المؤلفون: Alessandro Giollo, John Fitton, Maya H Buch

المصدر: Fitton, J, Giollo, A & Buch, M H 2018, ' GP2015 as a promising therapy for rheumatoid arthritis ', Expert opinion on biological therapy, vol. 18, no. 4, pp. 477-481 . https://doi.org/10.1080/14712598.2018.1452909Test

مصطلحات موضوعية: musculoskeletal diseases, Drug, medicine.medical_specialty, Receptors, Tumor Necrosis Factor/genetics, medicine.medical_treatment, media_common.quotation_subject, Clinical Biochemistry, Antibodies, Anti-Idiotypic/blood, Lymphotoxin-alpha/immunology, Receptors, Tumor Necrosis Factor, Etanercept, Arthritis, Rheumatoid, Double blind, 03 medical and health sciences, 0302 clinical medicine, Etanercept/adverse effects, Drug Discovery, medicine, Humans, Dermatologic Agents/adverse effects, skin and connective tissue diseases, Intensive care medicine, Biosimilar Pharmaceuticals, Lymphotoxin-alpha, media_common, 030203 arthritis & rheumatology, Pharmacology, Clinical Trials as Topic, Tumor Necrosis Factor-alpha, business.industry, Biosimilar, Tumor Necrosis Factor-alpha/immunology, medicine.disease, Reference drug, Antibodies, Anti-Idiotypic, TNF inhibitor, Arthritis, Rheumatoid/drug therapy, 030220 oncology & carcinogenesis, Rheumatoid arthritis, Pharmacodynamics, Biosimilar Pharmaceuticals/adverse effects, Dermatologic Agents, business, Half-Life, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc10b8a5e28ee9b2b92b7ca93635614fTest
https://doi.org/10.1080/14712598.2018.1452909Test

المؤلفون: Danielsson, O

المصدر: Medicinsk Vetenskap vid Karolinska Institutet. (1):23

المؤلفون: Nellie A. Martin, Viktor Molnar, Gabor T. Szilagyi, Maria L. Elkjaer, Arkadiusz Nawrocki, Justyna Okarmus, Agnieszka Wlodarczyk, Eva K. Thygesen, Miklos Palkovits, Ferenc Gallyas, Martin R. Larsen, Hans Lassmann, Eirikur Benedikz, Trevor Owens, Asa F. Svenningsen, Zsolt Illes

المصدر: Martin, N A, Molnar, V, Szilagyi, G T, Elkjaer, M L, Nawrocki, A, Okarmus, J, Wlodarczyk, A, Thygesen, E K, Palkovits, M, Gallyas, F, Larsen, M R, Lassmann, H, Benedikz, E, Owens, T, Svenningsen, A F & Illes, Z 2018, ' Experimental demyelination and axonal loss are reduced in MicroRNA-146a deficient mice ', Frontiers in Immunology, vol. 9, 490 . https://doi.org/10.3389/fimmu.2018.00490Test
Frontiers in Immunology
Frontiers in Immunology, Vol 9 (2018)

مصطلحات موضوعية: 0301 basic medicine, MiR-181b, Oligodendroglia/physiology, Axonal loss, Corpus callosum, multiple sclerosis, Receptors, Tumor Necrosis Factor, MiR-193a, Corpus Callosum, Transcriptome, Mice, 0302 clinical medicine, Immunology and Allergy, Chemokine CCL2, Original Research, Mice, Knockout, Microglia, Chemokine CCL2/genetics, Cell Differentiation, Oligodendroglia, medicine.anatomical_structure, Models, Animal, Tumor necrosis factor alpha, Female, demyelination, Demyelination, lcsh:Immunologic diseases. Allergy, Receptors, Tumor Necrosis Factor/genetics, Immunology, miR-181b, Demyelinating Diseases/genetics, miR-193a, CCL2, Biology, Multiple sclerosis, 03 medical and health sciences, Cuprizone, Corpus Callosum/physiology, medicine, Humans, Animals, Remyelination, Gene Expression Profiling, medicine.disease, Molecular biology, Axons, miR-146a, Mice, Inbred C57BL, MicroRNAs, MicroRNAs/genetics, 030104 developmental biology, remyelination, Axons/pathology, nervous system, MiR-146a, lcsh:RC581-607, 030217 neurology & neurosurgery, Demyelinating Diseases

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd4045fbbca3098e261b35feccb0ac8fTest
https://findresearcher.sdu.dk:8443/ws/files/137639610/Experimental_demyelination_and_axonal_loss_are_reduced_in_MicroRNA_146a_deficient_mice.pdfTest

المؤلفون: Bodo Grimbacher, Annette Schmitt-Graeff, A. D. B. Webster, Qiang Pan-Hammarström, Michael Schlesier, Jürgen K. Rockstroh, Ulrich Salzer, Pascal Schneider, Lennart Hammarström, Hans-Hartmut Peter, Alejandro A. Schäffer, Helen Chapel

المصدر: Nature Genetics, vol. 37, no. 8, pp. 820-828

مصطلحات موضوعية: Genetics, Candidate gene, Mutation, Common variable immunodeficiency, Transmembrane activator and CAML interactor, Biology, medicine.disease, medicine.disease_cause, Immunoglobulin class switching, Humoral immunity, Immunology, medicine, B-cell activating factor, Amino Acid Sequence, Antibody Formation, Cell Division/genetics, Cell Division/physiology, Common Variable Immunodeficiency/genetics, Female, Homozygote, Humans, Immunoglobulin M/physiology, Male, Membrane Proteins/chemistry, Membrane Proteins/genetics, Molecular Sequence Data, Nuclear Proteins/genetics, Nuclear Proteins/physiology, Pedigree, Receptors, Tumor Necrosis Factor/chemistry, Receptors, Tumor Necrosis Factor/genetics, Transmembrane Activator and CAML Interactor Protein, Immunodeficiency

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::042c023982477131bc66639caf3e4f71Test
https://doi.org/10.1038/ng1600Test

المؤلفون: Herman W. T. van Vlijmen, Timothy S. Zheng, Alexey Lugovskoy, Kay Hofmann, Max Dobles, Dian L. Olson, Aubry Tardivel, Jürg Tschopp, Dahai Gong, Linda C. Burkly, Beth Browning, Yen-Ming Hsu, Pascal Schneider, Sylvie Hertig

المصدر: Journal of Biological Chemistry, vol. 278, no. 7, pp. 5444-5454

مصطلحات موضوعية: Models, Molecular, Molecular Sequence Data, Biology, Vascular endothelial growth inhibitor, Biochemistry, Jurkat cells, Receptors, Tumor Necrosis Factor, Cell Line, Evolution, Molecular, TNF-Related Apoptosis-Inducing Ligand, Mice, Animals, Humans, Amino Acid Sequence, Decoy receptors, Receptor, Molecular Biology, Death domain, Membrane Glycoproteins, Apoptosis Regulatory Proteins, Chromosome Mapping, Membrane Glycoproteins/genetics, Membrane Glycoproteins/metabolism, Receptors, Tumor Necrosis Factor/genetics, Receptors, Tumor Necrosis Factor/metabolism, Sequence Analysis, Tumor Necrosis Factor-alpha/genetics, Tumor Necrosis Factor-alpha/metabolism, Tumor Necrosis Factor-alpha, HEK 293 cells, Cell Biology, Ligand (biochemistry), Molecular biology, Tumor necrosis factor alpha

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::170d88ceaee8e6baafc1febc57186cd6Test
https://doi.org/10.1074/jbc.m210783200Test

المؤلفون: Jeffrey Thompson, Sylvie Hertig, Jürg Tschopp, Jeffrey L. Browning, Kathy Strauch, Teresa G. Cachero, Luciana Trabach, Fang Qian, Christine Ambrose, Pascal Schneider, Colleen Mullen, Nils Holler, Paul Rennert

المصدر: The Journal of Experimental Medicine
Journal of Experimental Medicine, vol. 192, no. 11, pp. 1677-1684

مصطلحات موضوعية: Recombinant Fusion Proteins, Transmembrane Activator and CAML Interactor Protein, Tumor Necrosis Factor Ligand Superfamily Member 13, Immunology, Mice, Nude, Biology, medicine.disease_cause, Receptors, Tumor Necrosis Factor, 3T3 cells, Mice, Neoplasms, B-Cell Activating Factor, Tumor Cells, Cultured, medicine, Animals, Humans, Immunology and Allergy, B-Cell Maturation Antigen, B-cell activating factor, Receptor, Adaptor Proteins, Signal Transducing, Cell Line, Transformed, B-Lymphocytes, Tumor Necrosis Factor-alpha, Transmembrane activator and CAML interactor, Membrane Proteins, 3T3 Cells, Molecular biology, Cell Transformation, Neoplastic, medicine.anatomical_structure, Solubility, Cell culture, Commentary, Tumor necrosis factor alpha, Carrier Proteins, Carcinogenesis, HT29 Cells, Cell Division, B-Lymphocytes/physiology, Carrier Proteins/metabolism, Membrane Proteins/genetics, Membrane Proteins/metabolism, Neoplasms/therapy, Receptors, Tumor Necrosis Factor/genetics, Receptors, Tumor Necrosis Factor/metabolism, Recombinant Fusion Proteins/genetics, Recombinant Fusion Proteins/metabolism, Tumor Necrosis Factor-alpha/genetics, Tumor Necrosis Factor-alpha/metabolism

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a1b8b36932df694cc315beea9bc3993fTest
https://doi.org/10.1084/jem.192.11.1677Test

المؤلفون: Paul A. Overbeek, Pascal Schneider, Denis J. Headon, Ben Jackson, Chunyan Mou

المصدر: Proceedings of the National Academy of Sciences of the United States of America, vol. 103, no. 24, pp. 9075-9080

مصطلحات موضوعية: Animals, Body Patterning, Bone Morphogenetic Protein 4, Bone Morphogenetic Protein 7, Bone Morphogenetic Proteins/genetics, Bone Morphogenetic Proteins/metabolism, Edar Receptor, Embryo, Mammalian/anatomy & histology, Female, Gene Expression Regulation, Developmental, Hair Follicle/physiology, In Situ Hybridization, Male, Mice, Mice, Transgenic, Morphogenesis, Receptors, Ectodysplasin, Receptors, Tumor Necrosis Factor/genetics, Receptors, Tumor Necrosis Factor/metabolism, Signal Transduction/physiology, Skin/anatomy & histology, Skin/growth & development, Tissue Culture Techniques, Transcription, Genetic, Transforming Growth Factor beta/genetics, Transforming Growth Factor beta/metabolism, medicine.medical_specialty, Tissue Recombination, Biology, Bone morphogenetic protein, Receptors, Tumor Necrosis Factor, Dermis, stomatognathic system, Transforming Growth Factor beta, Internal medicine, medicine, Ectodysplasin A receptor, Skin, Multidisciplinary, integumentary system, Biological Sciences, Hair follicle, Embryo, Mammalian, Cell biology, medicine.anatomical_structure, Endocrinology, Bone morphogenetic protein 4, Ectodysplasins, Bone Morphogenetic Proteins, Hair Follicle, Signal Transduction

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33f6102e578b15d0f97efab963792abdTest
https://europepmc.org/articles/PMC1482568Test/

المؤلفون: Christine A. Kostek, Linda C. Burkly, Jeffrey A. Winkles, Pascal Schneider, Shawn Weng, Sharron A.N. Brown, Norm Allaire, Yen-Ming Hsu, Mahasweta Girgenrath, Richard A. Flavell, Timothy S. Zheng, Monica Wang, Martin L. Scott, Beth Browning, Hideo Yagita, Jennifer S. Michaelson, Jeffrey Boone Miller

المصدر: EMBO Journal, vol. 25, no. 24, pp. 5826-5839

مصطلحات موضوعية: Animals, Cell Cycle/drug effects, Cell Differentiation/drug effects, Cell Proliferation/drug effects, Cells, Cultured, Cobra Cardiotoxin Proteins/pharmacology, Gene Expression Regulation/drug effects, Humans, Inflammation, Mesenchymal Stem Cells/cytology, Mesenchymal Stem Cells/drug effects, Mice, Models, Biological, Muscle Development/drug effects, Muscle, Skeletal/cytology, Muscle, Skeletal/drug effects, Myoblasts/cytology, Myoblasts/drug effects, RNA, Messenger/genetics, RNA, Messenger/metabolism, Receptors, Tumor Necrosis Factor/deficiency, Receptors, Tumor Necrosis Factor/genetics, Regeneration/drug effects, Tumor Necrosis Factors/genetics, Tumor Necrosis Factors/metabolism, Cellular differentiation, Cobra Cardiotoxin Proteins, Biology, Muscle Development, General Biochemistry, Genetics and Molecular Biology, Receptors, Tumor Necrosis Factor, Article, Myoblasts, medicine, Myocyte, Regeneration, RNA, Messenger, Progenitor cell, Muscle, Skeletal, Molecular Biology, Cytokine TWEAK, Cell Proliferation, General Immunology and Microbiology, General Neuroscience, Regeneration (biology), Mesenchymal stem cell, Cell Cycle, Skeletal muscle, Cell Differentiation, Mesenchymal Stem Cells, Cell biology, medicine.anatomical_structure, Gene Expression Regulation, TWEAK Receptor, Tumor Necrosis Factors, C2C12

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::708b6dbd28ba8dfca62e27c87e25899bTest
https://pubmed.ncbi.nlm.nih.gov/17124496Test

المؤلفون: Pierre Vassalli, David M. Harlan, Pedro Luis Herrera

المصدر: Proceedings of the National Academy of Sciences, Vol. 97, No 1 (2000) pp. 279-284

مصطلحات موضوعية: Antigens, CD/metabolism, Fluorescent Antibody Technique, Apoptosis, Autoimmunity, Diabetes Mellitus/ genetics/immunology, CD8-Positive T-Lymphocytes, Receptors, Tumor Necrosis Factor, Diabetes mellitus genetics, Mice, Cytotoxic T cell, Insulin, Islets of Langerhans/immunology/pathology, Promoter Regions, Genetic, Tumor Necrosis Factor-alpha/ genetics, ddc:616, Mice, Knockout, Multidisciplinary, Membrane Glycoproteins, Membrane Glycoproteins/ genetics/immunology, Insulin/genetics, hemic and immune systems, Biological Sciences, Autoimmunity/ immunology, medicine.anatomical_structure, B7-1 Antigen, Tumor necrosis factor alpha, Signal Transduction, Pore Forming Cytotoxic Proteins, Antigens, CD95/ genetics/immunology, Receptors, Tumor Necrosis Factor/genetics, T cell, Antigens, CD80/genetics/immunology, Apoptosis/genetics/immunology, CD8-Positive T-Lymphocytes/ immunology, Mice, Transgenic, Pancreas/pathology, Biology, Islets of Langerhans, Immune system, Antigens, CD, medicine, Diabetes Mellitus, Animals, Receptors, Tumor Necrosis Factor, Type II, fas Receptor, Pancreas, Perforin, Tumor Necrosis Factor-alpha, medicine.disease, Disease Models, Animal, Microscopy, Electron, Immunology, biology.protein, Signal Transduction/genetics, Insulitis, CD80

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc2fdce90256c03f184fbc051ec843f5Test
https://pubmed.ncbi.nlm.nih.gov/10618409Test

المؤلفون: David Deperthes, Takao Kataoka, Jürgen Engel, Jacqueline Romero, Nils Holler, Pedro Romero, Pascal Schneider, Jean-Luc Bodmer, Jürg Tschopp

المصدر: Journal of Immunological Methods, vol. 237, no. 1-2, pp. 159-173

مصطلحات موضوعية: Fas Ligand Protein, Recombinant Fusion Proteins, CD40 Ligand, Immunology, Receptors, Fc, Cartilage Oligomeric Matrix Protein, Ligands, Lymphocyte Activation, Jurkat cells, Receptors, Tumor Necrosis Factor, Immunoglobulin G, Fas ligand, Jurkat Cells, Mice, Tumor Cells, Cultured, Animals, Humans, Matrilin Proteins, Immunology and Allergy, Avidity, fas Receptor, CD40 Antigens, Receptor, Glycoproteins, Mice, Knockout, Cartilage oligomeric matrix protein, B-Lymphocytes, Extracellular Matrix Proteins, Membrane Glycoproteins, CD40, biology, Chemistry, Virology, In vitro, Cell biology, Solubility, biology.protein, Antigens, CD40/metabolism, Antigens, CD95/metabolism, B-Lymphocytes/cytology, B-Lymphocytes/drug effects, Extracellular Matrix Proteins/genetics, Extracellular Matrix Proteins/metabolism, Glycoproteins/genetics, Glycoproteins/metabolism, Lymphocyte Activation/drug effects, Membrane Glycoproteins/antagonists & inhibitors, Membrane Glycoproteins/metabolism, Receptors, Fc/genetics, Receptors, Fc/metabolism, Receptors, Tumor Necrosis Factor/genetics, Receptors, Tumor Necrosis Factor/metabolism, Recombinant Fusion Proteins/genetics, Recombinant Fusion Proteins/metabolism

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c2d98abe1adc5f23e0e9aa537d56a090Test
https://serval.unil.ch/notice/serval:BIB_69A67AAF39A6Test