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1دورية أكاديمية
المؤلفون: Meyskens, Frank L, Jr, Gerner, Eugene W
المصدر: Cancer prevention research (Philadelphia, Pa.). 4(5)
مصطلحات موضوعية: Apoptosis, Biomimetics, Genes, Lethal, Humans, Intracellular Signaling Peptides and Proteins: antagonists & inhibitors, genetics, Lung Neoplasms: genetics, prevention & control, Mitochondrial Proteins: antagonists & inhibitors, genetics, Mutation: genetics, Proto-Oncogene Proteins: antagonists & inhibitors, genetics, TNF-Related Apoptosis-Inducing Ligand: antagonists & inhibitors, genetics, ras Proteins: antagonists & inhibitors, genetics, B Raf kinase inhibitor, eflornithine, imatinib, K ras protein, nicotinamide adenine dinucleotide adenosine diphosphate ribosyltransferase inhibitor, retinol acetate, retrovirus vector, second mitochondrial activator of caspase, sulindac, tumor necrosis factor related apoptosis inducing ligand, advanced cancer, breast cancer, carcinogenesis, chemoprophylaxis, colorectal cancer, combination chemotherapy, drug targeting, gene mutation, human, lung carcinogenesis, melanoma, metastasis, nonhuman, ovary cancer, practice guideline, priority journal, protein expression, questionnaire, review, viral gene delivery system, Apoptosis, Biomimetics, Genes, Lethal, Humans, Intracellular Signaling Peptides and Proteins, Lung Neoplasms, Mitochondrial Proteins, Mutation, Proto-Oncogene Proteins, ras Proteins, TNF-Related Apoptosis-Inducing Ligand
وصف الملف: application/pdf
الوصول الحر: https://escholarship.org/uc/item/5t1960spTest
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2
المؤلفون: Gary Gk Owens, Roberto Cuttano, Monica Corada, Ralf H. Adams, Marco Francesco Morini, Martin A. Schwartz, Maria Grazia Lampugnani, Elisabetta Dejana, Costanza Giampietro, Luigi Maddaluno, Nicolas Baeyens, Noemi Rudini, Mukesh Mk Jain, Eleanna Papa, Luca Bravi
المصدر: EMBO molecular medicine, 8 (1
Cuttano, R, Rudini, N, Bravi, L, Corada, M, Giampietro, C, Papa, E, Morini, M F, Maddaluno, L, Baeyens, N, Adams, R H, Jain, M K, Owens, G K, Schwartz, M, Lampugnani, M G & Dejana, E 2016, ' KLF4 is a key determinant in the development and progression of cerebral cavernous malformations ', EMBO Molecular Medicine, vol. 8, no. 1, PMID 26612856, pp. 6-24 . https://doi.org/10.15252/emmm.201505433Test
EMBO Molecular Medicine, 8 (1)
EMBO Molecular Medicineمصطلحات موضوعية: 0301 basic medicine, Hemangioma, Cavernous, Central Nervous System, Vascular Biology & Angiogenesis, Bone Morphogenetic Protein 6, Proto-Oncogene Proteins -- antagonists & inhibitors -- genetics -- metabolism, Microtubule-Associated Proteins -- antagonists & inhibitors -- genetics -- metabolism, TGF beta-BMP, Cardiovascular System, Mice, Transforming Growth Factor beta, EndMT, News & Views, KRIT1 Protein, Endothelial Cells -- cytology -- metabolism, Mice, Knockout, Basic Medicine, Sciences bio-médicales et agricoles, Phenotype, Hemangioma, Cavernous, Central Nervous System -- genetics -- metabolism -- pathology, KLF4, endothelial cells, 3. Good health, Bone morphogenetic protein 6, medicine.anatomical_structure, TGFβ‐BMP, embryonic structures, Disease Progression, Molecular Medicine, RNA Interference, Smad1 Protein -- metabolism, Signal transduction, Microtubule-Associated Proteins, Signal Transduction, CCM, Endothelial cells, TGFβ-BMP, Medicinska och farmaceutiska grundvetenskaper, Central nervous system, Kruppel-Like Transcription Factors, Mitogen-Activated Protein Kinase 7 -- metabolism, Biology, Bone morphogenetic protein, Smad1 Protein, 03 medical and health sciences, Kruppel-Like Factor 4, Kruppel-Like Transcription Factors -- antagonists & inhibitors -- genetics -- metabolism, Proto-Oncogene Proteins, medicine, Animals, Humans, Transforming Growth Factor beta -- metabolism, Mitogen-Activated Protein Kinase 7, Cell Proliferation, HEK 293 cells, Endothelial Cells, Transforming growth factor beta, Bone Morphogenetic Protein 6 -- antagonists & inhibitors -- genetics -- metabolism, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, HEK293 Cells, Mutation, Cancer research, biology.protein, Genetics, Gene Therapy & Genetic Disease
وصف الملف: application/pdf; 1 full-text file(s): application/pdf; application/application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40d09206d63d5e3952c0a878de00d86eTest
http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-276831Test -
3
المؤلفون: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, Ulrike Muscha, Sumners, C, Katovich, M J
المصدر: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, U M, Sumners, C & Katovich, M J 2015, ' Selective activation of angiotensin AT 2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis ', British Journal of Pharmacology, vol. 172, no. 9, pp. 2219-2231 . https://doi.org/10.1111/bph.13044Test
مصطلحات موضوعية: Male, Myocardium/metabolism, Pyridines, Angiotensin II Type 2 Receptor Blockers/pharmacology, Hypertension, Pulmonary, Pulmonary Fibrosis, Ventricular Dysfunction, Right, Proto-Oncogene Proteins/antagonists & inhibitors, Ventricular Remodeling/drug effects, Angiotensin II Type 2 Receptor Blockers, Vascular Remodeling, Proto-Oncogene Mas, Receptor, Angiotensin, Type 2, Receptors, G-Protein-Coupled, Rats, Sprague-Dawley, Signal Transduction/drug effects, Proto-Oncogene Proteins, Animals, Angiotensin II/analogs & derivatives, Ventricular Dysfunction, Right/chemically induced, Lung, Imidazoles/pharmacology, Pyridines/pharmacology, Receptors, G-Protein-Coupled/antagonists & inhibitors, Peptide Fragments/pharmacology, Monocrotaline, Lung/drug effects, Hypertrophy, Right Ventricular, Ventricular Remodeling, Angiotensin II, Myocardium, Ventricular Function, Right/drug effects, Hemodynamics, Imidazoles, Pulmonary Fibrosis/chemically induced, Hypertrophy, Right Ventricular/chemically induced, Cardiovascular Agents, Receptor, Angiotensin, Type 2/agonists, Research Papers, Fibrosis, Peptide Fragments, Disease Models, Animal, Hemodynamics/drug effects, Cardiovascular Agents/pharmacology, Hypertension, Pulmonary/chemically induced, Ventricular Function, Right, Vascular Remodeling/drug effects, Signal Transduction
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b3e044c48f01f109844d8f467705bbddTest
https://europepmc.org/articles/PMC4403089Test/ -
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المؤلفون: R. Xu, Deepa Sampath, Vinay K. Puduvalli, Janet M. Bruner, J. Nangia, A. P. Kyritsis
المصدر: Apoptosis. 10:233-243
مصطلحات موضوعية: Cancer Research, Apoptosis, MAP Kinase Kinase 4, Poly ADP ribose polymerase, Clinical Biochemistry, Pharmaceutical Science, Protein Serine-Threonine Kinases, Article, TNF-Related Apoptosis-Inducing Ligand, Proto-Oncogene Proteins/*antagonists & inhibitors, Downregulation and upregulation, Proto-Oncogene Proteins, Cell Line, Tumor, Glioma, medicine, Humans, Protein kinase B, PI3K/AKT/mTOR pathway, JNK Mitogen-Activated Protein Kinases/*metabolism, Mitogen-Activated Protein Kinase Kinases, Pharmacology, Glioma/*pathology, Membrane Glycoproteins, Tumor Necrosis Factor-alpha, Protein-Serine-Threonine Kinases/*antagonists & inhibitors, Chemistry, Biochemistry (medical), JNK Mitogen-Activated Protein Kinases, Membrane Glycoproteins/*physiology, Cell Biology, Mitogen-Activated Protein Kinase Kinases/*metabolism, medicine.disease, Cell biology, Enzyme Activation, Flip, Tumor Necrosis Factor-alpha/*physiology, Signal transduction, Apoptosis Regulatory Proteins, Proto-Oncogene Proteins c-akt, Signal Transduction
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c50b6c945b0afc07bface102bd19d31Test
https://doi.org/10.1007/s10495-005-6078-3Test -
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المؤلفون: Lorella Marselli, Daniel Andrade Da Cunha, Chantal Mathieu, Conny Gysemans, Decio L. Eizirik, Najib Naamane, Piero Marchetti, David Ron, Carla Maria Ramos C.M. Germano, Jean-Marie Vanderwinden, Mariana Igoillo-Esteve, Esteban Nicolas Gurzov, Ihsane Marhfour, Makiko Fukaya, Heather P. Harding, Miriam Cnop
المصدر: Diabetes
Diabetes (New York, N.Y.), 61 (11مصطلحات موضوعية: Male, Endocrinology, Diabetes and Metabolism, Palmitic Acid, Proto-Oncogene Proteins -- antagonists & inhibitors -- genetics -- metabolism, Apoptosis, Mitochondrion, ACTIVATION, Mice, Mitochondria -- metabolism, Puma, Insulin-Secreting Cells, Insulin-Secreting Cells -- cytology -- metabolism, RNA, Small Interfering, Cells, Cultured, Mice, Knockout, biology, FREE FATTY-ACIDS, Sciences bio-médicales et agricoles, Middle Aged, ER STRESS, Endoplasmic Reticulum Stress, Cell biology, Mitochondria, RNA Interference, Life Sciences & Biomedicine, Signal Transduction, medicine.medical_specialty, Programmed cell death, KAPPA-B, Cell Line, CONTRIBUTES, Endocrinology & Metabolism, Internal medicine, Proto-Oncogene Proteins, SELECTIVE-INHIBITION, PUMA, Internal Medicine, medicine, Animals, Humans, p53 upregulated modulator of apoptosis, RNA, Messenger, Protein kinase B, INDUCIBLE GENE, Aged, Science & Technology, Endoplasmic reticulum, Palmitic Acid -- adverse effects, Gene Expression Profiling, JNK PHOSPHORYLATION, biology.organism_classification, RNA, Messenger -- metabolism, Rats, Mice, Inbred C57BL, Endocrinology, DEPENDENT APOPTOSIS, biology.protein, Unfolded protein response, Apoptosis Regulatory Proteins, Apoptosis Regulatory Proteins -- antagonists & inhibitors -- genetics -- metabolism
وصف الملف: Print-Electronic; 1 full-text file(s): application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29a67a877b9bf77f1a8e7af91175467dTest
https://lirias.kuleuven.be/handle/123456789/669544Test