يعرض 1 - 5 نتائج من 5 نتيجة بحث عن '"drug effects [Recognition, Psychology]"', وقت الاستعلام: 1.33s تنقيح النتائج
  1. 1

    المساهمون: RS: MHeNs - R3 - Neuroscience, Psychiatrie & Neuropsychologie, Basic Neuroscience 2, Section Psychopharmacology, RS: FPN NPPP II, Cellular and Computational Neuroscience (SILS, FNWI)

    المصدر: Molecular and Cellular Neuroscience, 120:103719. Elsevier Science
    Molecular and cellular neuroscience, 120:103719. Academic Press Inc.
    Molecular and cellular neuroscience 120, 103719 (2022). doi:10.1016/j.mcn.2022.103719

    وصف الملف: application/pdf

  2. 2

    المصدر: Behavioural brain research 396, 112875-(2021). doi:10.1016/j.bbr.2020.112875
    Behavioral Brain Research, 396

  3. 3

    المصدر: Acta Neuropathologica
    Acta neuropathologica 130(5), 619-631 (2015). doi:10.1007/s00401-015-1483-3

    مصطلحات موضوعية: pathology [Tauopathies], 3-(1,3-benzodioxol-5-yl)-5-(3-bromophenyl)-1H-pyrazole, Male, drug effects [Hippocampus], Hippocampus, Protein aggregation, physiology [Cell Death], Frontotemporal dementia and parkinsonism linked to chromosome 17, Synapse, Random Allocation, Tau aggregation inhibitor, pathology [Neurons], drug therapy [Tauopathies], Gliosis, Neurons, Cell Death, Neurodegeneration, pathology [Gliosis], physiology [Neurons], Tauopathy, physiology [Motor Activity], Neuroprotective Agents, Tauopathies, Disease Progression, Female, medicine.symptom, drug effects [Recognition, Psychology], Alzheimer’s disease, Mapt protein, mouse, Genetically modified mouse, physiology [Recognition, Psychology], Clinical Neurology, pharmacology [Benzodioxoles], MAPT protein, human, Mice, Transgenic, tau Proteins, Biology, Motor Activity, Pathology and Forensic Medicine, Cellular and Molecular Neuroscience, Protein Aggregates, mental disorders, medicine, drug effects [Neurons], Animals, ddc:610, Benzodioxoles, Original Paper, pharmacology [Neuroprotective Agents], drug effects [Motor Activity], Anle138b, drug effects [Protein Aggregates], drug effects [Cell Death], Recognition, Psychology, medicine.disease, physiopathology [Gliosis], metabolism [tau Proteins], genetics [tau Proteins], Disease Models, Animal, pathology [Hippocampus], drug therapy [Gliosis], physiopathology [Hippocampus], Pyrazoles, Neurology (clinical), Tau, pharmacology [Pyrazoles], Neuroscience

    وصف الملف: application/pdf

  4. 4

    المؤلفون: Daniela A. Bota, Tami John, Naomi Lomeli

    المصدر: John, T; Lomeli, N; & Bota, DA. (2017). Systemic cisplatin exposure during infancy and adolescence causes impaired cognitive function in adulthood. BEHAVIOURAL BRAIN RESEARCH, 319, 200-206. doi: 10.1016/j.bbr.2016.11.013. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/2679s83kTest
    John, Tami; Lomeli, Naomi; & Bota, Daniela A. (2017). Systemic cisplatin exposure during infancy and adolescence causes impaired cognitive function in adulthood.. Behavioural brain research, 319, 200-206. UC Irvine: Institute for Clinical and Translational Science. Retrieved from: http://www.escholarship.org/uc/item/05x725jsTest

    مصطلحات موضوعية: Oncology, Male, Aging, drug effects [Recognition (Psychology)], medicine.medical_treatment, Medical and Health Sciences, Developmental psychology, chemically induced [Cognition Disorders], Rats, Sprague-Dawley, Behavioral Neuroscience, 0302 clinical medicine, Discrimination, Psychological, Chemotherapy-related cognitive impairment (CRCI), Discrimination, Conditioning, Psychological, Medicine and Health Sciences, Psychology, 2.1 Biological and endogenous factors, Fear conditioning, Aetiology, Cancer, Pediatric, Age Factors, Cognition, Hematology, Fear, drug effects [Aging], Cognitive test, Mental Health, medicine.anatomical_structure, 030220 oncology & carcinogenesis, medicine.drug, Cisplatin (CDDP), drug effects [Fear], Pediatric Research Initiative, medicine.medical_specialty, Childhood leukemia, Pediatric Cancer, Childhood Leukemia, Antineoplastic Agents, Basic Behavioral and Social Science, Amygdala, Article, toxicity [Antineoplastic Agents], drug effects [Discrimination (Psychology)], 03 medical and health sciences, Rare Diseases, Memory, Internal medicine, Behavioral and Social Science, Acquired Cognitive Impairment, medicine, Animals, Cisplatin, Chemotherapy, Neurology & Neurosurgery, Chemotherapy-related cognitive impairment, Psychology and Cognitive Sciences, Neurosciences, Neurotoxicity, Recognition, Psychology, Stem Cell Research, medicine.disease, toxicity [Cisplatin], Brain Disorders, Rats, Recognition, drug effects [Conditioning (Psychology)], Psychological, Sprague-Dawley, Cognition Disorders, 030217 neurology & neurosurgery, Conditioning

    وصف الملف: application/pdf

  5. 5

    المصدر: Acta Neuropathologica Communications
    Acta Neuropathologica Communications 4(1), 39 (2016). doi:10.1186/s40478-016-0310-y

    مصطلحات موضوعية: pharmacology [Protein Kinase Inhibitors], Time Factors, Pyridines, pharmacology [Enzyme Inhibitors], drug effects [Gene Expression Regulation], genetics [Gene Expression Regulation], Mice, pathology [Brain], genetics [Parkinson Disease], A53T mouse model, 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine, Fasudil, metabolism [alpha-Synuclein], Enzyme Inhibitors, therapeutic use [Pyridines], Brain, Parkinson Disease, therapeutic use [Enzyme Inhibitors], genetics [alpha-Synuclein], alpha-Synuclein, drug effects [Brain], SNCAIP protein, human, drug effects [Psychomotor Performance], drug effects [Recognition, Psychology], fasudil, Tyrosine 3-Monooxygenase, metabolism [Parkinson Disease], genetics [Mutation], Mice, Transgenic, Nerve Tissue Proteins, Protein Aggregates, Y 27632, Cell Line, Tumor, therapeutic use [Protein Kinase Inhibitors], Animals, Humans, analogs & derivatives [1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine], ddc:610, pharmacology [1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine], Protein Kinase Inhibitors, metabolism [Nerve Tissue Proteins], Research, drug effects [Protein Aggregates], Recognition, Psychology, pharmacology [Pyridines], Amides, drug therapy [Parkinson Disease], metabolism [Tyrosine 3-Monooxygenase], α-synuclein aggregation, Disease Models, Animal, Gene Expression Regulation, metabolism [Brain], therapeutic use [Amides], Mutation, Parkinson’s disease, pharmacology [Amides], therapeutic use [1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine], genetics [Protein Aggregates], Carrier Proteins, Psychomotor Performance, metabolism [Carrier Proteins]