يعرض 1 - 10 نتائج من 49 نتيجة بحث عن '"жировой обмен"', وقت الاستعلام: 0.83s تنقيح النتائج
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    دورية أكاديمية

    المؤلفون: Е. G. Zarubina, I. A. Gribanov

    المصدر: Вестник медицинского института «Реавиз»: Реабилитация, врач и здоровье, Vol 0, Iss 1, Pp 107-110 (2020)

    وصف الملف: electronic resource

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    المساهمون: This work was supported by State Budgeted Project 0324-2019-0041 and the Russian Foundation for Basic Research, project 20-04-00579.

    المصدر: Vavilov Journal of Genetics and Breeding; Том 24, № 4 (2020); 441-445 ; Вавиловский журнал генетики и селекции; Том 24, № 4 (2020); 441-445 ; 2500-3259 ; 2500-0462

    وصف الملف: application/pdf

    العلاقة: https://vavilov.elpub.ru/jour/article/view/2653/1399Test; Еремина М.А., Карпова Е.К., Раушенбах И.Ю., Пирожкова Д.С., Андреенкова О.В., Грунтенко Н.Е. Влияние мутаций генов инсулинового сигнального каскада на изменение уровня углеводов у самок Drosophila melanogaster при тепловом стрессе. Генетика. 2019;55(4):485-488. DOI 10.1134/S0016675819030068.; Ростовцев В.Р., Резник Г.Е. Количественное определение липидных фракций в крови. Лаб. дело. 1982;4:26-29.; Abdullah S., Davies S., Wall R. Spectrophotometric analysis of lipid used to examine the phenology of the tick Ixodes ricinus. Parasit Vectors. 2018;11:523. DOI 10.1186/s130-71-018-3102-3.; Al-Anzi B., Zinn K. Colorimetric measurement of triglycerides cannot provide an accurate measure of stored fat content in Drosophila. PLoS One. 2010;5(8):e12353. DOI 10.1371/journal.pone.0012353.; Álvarez-Rendón J.P., Salceda R., Riesgo-Escovar J.R. Drosophila melanogaster as a model for diabetes type 2 progression. BioMed. Res. 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The impact of FOXO on dopamine and octopamine metabolism in Drosophila under normal and heat stress conditions. Biol. Open. 2016;5:1706-1711. DOI 10.1242/bio.022038.; Gruntenko N.E., Rauschenbach I.Y. The role of insulin signalling in the endocrine stress response in Drosophila melanogaster: a minireview. Gen. Comp. Endocrinol. 2018;258:134-139. DOI 10.1016/j.ygcen.2017.05.019.; Hildebrandt A., Bickmeyer I., Kühnlein R.P. Reliable Drosophila body fat quantification by a coupled colorimetric assay. PLoS One. 2011; 6(9):e23796. DOI 10.1371/journal.pone.0023796.; Kleinert M., Clemmensen C., Hofmann S.M., Moore M.C., Renner S., Woods S.C., Huypens P., Beckers J., de Angelis M.H., Schürmann A., Bakhti M., Klingenspor M., Heiman M., Cherrington A.D., Ristow M., Lickert H., Wolf E., Havel P.J., Müller T.D., Tschöp M.H. Animal models of obesity and diabetes mellitus. Nat. Rev. Endocrinol. 2018;14(3):140-162. DOI 10.1038/nrendo.2017.161.; Knight J.A., Anderson S., Rawle J.M. Chemical basis of the sulfo-phospho-vanillin reaction for estimating total serum lipids. Clin. Chem. 1972;18(3):199-202.; Lee C.L. What we can learn from the energetic levels of insects: a guide and review. Ann. Entomol. Soc. Am. 2019;112(3):220-226. DOI 10.1093/aesa/say051.; Liu Z., Huang X. Lipid metabolism in Drosophila: development and disease. Acta Biochim. Biophys. Sin. (Shanghai). 2013;45(1):44-50. DOI 10.1093/abbs/gms105.; Lu Y., Ludsin S.A., Fanslow D.L., Pothoven S.A. Comparison of three microquantity techniques for measuring total lipids in fish. Can. J. Fish. Aquat. Sci. 2008;65:2233-2241. DOI 10.1139/F08-135.; Murillo-Maldonado J.M., Sánchez-Chávez G., Salgado L.M., Salceda R., Riesgo-Escovar J.R. Drosophila insulin pathway mutants affect visual physiology and brain function besides growth, lipid, and carbohydrate metabolism. Diabetes. 2011;60(5):1632-1636. DOI 10.2337/db10-1288.; Musselman L.P., Kühnlein R.P. Drosophila as a model to study obesity and metabolic disease. J. Exp. Biol. 2018;221(Suppl.1):jeb163881. DOI 10.1242/jeb.163881.; Park J., Jeong H.J., Yoon E.Y., Moon S.J. Easy and rapid quantification of lipid contents of marine dinoflagellates using the sulpho-phosphovanillin method. Algae. 2016;31(4):391-401. DOI 10.4490/algae.2016.31.12.7.; Patel A., Antonopoulou I., Enman J., Rova U., Christakopoulos P., Matsakas L. Lipids detection and quantification in oleaginous microorganisms: an overview of the current state of the art. BMC Chem. Eng. 2019;1:13. DOI 10.1186/s42480-019-0013-9.; Rauschenbach I.Yu., Karpova E.K., Burdina E.V., Adonyeva N.V., Bykov R.A., Ilinsky Y.Y., Menshanov P.N., Gruntenko N.E. Insulinlike peptide DILP6 regulates juvenile hormone and dopamine metabolism in Drosophila females. Gen. Comp. Endocrinol. 2016;243: 1-9. DOI 10.1016/j.ygcen.2016.11.0040016-6480.; Tennessen J.M., Barry W.E., Cox J., Thummel C.S. Methods for studying metabolism in Drosophila. Methods. 2014;68(1):105-115. DOI 10.1016/j.ymeth.2014.02.034.; Trinh I., Boulianne G.L. Modeling obesity and its associated disorders in Drosophila. Physiology. 2013;28:117-124. DOI 10.1152/physiol.00025.2012.; Van Handel E. Rapid determination of total lipids in mosquitoes. J. Am. Mosq. Control Assoc. 1985;1:302-304.; https://vavilov.elpub.ru/jour/article/view/2653Test

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    وصف الملف: application/pdf

    العلاقة: Scopus; Бродовская Т.О., Ковин Е.А., Баженова О.В., Гришина И.Ф., Перетолчина Т.Ф. Предикторы висцерального ожирения у пациентов с синдромом обструктивного апноэ сна и нормальной массой тела // Ожирение и метаболизм. — 2019. — Т.16. — №2. — С.29-35.; http://elib.usma.ru/handle/usma/7224Test

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    المصدر: Vavilov Journal of Genetics and Breeding; Том 22, № 2 (2018); 265-272 ; Вавиловский журнал генетики и селекции; Том 22, № 2 (2018); 265-272 ; 2500-3259 ; 2500-0462

    وصف الملف: application/pdf

    العلاقة: https://vavilov.elpub.ru/jour/article/view/1771/1146Test; An J.J., Rhee Y., Kim S.H., Kim D.M., Han D.H., Hwang J.H., Jin Y.J., Cha B.S., Baik J.H., Lee W.T., Lim S.K. Peripheral effect of α-melanocyte-stimulating hormone on fatty acid oxidation in skeletal muscle. J. Biol. Chem. 2007;282(5):2862-2870. DOI 10.1074/jbc. M603454200.; Bahzan N.M., Yakovleva T.V., Baginskaya N.V., Shevchenko A.Yu., Makarova E.N. Changes of lipid-carbohydrate metabolism during the development of melanocortin obesity in mice with the Agouti Yellow mutation. Rossiyskiy fiziologicheskiy zhurnal im. I.M. Sechenova = I.M. Sechenov Physiological Journal. 2005;91(12):1445-1453. (in Russian); Bonnefont J.P., Djouadi F., Prip-Buus C., Gobin S., Munnich A., Bastin J. Carnitine palmitoyltransferases 1 and 2: biochemical, molecular and medical aspects. Mol. Asp. Med. 2004;25(5):495-520. DOI 10.1016/j.mam.2004.06.004.; Brito M.N., Brito N.A., Baro D.J., Song C.K., Bartness T.J. Differential activation of the sympathetic innervation of adipose tissues by melanocortin receptor stimulation. Endocrinology. 2007;148(11):5339-5347. DOI 10.1210/en.2007-0621.; Cannon B., Nedergaard J.A.N. Brown adipose tissue: function and physiological significance. Physiol. Rev. 2004;84(1):277-359. DOI 10.1152/physrev.00015.2003.; Carmen G.-Y., Victor S.-M. Signalling mechanisms regulating lipolysis. Cell. Sign. 2006;18:401-408. DOI 10.1016/j.cellsig.2005.08.009. Carroll L., Voisey J., Van Daal A. Mouse models of obesity. Clin. Dermatol. 2004;22(4):345-349. DOI 10.1016/j.clindermatol.2004.01.004.; Chiu S., Fisler J.S., Espinal G.M., Havel P.J., Stern J.S., Warden C.H. The yellow agouti mutation alters some but not all responses to diet and exercise. Obes. Res. 2004;12(8):1243-1255.; Claycombe K.J., Xue B.Z., Mynatt R.L., Zemel M.B., Moustaid-Moussa N. Regulation of leptin by agouti. Physiol. Genomics. 2000;2(3): 101-105.; Cummings D.E., Merriam G.R. 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Signal transducer and activator of transcription-3 is required in hypothalamic agouti-related protein/neuropeptide Y neurons for normal energy homeostasis. Endocrinology. 2008;149(7):3346-3354. DOI 10.1210/en.2007-0945.; Im S.S., Kwon S.K., Kim T.H., Kim H.I., Ahn Y.H. Regulation of glucose transporter type 4 isoform gene expression in muscle and adipocytes. IUBMB Life. 2007;59(3):134-145. DOI 10.1080/ 15216540701313788.; Karpe F., Dickmann J.R., Frayn K.N. Fatty acids, obesity, and insulin resistance: time for a reevaluation. Diabetes. 2011;60(10):2441- 2449. DOI 10.2337/db11-0425.; Kelly D.M., Jones T.H. Testosterone: A metabolic hormone in health and disease. J. Endocrin. 2013;217(3):R25-R45. DOI 10.1530/JOE12-0455.; Kersten S. Physiological regulation of lipoprotein lipase. Biochim. Biophys. Acta (BBA)-Molec. Сell. Biol. Lipids. 2014;1841(7):919-933. DOI 10.1016/j.bbalip.2014.03.013.; Kim J.Y., Tillison K., Lee J.-H., Rearick D.A., Smas C.M. The adipose tissue triglyceride lipase ATGL/PNPLA2 is downregulated by insulin and TNF-α in 3T3-L1 adipocytes and is a target for transactivation by PPARγ. Am. J. Physiol. Endocr. Metab. 2006;291:E115- E127. DOI 10.1152/ajpendo.00317.2005.; Kim T., He L., Johnson M.S., Li Y., Zeng L., Ding Y., Long Q., Moore J.F., Sharer J.D., Nagy T.R., Young M.E., Wood P.A., Yang Q. Carnitine palmitoyltransferase 1b deficiency protects mice from diet-induced insulin resistance. J. Diabetes Metab. 2014;5(4):361. DOI 10.4172/2155-6156.1000361.; Lass A., Zimmermann R., Oberer M., Zechner R. Lipolysis – а highly regulated multi-enzyme complex mediates the catabolism of cellular fat stores. Prog. Lipid. Res. 2011;50:14-27. DOI 10.1016/j.plipres. 2010.10.004.; Lee M.S., Kim I.H., Kim Y. Effects of eicosapentaenoic acid and docosahexaenoic acid on uncoupling protein 3 gene expression in C2C12 muscle cells. Nutrients. 2013;5(5):1660-1671. DOI 10.3390/ nu5051660.; Lee Y.S. 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Richelsen B., Pedersen S.B., Kristensen K., Børglum J.D., Nørrelund H., Christiansen J.S., Jørgensen J.O. Regulation of lipoprotein lipase and hormone-sensitive lipase activity and gene expression in adipose and muscle tissue by growth hormone treatment during weight loss in obese patients. Metab. Clin. Exp. 2000;49(7):906- 911. DOI 10.1053/meta.2000.6738.; Shaw A.M., Irani B.G., Moore M.C., Haskell-Luevano C., Millard W.J. Ghrelin-induced food intake and growth hormone secretion are altered in melanocortin 3 and 4 receptor knockout mice. Peptides. 2005;26(10):1720-1727.; Shen Y., Xu X., Yue K., Xu G. Effect of different exercise protocols on metabolic profiles and fatty acid metabolism in skeletal muscle in high-fat diet-fed rats. Obesity. 2015;23(5):1000-1006. DOI 10.1002/ oby.21056.; Shi W., Hu S., Wang W., Zhou X., Qiu W. Skeletal muscle-specific CPT1 deficiency elevates lipotoxic intermediates but preserves insulin sensitivity. J. Diabetes. Res. 2013;2013:163062. DOI 10.1155/ 2013/163062.; Silva A.A., Carmo J.M., Wang Z., Hall E.J. The brain melanocortin system, sympathetic control, and obesity hypertension. Physiology. 2014;29(3):196-202. DOI 10.1152/physiol.00061.2013.; Slocum N., Durrant J.R., Bailey D., Yoon L., Jordan H., Barton J., Brown R., Clifton H., Milliken L., Harrington T., Kimbrough W., Faber C., Cariello C.A., Elangb. N. Responses of brown adipose tissue to diet-induced obesity, exercise, dietary restriction and ephedrine treatment. Exp. Toxicol. Pathol. 2013;65(5):549-557. DOI 10.1016/j.etp.2012.04.001.; Talbot D.A., Lambert A.J., Brand M.D. Production of endogenous matrix superoxide from mitochondrial complex I leads to activation of uncoupling protein 3. FEBS Lett. 2004;556(1):111-115.; Tare R.S., Oreffo R.O., Sato K., Rauvala H., Clarke N.M., Roach H.I. Effects of targeted overexpression of pleiotrophin in postnatal bone development. Biochem. Biophys. Res. Com. 2002;298(3):324-332.; Varlamov O., Bethea C.L., Roberts C.T. Sex-specific differences in lipid and glucose metabolism. Front. Endocrinol. 2014;5:241. DOI 10.3389/fendo.2014.00241.; Waalen J. The genetics of human obesity. Transl. Res. 2014;164(4):293- 301. DOI 10.1038/nrg1556.; Wolff G.L., Kodell R.L., Kaput J.A., Visek W.J. Caloric restriction abolishes enhanced metabolic efficiency induced by ectopic agouti protein in yellow mice. Exp. Biol. Med. 1999a;221(2):99-104.; Wolff G.L., Roberts D.W., Mountjoy K.G. Physiological consequences of ectopic agouti gene expression: the yellow obese mouse syndrome. Physiol. Genomics. 1999b;1(3):151-163.; Yakar S., Isaksson O. Regulation of skeletal growth and mineral acquisition by the GH/IGF-1 axis. Lessons from mouse models. Growth Horm. IGF Res. 2016;28:26-42. DOI 10.1016/j.ghir.2015.09.004.; Yang J.Y., Koo J.H., Yoon H.Y., Lee J.H., Park B.H., Kim J.S., Chi M.S., Park J.W. Effect of scopoletin on lipoprotein lipase activity in 3T3- L1 adipocytes. Int. J. Mol. 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    المصدر: Vavilov Journal of Genetics and Breeding; Том 20, № 6 (2016); 918-924 ; Вавиловский журнал генетики и селекции; Том 20, № 6 (2016); 918-924 ; 2500-3259 ; 2500-0462

    وصف الملف: application/pdf

    العلاقة: https://vavilov.elpub.ru/jour/article/view/872/868Test; Ashrafi S., Betley J.N., Comer J.D., Brenner-Morton S., Bar V., Shimoda Y., Watanabe K., Peles E., Jessell T.M., Kaltschmidt J.A. Neuronal Ig/Caspr recognition promotes the formation of axoaxonic synapses in mouse spinal cord. Neuron. 2014;81(1):120-129.; Burbach J.P.H., van der Zwaag B. Contact in the genetics of autism and schizophrenia. Trends Neurosci. 2009;32:69-72.; Burkov I., Serova I., Battulin N., Smirnov A., Babkin I., Andreeva L., Dvoryanchikov G., Serov O. Expression of the human granulocytemacrophage colony stimulating factor (hGM-CSF) gene under control of the 5′-regulatory sequence of the goat alpha-S1-casein gene with and without a MAR element in transgenic mice. Transgenic Res. 2013;22(5):949-964.; Gogliotti R.G., Lutz C., Jorgensen M., Huebsch K., Koh S., Didonato C.J. Characterization of a commonly used mouse model of SMA reveals increased seizure susceptibility and heightened fear response in FVB/N mice. Neurobiol. Dis. 2011;43(1):142-151. DOI 10.1016/j.nbd.2011.03.002.; Fernandez T., Morgan T., Davis N., Klin A., Morris A., Farhi A., Lifton R.P., State M.W. Disruption of Contactin 4 (CNTN4) results in developmental delay and other features of 3p deletion syndrome. Am. J. Hum. Genet. 2008;82:1385.; Fu C., Begum K., Overbeek P.A. Primary ovarian insufficiency induced by Fanconi anemia E mutation in a mouse model. PLoS ONE. 2016;11(3):e0144285. DOI 10.1371/journal.pone.0144285.; Kleijer K.T., Zuko A., Shimoda Y., Watanabe K., Burbach J.P. Contactin-5 expression during development and wiring of the thalamocortical system. Neuroscience. 2015;310:106-113.; Li H., Takeda Y., Niki H., Ogawa J., Kobayashi S., Kai N., Akasaka K., Asano M., Sudo K., Iwakura Y., Watanabe K. Aberrant responses to acoustic stimuli in mice deficient for neural recognition molecule NB-2. Eur. J. Neurosci. 2003;17(5):929-936.; Lionel A.C., Crosbie J., Barbosa N., Goodale T., Thiruvahindrapuram B., Rickaby J., Gazzellone M., Carson A.R., Howe J.L., Wang Z., Wei J., Stewart A.F., Roberts R., McPherson R., Fiebig A., Franke A., Schreiber S., Zwaigenbaum L., Fernandez B.A., Roberts W., Arnold P.D., Szatmari P., Marshall C.R., Schachar R., Scherer S.W. Rare copy number variation discovery and cross- disorder comparisons identify risk genes for ADHD. Sci. Transl. Med. 2011;3(95):95ra75. DOI 10.1126/scitranslmed.3002464.; Mattson D.L. Comparison of arterial blood pressure in different strains of mice. Am. J. Hypertens. 2001;14:405-408.; Morrow E.M., Yoo S.Y., Flavell S.W., Kim T.K., Lin Y., Hill R.S., Mukaddes N.M., Balkhy S., Gascon G., Hashmi A., Al-Saad S., Ware J., Joseph R.M., Greenblatt R., Gleason D., Ertelt J.A., Apse K.A., Bodell A., Partlow J.N., Barry B., Yao H., Markianos K., Ferland R.J., Greenberg M.E., Walsh C.A. Identifying autism loci and genes by tracing recent shared ancestry. Science. 2008;321: 218-223.; Nakabayashi K., Komaki G., Tajima A., Ando T., Ishikawa M., Nomoto J., Hata K., Oka A., Inoko H., Sasazuki T., Shirasawa S. Identification of novel candidate loci for anorexia nervosa at 1q41 and 11q22 in Japanese by a genome-wide association analysis with microsatellite markers. J. Hum. Genet. 2009;54:531-537.; Nava C., Keren B., Mignot C., Rastetter A., Chantot-Bastaraud S., Faudet A., Fonteneau E., Amiet C., Laurent C., Jacquette A., Whalen S., Afenjar A., Périsse D., Doummar D., Dorison N., Leboyer M., Siffroi J.P., Cohen D., Brice A., Héron D., Depienne C. Prospective diagnostic analysis of copy number variants using SNP microarrays in individuals with autism spectrum disorders. Eur. J. Hum. Genet. 2013;975:1-8.; Nikpay M., Seda O., Tremblay J., Petrovich M., Gaudet D., Kotchen T.A., Cowley A.W., Hamet P. Genetic mapping of habitual substance use, obesity-related traits, responses to mental and physical stress, and heart rate and blood pressure measurements reveals shared genes that are overrepresented in the neural synapse. Hypertens. Res. 2012;35:585-591.; Ogawa J., Kaneko H., Masuda T., Nagata S., Hosoya H., Watanabe K. Novel neural adhesion molecules in the Contactin/F3 subgroup of the immunoglobulin superfamily: isolation and characterization of cDNAs from rat brain. Neurosci. Lett. 1996;218(3):173-176.; Roohi J., Montagna C., Tegay D.H., Palmer L.E., DeVincent C., Pomeroy J.C., Christian S.L., Nowak N., Hatchwell E. Disruption of contactin 4 in three subjects with autism spectrum disorder. J. Med. Genet. 2009;46:176-182.; Van Daalen E., Kemner C., Verbeek N.E., van der Zwaag B., Dijkhuizen T., Rump P., Houben R., van’t Slot R., de Jonge M.V., Staal W.G., Beemer F.A., Vorstman J.A.S., Burbach J.P.H., van Amstel H.K.P., Hochstenbach R., Brilstra E.H., Poot M. Social responsiveness scale-aided analysis of the clinical impact of copy number variations in autism. Neurogenetics. 2011;12:315-323.; Zuko A., Bouyain S., van der Zwaag B., Burbach J.P.H. Contactins: structural aspects in relation to developmental functions in brain disease. Adv. Protein Chem. Struct. Biol. 2011;84:143-180.; Zuko A., Kleijer K.T., Oguro-Ando A., Kas M.J., van Daalen E., van der Zwaag B., Burbach J.P. Contactins in the neurobiology of autism. Eur. J. Pharmacol. 2013;719(1-3):63-74.; https://vavilov.elpub.ru/jour/article/view/872Test

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