نتائج البحث - "Katja Seipel"

1

دورية أكاديمية

Correlation of Peripheral Chimeric Antigen Receptor T-cell (CAR-T Cell) mRNA Expression Levels with Toxicities and Outcomes in Patients with Diffuse Large B-cell Lymphoma

المؤلفون: Christian Messerli, Gertrud Wiedemann, Naomi Porret, Michael Nagler, Katja Seipel, Barbara Jeker, Urban Novak, Sacha Zeerleder, Ulrike Bacher, Thomas Pabst

المصدر: Turkish Journal of Hematology, Vol 40, Iss 3, Pp 187-196 (2023)

مصطلحات موضوعية: non-hodgkin lymphoma, lymphomas, neoplasia, transplant-related toxicity, stem cell transplantation, molecular biology, Diseases of the blood and blood-forming organs, RC633-647.5

وصف الملف: electronic resource

العلاقة: https://jag.journalagent.com/z4/download_fulltext.asp?pdir=tjh&un=TJH-63625Test; https://doaj.org/toc/1308-5263Test

الوصول الحر: https://doaj.org/article/9a24576f2c1a4135973771d15a06c133Test

View record in DOAJ

النص الكامل

2

CAR T-Cell Persistence Correlates with Improved Outcome in Patients with B-Cell Lymphoma

المؤلفون: Valerie Wittibschlager, Ulrike Bacher, Katja Seipel, Naomi Porret, Gertrud Wiedemann, Claudia Haslebacher, Michèle Hoffmann, Michael Daskalakis, Dilara Akhoundova, Thomas Pabst

المصدر: International Journal of Molecular Sciences; Volume 24; Issue 6; Pages: 5688
Wittibschlager, Valerie; Bacher, Ulrike; Seipel, Katja; Porret, Naomi; Wiedemann, Gertrud; Haslebacher, Claudia; Hoffmann, Michèle; Daskalakis, Michael; Akhoundova, Dilara; Pabst, Thomas (2023). CAR T-Cell Persistence Correlates with Improved Outcome in Patients with B-Cell Lymphoma. International journal of molecular sciences, 24(6) MDPI 10.3390/ijms24065688 <http://dx.doi.org/10.3390/ijms24065688Test>

مصطلحات موضوعية: Inorganic Chemistry, CAR T-cell persistence, CAR T-cell therapy, B-cell lymphoma, ddPCR, Organic Chemistry, 610 Medicine & health, General Medicine, Physical and Theoretical Chemistry, 610 Medizin und Gesundheit, Molecular Biology, Spectroscopy, Catalysis, Computer Science Applications

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9528ae888c6d3aae4fe276060a7632e5Test
https://doi.org/10.3390/ijms24065688Test

3

Rationale for Combining the BCL2 Inhibitor Venetoclax with the PI3K Inhibitor Bimiralisib in the Treatment of IDH2- and FLT3-Mutated Acute Myeloid Leukemia

المؤلفون: Katja Seipel, Yvo Brügger, Harpreet Mandhair, Ulrike Bacher, Thomas Pabst

المصدر: International Journal of Molecular Sciences; Volume 23; Issue 20; Pages: 12587
Seipel, Katja; Brügger, Yvo; Mandhair, Harpreet; Bacher, Ulrike; Pabst, Thomas (2022). Rationale for Combining the BCL2 Inhibitor Venetoclax with the PI3K Inhibitor Bimiralisib in the Treatment of IDH2-and FLT3-Mutated Acute Myeloid Leukemia. International journal of molecular sciences, 23(20) MDPI 10.3390/ijms232012587 <http://dx.doi.org/10.3390/ijms232012587Test>

مصطلحات موضوعية: Adult, Mitogen-Activated Protein Kinase Kinases, acute myeloid leukemia (AML), B-cell lymphoma-2 (BCL2), venetoclax (ABT-199), isocitrate dehydrogenase 2 (IDH2), bimiralisib (PQR309), mammalian target of rapamycin (mTOR), phosphoinositide 3-kinase (PI3K), Fms-related receptor tyrosine kinase 3 (FLT3), protein tyrosine phosphatase non-receptor type 11 (PTPN11), TOR Serine-Threonine Kinases, Organic Chemistry, 610 Medicine & health, Antineoplastic Agents, General Medicine, Bridged Bicyclo Compounds, Heterocyclic, Catalysis, Computer Science Applications, Inorganic Chemistry, Phosphatidylinositol 3-Kinases, Leukemia, Myeloid, Acute, Proto-Oncogene Proteins c-bcl-2, fms-Like Tyrosine Kinase 3, Humans, Myeloid Cell Leukemia Sequence 1 Protein, Physical and Theoretical Chemistry, Protein Kinase Inhibitors, Molecular Biology, Spectroscopy

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9891a6780c13de04faa020ec477f25aaTest

4

Rationale for a Combination Therapy with the STAT5 Inhibitor AC-4-130 and the MCL1 Inhibitor S63845 in the Treatment of FLT3-Mutated or TET2-Mutated Acute Myeloid Leukemia

المؤلفون: Ulrike Bacher, Laura Flückiger, Thomas Pabst, Carolyn Graber, Katja Seipel

المصدر: International Journal of Molecular Sciences, Vol 22, Iss 8092, p 8092 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 15
Seipel, Katja; Graber, Carolyn; Flückiger, Laura; Bacher, Vera; Pabst, Thomas (2021). Rationale for a Combination Therapy with the STAT5 Inhibitor AC-4-130 and the MCL1 Inhibitor S63845 in the Treatment of FLT3-Mutated or TET2-Mutated Acute Myeloid Leukemia. International journal of molecular sciences, 22(15) MDPI 10.3390/ijms22158092 <http://dx.doi.org/10.3390/ijms22158092Test>

مصطلحات موضوعية: 0301 basic medicine, chemistry.chemical_compound, 0302 clinical medicine, fluids and secretions, hemic and lymphatic diseases, Antineoplastic Combined Chemotherapy Protocols, STAT5 Transcription Factor, Medicine, MCL1, Midostaurin, Biology (General), Spectroscopy, Trametinib, Kinase, ten-eleven translocation-2 (TET2), Myeloid leukemia, hemic and immune systems, General Medicine, 3. Good health, Computer Science Applications, DNA-Binding Proteins, Leukemia, Myeloid, Acute, Chemistry, myeloid leukemia cell differentiation protein (MCL1), 030220 oncology & carcinogenesis, embryonic structures, FLT3 Inhibitor, Combination therapy, QH301-705.5, tumor suppressor p53 (TP53), 610 Medicine & health, Thiophenes, Article, Catalysis, Dioxygenases, Inorganic Chemistry, 03 medical and health sciences, signal transducer and activator of transcription 5 (STAT5), acute myeloid leukemia (AML), Cell Line, Tumor, Proto-Oncogene Proteins, Humans, hematological malignancies, Physical and Theoretical Chemistry, Molecular Biology, QD1-999, FMS-like tyrosine kinase 3 (FLT3), business.industry, Venetoclax, Tumor Suppressor Proteins, Organic Chemistry, Pyrimidines, 030104 developmental biology, fms-Like Tyrosine Kinase 3, chemistry, Cancer research, business

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a86caaa2cbf92ffdc34d64b704e8471Test
https://www.mdpi.com/1422-0067/22/15/8092Test

5

(2R,3S)-Dihydroxybutanoic Acid Synthesis as a Novel Metabolic Function of Mutant Isocitrate Dehydrogenase 1 and 2 in Acute Myeloid Leukemia

المؤلفون: Jeffrey R. Idle, Diren Beyoğlu, Thomas Pabst, Ulrike Bacher, Katja Seipel

المصدر: Cancers
Volume 12
Issue 10
Cancers, Vol 12, Iss 2842, p 2842 (2020)
Idle, Jeffrey R; Seipel, Katja; Bacher, Ulrike; Pabst, Thomas; Beyoğlu, Diren (2020). (2R,3S)-Dihydroxybutanoic Acid Synthesis as a Novel Metabolic Function of Mutant Isocitrate Dehydrogenase 1 and 2 in Acute Myeloid Leukemia. Cancers, 12(10) MDPI AG 10.3390/cancers12102842 <http://dx.doi.org/10.3390/cancers12102842Test>

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, IDH1, Metabolite, Mutant, 610 Medicine & health, acute myeloid leukemia, medicine.disease_cause, IDH2, lcsh:RC254-282, Article, isocitrate dehydrogenase 2, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, isocitrate dehydrogenase 1, hemic and lymphatic diseases, medicine, (2R,3S)-dihydroxybutanoic acid, Threonine, (2R)-hydroxyglutaric acid, neoplasms, Mutation, Chemistry, Myeloid leukemia, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Molecular biology, metabolomics, oncometabolite, 030104 developmental biology, Isocitrate dehydrogenase, Oncology, 030220 oncology & carcinogenesis, biomarker