يعرض 1 - 2 نتائج من 2 نتيجة بحث عن '"Lama, V."', وقت الاستعلام: 0.63s تنقيح النتائج
  1. 1
    دورية أكاديمية

    وصف الملف: application/pdf

    العلاقة: Cantu, E.; Suzuki, Y.; Diamond, J. M.; Ellis, J.; Tiwari, J.; Beduhn, B.; Nellen, J. R.; Shah, R.; Meyer, N. J.; Lederer, D. J.; Kawut, S. M.; Palmer, S. M.; Snyder, L. D.; Hartwig, M. G.; Lama, V. N.; Bhorade, S.; Crespo, M.; Demissie, E.; Wille, K.; Orens, J.; Shah, P. D.; Weinacker, A.; Weill, D.; Wilkes, D.; Roe, D.; Ware, L. B.; Wang, F.; Feng, R.; Christie, J. D. (2016). "Protein Quantitative Trait Loci Analysis Identifies Genetic Variation in the Innate Immune Regulator TOLLIP in Post–Lung Transplant Primary Graft Dysfunction Risk." American Journal of Transplantation (3): 833-840.; https://hdl.handle.net/2027.42/134189Test; American Journal of Transplantation; Christie JD, Kotloff RM, Ahya VN, et al. The effect of primary graft dysfunction on survival after lung transplantation. Am J Respir Crit Care Med 2005; 171: 1312 – 1316.; Lee JC, Christie JD, Keshavjee S. Primary graft dysfunction: Definition, risk factors, short‐ and long‐term outcomes. Semin Respir Crit Care Med 2010; 31: 161 – 171.; Klarstrom Engstrom K, Khalaf H, Kalvegren H, Bengtsson T. The role of Porphyromonas gingivalis gingipains in platelet activation and innate immune modulation. Mol Oral Microbiol 2015; 30: 62 – 73.; Shetty S, Padijnayayveetil J, Tucker T, et al. The fibrinolytic system and the regulation of lung epithelial cell proteolysis, signaling, and cellular viability. Am J Physiol Lung Cell Mol Physiol 2008; 295: L967 – l975.; Sapru A, Curley MA, Brady S, Matthay MA, Flori H. Elevated PAI‐1 is associated with poor clinical outcomes in pediatric patients with acute lung injury. Intensive Care Med 2010; 36: 157 – 163.; Diamond JM, Lederer DJ, Kawut SM, et al. Elevated plasma long pentraxin‐3 levels and primary graft dysfunction after lung transplantation for idiopathic pulmonary fibrosis. Am J Transplant 2011; 11: 2517 – 2522.; Shah JA, Vary JC, Chau TT, et al. 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Semin Respir Crit Care Med 2013; 34: 305 – 319.; Yusen RD, Edwards LB, Kucheryavaya AY, et al. The Registry of the International Society for Heart and Lung Transplantation: Thirty‐First Adult Lung and Heart‐Lung Transplant Report—2014; Focus Theme: Retransplantation. J Heart Lung Transplant 2014; 33: 1009 – 1024.; Diamond JM, Lee JC, Kawut SM, et al. Clinical risk factors for primary graft dysfunction after lung transplantation. Am J Respir Crit Care Med 2013; 187: 527 – 534.; Katoh S, Honda S, Watanabe T, et al. Atrial endothelial impairment through Toll‐like receptor 4 signaling causes atrial thrombogenesis. Heart Vessels 2014; 29: 263 – 272.; Lo YL, Beckhouse AG, Boulus SL, Wells CA. Diversification of TOLLIP isoforms in mouse and man. Mamm Genome 2009; 20: 305 – 314.; Dowling JK, O’Neill LA. Biochemical regulation of the inflammasome. Crit Rev Biochem Mol Biol 2012; 47: 424 – 443.; Capelluto DG. Tollip: A multitasking protein in innate immunity and protein trafficking. Microbes Infect 2012; 14: 140 – 147.; Jeong E, Lee JY. Intrinsic and extrinsic regulation of innate immune receptors. Yonsei Med J 2011; 52: 379 – 392.; Cantu E, Lederer DJ, Meyer K, et al. Gene set enrichment analysis identifies key innate immune pathways in primary graft dysfunction after lung transplantation. Am J Transplant 2013; 13: 1898 – 1904.; Meyer NJ, Li M, Feng R, et al. ANGPT2 genetic variant is associated with trauma‐associated acute lung injury and altered plasma angiopoietin‐2 isoform ratio. Am J Respir Crit Care Med 2011; 183: 1344 – 1353.; Diamond JM, Meyer NJ, Feng R, et al. Variation in PTX3 is associated with primary graft dysfunction after lung transplantation. Am J Respir Crit Care Med 2012; 186: 546 – 552.; Diamond JM, Akimova T, Kazi A, et al. Genetic variation in the prostaglandin E2 pathway is associated with primary graft dysfunction. Am J Respir Crit Care Med 2014; 189: 567 – 575.; Noth I, Zhang Y, Ma SF, et al. Genetic variants associated with idiopathic pulmonary fibrosis susceptibility and mortality: A genome‐wide association study. Lancet Respir Med 2013; 1: 309 – 317.; Johnson AD, Handsaker RE, Pulit SL, Nizzari MM, O’Donnell CJ, de Bakker PI. SNAP: A web‐based tool for identification and annotation of proxy SNPs using HapMap. Bioinformatics 2008; 24: 2938 – 2939.; Carlson CS, Eberle MA, Rieder MJ, et al. Selecting a maximally informative set of single‐nucleotide polymorphisms for association analyses using linkage disequilibrium. Am J Hum Genet 2004; 74: 106 – 120.; Meyer NJ, Feng R, Li M, et al. IL1RN coding variant is associated with lower risk of acute respiratory distress syndrome and increased plasma IL‐1 receptor antagonist. Am J Respir Crit Care Med 2013; 187: 950 – 959.; Christie JD, Wurfel MM, Feng R, et al. Genome wide association identifies PPFIA1 as a candidate gene for acute lung injury risk following major trauma. PLoS One 2012; 7: e28268.; Price AL, Patterson NJ, Plenge RM, Weinblatt ME, Shadick NA, Reich D. Principal components analysis corrects for stratification in genome‐wide association studies. Nat Genet 2006; 38: 904 – 909.; Satagopan JM, Elston RC. Optimal two‐stage genotyping in population‐based association studies. Genet Epidemiol 2003; 25: 149 – 157.; Keating BJ, Tischfield S, Murray SS, et al. Concept, design and implementation of a cardiovascular gene‐centric 50 k SNP array for large‐scale genomic association studies. PLoS ONE 2008; 3: e3583.; Shah RJ, Diamond JM, Cantu E, et al. Latent class analysis identifies distinct phenotypes of primary graft dysfunction after lung transplantation. Chest 2013; 144: 616 – 622.; Ware LB, Matthay MA, Parsons PE, et al. Pathogenetic and prognostic significance of altered coagulation and fibrinolysis in acute lung injury/acute respiratory distress syndrome. Crit Care Med 2007; 35: 1821 – 1828.; Horrevoets AJ. Plasminogen activator inhibitor 1 (PAI‐1): In vitro activities and clinical relevance. Br J Haematol 2004; 125: 12 – 23.; Shah RJ, Bellamy SL, Localio AR, et al. A panel of lung injury biomarkers enhances the definition of primary graft dysfunction (PGD) after lung transplantation. J Heart Lung Transplant 2012; 31: 942 – 949.; Schliekelman P. Statistical power of expression quantitative trait loci for mapping of complex trait loci in natural populations. Genetics 2008; 178: 2201 – 2216.; Schadt EE, Monks SA, Drake TA, et al. Genetics of gene expression surveyed in maize, mouse and man. Nature 2003; 422: 297 – 302.

  2. 2
    دورية أكاديمية

    المساهمون: Division of Allergy, Pulmonary and Critical Care Medicine, University of Michigan, Ann Arbor, MI, Division of Allergy, Pulmonary and Critical Care Medicine, Department of Medicine, Vanderbilt University, Nashville, TN, Division of Allergy, Pulmonary and Critical Care Medicine, Columbia University, College of Physicians and Surgeons, New York, NY, Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, Division of Pulmonary and Critical Care Medicine, University of Alabama, Birmingham, AL, Division of Pulmonary and Critical Care Medicine, Stanford University, Stanford, CA, Division of Allergy, Pulmonary and Critical Care Medicine, Department of Medicine, Johns Hopkins University Hospital, Baltimore, MD, Department of Biostatistics and Epidemiology and, Division of Allergy, Pulmonary and Critical Care Medicine, University of Pennsylvania School of Medicine, Philadelphia, PA

    وصف الملف: 129593 bytes; 3109 bytes; application/pdf; text/plain

    العلاقة: Covarrubias, M.; Ware, L. B.; Kawut, S. M.; De Andrade, J.; Milstone, A.; Weinacker, A.; Orens, J.; Lama, V.; Wille, K.; Bellamy, S.; Shah, C.; Demissie, E.; Christie, J. D. (2007). "Plasma Intercellular Adhesion Molecule-1 and von Willebrand Factor in Primary Graft Dysfunction After Lung Transplantation." American Journal of Transplantation 7(11): 2573-2578.; https://hdl.handle.net/2027.42/75029Test; http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=17908278&dopt=citationTest; American Journal of Transplantation; Carter YM, Davis RD. Primary graft dysfunction in lung transplantation. Semin Respir Crit Care Med 2006; 27: 501 – 507.; Christie JD, Carby M, Bag R, Corris P, Hertz M, Weill D. Report of the ISHLT Working Group on Primary Lung Graft Dysfunction part II: Definition. A consensus statement of the International Society for Heart and Lung Transplantation. J Heart Lung Transplant 2005; 24: 1454 – 1459.; Christie JD, Van Raemdonck D, de Perrot M et al. Report of the ISHLT Working Group on Primary Lung Graft Dysfunction part I: Introduction and methods. J Heart Lung Transplant 2005; 24: 1451 – 1453.; Trulock EP, Edwards LB, Taylor DO, Boucek MM, Keck BM, Hertz MI. Registry of the International Society for Heart and Lung Transplantation: Twenty-third official adult lung and heart-lung transplantation report—2006. J Heart Lung Transplant 2006; 25: 880 – 892.; Barr ML, Kawut SM, Whelan TP et al. Report of the ISHLT Working Group on Primary Lung Graft Dysfunction part IV: Recipient-related risk factors and markers. J Heart Lung Transplant 2005; 24: 1468 – 1482.; Christie JD, Kotloff RM, Pochettino A et al. Clinical risk factors for primary graft failure following lung transplantation. Chest 2003; 124: 1232 – 1241.; de Perrot M, Bonser RS, Dark J et al. Report of the ISHLT Working Group on Primary Lung Graft Dysfunction part III: Donor-related risk factors and markers. J Heart Lung Transplant 2005; 24: 1460 – 1467.; Thabut G, Mal H, Cerrina J, et al. Influence of donor characteristics on outcome after lung transplantation: A multicenter study. J Heart Lung Transplant 2005; 24: 1347 – 1353.; Conner ER, Ware LB, Modin G, Matthay MA. Elevated pulmonary edema fluid concentrations of soluble intercellular adhesion molecule-1 in patients with acute lung injury: Biological and clinical significance. Chest 1999; 116 ( 1 Suppl ): 83S – 84S.; Flori HR, Ware LB, Glidden D, Matthay MA. Early elevation of plasma soluble intercellular adhesion molecule-1 in pediatric acute lung injury identifies patients at increased risk of death and prolonged mechanical ventilation. Pediatr Crit Care Med 2003; 4: 315 – 321.; Folsom AR, Wu KK, Rosamond WD, Sharrett AR, Chambless LE. Prospective study of hemostatic factors and incidence of coronary heart disease: The Atherosclerosis Risk in Communities (ARIC) Study. Circulation 1997; 96: 1102 – 1108.; Kawut SM, Horn EM, Berekashvili KK, Widlitz AC, Rosenzweig EB, Barst RJ. von Willebrand factor independently predicts long-term survival in patients with pulmonary arterial hypertension. Chest 2005; 128: 2355 – 2362.; Morange PE, Simon C, Alessi MC et al. Endothelial cell markers and the risk of coronary heart disease: The Prospective Epidemiological Study of Myocardial Infarction (PRIME) study. Circulation 2004; 109: 1343 – 1348.; Rubin DB, Wiener-Kronish JP, Murray JF et al. Elevated von Willebrand factor antigen is an early plasma predictor of acute lung injury in nonpulmonary sepsis syndrome. J Clin Invest 1990; 86: 474 – 480.; Ware LB, Conner ER, Matthay MA. von Willebrand factor antigen is an independent marker of poor outcome in patients with early acute lung injury. Crit Care Med 2001; 29: 2325 – 2331.; Ware LB, Eisner MD, Thompson BT, Parsons PE, Matthay MA. Significance of von Willebrand factor in septic and nonseptic patients with acute lung injury. Am J Respir Crit Care Med 2004; 170: 766 – 772.; Christie JD, Robinson N, Ware LB et al. Association of protein C and type 1 plasminogen activator inhibitor with primary graft dysfunction. Am J Respir Crit Care Med 2007; 175: 69 – 74.; Egan TM, Thomas Y, Gibson D et al. Trigger for intercellular adhesion molecule-1 expression in rat lungs transplanted from non-heart-beating donors. Ann Thorac Surg 2004; 77: 1048 – 1055.; Toda K, Kayano K, Karimova A et al. Antisense intercellular adhesion molecule-1 (ICAM-1) oligodeoxyribonucleotide delivered during organ preservation inhibits posttransplant ICAM-1 expression and reduces primary lung isograft failure. Circ Res 2000; 86: 166 – 174.; Kato GJ, Martyr S, Blackwelder WC et al. Levels of soluble endothelium-derived adhesion molecules in patients with sickle cell disease are associated with pulmonary hypertension, organ dysfunction, and mortality. Br J Haematol 2005; 130: 943 – 953.; Ware LB, Golden JA, Finkbeiner WE, Matthay MA. Alveolar epithelial fluid transport capacity in reperfusion lung injury after lung transplantation. Am J Respir Crit Care Med 1999; 159: 980 – 988.; Prekker ME, Nath DS, Walker AR et al. Validation of the proposed International Society for Heart and Lung Transplantation grading system for primary graft dysfunction after lung transplantation. J Heart Lung Transplant 2006; 25: 371 – 378.; Krenn K, Klepetko W, Taghavi S, Lang G, Schneider B, Aharinejad S. Recipient vascular endothelial growth factor serum levels predict primary lung graft dysfunction. Am J Transplant 2007; 7: 700 – 706.