نتائج البحث - "Akt Inhibitor MK2206"

المصدر: International Journal of Molecular Sciences, Vol 22, Iss 10954, p 10954 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 20

مصطلحات موضوعية: Afatinib, androgen receptor (AR), 1607 Spectroscopy, Ligands, Receptor tyrosine kinase, Akt Inhibitor MK2206, AR antagonist, Epidermal growth factor receptor, Phosphorylation, Biology (General), Spectroscopy, biology, Kinase, Chemistry, Brain Neoplasms, General Medicine, epidermal growth factor receptor (EGFR), Computer Science Applications, ErbB Receptors, Receptors, Androgen, Benzamides, 1606 Physical and Theoretical Chemistry, medicine.drug, Signal Transduction, QH301-705.5, 1503 Catalysis, 610 Medicine & health, Catalysis, Article, Inorganic Chemistry, protein kinase B (AKT), Cell Line, Tumor, Nitriles, Phenylthiohydantoin, medicine, Androgen Receptor Antagonists, 1312 Molecular Biology, 1706 Computer Science Applications, Humans, Physical and Theoretical Chemistry, Molecular Biology, Protein kinase B, Protein Kinase Inhibitors, QD1-999, 1604 Inorganic Chemistry, Organic Chemistry, glioblastoma, 10040 Clinic for Neurology, Androgen receptor, Gene Expression Regulation, Cancer research, biology.protein, Proto-Oncogene Proteins c-akt, 1605 Organic Chemistry

وصف الملف: ijms-22-10954-v2.pdf - application/pdf; application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1080f31d0c67a422fa343d09473a68caTest
https://www.zora.uzh.ch/id/eprint/209437Test/

9

A PLCB1-PI3K-AKT Signaling Axis Activates EMT to Promote Cholangiocarcinoma Progression

المؤلفون: Wei Wang, Yifeng Cui, Hongrui Guo, Dehai Wu, Yao Liu, Ning Zhang, Lianxin Liu, Bo Zhang, Shuhang Liang, Mengyang Xin, Jiabei Wang, Xianying Li, Linmao Sun, Yufeng Liu, Shugeng Zhang, Huanran Zhou, Yiqi Wang, Kun Ma

المصدر: Cancer research. 81(23)

مصطلحات موضوعية: Male, Cancer Research, Epithelial-Mesenchymal Transition, Carcinogenesis, Phospholipase C beta, Mice, Nude, Apoptosis, Biology, Phospholipase, medicine.disease_cause, Deoxycytidine, Cholangiocarcinoma, Mice, Phosphatidylinositol 3-Kinases, Downregulation and upregulation, PABPC1, Cell Movement, parasitic diseases, Antineoplastic Combined Chemotherapy Protocols, Akt Inhibitor MK2206, medicine, Biomarkers, Tumor, Tumor Cells, Cultured, Animals, Humans, Neoplasm Invasiveness, Protein kinase B, PI3K/AKT/mTOR pathway, Cell Proliferation, Cisplatin, Mice, Inbred BALB C, fungi, Prognosis, Xenograft Model Antitumor Assays, Gemcitabine, Gene Expression Regulation, Neoplastic, Oncology, Bile Duct Neoplasms, Drug Resistance, Neoplasm, Cancer research, Proto-Oncogene Proteins c-akt, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3b4d34cfaee8879ad6a1ed2d36b747d2Test
https://pubmed.ncbi.nlm.nih.gov/34580062Test

10

LY‑294002 enhances the chemosensitivity of liver cancer to oxaliplatin by blocking the PI3K/AKT/HIF‑1α pathway

المؤلفون: Ruyue Xu, Yongfang Ma, Amin Li, Li Song, Weiya Cao, Yinghai Xie, Wenpeng Cai, Xiaolong Tang, Yinci Zhang, Shuping Zhou

المصدر: Molecular Medicine Reports

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Apoptosis, Cell Cycle Proteins, Biochemistry, Phosphatidylinositol 3-Kinases, chemistry.chemical_compound, 0302 clinical medicine, Akt Inhibitor MK2206, hypoxia inducible factor-1α, Enzyme Inhibitors, Chemistry, TOR Serine-Threonine Kinases, Cell Cycle, Liver Neoplasms, Ribosomal Protein S6 Kinases, 70-kDa, Drug Synergism, Articles, hepatocellular carcinoma, Hep G2 Cells, MK-2206, Oncology, 030220 oncology & carcinogenesis, Molecular Medicine, Liver cancer, Heterocyclic Compounds, 3-Ring, Signal Transduction, Morpholines, Antineoplastic Agents, LY-294002, 03 medical and health sciences, Genetics, medicine, Humans, Molecular Biology, Protein kinase B, PI3K/AKT/mTOR pathway, Adaptor Proteins, Signal Transducing, Cell Proliferation, PI3K/AKT, Akt/PKB signaling pathway, oxaliplatin, Cancer, Hypoxia-Inducible Factor 1, alpha Subunit, medicine.disease, 030104 developmental biology, Chromones, Drug Resistance, Neoplasm, Cancer cell, Cancer research, Proto-Oncogene Proteins c-akt

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a5f4d2a3fccc03d45824d66ad2453ccTest
https://doi.org/10.3892/mmr.2021.12147Test

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