نتائج البحث - "deficiency [Nerve Tissue Proteins]"

المصدر: Annals of Clinical and Translational Neurology, Vol 7, Iss 1, Pp 46-58 (2020)
Annals of Clinical and Translational Neurology
Annals of Clinical and Translational Neurology 7(1), 46-58 (2019). doi:10.1002/acn3.50959

مصطلحات موضوعية: Adult, Male, genetics [Chromosome Disorders], physiopathology [Autism Spectrum Disorder], Adolescent, Autism Spectrum Disorder, Chromosomes, Human, Pair 22, pathology [Chromosome Disorders], Chromosome Disorders, Nerve Tissue Proteins, Neurosciences. Biological psychiatry. Neuropsychiatry, pathology [Autism Spectrum Disorder], diagnostic imaging [White Matter], Translational Research, Biomedical, Young Adult, physiopathology [Chromosome Disorders], pathology [Gray Matter], pathology [White Matter], pathology [Neurodevelopmental Disorders], Animals, Humans, ddc:610, Gray Matter, Child, RC346-429, Research Articles, Mice, Knockout, deficiency [Nerve Tissue Proteins], genetics [Neurodevelopmental Disorders], Microfilament Proteins, diagnostic imaging [Gray Matter], diagnostic imaging [Chromosome Disorders], Middle Aged, physiopathology [Neurodevelopmental Disorders], White Matter, diagnostic imaging [Neurodevelopmental Disorders], Mice, Inbred C57BL, Disease Models, Animal, Diffusion Tensor Imaging, Neurodevelopmental Disorders, Child, Preschool, genetics [Chromosomes, Human, Pair 22], genetics [Autism Spectrum Disorder], Female, Neurology. Diseases of the nervous system, Chromosome Deletion, diagnostic imaging [Autism Spectrum Disorder], Research Article, RC321-571

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::890d464721db1e01600b653e2d75b2fdTest

5

Disturbed Prefrontal Cortex Activity in the Absence of Schizophrenia-Like Behavioral Dysfunction in Arc/Arg3.1 Deficient Mice

المؤلفون: Dietmar Kuhl, Matthias Kneussel, Ora Ohana, Xiaoyan Gao, Ute Süsens, Jasper Grendel, Mary Muhia, Sergio Castro-Gomez, Dirk Isbrandt

المصدر: The journal of neuroscience 39(41), 8149-8163 (2019). doi:10.1523/JNEUROSCI.0623-19.2019

مصطلحات موضوعية: 0301 basic medicine, Male, Patch-Clamp Techniques, deficiency [Cytoskeletal Proteins], drug effects [Reflex, Startle], Biology, 03 medical and health sciences, Mice, 0302 clinical medicine, chemically induced [Seizures], medicine, Animals, ddc:610, physiopathology [Prefrontal Cortex], Prefrontal cortex, Social Behavior, Evoked Potentials, drug effects [Memory, Short-Term], genetics [Nerve Tissue Proteins], genetics [Cytoskeletal Proteins], Mice, Knockout, Neurons, Arc (protein), deficiency [Nerve Tissue Proteins], Working memory, General Neuroscience, Dopaminergic Neurons, drug effects [Motor Activity], Dopaminergic, Environmental stressor, Excitatory Postsynaptic Potentials, Sensory Gating, medicine.disease, genetics [Seizures], 030104 developmental biology, Schizophrenia, drug effects [Electroencephalography], Synaptic plasticity, Female, Schizophrenic Psychology, Immediate early gene, Neuroscience, 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4f15ba2ee1906ae224670aecd7fb8eddTest
https://pub.dzne.de/record/141538Test

6

Streptozotocin-induced β-cell damage, high fat diet, and metformin administration regulate Hes3 expression in the adult mouse brain

المؤلفون: Nikolakopoulou, Polyxeni, Chatzigeorgiou, Antonios, Consortium, German Mouse Clinic, Wolf, Eckhard, Klingenspor, Martin, Ollert, Markus, Schmidt-Weber, Carsten, Fuchs, Helmut, Gailus-Durner, Valerie, Hrabe de Angelis, Martin, Tsata, Vasiliki, Monasor, Laura Sebastian, Kourtzelis, Ioannis, Troullinaki, Maria, Witt, Anke, Anastasiou, Vivian, Chrousos, George, Yi, Chun-Xia, García-Cáceres, Cristina, Tschöp, Matthias H, Bornstein, Stefan R, Androutsellis-Theotokis, Andreas, Becker, Lore, Toutouna, Louiza, Klopstock, Thomas, Treise, Irina, Busch, Dirk H, Beckers, Johannes, Moreth, Kristin, Bekeredjian, Raffi, Garrett, Lillian, Hölter, Sabine M, Zimprich, Annemarie, Wurst, Wolfgang, Masjkur, Jimmy, Brommage, Robert, Amarie, Oana, Graw, Jochen, Calzada-Wack, Julia, Neff, Frauke, Zimmer, Andreas, Östereicher, Manuela, Steinkamp, Ralph, Lengger, Christoph, Maier, Holger, Arps-Forker, Carina, Stoeger, Claudia, Leuchtenberger, Stefanie, Poser, Steven W, Rozman, Jan, Rathkolb, Birgit, Aguilar-Pimentel, Juan Antonio

المساهمون: University of Zurich, Androutsellis-Theotokis, Andreas, APH - Aging & Later Life, Amsterdam Neuroscience - Cellular & Molecular Mechanisms, Laboratory for Endocrinology, AGEM - Endocrinology, metabolism and nutrition, ACS - Diabetes & metabolism

المصدر: Scientific reports 8(1), 11335 (2018). doi:10.1038/s41598-018-29434-2
Scientific Reports, Vol 8, Iss 1, Pp 1-15 (2018)
Scientific reports, 8(1):11335. Nature Publishing Group
German Mouse Clinic Consortium 2018, ' Streptozotocin-induced β-cell damage, high fat diet, and metformin administration regulate Hes3 expression in the adult mouse brain ', Scientific Reports, vol. 8, 11335 . https://doi.org/10.1038/s41598-018-29434-2Test
Sci. Rep. 8:11335 (2018)
Scientific Reports

مصطلحات موضوعية: Male, 0301 basic medicine, Aging, medicine.medical_treatment, 10265 Clinic for Endocrinology and Diabetology, lcsh:Medicine, drug effects [Gene Expression Regulation], Type 2 diabetes, Insulin-Secreting Cells, pathology [Insulin-Secreting Cells], Basic Helix-Loop-Helix Transcription Factors, Medicine, lcsh:Science, genetics [Nerve Tissue Proteins], Multidisciplinary, Brain, toxicity [Streptozocin], Metformin, Phenotype, Hes3 protein, mouse, medicine.drug, medicine.medical_specialty, genetics [Basic Helix-Loop-Helix Transcription Factors], Nerve Tissue Proteins, 610 Medicine & health, Diet, High-Fat, Article, Streptozocin, 03 medical and health sciences, Insulin resistance, Diabetes mellitus, Internal medicine, Animals, metabolism [Aging], Progenitor cell, 1000 Multidisciplinary, Type 1 diabetes, metabolism [Nerve Tissue Proteins], deficiency [Nerve Tissue Proteins], business.industry, Insulin, lcsh:R, administration & dosage [Metformin], metabolism [Insulin-Secreting Cells], medicine.disease, Streptozotocin, Repressor Proteins, Mice, Inbred C57BL, metabolism [Basic Helix-Loop-Helix Transcription Factors], 030104 developmental biology, Endocrinology, Gene Expression Regulation, deficiency [Basic Helix-Loop-Helix Transcription Factors], metabolism [Brain], lcsh:Q, business, ddc:600, drug effects [Insulin-Secreting Cells]

وصف الملف: s41598-018-29434-2.pdf - application/pdf; application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b14530a6b1ab6f563ab8acda07ceaa8eTest
https://doi.org/10.1038/s41598-018-29434-2Test

7

Semaphorin 4C and 4G are ligands of Plexin-B2 required in cerebellar development

المؤلفون: Noriko Takegahara, Roland H. Friedel, Christine Jolicoeur, Atsushi Kumanogoh, Wolfgang Wurst, Viola Maier, Hitoshi Kikutani, Helen Rayburn, Marc Tessier-Lavigne

المصدر: Molecular and cellular neuroscience 46(2), 419-431 (2011). doi:10.1016/j.mcn.2010.11.005

مصطلحات موضوعية: Cerebellum, Organogenesis, deficiency [Semaphorins], Sema4c protein, mouse, Gene Expression, metabolism [Neural Stem Cells], Semaphorins, Exencephaly, Ligands, Plxnb2 protein, mouse, Mice, Neural Stem Cells, Cell Movement, In Situ Hybridization, genetics [Nerve Tissue Proteins], Neurons, Mice, Knockout, biology, Gene Expression Regulation, Developmental, metabolism [Cerebellum], Immunohistochemistry, Neural stem cell, genetics [Organogenesis], genetics [Semaphorins], metabolism [Semaphorins], medicine.anatomical_structure, metabolism [Neurons], Cerebellar cortex, embryonic structures, animal structures, Blotting, Western, embryology [Cerebellum], Nerve Tissue Proteins, Article, Cellular and Molecular Neuroscience, Sema4g protein, mouse, Semaphorin, medicine, Animals, ddc:610, Molecular Biology, metabolism [Nerve Tissue Proteins], deficiency [Nerve Tissue Proteins], Plexin, Neural tube, Cell Biology, medicine.disease, Granule cell, Molecular biology, Mice, Inbred C57BL, nervous system, biology.protein

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5304a983cfee1ab62cc52a7e16be2635Test
https://pub.dzne.de/record/136174Test

8

The novel membrane protein TMEM59 modulates complex glycosylation, cell surface expression, and secretion of the amyloid precursor protein

المؤلفون: Jörg Tatzelt, Stephanie Neumann, Anna Münch, Sylvia Ullrich, Stefan F. Lichtenthaler, Elisabeth Kremmer

المصدر: J. Biol. Chem. 285, 20664-20674 (2010)
The journal of biological chemistry 285(27), 20664-20674 (2010). doi:10.1074/jbc.M109.055608

مصطلحات موضوعية: N-linked glycosylation, Trans-golgi network, Alzheimers-disease, Gamma-secretase, Alpha-Secretase, Beta-secretase, Intramembrane proteolysis, Mediated endocytosis, Converting-enzyme, Cleaving enzyme, metabolism [Receptors, Cell Surface], genetics [RNA, Small Interfering], Kidney, Biochemistry, APP protein, human, chemistry.chemical_compound, Amyloid beta-Protein Precursor, Genes, Reporter, Cricetinae, metabolism [Amyloid beta-Protein Precursor], Chlorocebus aethiops, Amyloid precursor protein, Polylysine, RNA, Small Interfering, genetics [Nerve Tissue Proteins], P3 peptide, Brain, Cell biology, genetics [Membrane Proteins], Ectodomain, Alpha secretase, TMEM59L protein, human, Protein Synthesis and Degradation, genetics [Amyloid beta-Protein Precursor], Gene Knockdown Techniques, ddc:540, COS Cells, symbols, Glycosylation, Endosome, Nerve Tissue Proteins, Receptors, Cell Surface, CHO Cells, Biology, Cell Line, symbols.namesake, Cricetulus, mental disorders, Animals, Humans, Molecular Biology, deficiency [Membrane Proteins], metabolism [Nerve Tissue Proteins], deficiency [Nerve Tissue Proteins], TMEM59, Membrane Proteins, Cell Biology, Golgi apparatus, Blotting, Northern, DCF1 protein, human, Protease Nexins, chemistry, metabolism [Brain], biology.protein, metabolism [Membrane Proteins]

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1cab426c5d1f66efd0b982779e4c4bb4Test
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=5924Test

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