التفاصيل البيبلوغرافية
| العنوان: |
Communicating Hydrocephalus in Systemic Lupus Erythematosus. |
| المؤلفون: |
Albayrak, Ramazan, Albayram, Sait, Selçuk, Hakan, Saİp, Sebahattin, Yilmaz, Halit, Deǧirmenci, Bumin, IŘlak, Civan |
| المصدر: |
Turkish Journal of Medical Sciences; 2006, Vol. 36 Issue 5, p323-326, 4p |
| مصطلحات موضوعية: |
HYDROCEPHALUS, BRAIN diseases, CENTRAL nervous system diseases, SYSTEMIC lupus erythematosus, AUTOIMMUNE diseases, VASCULAR diseases |
| الملخص (بالإنجليزية): |
Central nervous system (CNS) involvement has been recognized as the second leading cause of death in patients with systemic lupus erythematosus (SLE), although hydrocephalus is seen very rarely. We report a case of communicating hydrocephalus in a 24-year-old woman having previously diagnosed SLE without antiphospholipid antibody syndrome or cerebral venous angiographic abnormality. We propose direct damagethrombosis of small sized venous structures or immune complex deposition within arachnoid villi, which impair cerebrospinal fluid flow, as a possible, yet unproven, pathophysiologic mechanism for hydrocephalus in SLE. [ABSTRACT FROM AUTHOR] |
| Abstract (Turkish): |
Sistemik lupus eritematosumda ölüm sebebi olarak santral sinir sistemi tutulumu ikinci sıklıkta tanımlanmasına raǧmen hidrosefali oldukşa nadir görülebilir. Daha önce SLE tanısı konan, antifosfolipid antikor sendromu olmayan ve serebral venöz anjiografisi normal olan 24 yaşındaki bir hastada komminikan hidrosefali vakasını sunduk. Araknoid villi işerisindeki immune complex birikimi yada küşük şaplı venlerin trombozu beyin omurilik sıvısının akımını bozabilir. Bu durum SLE'de izlenen hidrosefalinin patofizyolojik mekanizması aşıklayabilir. [ABSTRACT FROM AUTHOR] |
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| قاعدة البيانات: |
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