نتائج البحث - "Daniela Pucciarelli"

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دورية أكاديمية

A high-throughput screen of real-time ATP levels in individual cells reveals mechanisms of energy failure.

المؤلفون: Bryce A Mendelsohn, Neal K Bennett, Maxwell A Darch, Katharine Yu, Mai K Nguyen, Daniela Pucciarelli, Maxine Nelson, Max A Horlbeck, Luke A Gilbert, William Hyun, Martin Kampmann, Jean L Nakamura, Ken Nakamura

المصدر: PLoS Biology, Vol 16, Iss 8, p e2004624 (2018)

مصطلحات موضوعية: Biology (General), QH301-705.5

وصف الملف: electronic resource

العلاقة: http://europepmc.org/articles/PMC6110572?pdf=renderTest; https://doaj.org/toc/1544-9173Test; https://doaj.org/toc/1545-7885Test

الوصول الحر: https://doaj.org/article/e811f8fca40f46dda21b805657c1f66eTest

View record in DOAJ

النص الكامل

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Fig. S2 from Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Jean L. Nakamura, Gary L. Johnson, Kevin Shannon, D. Wade Clapp, Sourav Bandyopadhyay, Ganesh Krishnamurthi, Hiroki J. Nakaoka, Chi Zhang, Benjamin Huang, Steven P. Angus, Daniela Pucciarelli

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9059e9dd951ee033455a93e1ba63da9fTest
https://doi.org/10.1158/1535-7163.22518777.v1Test

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Supplementary Table 2 from Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Jean L. Nakamura, Gary L. Johnson, Kevin Shannon, D. Wade Clapp, Sourav Bandyopadhyay, Ganesh Krishnamurthi, Hiroki J. Nakaoka, Chi Zhang, Benjamin Huang, Steven P. Angus, Daniela Pucciarelli

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ce466ec2f39826c2bcb41e0c1a2c4218Test
https://doi.org/10.1158/1535-7163.22518738Test

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Supplementary Materials and Methods from Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Jean L. Nakamura, Gary L. Johnson, Kevin Shannon, D. Wade Clapp, Sourav Bandyopadhyay, Ganesh Krishnamurthi, Hiroki J. Nakaoka, Chi Zhang, Benjamin Huang, Steven P. Angus, Daniela Pucciarelli

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1cd937f798c4b530b4b075f6af4fdb40Test
https://doi.org/10.1158/1535-7163.22518744.v1Test

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Data from Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Jean L. Nakamura, Gary L. Johnson, Kevin Shannon, D. Wade Clapp, Sourav Bandyopadhyay, Ganesh Krishnamurthi, Hiroki J. Nakaoka, Chi Zhang, Benjamin Huang, Steven P. Angus, Daniela Pucciarelli

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::77b9200fd99429f0de8d13ec16a7d102Test
https://doi.org/10.1158/1535-7163.c.6542103Test

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Supplementary Table 1 from Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Jean L. Nakamura, Gary L. Johnson, Kevin Shannon, D. Wade Clapp, Sourav Bandyopadhyay, Ganesh Krishnamurthi, Hiroki J. Nakaoka, Chi Zhang, Benjamin Huang, Steven P. Angus, Daniela Pucciarelli

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::83c4b691074ad3341b8587cc857033d3Test
https://doi.org/10.1158/1535-7163.22518741.v1Test

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Differentiating Radiation Necrosis from Brain Tumor Using Hyperpolarized Carbon-13 MR Metabolic Imaging

المؤلفون: Juhyun Song, Ilwoo Park, Yun Hyeon Kim, Kyung-Hwa Lee, Jin Myung Choi, Seul Kee Kim, Daniela Pucciarelli, Woong Yoon, Jean L. Nakamura, Shin Jung

المصدر: Molecular Imaging and Biology. 23:417-426

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::68f9df9c6ea34b14d89470752c95dd75Test
https://doi.org/10.1007/s11307-020-01574-wTest

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Nf1-Mutant Tumors Undergo Transcriptome and Kinome Remodeling after Inhibition of either mTOR or MEK

المؤلفون: Gary L. Johnson, Kevin Shannon, Jean L. Nakamura, Ganesh Krishnamurthi, Daniela Pucciarelli, Steven P. Angus, Sourav Bandyopadhyay, Benjamin J. Huang, Hiroki J. Nakaoka, Chi Zhang, D. Wade Clapp

المصدر: Molecular cancer therapeutics, vol 19, iss 11
Mol Cancer Ther

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Drug Resistance, Drug resistance, Loss of heterozygosity, Transcriptome, Mice, 0302 clinical medicine, Models, Kinome, Cancer, Tumor, Neurofibromin 1, Effector, High-Throughput Nucleotide Sequencing, Pharmacology and Pharmaceutical Sciences, Phenotype, Oncology, 5.1 Pharmaceuticals, 030220 oncology & carcinogenesis, Development of treatments and therapeutic interventions, Signal Transduction, congenital, hereditary, and neonatal diseases and abnormalities, Neurofibromatosis 1, Oncology and Carcinogenesis, Context (language use), Biology, Article, Cell Line, Neurofibromatosis, 03 medical and health sciences, Rare Diseases, Genetics, Animals, Humans, Oncology & Carcinogenesis, Protein Kinase Inhibitors, PI3K/AKT/mTOR pathway, Animal, Gene Expression Profiling, Neurosciences, Computational Biology, Biological, nervous system diseases, Orphan Drug, Good Health and Well Being, 030104 developmental biology, Disease Models, Mutation, Cancer research, Neoplasm, Protein Kinases

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd615db499ff0a87ce90f0128a2ca35fTest
https://doi.org/10.1158/1535-7163.mct-19-1017Test

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A high-throughput screen of real-time ATP levels in individual cells reveals mechanisms of energy failure

المؤلفون: Katharine Yu, Maxwell A. Darch, Maxine Nelson, Max A. Horlbeck, Neal K. Bennett, Mai K. Nguyen, William C. Hyun, Martin Kampmann, Ken Nakamura, Jean L. Nakamura, Luke A. Gilbert, Bryce A. Mendelsohn, Daniela Pucciarelli

المساهمون: Tian, Rong

المصدر: PLoS biology, vol 16, iss 8
PLoS Biology, Vol 16, Iss 8, p e2004624 (2018)

مصطلحات موضوعية: 0301 basic medicine, Mitochondrial DNA, QH301-705.5, Ubiquinone, 1.1 Normal biological development and functioning, Mitochondrion, Biology, Medical and Health Sciences, General Biochemistry, Genetics and Molecular Biology, Mitochondrial Proteins, 03 medical and health sciences, chemistry.chemical_compound, Adenosine Triphosphate, Single-cell analysis, Affordable and Clean Energy, Underpinning research, Complementary and Integrative Health, Genetics, Humans, 2.1 Biological and endogenous factors, Glycolysis, Clustered Regularly Interspaced Short Palindromic Repeats, Biology (General), Aetiology, Gene, Nutrition, Gene knockdown, General Immunology and Microbiology, Agricultural and Veterinary Sciences, General Neuroscience, Biological Sciences, Cell biology, Mitochondria, High-Throughput Screening Assays, 030104 developmental biology, chemistry, Generic health relevance, Single-Cell Analysis, General Agricultural and Biological Sciences, Energy Metabolism, Adenosine triphosphate, Genetic screen, Biotechnology, Developmental Biology

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e71f5fc79788a041bd562e769a5b30aTest
https://escholarship.org/uc/item/1vp7g6cnTest

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Hypoxia increases the heterogeneity of melanoma cell populations and affects the response to vemurafenib

المؤلفون: Maria Bartosova, Christine Hafner, Nina Lengger, Martina Takacova, Heimo Breiteneder, Silvia Pastorekova, Lucia Csaderova, Daniela Pucciarelli

المصدر: Molecular Medicine Reports. 13:3281-3288

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Indoles, Biochemistry, Phosphatidylinositol 3-Kinases, 0302 clinical medicine, Cell Movement, RNA, Small Interfering, Extracellular Signal-Regulated MAP Kinases, Vemurafenib, Melanoma, Sulfonamides, Cell cycle, Cell Hypoxia, Oncology, Hypoxia-inducible factors, 030220 oncology & carcinogenesis, Molecular Medicine, RNA Interference, Signal transduction, Signal Transduction, medicine.drug, Proto-Oncogene Proteins B-raf, medicine.medical_specialty, Antineoplastic Agents, Biology, Real-Time Polymerase Chain Reaction, 03 medical and health sciences, Oxygen Consumption, Antigens, Neoplasm, Cell Line, Tumor, Internal medicine, Genetics, medicine, Humans, Carbonic Anhydrase IX, Protein kinase A, Molecular Biology, Protein kinase B, Cell Proliferation, Cell growth, JNK Mitogen-Activated Protein Kinases, Hypoxia-Inducible Factor 1, alpha Subunit, medicine.disease, 030104 developmental biology, Endocrinology, Cancer research, Proto-Oncogene Proteins c-akt

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1a3bb2a7b07f7a036775701abffd1b2cTest
https://doi.org/10.3892/mmr.2016.4888Test

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