يعرض 1 - 10 نتائج من 10 نتيجة بحث عن '"Polymorphism, Genetic/*physiology"', وقت الاستعلام: 0.97s تنقيح النتائج
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    دورية أكاديمية
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    المصدر: Circulation
    Circulation, Vol. 124, No 8 (2011) pp. 930-9

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    المصدر: Heart and Vessels. 22:1-8

    مصطلحات موضوعية: Carotid Artery Diseases, Male, angiocardiography, AT1R A/C1166 polymorphism, Adult, Aged, Alleles, Carotid Artery Diseases/*genetics/*physiopathology, Coronary Angiography, Coronary Vessels/*physiopathology, Female, Humans, Middle Aged, Peptidyl-Dipeptidase A/*genetics, Polymorphism, Genetic/*physiology, Regional Blood Flow, Renin-Angiotensin System, Tunica Intima/pathology, Tunica Media/pathology, Intima-media thickness, correlation analysis, genotype, Renin-angiotensin gene system, image display, Coronary artery disease, cardiovascular disease, Carotid atherosclerosis, coronary artery blood flow, renin angiotensin aldosterone system, Myocardial infarction, Common carotid artery, statistical significance, clinical article, biology, adult, pathogenesis, artery intima, allele, article, vascular disease, Coronary Vessels, transducer, ACEI/D polymorphism, aged, female, medicine.anatomical_structure, priority journal, risk factor, Cardiology, Tunica Media, Cardiology and Cardiovascular Medicine, coronary artery disease, gene insertion, TIMI, medicine.medical_specialty, regulatory mechanism, angiotensin 1 receptor, dipeptidyl carboxypeptidase, common carotid artery, Peptidyl-Dipeptidase A, artery media, Internal medicine, medicine.artery, medicine, inheritance, controlled study, human, cardiovascular diseases, artery wall, Polymorphism, Genetic, Slow coronary flow, carotid artery, gene deletion, business.industry, disease association, echography, Angiotensin-converting enzyme, medicine.disease, Angiotensin II, thickness, Coronary arteries, enzyme polymorphism, biology.protein, Gene polymorphism, atherosclerosis, Tunica Intima, business

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    المساهمون: Gouilleux, Fabrice, Epidémiologie des maladies chroniques: impact des intéractions gène environnement sur la santé des populations, Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille, Droit et Santé-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Institut de Génétique et de Biologie Moléculaire et Cellulaire (IGBMC), Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), CHU Amiens-Picardie, Université de Picardie Jules Verne (UPJV), Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), ARC, Fondation De France, and Ligand Pharmaceuticals supported this work. The WHO-MONICA population study developed in the North of France was supported by grants from the Conseil Régional du Nord-Pas de Calais, the Fondation pour la Recherche Médicale, ONIVINS, the Parke-Davis Laboratory, the Mutuelle Générale de l’Education Nationale (MGEN), the Réseau National de Santé Publique, the Direction Générale de La Santé, the Institut National de la Santé Et de la Recherche Médicale (INSERM), the Institut Pasteur de Lille and the Unité d’Evaluation du Center Hospitalier et Universitaire de Lille., Antonio Vidal-Puig and Giles S.H. Yeo are acknowledged for the lecture of the manuscript and helpful comments. Odile Vidal is acknowledged for plasmid preparation and Xavier Hermant and Valérie Codron for technical assistance.

    المصدر: Arteriosclerosis, Thrombosis, and Vascular Biology
    Arteriosclerosis, Thrombosis, and Vascular Biology, 2003, 23 (2), pp.289-294. ⟨10.1161/01.atv.0000051382.28752.fe⟩
    Arteriosclerosis, Thrombosis, and Vascular Biology, American Heart Association, 2003, 23 (2), pp.289-294. ⟨10.1161/01.atv.0000051382.28752.fe⟩

    مصطلحات موضوعية: Male, Candidate gene, obesity, [SDV]Life Sciences [q-bio], Peroxisome proliferator-activated receptor, Receptors, Cytoplasmic and Nuclear, PPAR, Linkage Disequilibrium, Exon, Transactivation, 0302 clinical medicine, MESH: DNA-Binding Proteins/genetics, Gene Frequency, MESH: Lipid Metabolism, Inborn Errors/metabolism, STAT5 Transcription Factor, Promoter Regions, Genetic, chemistry.chemical_classification, 0303 health sciences, education.field_of_study, MESH: Middle Aged, STAT, Middle Aged, Milk Proteins, MESH: Body Height/genetics, [SDV] Life Sciences [q-bio], DNA-Binding Proteins, MESH: Receptors, Cytoplasmic and Nuclear/genetics, 030220 oncology & carcinogenesis, Female, Cardiology and Cardiovascular Medicine, MESH: Polymorphism, Genetic/physiology, Adult, medicine.medical_specialty, Population, Molecular Sequence Data, Biology, Lipid Metabolism, Inborn Errors, 03 medical and health sciences, MESH: Transcription Factors/genetics, Internal medicine, medicine, MESH: Transcription Factors/physiology, MESH: DNA-Binding Proteins/physiology, Humans, MESH: Trans-Activators/genetics, MESH: Receptors, Cytoplasmic and Nuclear/physiology, education, Gene, MESH: Linkage Disequilibrium/genetics, 030304 developmental biology, MESH: Trans-Activators/physiology, MESH: Lipid Metabolism, Inborn Errors/genetics, MESH: Humans, MESH: Molecular Sequence Data, Polymorphism, Genetic, MESH: STAT5 Transcription Factor, cholesterol, Promoter, Lipid metabolism, MESH: Adult, MESH: Milk Proteins, MESH: Male, MESH: Promoter Regions, Genetic/physiology, Body Height, Endocrinology, chemistry, growth hormone, Trans-Activators, MESH: Gene Frequency/genetics, MESH: Female, Transcription Factors

    وصف الملف: application/pdf

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