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المصدر: International Journal of Molecular Sciences, Vol 22, Iss 13491, p 13491 (2021)
International Journal of Molecular Sciences; Volume 22; Issue 24; Pages: 13491
International Journal of Molecular Sciences

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f90fecd197369dd6b0e8cf23ae2a45d8Test
https://www.mdpi.com/1422-0067/22/24/13491Test

4

The GEF Trio controls endothelial cell size and arterial remodeling downstream of Vegf signaling in both zebrafish and cell models

المؤلفون: Klems, Alina, Rijssel, Jos Van, Ramms, Anne S., Wild, Raphael, Hammer, Julia, Merkel, Melanie, Derenbach, Laura, Préau, Laetitia, Hinkel, Rabea, Suarez-Martinez, Irina, Schulte-Merker, Stefan, Vidal, Ramon, Sauer, Sascha, Kivelä, Riikka, Alitalo, Kari, Kupatt, Christian, Buul, Jaap D. Van, Noble, Ferdinand Le

المساهمون: Landsteiner Laboratory, HUSLAB, CAN-PRO - Translational Cancer Medicine Program, Kari Alitalo / Principal Investigator, Research Programs Unit, Faculty of Medicine, University of Helsinki

المصدر: Nature Communications
Nature communications, 11(1):5319. Nature Publishing Group
Nature Communications, Vol 11, Iss 1, Pp 1-20 (2020)
Nature Communications, 11 (1), Art.-Nr. 5319

مصطلحات موضوعية: Life sciences, biology, Vascular Endothelial Growth Factor A, rac1 GTP-Binding Protein, GROWTH-FACTOR, MIGRATION, Science, CADHERIN, ALK1, Protein Serine-Threonine Kinases, Vascular Remodeling, Article, Animals, Genetically Modified, ddc:570, Human Umbilical Vein Endothelial Cells, Animals, Guanine Nucleotide Exchange Factors, Humans, SPECIFICATION, lcsh:Science, Cells, Cultured, Zebrafish, Cell Size, Placenta Growth Factor, Vascular Endothelial Growth Factor Receptor-1, MECHANOTRANSDUCTION, Models, Cardiovascular, Endothelial Cells, Health sciences, JUNCTIONS, Zebrafish Proteins, ARTERIOGENESIS, lcsh:Q, 3111 Biomedicine, Angiogenesis, Technology Platforms, COLLATERAL CIRCULATION, Signal Transduction

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::616c7ace4fe9720b95e1c27f26017b63Test
http://edoc.mdc-berlin.de/19505/7/19505suppl.zipTest

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Endoplasmic Reticulum Associated Protein Degradation (ERAD) in the Pathology of Diseases Related to TGFβ Signaling Pathway: Future Therapeutic Perspectives

المؤلفون: Nesrin Gariballa, Bassam R. Ali

المصدر: Frontiers in Molecular Biosciences, Vol 7 (2020)
Frontiers in Molecular Biosciences

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04f39e553bf27151d487a3897133f698Test
https://www.frontiersin.org/articles/10.3389/fmolb.2020.575608/fullTest

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Bioreactor-Controlled Physoxia Regulates TGF-β Signaling to Alter Extracellular Matrix Synthesis by Human Chondrocytes

المؤلفون: Sven Nebelung, Thomas Pufe, Seval Gunes, Holger Jahr, Annika-Ricarda Kuhn

المصدر: International Journal of Molecular Sciences, 20(7):1715. Multidisciplinary Digital Publishing Institute (MDPI)
International Journal of Molecular Sciences, Vol 20, Iss 7, p 1715 (2019)
International Journal of Molecular Sciences
International journal of molecular sciences 20(7), 1715 (2019). doi:10.3390/ijms20071715 special issue: "Special Issue "Osteoarthritis and Cartilage Regeneration: Focus on Pathophysiology and Molecular Mechanisms" / Special Issue Editors: Prof. Dr. Susanne Grässel, Guest Editor; PD Dr. Attila Aszodi, Guest Editor"
Volume 20
Issue 7

مصطلحات موضوعية: Vascular Endothelial Growth Factor A, 0301 basic medicine, physiological oxygen, medicine.medical_treatment, mesenchymal stem-cells, Smad2 Protein, ALK1, hypoxia-inducible factors, TGF- receptors, ALK5, Extracellular matrix, lcsh:Chemistry, Bioreactors, 0302 clinical medicine, Transforming Growth Factor beta, Gene expression, Protein Isoforms, Aggrecans, chondrogenic differentiation, lcsh:QH301-705.5, Spectroscopy, TGF-β superfamily, TGF-β receptors, type I, type II, type III, GENE-EXPRESSION, endoglin, HUMAN ARTICULAR CHONDROCYTES, Chemistry, type II, SOX9 Transcription Factor, General Medicine, progenitor cells, Extracellular Matrix, Computer Science Applications, Cell biology, Oxygen tension, Hypoxia-inducible factors, 030220 oncology & carcinogenesis, Signal Transduction, Smad5 Protein, TGF- superfamily, GROWTH-FACTOR, type III, BONE-MARROW, type I, in-vitro, Article, Catalysis, Hypoxia-Inducible Factor-Proline Dioxygenases, Smad1 Protein, Sp1, Inorganic Chemistry, Transforming Growth Factor beta2, ALK1, ALK5, 03 medical and health sciences, Chondrocytes, medicine, Humans, Smad3 Protein, Physical and Theoretical Chemistry, physoxia, Collagen Type II, Molecular Biology, Transcription factor, betaglycan, hypertrophic differentiation, Growth factor, Organic Chemistry, Mesenchymal stem cell, Endoglin, Collagen Type III, 030104 developmental biology, lcsh:Biology (General), lcsh:QD1-999, Smad8 Protein, Sp1/3, chondrocytic marker expression

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::48e2ddd41f80f96729e5e67022b4369eTest
https://cris.maastrichtuniversity.nl/en/publications/8a44354e-d752-469e-a65b-452327fb8fdaTest

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A signaling crosstalk between BMP9 and HGF/c-met regulates mouse adult liver progenitor cell survival

المؤلفون: Nerea Lazcanoiturburu, Isabel Fabregat, Laura Almalé, María García-Álvaro, Peter ten Dijke, Rebeca Méndez, Aránzazu Sánchez, Annalisa Addante, C. Roncero, Blanca Herrera

المصدر: Cells, 9(3). MDPI
Dipòsit Digital de la UB
Universidad de Barcelona
Cells, Vol 9, Iss 3, p 752 (2020)
E-Prints Complutense. Archivo Institucional de la UCM
instname
E-Prints Complutense: Archivo Institucional de la UCM
Universidad Complutense de Madrid
Cells
Volume 9
Issue 3

مصطلحات موضوعية: 0301 basic medicine, Aging, Activin Receptors, Type II, SMAD1, Smad Proteins, Apoptosis, SMAD, ALK1, p38 Mitogen-Activated Protein Kinases, chemistry.chemical_compound, Mice, 0302 clinical medicine, Growth Differentiation Factor 2, HGF, lcsh:QH301-705.5, Biología molecular, Hepatocyte Growth Factor, Stem Cells, Malalties del fetge, apoptosis, General Medicine, Proto-Oncogene Proteins c-met, Liver regeneration, 3. Good health, Cell biology, Crosstalk (biology), Liver, 030220 oncology & carcinogenesis, p38MAPK, Hepatocyte growth factor, Mothers against decapentaplegic, medicine.drug, Signal Transduction, Bioquímica, C-Met, Cell Survival, Biology, signaling crosstalk, Bone morphogenetic protein, BMP9, Article, Cell Line, 03 medical and health sciences, hepatic oval cell, medicine, Animals, Humans, Progenitor cell, Liver diseases, Apoptosi, Enzyme Activation, 030104 developmental biology, chemistry, lcsh:Biology (General)