مصطلحات الفهرس: Western blotting, glucocorticoid receptor/ec [Endogenous Compound], interleukin 10/ec [Endogenous Compound], interleukin 1beta/ec [Endogenous Compound], interleukin 33/ec [Endogenous Compound], interleukin 6/ec [Endogenous Compound], macrophage elastase/ec [Endogenous Compound], mineralocorticoid receptor/ec [Endogenous Compound], prostaglandin synthase/ec [Endogenous Compound], toll like receptor 4/ec [Endogenous Compound], macrophage, mouse, animal cell, animal experiment, animal model, animal tissue, article, bone marrow derived macrophage, carditis/et [Etiology], controlled study, DNA binding, genotype, heart muscle fibrosis/et [Etiology], male, macrophage cell line, nonhuman, priority journal, promoter region, signal transduction, transactivation assay, transcription regulation, Article

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/29150Test
Journal of Endocrinology
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مصطلحات الفهرس: Western blotting, glucocorticoid receptor/ec [Endogenous Compound], interleukin 10/ec [Endogenous Compound], interleukin 1beta/ec [Endogenous Compound], interleukin 33/ec [Endogenous Compound], interleukin 6/ec [Endogenous Compound], macrophage elastase/ec [Endogenous Compound], mineralocorticoid receptor/ec [Endogenous Compound], prostaglandin synthase/ec [Endogenous Compound], toll like receptor 4/ec [Endogenous Compound], macrophage, mouse, animal cell, animal experiment, animal model, animal tissue, article, bone marrow derived macrophage, carditis/et [Etiology], controlled study, DNA binding, genotype, heart muscle fibrosis/et [Etiology], male, macrophage cell line, nonhuman, priority journal, promoter region, signal transduction, transactivation assay, transcription regulation, Article

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/29150Test
Journal of Endocrinology
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مصطلحات الفهرس: podocyte, cardiac muscle cell, chronic kidney failure/dt [Drug Therapy], chronic kidney failure/et [Etiology], chronic kidney failure/th [Therapy], disease course, drug targeting, endothelium cell, fibroblast, fibrogenesis, gene deletion, heart failure/dt [Drug Therapy], heart failure/et [Etiology], heart muscle fibrosis/et [Etiology], heart protection, hormone action, human, hyperkalemia/co [Complication], in vitro study, inflammation, intracellular signaling, kidney fibrosis/dt [Drug Therapy], kidney fibrosis/et [Etiology], kidney injury, kidney tubule cell, macrophage, mesangium cell, nonhuman, oxidative stress, pathogenesis, renal protection, renin angiotensin aldosterone system, short survey, vascular smooth muscle cell, aldosterone/ec [Endogenous Compound], dipeptidyl carboxypeptidase inhibitor, mineralocorticoid antagonist/dt [Drug Therapy], mineralocorticoid receptor/ec [Endogenous Compound], Short Survey

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/39468Test
Frontiers in Pharmacology
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مصطلحات الفهرس: podocyte, cardiac muscle cell, chronic kidney failure/dt [Drug Therapy], chronic kidney failure/et [Etiology], chronic kidney failure/th [Therapy], disease course, drug targeting, endothelium cell, fibroblast, fibrogenesis, gene deletion, heart failure/dt [Drug Therapy], heart failure/et [Etiology], heart muscle fibrosis/et [Etiology], heart protection, hormone action, human, hyperkalemia/co [Complication], in vitro study, inflammation, intracellular signaling, kidney fibrosis/dt [Drug Therapy], kidney fibrosis/et [Etiology], kidney injury, kidney tubule cell, macrophage, mesangium cell, nonhuman, oxidative stress, pathogenesis, renal protection, renin angiotensin aldosterone system, short survey, vascular smooth muscle cell, aldosterone/ec [Endogenous Compound], dipeptidyl carboxypeptidase inhibitor, mineralocorticoid antagonist/dt [Drug Therapy], mineralocorticoid receptor/ec [Endogenous Compound], Short Survey

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/39468Test
Frontiers in Pharmacology
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مصطلحات الفهرس: heart weight, hypertension/et [Etiology], immunohistochemistry, inflammatory cell, intracellular signaling, macrophage, male, mouse, myofibroblast, nonhuman, priority journal, systolic blood pressure, wild type, aldosterone/ec [Endogenous Compound], alpha smooth muscle actin/ec [Endogenous Compound], angiotensin II/ec [Endogenous Compound], brain natriuretic peptide/ec [Endogenous Compound], calvasculin/ec [Endogenous Compound], CD3 antigen/ec [Endogenous Compound], cell marker/ec [Endogenous Compound], chemokine receptor CCR5/ec [Endogenous Compound], chlordane/ec [Endogenous Compound], collagen/ec [Endogenous Compound], connective tissue growth factor/ec [Endogenous Compound], corticosterone/ec [Endogenous Compound], deoxycorticosterone, endothelin 1/ec [Endogenous Compound], gelatinase A/ec [Endogenous Compound], gelatinase B/ec [Endogenous Compound], incyclinide/ec [Endogenous Compound], interleukin 23/ec [Endogenous Compound], mineralocorticoid receptor, monocyte chemotactic protein 1/ec [Endogenous Compound], osteopontin/ec [Endogenous Compound], plasminogen activator inhibitor 1/ec [Endogenous Compound], platelet derived growth factor beta receptor/ec [Endogenous Compound], RANTES/ec [Endogenous Compound], Rho guanine nucleotide dissociation inhibitor 2/ec [Endogenous Compound], transforming growth factor beta1/ec [Endogenous Compound], tumor necrosis factor alpha, fibroblast, aldosterone blood level, angiotensin blood level, animal experiment, animal model, animal tissue, article, cardiovascular inflammation/et [Etiology], CD3+ T lymphocyte, cell infiltration, controlled study, corticosterone blood level, enzyme activity, gene expression, gene inactivation, heart muscle fibrosis/et [Etiology], heart ventricle hypertrophy, Article

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/41790Test
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مصطلحات الفهرس: heart weight, hypertension/et [Etiology], immunohistochemistry, inflammatory cell, intracellular signaling, macrophage, male, mouse, myofibroblast, nonhuman, priority journal, systolic blood pressure, wild type, aldosterone/ec [Endogenous Compound], alpha smooth muscle actin/ec [Endogenous Compound], angiotensin II/ec [Endogenous Compound], brain natriuretic peptide/ec [Endogenous Compound], calvasculin/ec [Endogenous Compound], CD3 antigen/ec [Endogenous Compound], cell marker/ec [Endogenous Compound], chemokine receptor CCR5/ec [Endogenous Compound], chlordane/ec [Endogenous Compound], collagen/ec [Endogenous Compound], connective tissue growth factor/ec [Endogenous Compound], corticosterone/ec [Endogenous Compound], deoxycorticosterone, endothelin 1/ec [Endogenous Compound], gelatinase A/ec [Endogenous Compound], gelatinase B/ec [Endogenous Compound], incyclinide/ec [Endogenous Compound], interleukin 23/ec [Endogenous Compound], mineralocorticoid receptor, monocyte chemotactic protein 1/ec [Endogenous Compound], osteopontin/ec [Endogenous Compound], plasminogen activator inhibitor 1/ec [Endogenous Compound], platelet derived growth factor beta receptor/ec [Endogenous Compound], RANTES/ec [Endogenous Compound], Rho guanine nucleotide dissociation inhibitor 2/ec [Endogenous Compound], transforming growth factor beta1/ec [Endogenous Compound], tumor necrosis factor alpha, fibroblast, aldosterone blood level, angiotensin blood level, animal experiment, animal model, animal tissue, article, cardiovascular inflammation/et [Etiology], CD3+ T lymphocyte, cell infiltration, controlled study, corticosterone blood level, enzyme activity, gene expression, gene inactivation, heart muscle fibrosis/et [Etiology], heart ventricle hypertrophy, Article

URL: https://repository.monashhealth.org/monashhealthjspui/handle/1/41790Test
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