نتائج البحث - "Oliwia W. Zurek"

المساهمون: Groupe Interdisciplinaire de Génoprotéomique Appliquée (GIGA-Research), Université de Liège, Department of Pathology [Stanford], Stanford Medicine, Stanford University-Stanford University, Sean N. Parker Center for Allergy and Asthma Research [Stanford], Institut de Génétique et de Biologie Moléculaire et Cellulaire (IGBMC), Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Technische Universität Dresden = Dresden University of Technology (TU Dresden), Cell and Developmental Biology [San Diego], School of Biological Sciences [Univ California San Diego] (UC San Diego), University of California [San Diego] (UC San Diego), University of California (UC)-University of California (UC)-University of California [San Diego] (UC San Diego), University of California (UC)-University of California (UC), Environmental Molecular Biology Laboratory (RIKEN), RIKEN - Institute of Physical and Chemical Research [Japon] (RIKEN), Walloon Excellence in Life sciences and BIOtechnology [Liège] (WELBIO), Stanford School of Medicine [Stanford], Department of Microbiology and Immunology [Stanford], Pistre, Karine

المصدر: Journal of Clinical Investigation
Journal of Clinical Investigation, 2016, 126 (12), pp.4497-4515. ⟨10.1172/JCI86359⟩

مصطلحات موضوعية: Keratinocytes, 0301 basic medicine, MESH: Guanine Nucleotide Exchange Factors / genetics, [SDV]Life Sciences [q-bio], MESH: Myeloid Differentiation Factor 88 / immunology, MESH: Dermatitis, Contact / immunology, Dermatitis, Contact, MESH: Mice, Knockout, Mice, 0302 clinical medicine, MESH: Humans Keratinocytes / immunology, Guanine Nucleotide Exchange Factors, MESH: Animals, MESH: NF-kappa B / genetics, Mice, Knockout, MESH: Dermatitis, Contact / pathology, integumentary system, MESH: Signal Transduction / immunology, MESH: Guanine Nucleotide Exchange Factors / immunology, Chemistry, NF-kappa B, MESH: Epidermis / pathology, General Medicine, Atopic dermatitis, Phenotype, Cell biology, [SDV] Life Sciences [q-bio], medicine.anatomical_structure, MESH: NF-kappa B / immunology, 030220 oncology & carcinogenesis, MESH: Dermatitis, Contact / genetics, [SDV.IMM]Life Sciences [q-bio]/Immunology, Female, Guanine nucleotide exchange factor, MESH: Signal Transduction / genetics Signal Transduction / immunology, Signal transduction, medicine.symptom, Keratinocyte, Signal Transduction, Research Article, [SDV.IMM] Life Sciences [q-bio]/Immunology, Inflammation, [SDV.BC]Life Sciences [q-bio]/Cellular Biology, 03 medical and health sciences, MESH: Keratinocytes / pathology, medicine, Animals, Humans, MESH: Receptors, Interleukin-1 Type I / immunology, MESH: Mice, [SDV.BC] Life Sciences [q-bio]/Cellular Biology, Allergic contact dermatitis, Receptors, Interleukin-1 Type I, medicine.disease, 030104 developmental biology, Myeloid Differentiation Factor 88, Rab, Epidermis, MESH: Receptors, Interleukin-1 Type I / genetics, MESH: Female, MESH: Myeloid Differentiation Factor 88 / genetics

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::542066fd135b8c7de9d713b77f413dc3Test
https://doi.org/10.1172/jci86359Test

4

Transcription of the Streptococcus pyogenes Hyaluronic Acid Capsule Biosynthesis Operon Is Regulated by Previously Unknown Upstream Elements

المؤلفون: Jovanka M. Voyich, Hadeel Ali, Robert W. Reed, Marina Falaleeva, Natalia Korotkova, Robert L. Watkins, Oliwia W. Zurek, Paul Sumby

المصدر: Infection and Immunity. 82:5293-5307

مصطلحات موضوعية: Blood Bactericidal Activity, Transcription, Genetic, Streptococcus pyogenes, Operon, Sequence analysis, Immunology, Electrophoretic Mobility Shift Assay, Biology, Real-Time Polymerase Chain Reaction, medicine.disease_cause, Microbiology, Genes, Reporter, Transcription (biology), Streptococcal Infections, medicine, Animals, Humans, Electrophoretic mobility shift assay, Regulatory Elements, Transcriptional, Hyaluronic Acid, Gene, Bacterial Capsules, Immune Evasion, Sequence Deletion, Mice, Inbred BALB C, Virulence, Gene Expression Profiling, Genetic Variation, Promoter, Molecular Pathogenesis, Molecular biology, Disease Models, Animal, Infectious Diseases, Female, Parasitology, Terminator (franchise)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b96cc190032c2d8c153fe597849f268bTest
https://doi.org/10.1128/iai.02035-14Test

5

A TNFRSF14-FcɛRI-mast cell pathway contributes to development of multiple features of asthma pathology in mice

المؤلفون: Mindy Tsai, Joseph D. Hernandez, Mang Yu, Sonja Zahner, Stephen B. Montgomery, Axel Roers, Oliwia W Zurek, Nicolas Gaudenzio, Philipp Starkl, Stephen J. Galli, Mitchell Kronenberg, Laurent L. Reber, Marianne K. DeGorter, Riccardo Sibilano

المساهمون: Department of Pathology [Stanford], Stanford Medicine, Stanford University-Stanford University, Department of Genetics [Stanford], Anticorps en thérapie et pathologie - Antibodies in Therapy and Pathology, Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM), Department of Pediatrics [Stanford], Research Center for Molecular Medicine of the Austrian Academy of Sciences [Vienna, Austria] (CeMM ), Austrian Academy of Sciences (OeAW), La Jolla Institute for Immunology [La Jolla, CA, États-Unis], Technische Universität Dresden = Dresden University of Technology (TU Dresden), Sean N. Parker Center for Allergy and Asthma Research [Stanford], This work was supported by US National Institutes of Health (NIH) grants to S.J.G. (U19AI104209 and R01AR067145), M.K. (R01AI61516) and L.L.R. (K99AI110645), fellowships from the Lucile Packard Foundation for Children’s Health to R.S. (UL1 RR025744) and J.D.H. (UL1 TR001085), the Fondation pour la Recherche Medicale (FRM) SPE20130326582 and Philippe foundation to N.G., a Schroedinger Fellowship of the Austrian Science Fund (FWF) J3399-B21 to P.M.S., an NIH postdoctoral fellowship (2T32AI007290-31) to O.W.Z., the Department of Pathology and the Sean N. Parker Center for Allergy and Asthma Research, Stanford University., Pistre, Karine

المصدر: Nature Communications
Nature Communications, 2016, 7, pp.13696. ⟨10.1038/ncomms13696⟩
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)

مصطلحات موضوعية: 0301 basic medicine, Pathology, General Physics and Astronomy, Immunoglobulin E, MESH: Mice, Knockout, MESH: Ovalbumin / immunology, Immunoglobulin G, MESH: Receptors, IgE / metabolism, MESH: Genotype, MESH: Ovalbumin / toxicity, Mice, 0302 clinical medicine, Antigen Sensitization, MESH: Asthma / pathology, MESH: Animals, MESH: Mast Cells / physiology, Mast Cells, Mice, Knockout, MESH: Immunoglobulin G, Multidisciplinary, biology, MESH: Gene Expression Regulation / drug effects, MESH: Bronchoalveolar Lavage Fluid / cytology, MESH: Immunoglobulin E, Chronic inflammation, Mast cell, MESH: Antigens, Dermatophagoides / toxicity, 3. Good health, medicine.anatomical_structure, MESH: Receptors, IgE / genetics, Airway Remodeling, Mucosal immunology, [SDV.IMM]Life Sciences [q-bio]/Immunology, Female, Tumor necrosis factor alpha, Antibody, medicine.symptom, [SDV.IMM.IMM] Life Sciences [q-bio]/Immunology/Immunotherapy, Bronchoalveolar Lavage Fluid, Receptors, Tumor Necrosis Factor, Member 14, MESH: Antigens, Dermatophagoides / immunology, medicine.medical_specialty, Genotype, [SDV.IMM] Life Sciences [q-bio]/Immunology, Ovalbumin, Science, MESH: Asthma / metabolism, Inflammation, Article, Antibodies, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, Mediator, medicine, Animals, Antigens, Dermatophagoides, MESH: Mice, Receptors, IgE, business.industry, MESH: Asthma / chemically induced, MESH: Antibodies, General Chemistry, [SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/Immunotherapy, MESH: Receptors, Tumor Necrosis Factor, Member 14 / genetics, Asthma, respiratory tract diseases, MESH: Airway Remodeling, 030104 developmental biology, Gene Expression Regulation, Immunology, biology.protein, MESH: Receptors, Tumor Necrosis Factor, Member 14 / metabolism, business, MESH: Female, 030215 immunology