المؤلفون: ERGÜN, UFUK, ARSAVA, ETHEM MURAT, KARABUDAK, RANA, TEZER FİLİK, FADİME İRSEL, TOPÇUOĞLU, MEHMET AKİF, ALAGÖZ, AYBALA NESLİHAN, Acar, Turkan, Demirel, Esra Aciman, AFSAR, Nazire, AKÇALI, AYLİN, Demir, Gulsen Akman, Mengi, Tugce Angin, Ayta, Semih, Bebek, Nerses, Bilgic, Basar, BOZ, CAVİT, Cakar, Arman, ÇELEBİSOY, NEŞE, ÇEVİK, MEHMET UĞUR, Delen, Firuze, Tekce, Hacer Durmus, Ekmekci, Hakan, Elmali, Ayse Deniz, Erdinc, Oguz Osman, ERDOĞAN, FÜSUN FERDA, Eren, Fettah, Parman, Yesim Gulsen, GÜMÜŞ, HALUK, ALGIN, DEMET İLHAN, Karadas, Omer, Yildiz, Ozlem Kayim, Koc, Emine Rabia, Adapinar, Demet Ozbabalik, ÖZDEMİR, ATİLLA ÖZCAN, ÖZTÜRK, ŞEREFNUR, Kocaman, Ayse Sagduyu, ŞAHİN, ŞEVKİ, Topcuoglu, Esen Saka, ŞENER, ÖZDEN, Togrol, Rifat Erdem, Tokcaer, Ayse Bora, Tuncer, Nese, UCA, ALİ ULVİ, ULUÇ, KAYIHAN, YAKA, ERDEM, Yon, Mehmet Ilker

العلاقة: 9510c5c3-6dce-4826-abad-bfa6de07d635; https://avesis.kocaeli.edu.tr/publication/details/9510c5c3-6dce-4826-abad-bfa6de07d635/oaiTest

الإتاحة: https://doi.org/10.4274/tnd.2020.73669Test
https://avesis.kocaeli.edu.tr/publication/details/9510c5c3-6dce-4826-abad-bfa6de07d635/oaiTest

المؤلفون: ÇEVİK, MEHMET UĞUR, Eren, Fettah, ERGÜN, UFUK, Parman, Yesim Gulsen, GÜMÜŞ, HALUK, ALGIN, DEMET İLHAN, KARABUDAK, RANA, Karadas, Omer, Yildiz, Ozlem Kayim, Koc, Emine Rabia, Adapinar, Demet Ozbabalik, ÖZDEMİR, ATİLLA ÖZCAN, ÖZTÜRK, ŞEREFNUR, Kocaman, Ayse Sagduyu, ŞAHİN, ŞEVKİ, Topcuoglu, Esen Saka, ŞENER, ÖZDEN, TEZER FİLİK, FADİME İRSEL, Togrol, Rifat Erdem, Tokcaer, Ayse Bora, TOPÇUOĞLU, MEHMET AKİF, Tuncer, Nese, UCA, ALİ ULVİ, ULUÇ, KAYIHAN, YAKA, ERDEM, Yon, Mehmet Ilker, Cakar, Arman, Bilgic, Basar, Acar, Turkan, Demirel, Esra Aciman, AFSAR, Nazire, AKÇALI, AYLİN, Demir, Gulsen Akman, ALAGÖZ, AYBALA NESLİHAN, Mengi, Tugce Angin, ARSAVA, ETHEM MURAT, Ayta, Semih, Bebek, Nerses, BOZ, CAVİT, ÇELEBİSOY, NEŞE, Delen, Firuze, Tekce, Hacer Durmus, Ekmekci, Hakan, Elmali, Ayse Deniz, Erdinc, Oguz Osman, ERDOĞAN, FÜSUN FERDA

المساهمون: Sakarya Üniversitesi ,, 2283675

مصطلحات موضوعية: Tıp, Sağlık Bilimleri, Dahili Tıp Bilimleri, Nöroloji, KLİNİK NEUROLOJİ, Klinik Tıp (MED), Klinik Tıp

العلاقة: TURKISH JOURNAL OF NEUROLOGY; Acar T., Demirel E. A. , AFSAR N., AKÇALI A., Demir G. A. , ALAGÖZ A. N. , Mengi T. A. , ARSAVA E. M. , Ayta S., Bebek N., et al., "The COVID-19 from Neurological Overview", TURKISH JOURNAL OF NEUROLOGY, cilt.26, ss.58-108, 2020; av_9510c5c3-6dce-4826-abad-bfa6de07d635; vv_1032021; http://hdl.handle.net/20.500.12627/100395Test; https://doi.org/10.4274/tnd.2020.73669Test; 26; 58; 108

الإتاحة: https://doi.org/20.500.12627/100395Test
https://doi.org/10.4274/tnd.2020.73669Test
https://hdl.handle.net/20.500.12627/100395Test

المؤلفون: ARSAVA, ETHEM MURAT, TOPÇUOĞLU, MEHMET AKİF, TEZER FİLİK, FADİME İRSEL, KARABUDAK, RANA, İLHAN ALGIN, DEMET, ERDOĞAN, FÜSUN FERDA, ÖZDEMİR, ATİLLA ÖZCAN, ERDİNÇ, OĞUZ OSMAN, Tekce, Hacer Durmus, ŞENER, ÖZDEN, Togrol, Rifat Erdem, Yon, Mehmet Ilker, Tokcaer, Ayse Bora, Tuncer, Nese, Ayta, Semih, UCA, ALİ ULVİ, ULUÇ, KAYIHAN, ALAGÖZ, AYBALA NESLİHAN, YAKA, ERDEM, Mengi, Tugce Angin, Eren, Fettah, Demir, Gulsen Akman, ERGÜN, UFUK, Parman, Yesim Gulsen, AKÇALI, AYLİN, AFSAR, Nazire, BOZ, CAVİT, Cakar, Arman, GÜMÜŞ, HALUK, Karadas, Omer, Demirel, Esra Aciman, Yildiz, Ozlem Kayim, KOÇ, EMİNE RABİA, Elmali, Ayse Deniz, Acar, Turkan, Adapinar, Demet Ozbabalik, ÖZTÜRK, ŞEREFNUR, Kocaman, Ayse Sagduyu, Bilgic, Basar, ÇELEBİSOY, NEŞE, Bebek, Nerses, ÇEVİK, MEHMET UĞUR, ŞAHİN, ŞEVKİ, Topcuoglu, Esen Saka, Delen, Firuze, Ekmekci, Hakan

العلاقة: 9510c5c3-6dce-4826-abad-bfa6de07d635; https://avesis.gazi.edu.tr/publication/details/9510c5c3-6dce-4826-abad-bfa6de07d635/oaiTest

الإتاحة: https://doi.org/10.4274/tnd.2020.73669Test
https://avesis.gazi.edu.tr/publication/details/9510c5c3-6dce-4826-abad-bfa6de07d635/oaiTest

المؤلفون: Hacer Durmus, Yesim Parman, Volker Straub, Ana Töpf, Magdalena Mroczek

المصدر: Genes
Volume 11
Issue 7
Genes, Vol 11, Iss 716, p 716 (2020)

مصطلحات موضوعية: Adult, 0301 basic medicine, Weakness, Pathology, medicine.medical_specialty, Adolescent, lcsh:QH426-470, plectin, Electromyography, Article, PLEC, 03 medical and health sciences, 0302 clinical medicine, Ptosis, Genetics, medicine, Humans, Repetitive nerve stimulation, Muscular dystrophy, Child, Genetics (clinical), Myasthenic Syndromes, Congenital, medicine.diagnostic_test, business.industry, limb-girdle muscular dystrophy, Homozygote, Facial weakness, Exons, medicine.disease, myasthenia, lcsh:Genetics, Phenotype, 030104 developmental biology, Muscular Dystrophies, Limb-Girdle, Mutation, Female, medicine.symptom, business, 030217 neurology & neurosurgery, Limb-girdle muscular dystrophy, Muscle cramp

الوصف: We identified the known c.1_9del mutation in the PLEC gene in four unrelated females from consanguineous families of Turkish origin. All individuals presented with slowly progressive limb-girdle weakness without any dermatological findings, and dystrophic changes observed in their muscle biopsies. Additionally, the neurological examination revealed ptosis, facial weakness, fatigability, and muscle cramps in all four cases. In two patients, repetitive nerve stimulation showed a borderline decrement and a high jitter was detected in all patients by single-fiber electromyography. Clinical improvement was observed after treatment with pyridostigmine and salbutamol was started. We further characterize the phenotype of patients with limb-girdle muscular dystrophy R17 clinically, by muscle magnetic resonance imaging (MRI) features and by describing a common 3.8 Mb haplotype in three individuals from the same geographical region. In addition, we review the neuromuscular symptoms associated with PLEC mutations and the role of plectin in the neuromuscular junction.

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56b4f36d8f03d6adf456c1ba436f19ffTest

المؤلفون: Yesim Parman, Vuslat Yilmaz, Güher Saruhan-Direskeneli, Hacer Durmus, Piraye Oflazer, Fikret Aysal, Erdem Tüzün, Ozlem Gungor-Tuncer, Feza Deymeer

المصدر: Journal of neuroimmunology. 349

مصطلحات موضوعية: 0301 basic medicine, Adult, Male, Adolescent, medicine.medical_treatment, Immunology, CD38, CD19, 03 medical and health sciences, Young Adult, 0302 clinical medicine, Myasthenia Gravis, medicine, Immunology and Allergy, Humans, B-cell activating factor, Child, B cell, Aged, B-Lymphocytes, biology, business.industry, Immunosuppression, Middle Aged, medicine.disease, Myasthenia gravis, Pathophysiology, 030104 developmental biology, medicine.anatomical_structure, Treatment Outcome, Neurology, biology.protein, Female, Neurology (clinical), Antibody, business, 030217 neurology & neurosurgery, Biomarkers, Immunosuppressive Agents

الوصف: B cells play a major role in the pathophysiology of myasthenia gravis (MG) with their ability to produce disease specific, pathogenic antibodies. However, their status during disease development and follow-up stages of the disease in the peripheral blood may need further studies to determine useful markers. In this study, we aimed to detect B cell associated factors concerning immunosuppressive treatment in generalized non-thymomatous MG patients. Although CD19(+) B cell distribution did not vary among disease subgroups, expressions of both CD38 and BAFFR were altered on B cells in MG patients under immunosuppressive therapy. Serum levels of BAFF were elevated in untreated MG patients as compared to treated MG patients and healthy controls. B cell activation factors may show profound alterations due to immunosuppression.

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5ce74a658149bb47de6d62c23141cf3Test
https://pubmed.ncbi.nlm.nih.gov/32977248Test

المؤلفون: Melih Tutuncu, Oner Dogan, Fikret Aysal, Merve Cebi, Arman Çakar, Yesim Parman, Berker Özkan, Mehmet Hocaoglu, Metin Mercan, Vildan Yayla, Onur Akan, Güher Saruhan-Direskeneli, Gizem Engin Gül, Sibel P. Yentür, Hacer Durmus

المساهمون: İÜC, Cerrahpaşa Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü

المصدر: Frontiers in Immunology
Frontiers in Immunology, Vol 11 (2020)

مصطلحات موضوعية: 0301 basic medicine, CD4-Positive T-Lymphocytes, Male, Chemokine, medicine.medical_treatment, Programmed Cell Death 1 Receptor, Chemokine receptor, 0302 clinical medicine, PD-1, IL-21, Immunology and Allergy, Receptors, Cholinergic, Original Research, biology, Chemistry, Interleukin-17, Middle Aged, IL-17, Cytokine, medicine.anatomical_structure, Female, Interleukin 17, Antibody, Immunosuppressive Agents, Signal Transduction, lcsh:Immunologic diseases. Allergy, Adult, Adolescent, T Follicular Helper Cells, T cell, Immunology, Inducible T-Cell Co-Stimulator Protein, 03 medical and health sciences, Young Adult, Myasthenia Gravis, medicine, Humans, Interleukin 4, Aged, Autoantibodies, Immunosuppression Therapy, myasthenia gravis, Interleukins, IL-4, CXCR5, Thymectomy, 030104 developmental biology, ICOS, biology.protein, T follicular helper cells, Interleukin-4, lcsh:RC581-607, 030215 immunology

الوصف: WOS:000538882900001 PubMed ID: 32508812 Myasthenia gravis (MG) is an autoimmune disease mediated by autoantibodies predominantly against the acetylcholine receptor (AChR). Specific T cell subsets are required for long-term antibody responses, and cytokines secreted mainly from CD4(+) T cells regulate B cell antibody production. The aim of this study was to assess the differences in the cytokine expressions of CD4(+) T cells in MG patients with AChR antibodies (AChR-MG) and the effect of immunosuppressive (IS) therapy on cytokine activity and to test these findings also in MG patients without detectable antibodies (SN-MG). Clinically diagnosed AChR-MG and SN-MG patients were included. The AChR-MG patients were grouped as IS-positive and -negative and compared with age- and sex-matched healthy controls. Peripheral blood mononuclear cells were used for ex vivo intracellular cytokine production, and subsets of CD4(+) T cells and circulating follicular helper T (cTfh) cells were detected phenotypically by the expression of the chemokine and the costimulatory receptors. Thymocytes obtained from patients who had thymectomy were also analyzed. IL-21, IL-4, IL-10, and IL-17A productions in CD4(+) T cells were increased in AChR-MG compared to those in healthy controls. IS treatment enhanced IL-10 and reduced IFN-gamma production in AChR-MG patients compared to those in IS-negative patients. Increased IL-21 and IL-4 productions were also demonstrated in SN-MG patients. Among CD4(+) T cells, Th17 cells were increased in both disease subgroups. Treatment induced higher proportions of Th2 cells in AChR-MG patients. Both CXCR5(+) and CXCR5(-) CD4(+) T cells expressed higher programmed cell death protein 1 (PD-1) and inducible costimulatory (ICOS) in AChR-MG and SN-MG groups, mostly irrespective of the treatment. Based on chemokine receptors on CXCR5(+)PD-1(+) in CD4(+) T (cTfh) cells, in AChR-MG patients without treatment, the proportions of Tfh17 cells were higher than those in the treated group, whereas the Tfh1 cells were decreased compared with those in the controls. The relevance of CXCR5 and PD-1 in the pathogenesis of AChR-MG was also suggested by the increased presence of these molecules on mature CD4 single-positive thymocytes from the thymic samples. The study provides further evidence for the importance of IL-21, IL-17A, IL-4, and IL-10 in AChR-MG. Disease-related CD4(+)T cells are identified mainly as PD-1(+) or ICOS+ with or without CXCR5, resembling cTfh cells in the circulation or probably in the thymus. AChR-MG and SN-MG seem to have some similar characteristics. IS treatment has distinctive effects on cytokine expression. TUBITAKTurkiye Bilimsel ve Teknolojik Arastirma Kurumu (TUBITAK) [116S317]; Istanbul University Research FundIstanbul University This study was supported by TUBITAK (116S317) and Istanbul University Research Fund.

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::273c967f5cde2dabc6846cda979ed160Test
https://hdl.handle.net/20.500.12511/5437Test

المؤلفون: Ana Töpf, Katherine Johnson, Adam Bates, Lauren Phillips, Katherine R. Chao, Eleina M. England, Kristen M. Laricchia, Thomas Mullen, Elise Valkanas, Liwen Xu, Marta Bertoli, Alison Blain, Ana B. Casasús, Jennifer Duff, Magdalena Mroczek, Sabine Specht, Monkol Lek, Monica Ensini, Daniel G. MacArthur, Ela Akay, Jorge Alonso-Pérez, Jonathan Baets, Nina Barisic, Alexandra Bastian, Sabine Borell, Teodora Chamova, Kristl Claeys, Jaume Colomer, Sandra Coppens, Nicolas Deconinck, Willem de Ridder, Jordi Díaz-Manera, Cristina Domínguez-González, Alexis Duncan, Hacer Durmus, Nagia A. Fahmy, Maria Elena Farrugia, Roberto Fernández-Torrón, Lidia Gonzalez-Quereda, Jana Haberlova, Maja von der Hagen, Andreas Hahn, Antonia Jakovčević, Ivonne Jerico Pascual, Solange Kapetanovic, Viktorija Kenina, Janbernd Kirschner, Andrea Klein, Heike Kölbel, Anna Kostera-Pruszczyk, Richa Kulshrestha, Jaana Lähdetie, Mahsa Layegh, Cheryl Longman, Adolfo López de Munain, Wolfgang Loscher, Anna Lusakowska, Paul Maddison, Armelle Magot, Anirban Majumdar, Pilar Martí, Amaia Martínez Arroyo, Radim Mazanec, Sandra Mercier, Tiziana Mongini, Nuria Muelas, Andrés Nascimento, Shahriar Nafissi, Shirin Omidi, Carlos Ortez, Stéphanie Paquay, Yann Pereon, Stojan Perić, Valentina Ponzalino, Vidosava Rakočević Stojanović, Gauthier Remiche, Aida Rodríguez Sainz, Sabine Rudnik, Iciar Sanchez Albisua, Manuela Santos, Ulrike Schara, Andriy Shatillo, Jadranka Sertić, Ulrich Stephani, Sonja Strang-Karlsson, Yves Sznajer, Ani Tanev, Ivailo Tournev, Peter Van den Bergh, Vinciane Van Parijs, Juan Vílchez, Katharina Vill, John Vissing, Carina Wallgren-Pettersson, Julia Wanschitz, Tracey Willis, Nanna Witting, Miren Zulaica, Volker Straub

المساهمون: MYO-SEQ Consortium, HUSLAB, HUS Children and Adolescents, Clinicum, Medicum, Claeys, Kristl

المصدر: Genetics in medicine
Genetics in Medicine
GENETICS IN MEDICINE
r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau
instname

مصطلحات موضوعية: 0301 basic medicine, targeted exome analysis, Neuromuscular disease, Medizin, Anoctamins, 030105 genetics & heredity, Bioinformatics, 3124 Neurology and psychiatry, DNA sequencing, Article, 03 medical and health sciences, genetic diagnosis, limb-girdle weakness, neuromuscular disease, next-generation sequencing, 3123 Gynaecology and paediatrics, Exome Sequencing, Medicine, Humans, Exome, Gene, Genetics (clinical), Exome sequencing, SGCA, RYR1, Genetic heterogeneity, business.industry, Sciences bio-médicales et agricoles, medicine.disease, Phenotype, 3. Good health, 030104 developmental biology, Muscular Dystrophies, Limb-Girdle, Glucosyltransferases, Human medicine, business

الوصف: Several hundred genetic muscle diseases have been described, all of which are rare. Their clinical and genetic heterogeneity means that a genetic diagnosis is challenging. We established an international consortium, MYO-SEQ, to aid the work-ups of muscle disease patients and to better understand disease etiology.
info:eu-repo/semantics/published

وصف الملف: 1 full-text file(s): application/pdf; Print-Electronic

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5eb0b77bdff412d8ddab3c132a95c157Test
https://repository.uantwerpen.be/docstore/d:irua:4405Test

المؤلفون: Hasan Demirci, Hasmet Hanagasi, Atilla Uslu, Güven Toksoy, Hacer Durmus, Yesim Parman

المصدر: MusclenerveREFERENCES. 62(6)

مصطلحات موضوعية: musculoskeletal diseases, 0301 basic medicine, Adult, Heterozygote, Physiology, Duchenne muscular dystrophy, Mothers, 030105 genetics & heredity, Neuropsychological Tests, 03 medical and health sciences, Cellular and Molecular Neuroscience, Executive Function, 0302 clinical medicine, Cognition, Spatial Processing, Physiology (medical), Medicine, Humans, Attention, Cognitive Dysfunction, Cognitive impairment, Immediate verbal memory, medicine.diagnostic_test, business.industry, Working memory, Neuropsychology, Neuropsychological test, Middle Aged, medicine.disease, Executive functions, Muscular Dystrophy, Duchenne, Memory, Short-Term, Case-Control Studies, Female, Neurology (clinical), business, 030217 neurology & neurosurgery, Clinical psychology

الوصف: Duchenne muscular dystrophy (DMD) has been found to be associated with cognitive impairment. However, few studies have addressed cognitive impairment among mothers of children with DMD. In the present study, the neuropsychological profiles of both carrier mothers (C-Ms) and noncarrier mothers (NC-Ms) were examined, and the findings were compared with healthy control mothers (HC-Ms). There were 90 participants, consisting of 31 C-Ms, 24 NC-Ms, and 35 HC-Ms, each of whom completed a neuropsychological test battery. C-Ms had poorer cognition performance in attention, working memory, immediate verbal memory, visuospatial skills, and executive functions than NC-Ms, and HC-Ms. This study provides evidence that there may be cognitive impairment in mothers of patients with DMD. The cognitive impairment of C-Ms has similarities to that seen in children with DMD.

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b518b85efe296824774b6f659729495eTest
https://pubmed.ncbi.nlm.nih.gov/32893363Test

المؤلفون: Bülent Kara, Piraye Serdaroglu-Oflazer, Xin Ming Shen, Yesim Parman-Gulsen, Joan M. Brengman, Coşkun Özdemir, Andrew G. Engel, Feza Deymeer, Hacer Durmus

المصدر: Neuromuscular Disorders. 28:315-322

مصطلحات موضوعية: Male, 0301 basic medicine, medicine.medical_specialty, Pathology, Adolescent, Neuromuscular Junction, Muscle Proteins, Disease, Gastroenterology, Article, Neuromuscular junction, Young Adult, 03 medical and health sciences, 0302 clinical medicine, health services administration, Internal medicine, COLQ, medicine, Humans, CHRNE, Receptors, Cholinergic, health care economics and organizations, Genetics (clinical), Retrospective Studies, Acetylcholine receptor, Myasthenic Syndromes, Congenital, biology, business.industry, Muscle weakness, Prognosis, Choline acetyltransferase, Phenotype, 030104 developmental biology, medicine.anatomical_structure, Neurology, Pyridostigmine, Mutation, Pediatrics, Perinatology and Child Health, Acetylcholinesterase, biology.protein, Female, Collagen, Neurology (clinical), medicine.symptom, business, 030217 neurology & neurosurgery, Follow-Up Studies, medicine.drug

الوصف: Congenital myasthenic syndromes (CMS) are a group of hereditary disorders affecting the neuromuscular junction. Here, we present clinical, electrophysiological and genetic findings of 69 patients from 51 unrelated kinships from Turkey. Genetic tests of 60 patients were performed at Mayo Clinic. Median follow-up time was 9.8 years (range 1-22 years). The most common CMS was primary acetylcholine receptor (AChR) deficiency (31/51) and the most common mutations in AChR were c.1219 + 2T G (12/51) and c.1327delG (6/51) in CHRNE. Four of our 5 kinships with AChE deficiency carried p.W148X that truncates the collagen domain of COLQ, and was previously reported only in patients from Turkey. These were followed by GFPT1 deficiency (4/51), DOK7 deficiency (3/51), slow channel CMS (3/51), fast channel CMS (3/51), choline acetyltransferase deficiency (1/51) and a CMS associated with desmin deficiency (1/51). Distribution of muscle weakness was sometimes useful in giving a clue to the CMS subtype. Presence of repetitive compound muscle action potentials pointed to AChE deficiency or slow channel CMS. Our experience confirms that one needs to be cautious using pyridostigmine, since it can worsen some types of CMS. Ephedrine/salbutamol were very effective in AChE and DOK7 deficiencies and were useful as adjuncts in other types of CMS. Long follow-up gave us a chance to assess progression of the disease, and to witness 12 mainly uneventful pregnancies in 8 patients. In this study, we describe some new phenotypes and detail the clinical features of the well-known CMS.

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c8a17c575429d766f1cd587ba6a605aTest
https://doi.org/10.1016/j.nmd.2017.11.013Test

المؤلفون: Sema, Icoz, Mefkure, Eraksoy, Erdem, Tuzun, Murat, Kurtuncu, Hacer, Durmus, Omer, Birisik, Angela, Vincent, Gulse, Akman-Demir

المساهمون: İstanbul Üniversitesi ,, 137541

مصطلحات موضوعية: Yaşam Bilimleri, Temel Bilimler, İmmünoloji, Yaşam Bilimleri (LIFE), NEUROSCIENCES, Sinirbilim ve Davranış

العلاقة: Erdem T., Hacer D., Murat K., Sema I., Omer B., Mefkure E., Angela V., Gulse A., "Increased anti-neuronal antibody positivity in thyroid antibody positive individuals irrespective of neurological findings", 10th Congress of the International-Society-of-Neuroimmunology (ISNI), Sitges, İspanya, 26 - 30 Ekim 2010, cilt.228, ss.159; vv_1032021; av_5af14ecb-c311-4043-a62b-debd0c2c4aa7; http://hdl.handle.net/20.500.12627/63872Test; 228

الإتاحة: https://doi.org/20.500.12627/63872Test
https://hdl.handle.net/20.500.12627/63872Test