-
1دورية أكاديمية
المؤلفون: Migliorini, Domenico, Bogaerts, Sven, Defever, Dieter, Vyas, Rajesh, Denecker, Geertrui, Radaelli, Enrico, Zwolinska, Aleksandra, Depaepe, Vanessa, Hochepied, Tino, Skarnes, William, Marine, Jean-Christophe
المصدر: The Journal of clinical investigation, 121 (4
مصطلحات موضوعية: Sciences biomédicales, Animals, Base Sequence, Cell Proliferation, Female, Heterozygote, Humans, JNK Mitogen-Activated Protein Kinases -- genetics -- metabolism, MAP Kinase Signaling System, Male, Mice, 129 Strain, Inbred C57BL, Mutant Strains, Transgenic, Neoplasms -- etiology -- genetics -- metabolism, Nuclear Proteins -- deficiency -- genetics -- metabolism, Pregnancy, Protein Stability, RNA, Small Interfering -- genetics, Transcription Factor AP-1 -- genetics -- metabolism, Tumor Suppressor Protein p53 -- metabolism, Tumor Suppressor Proteins -- deficiency -- genetics -- metabolism, Ubiquitin-Protein Ligases -- deficiency -- genetics -- metabolism
وصف الملف: 1 full-text file(s): application/pdf
العلاقة: uri/info:doi/10.1172/JCI45784; uri/info:pii/45784; uri/info:pmid/21403399; uri/info:pmcid/PMC3070608; https://dipot.ulb.ac.be/dspace/bitstream/2013/124773/4/PMC3070608.pdfTest; http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/124773Test
-
2دورية أكاديمية
المؤلفون: Jaeger, VM, Wehnert, M, Munck, M, Lu, W, Müller, CR, Neumann, S, Kleinhenz, T, Zhou, Z, Noegel, AA, Lüke, Y, Kandert, S, Karakesisoglou, I, Dabauvalle, MC
مصطلحات موضوعية: Nuclear Proteins - deficiency - genetics - metabolism, Molecular Sequence Data, Nuclear Envelope - metabolism - pathology, Phenotype, Point Mutation, Progeria - genetics - metabolism - pathology, Sequence Homology, Amino Acid, Transcription, Genetic, Microfilament Proteins - deficiency - genetics - metabolism, Nerve Tissue Proteins - deficiency - genetics - metabolism, Amino Acid Sequence, Base Sequence, Cells, Cultured, Chromatin - metabolism, DNA Primers - genetics, Female, Fibroblasts - metabolism - pathology, Gene Deletion, Humans, Lamin Type A - chemistry - genetics - metabolism, Membrane Proteins - deficiency - genetics, Metalloendopeptidases - deficiency - genetics
العلاقة: Human Molecular Genetics; http://www.scopus.com/mlt/select.url?eid=2-s2.0-35748967561&selection=ref&src=s&origin=recordpageTest; Human Molecular Genetics, 2007, v. 16 n. 23, p. 2944-2959; 2959; 140619; WOS:000251036400015; 23; http://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0964-6906&volume=16&spage=2944&epage=59&date=2007&atitle=Nesprin-2+giant+safeguardsTest+nuclear+envelope+architecture+in+LMNA+S143F+progeria+cells; eid_2-s2.0-35748967561; 2944; http://hdl.handle.net/10722/68192Test; 16
الإتاحة: https://doi.org/10.1093/hmg/ddm255Test
http://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0964-6906&volume=16&spage=2944&epage=59&date=2007&atitle=Nesprin-2+giant+safeguardsTest+nuclear+envelope+architecture+in+LMNA+S143F+progeria+cells
http://hdl.handle.net/10722/68192Test -
3مورد إلكتروني
المصدر: The Journal of clinical investigation, 121 (4
مصطلحات الفهرس: Sciences biomédicales, Animals, Base Sequence, Cell Proliferation, Female, Heterozygote, Humans, JNK Mitogen-Activated Protein Kinases -- genetics -- metabolism, MAP Kinase Signaling System, Male, Mice, Mice, 129 Strain, Mice, Inbred C57BL, Mice, Mutant Strains, Mice, Transgenic, Neoplasms -- etiology -- genetics -- metabolism, Nuclear Proteins -- deficiency -- genetics -- metabolism, Pregnancy, Protein Stability, RNA, Small Interfering -- genetics, Transcription Factor AP-1 -- genetics -- metabolism, Tumor Suppressor Protein p53 -- metabolism, Tumor Suppressor Proteins -- deficiency -- genetics -- metabolism, Ubiquitin-Protein Ligases -- deficiency -- genetics -- metabolism, info:eu-repo/semantics/article, info:ulb-repo/semantics/articlePeerReview, info:ulb-repo/semantics/openurl/article
-
4مورد إلكتروني
المصدر: The Journal of clinical investigation, 121 (4
مصطلحات الفهرس: Sciences biomédicales, Animals, Base Sequence, Cell Proliferation, Female, Heterozygote, Humans, JNK Mitogen-Activated Protein Kinases -- genetics -- metabolism, MAP Kinase Signaling System, Male, Mice, Mice, 129 Strain, Mice, Inbred C57BL, Mice, Mutant Strains, Mice, Transgenic, Neoplasms -- etiology -- genetics -- metabolism, Nuclear Proteins -- deficiency -- genetics -- metabolism, Pregnancy, Protein Stability, RNA, Small Interfering -- genetics, Transcription Factor AP-1 -- genetics -- metabolism, Tumor Suppressor Protein p53 -- metabolism, Tumor Suppressor Proteins -- deficiency -- genetics -- metabolism, Ubiquitin-Protein Ligases -- deficiency -- genetics -- metabolism, info:eu-repo/semantics/article, info:ulb-repo/semantics/articlePeerReview, info:ulb-repo/semantics/openurl/article
النص الكامل