يعرض 81 - 90 نتائج من 562 نتيجة بحث عن '"Flaherty, Kevin R."', وقت الاستعلام: 1.66s تنقيح النتائج
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    العلاقة: American Journal Of Respiratory And Critical Care Medicine; Makale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanı; https://doi.org/10.1164/rccm.202106-1485OCTest; https://hdl.handle.net/11454/77687Test; Martinez, Fernando/0000-0002-2412-3182; Chambers, Daniel/0000-0002-9553-5870; Kolb, Martin/0000-0003-3837-1467; Maher, Toby/0000-0001-7192-9149; 205; 1084; 1092

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    وصف الملف: application/pdf

    العلاقة: Fang, Chuling; Rinke, Andrew E; Wang, Jing; Flaherty, Kevin R; Phan, Sem H; Liu, Tianju (2022). "B7H3 expression and significance in idiopathic pulmonary fibrosis." The Journal of Pathology 256(3): 310-320.; https://hdl.handle.net/2027.42/171883Test; The Journal of Pathology; Chen X, Li Y, Blankson S, et al. B7‐H3 augments inflammatory responses and exacerbates brain damage via amplifying NF‐κB p65 and MAPK p38 activation during experimental pneumococcal meningitis. PLoS One 2017; 12: e0171146.; Inamura K, Amori G, Yuasa T, et al. Relationship of B7‐H3 expression in tumor cells and tumor vasculature with FOXP3+ regulatory T cells in renal cell carcinoma. Cancer Manag Res 2019; 11: 7021 – 7030.; Chen L, Zhang G, Sheng S, et al. Upregulation of soluble B7‐H3 in NSCLC‐derived malignant pleural effusion: a potential diagnostic biomarker correlated with NSCLC staging. Clin Chim Acta 2016; 457: 81 – 85.; Yoon BR, Chung YH, Yoo SJ, et al. Preferential induction of the T cell auxiliary signaling molecule B7‐H3 on synovial monocytes in rheumatoid arthritis. J Biol Chem 2016; 291: 4048 – 4057.; Nakashima T, Liu T, Hu B, et al. Role of B7H3/IL‐33 signaling in pulmonary fibrosis‐induced profibrogenic alterations in bone marrow. Am J Respir Crit Care Med 2019; 200: 1032 – 1044.; Raghu G, Remy‐Jardin M, Myers JL, et al. Diagnosis of idiopathic pulmonary fibrosis. An official ATS/ERS/JRS/ALAT clinical practice guideline. Am J Respir Crit Care Med 2018; 198: e44 – e68.; Hashimoto N, Jin H, Liu T, et al. Bone marrow‐derived progenitor cells in pulmonary fibrosis. J Clin Invest 2004; 113: 243 – 252.; Liu T, Baek HA, Yu H, et al. FIZZ2/RELM‐β induction and role in pulmonary fibrosis. J Immunol 2011; 187: 450 – 461.; Livak KJ, Schmittgen TD. Analysis of relative gene expression data using real‐time quantitative PCR and the 2(−Delta Delta C(T)) method. Methods 2001; 25: 402 – 408.; Ding L, Liu T, Wu Z, et al. Bone marrow CD11c + cell‐derived amphiregulin promotes pulmonary fibrosis. J Immunol 2016; 197: 303 – 312.; Luo L, Zhu G, Xu H, et al. B7‐H3 promotes pathogenesis of autoimmune disease and inflammation by regulating the activity of different T cell subsets. PLoS One 2015; 10: e0130126.; Janick‐Buckner D, Ranges GE, Hacker MP. Alteration of bronchoalveolar lavage cell populations following bleomycin treatment in mice. Toxicol Appl Pharmacol 1989; 100: 465 – 473.; Moore BB, Hogaboam CM. Murine models of pulmonary fibrosis. Am J Physiol Lung Cell Mol Physiol 2008; 294: L152 – L160.; Iida K, Miyake M, Onishi K, et al. Prognostic impact of tumor‐infiltrating CD276/Foxp3‐positive lymphocytes and associated circulating cytokines in patients undergoing radical nephrectomy for localized renal cell carcinoma. Oncol Lett 2019; 17: 4004 – 4010.; Emura I, Usuda H. Acute exacerbation of IPF has systemic consequences with multiple organ injury, with SRA + and TNF‐α + cells in the systemic circulation playing central roles in multiple organ injury. BMC Pulm Med 2016; 16: 138.; Schupp JC, Binder H, Jäger B, et al. Macrophage activation in acute exacerbation of idiopathic pulmonary fibrosis. PLoS One 2015; 10: e0116775.; Fukushima A, Sumi T, Fukuda K, et al. B7‐H3 regulates the development of experimental allergic conjunctivitis in mice. Immunol Lett 2007; 113: 52 – 57.; Gu W, Zhang X, Yan Y, et al. B7‐H3 participates in the development of asthma by augmentation of the inflammatory response independent of TLR2 pathway. Sci Rep 2017; 7: 40398.; Altan M, Pelekanou V, Schalper KA, et al. B7‐H3 expression in NSCLC and its association with B7‐H4, PD‐L1 and tumor‐infiltrating lymphocytes. Clin Cancer Res 2017; 23: 5202 – 5209.; Benzon B, Zhao SG, Haffner MC, et al. Correlation of B7‐H3 with androgen receptor, immune pathways and poor outcome in prostate cancer: an expression‐based analysis. Prostate Cancer Prostatic Dis 2017; 20: 28 – 35.; Chen JT, Chen CH, Ku KL, et al. Glycoprotein B7‐H3 overexpression and aberrant glycosylation in oral cancer and immune response. Proc Natl Acad Sci U S A 2015; 112: 13057 – 13062.; Fernandez IE, Greiffo FR, Frankenberger M, et al. Peripheral blood myeloid‐derived suppressor cells reflect disease status in idiopathic pulmonary fibrosis. Eur Respir J 2016; 48: 1171 – 1183.; Wang L, Cao NN, Wang S, et al. Roles of coinhibitory molecules B7‐H3 and B7‐H4 in esophageal squamous cell carcinoma. Tumour Biol 2016; 37: 2961 – 2971.; Leitner J, Klauser C, Pickl WF, et al. B7‐H3 is a potent inhibitor of human T‐cell activation: no evidence for B7‐H3 and TREML2 interaction. Eur J Immunol 2009; 39: 1754 – 1764.; Wynn TA. Integrating mechanisms of pulmonary fibrosis. J Exp Med 2011; 208: 1339 – 1350.; Sanaei MJ, Salimzadeh L, Bagheri N. Crosstalk between myeloid‐derived suppressor cells and the immune system in prostate cancer: MDSCs and immune system in prostate cancer. J Leukoc Biol 2020; 107: 43 – 56.; Zhang X, Zhao X, Sun H, et al. The role of miR‐29c/B7‐H3 axis in children with allergic asthma. J Transl Med 2018; 16: 218.; The Idiopathic Pulmonary Fibrosis Clinical Research Network, Raghu G, Anstrom KJ, et al. Prednisone, azathioprine, and N ‐acetylcysteine for pulmonary fibrosis. N Engl J Med 2012; 366: 1968 – 1977.; Raghu G. Idiopathic pulmonary fibrosis: new evidence and an improved standard of care in 2012. Lancet 2012; 380: 699 – 701.; Magnini D, Montemurro G, Iovene B, et al. Idiopathic pulmonary fibrosis: molecular endotypes of fibrosis stratifying existing and emerging therapies. Respiration 2017; 93: 379 – 395.; Fernandez IE, Kass DJ. Do circulating monocytes promote and predict idiopathic pulmonary fibrosis progression? 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Am J Respir Crit Care Med 2013; 187: 180 – 188.; Yang S, Wei W, Zhao Q. B7‐H3, a checkpoint molecule, as a target for cancer immunotherapy. Int J Biol Sci 2020; 16: 1767 – 1773.; Zhang G, Hou J, Shi J, et al. Soluble CD276 (B7‐H3) is released from monocytes, dendritic cells and activated T cells and is detectable in normal human serum. Immunology 2008; 123: 538 – 546.; Greenwald RJ, Freeman GJ, Sharpe AH. The B7 family revisited. Annu Rev Immunol 2005; 23: 515 – 548.; Zhang SS, Tang J, Yu SY, et al. Expression levels of B7‐H3 and TLT‐2 in human oral squamous cell carcinoma. Oncol Lett 2015; 10: 1063 – 1068.; Stanciu LA, Bellettato CM, Laza‐Stanca V, et al. Expression of programmed death‐1 ligand (PD‐L) 1, PD‐L2, B7‐H3, and inducible costimulator ligand on human respiratory tract epithelial cells and regulation by respiratory syncytial virus and type 1 and 2 cytokines. J Infect Dis 2006; 193: 404 – 412.; Suh WK, Wang SX, Jheon AH, et al. The immune regulatory protein B7‐H3 promotes osteoblast differentiation and bone mineralization. Proc Natl Acad Sci U S A 2004; 101: 12969 – 12973.; Hashiguchi M, Kobori H, Ritprajak P, et al. Triggering receptor expressed on myeloid cell‐like transcript 2 (TLT‐2) is a counter‐receptor for B7‐H3 and enhances T cell responses. Proc Natl Acad Sci U S A 2008; 105: 10495 – 10500.

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    المساهمون: Richeldi, Luca, du Bois, Roland M, Raghu, Ganesh, Azuma, Arata, Brown, Kevin K, Costabel, Ulrich, Cottin, Vincent, Flaherty, Kevin R, Hansell, David M, Inoue, Yoshikazu, Kim, Dong Soon, Kolb, Martin, Nicholson, Andrew G, Noble, Paul W, Selman, Moisé, Taniguchi, Hiroyuki, Brun, Michèle, Le Maulf, Florence, Girard, Mannaïg, Stowasser, Susanne, Schlenker Herceg, Rozsa, Disse, Bernd, Collard, Harold R, M. D., for the INPULSIS Trial Investigator, Rottoli, Paola

    وصف الملف: STAMPA

    العلاقة: info:eu-repo/semantics/altIdentifier/pmid/24836310; info:eu-repo/semantics/altIdentifier/wos/WOS:000336434000005; volume:370; issue:22; firstpage:2071; lastpage:2082; numberofpages:12; journal:NEW ENGLAND JOURNAL OF MEDICINE; http://hdl.handle.net/11365/974374Test; info:eu-repo/semantics/altIdentifier/scopus/2-s2.0-84901810710

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    المساهمون: Ding, Qiang, National Institutes of Health, National Institute of Allergy and Infectious Diseases, United Negro College Fund/MERCK

    المصدر: PLOS ONE ; volume 11, issue 8, page e0159878 ; ISSN 1932-6203

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