeldt-Jakob Syndrome], Amyloid beta-Peptides, Aif1 protein, mouse, ROCK2 protein, human, Calcium-Binding Proteins, LIMK1 protein, human, metabolism [Microfilament Proteins], metabolism [Calcium-Binding Proteins], VV2, Actins, Creutzfeldt-Jakob disease, 030104 developmental biology, Metabolism, Prion protein, Rock, biology.protein, APP, 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::decd9daddee4f10c42df04db2b84a74cTest
http://hdl.handle.net/2445/141810Test

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