نتائج البحث - "Hyperphosphorylated tau"

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دورية أكاديمية

Fasting-mimicking diet cycles reduce neuroinflammation to attenuate cognitive decline in Alzheimer’s models

المصدر: Cell Reports. 40(13)

مصطلحات موضوعية: Biochemistry and Cell Biology, Biological Sciences, Neurosciences, Brain Disorders, Alzheimer's Disease, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), Nutrition, Acquired Cognitive Impairment, Neurodegenerative, Aging, Stem Cell Research, Dementia, Stem Cell Research - Nonembryonic - Non-Human, Neurological, Alzheimer Disease, Amyloid beta-Peptides, Animals, Cognitive Dysfunction, Disease Models, Animal, Fasting, Mice, Mice, Transgenic, NADPH Oxidases, Neuroinflammatory Diseases, Superoxides, tau Proteins, Alzheimer’s disease, CP: Metabolism, CP: Neuroscience, NADPH oxidase, amyloid beta, fasting, fasting-mimicking diet, hyperphosphorylated tau, microglia, neuroinflammation, protein restriction, superoxide, Medical Physiology, Biological sciences

وصف الملف: application/pdf

الوصول الحر: https://escholarship.org/uc/item/6s41r9ktTest

View record in eScholarship

2

C. elegans detects toxicity of traumatic brain injury generated tau

المؤلفون: Luca Russo, Luana Fioriti, Carmina Natale, Elisa R. Zanier, Roberto Chiesa, Luisa Diomede, Maria Monica Barzago, Elena Restelli, Gloria Vegliante, Laura Colombo, Luca Colnaghi, Edoardo Micotti, Margherita Romeo, Ilaria Bertani

المصدر: Neurobiology of Disease, Vol 153, Iss, Pp 105330-(2021)
Neurobiology of Disease

مصطلحات موضوعية: 0301 basic medicine, Genetically modified mouse, Aβ, amyloid β, Traumatic brain injury, Neuromuscular Junction, Neuromuscular transmission, tau Proteins, Motor Activity, Biology, Article, TBI, traumatic brain injury, lcsh:RC321-571, TauP301L, recombinant tau with the P301L mutation, Mice, 03 medical and health sciences, 0302 clinical medicine, Brain Injuries, Traumatic, medicine, Animals, Neuromuscular synaptic transmission, Cognitive decline, NGM, nematode growth medium, Caenorhabditis elegans, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, p-tau, hyperphosphorylated tau, KO, knockout, PK, proteinase-K, SNAP, simple neuroassessment of asymmetric impairment, Non-Tg, non-transgenic, Neurodegeneration, TBIcontra, tissue from contralateral brain area of TBI mice, Neurodegenerative Diseases, medicine.disease, WT, wild-type, Cell biology, Tauopathy, 030104 developmental biology, Tauopathies, Neurology, Toxicity, tauTBI, TBI-induced abnormal form of tau, TauWT, recombinant wild-type tau, TBIipsi, tissue from ipsilateral brain area of TBI mice, Tau, 030217 neurology & neurosurgery, Contra, contralateral

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::241bd53568f58354b57cccba9cff43e8Test
http://www.sciencedirect.com/science/article/pii/S0969996121000796Test

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Fasting-mimicking diet cycles reduce neuroinflammation to attenuate cognitive decline in Alzheimer's models

المصدر: Cell reports, vol 40, iss 13

مصطلحات موضوعية: Aging, fasting, Medical Physiology, microglia, Mice, Transgenic, tau Proteins, Neurodegenerative, Alzheimer's Disease, General Biochemistry, Genetics and Molecular Biology, Transgenic, neuroinflammation, Mice, Alzheimer Disease, Superoxides, Acquired Cognitive Impairment, Animals, protein restriction, Cognitive Dysfunction, Nutrition, Amyloid beta-Peptides, NADPH oxidase, Animal, Neuroscience [CP], Neurosciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), NADPH Oxidases, Fasting, Stem Cell Research, CP: Metabolism, fasting-mimicking diet, Brain Disorders, amyloid beta, Disease Models, Animal, Metabolism [CP], CP: Neuroscience, Disease Models, Neurological, Neuroinflammatory Diseases, hyperphosphorylated tau, Dementia, Stem Cell Research - Nonembryonic - Non-Human, superoxide, Biochemistry and Cell Biology, Alzheimer’s disease

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cc0b3f9d83e2f452146e6103295d13c7Test
https://pubmed.ncbi.nlm.nih.gov/36170815Test

4

Surfactant Protein-G in Wildtype and 3xTg-AD Mice: Localization in the Forebrain, Age-Dependent Hippocampal Dot-like Deposits and Brain Content

المؤلفون: Anton Meinicke, Wolfgang Härtig, Karsten Winter, Joana Puchta, Bianca Mages, Dominik Michalski, Alexander Emmer, Markus Otto, Karl-Titus Hoffmann, Willi Reimann, Matthias Krause, Stefan Schob

المصدر: Biomolecules; Volume 12; Issue 1; Pages: 96
Biomolecules
Biomolecules, Vol 12, Iss 96, p 96 (2022)

مصطلحات موضوعية: 3xTg mouse, hippocampus, Mice, Transgenic, tau Proteins, Microbiology, Biochemistry, Article, Mice, Surface-Active Agents, Alzheimer Disease, ddc:570, SP-G, Animals, Humans, Alzheimer’s disease, SFTA2, habenula, Reelin, β-amyloid, hyperphosphorylated tau, Molecular Biology, Amyloid beta-Peptides, Pulmonary Surfactant-Associated Protein A, Brain, QR1-502, Disease Models, Animal

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::81ea76c82f477a1cb4d0724868fe4787Test
https://doi.org/10.3390/biom12010096Test

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Hyperexcitability and epileptic seizures in a model of frontotemporal dementia

المؤلفون: Rosa Guerrero-López, Beatriz G. Giráldez, Marina P. Sánchez, María Llorens-Martín, José M. Serratosa, Ana M. Garcia-Cabrero, Jesús Avila

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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Neurobiology of Disease, Vol 58, Iss, Pp 200-208 (2013)

مصطلحات موضوعية: Hyperphosphorylated tau aggregates, Video Recording, Polycomb-Group Proteins, Hippocampus, Convulsants, Mice, Transgenic, tau Proteins, FTDP-17 tau mutations, Presenilin, lcsh:RC321-571, Mice, Epilepsy, Glial Fibrillary Acidic Protein, mental disorders, medicine, Animals, Humans, Dementia, EEG, Pentylenetetrazol, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, Parkinsonism, Calcium-Binding Proteins, Microfilament Proteins, Brain, Electroencephalography, medicine.disease, Microgliosis, Chemoconvulsant, DNA-Binding Proteins, Disease Models, Animal, Astrocytosis, Neurology, Frontotemporal Dementia, Pentylenetetrazole, Alzheimer's disease, Psychology, Neuroscience, Transcription Factors, medicine.drug, Frontotemporal dementia

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40f5beccad206190647b5cd58ca0dfc2Test
https://doi.org/10.1016/j.nbd.2013.06.005Test

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Abnormal processing of tau in the brain of aged TgCRND8 mice

المؤلفون: Ilaria Luccarini, Fiorella Casamenti, Maria Cristina Rosi, Anna Fiorentini, Cristina Grossi, Arianna Bellucci

المصدر: Neurobiology of Disease, Vol 27, Iss 3, Pp 328-338 (2007)

مصطلحات موضوعية: Male, Aging, Pathology, medicine.medical_specialty, Amyloid, MAP Kinase Kinase 4, Immunoblotting, Tau protein, Hippocampus, Nitrosative stress, Mice, Transgenic, Plaque, Amyloid, tau Proteins, Hippocampal formation, lcsh:RC321-571, Mice, chemistry.chemical_compound, mental disorders, medicine, Animals, Humans, Immunoprecipitation, Hyperphosphorylated tau, Phosphorylation, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, TgCRND8, Inclusion Bodies, Neocortex, biology, Chemistry, Nitrotyrosine, Amyloidosis, SAPK/JNK, Brain, Alzheimer's disease, medicine.disease, Immunohistochemistry, medicine.anatomical_structure, Neurology, biology.protein, Tyrosine, Female, Tauopathy, Double APP transgenic mouse

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::32c8637f932d436f0ab854361619db8cTest
https://doi.org/10.1016/j.nbd.2007.06.008Test

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Inhibitors of protein phosphatase-2A from human brain structures, immunocytological localization and activities towards dephosphorylation of the Alzheimer type hyperphosphorylated tau

المؤلفون: Jiliu Xu, Khalid Iqbal, Ichiro Tsujio, Leszek Kotula, Inge Grundke-Iqbal, Tanweer Zaidi

المصدر: FEBS Letters. 579:363-372

مصطلحات موضوعية: Chromosomal Proteins, Non-Histone, PHAP, Gene Expression, PC12 Cells, environment and public health, Biochemistry, Mice, 0302 clinical medicine, Structural Biology, Phosphoprotein Phosphatases, Protein Phosphatase 2, Phosphorylation, Neurons, 0303 health sciences, Stem Cells, Intracellular Signaling Peptides and Proteins, Brain, Nuclear Proteins, RNA-Binding Proteins, Transfection, 3. Good health, DNA-Binding Proteins, medicine.anatomical_structure, embryonic structures, Alzheimer disease, Microtubule-associated protein, Recombinant Fusion Proteins, Green Fluorescent Proteins, Molecular Sequence Data, Phosphatase, Biophysics, tau Proteins, macromolecular substances, Biology, Dephosphorylation, 03 medical and health sciences, Genetics, medicine, Animals, Humans, Histone Chaperones, Amino Acid Sequence, RNA, Messenger, Molecular Biology, 030304 developmental biology, Brain Chemistry, Cell Nucleus, Template activating factor, Protein phosphatase-2A inhibitor, Proteins, Cell Biology, Protein phosphatase 2, Molecular biology, Rats, enzymes and coenzymes (carbohydrates), Cell nucleus, Cytoplasm, LANP, NIH 3T3 Cells, Abnormally hyperphosphorylated tau, 030217 neurology & neurosurgery, Transcription Factors

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d1d2bb85c1b4f6d16f310bb7f67c2a6fTest
https://doi.org/10.1016/j.febslet.2004.11.097Test

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Hyperphosphorylated tau and paired helical filament-like structures in the brains of mice carrying mutant amyloid precursor protein and mutant presenilin-1 transgenes

المؤلفون: M. Kidd, D.C. Davies, M.A. Kurt, David R. Howlett, Karen Duff

المساهمون: Uludağ Üniversitesi/Tıp Fakültesi., Kurt, M. Ayberk, AAR-4341-2020

المصدر: Neurobiology of Disease, Vol 14, Iss 1, Pp 89-97 (2003)

مصطلحات موضوعية: Pathology, Mutant, Hippocampus, Paired helical filaments, Beta-amyloid, Gene, Amyloid beta-Protein Precursor, Mice, Amyloid precursor protein, Senile plaques, Transgenes, Phosphorylation, Aged, 80 and over, biology, Chemistry, Age Factors, Brain, Immunogold labelling, Alzheimer's disease, Middle Aged, Neurology, β-Amyloid, Alzheimer’s disease, Neurofibrillary tangles, Adult, medicine.medical_specialty, A-beta amyloidosis, Immunocytochemistry, Mice, Transgenic, tau Proteins, Neurosciences & neurology, Dystrophic neurites, Presenilin, lcsh:RC321-571, Accumulation, Alzheimer Disease, mental disorders, Electron microscopy, medicine, Presenilin-1, Animals, Humans, Hyperphosphorylated tau, Transgenic mouse brain, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, Aged, Neurosciences, Membrane Proteins, Molecular biology, nervous system diseases, Cortex (botany), Missense mutations, Mutation, biology.protein, Familial alzheimers-disease

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a770a75c3c38bfafa75b2662a0a9c6bTest
https://pubmed.ncbi.nlm.nih.gov/13678670Test

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