يعرض 1 - 10 نتائج من 328 نتيجة بحث عن '"Steroidogenic factor 1"', وقت الاستعلام: 1.00s تنقيح النتائج
  1. 1
    Contribution of the co-chaperone FKBP51 in the ventromedial hypothalamus to metabolic homeostasis in male and female mice

    المؤلفون: Lea M. Brix, Irmak Toksöz, London Aman, Veronika Kovarova, Margherita Springer, Joeri Bordes, Lotte van Doeselaar, Clara Engelhardt, Alexander S. Häusl, Sowmya Narayan, Vera Sterlemann, Huanqing Yang, Jan M. Deussing, Mathias V. Schmidt

    المصدر: Molecular Metabolism. 65:101579

    مصطلحات موضوعية: Male, Tacrolimus Binding Proteins, Mice, Diabetes Mellitus, Type 2, Ventromedial Hypothalamic Nucleus, Hypothalamus, Animals, Homeostasis, Female, Cell Biology, Energy Metabolism, Steroidogenic Factor 1, Molecular Biology

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::92ab01b36b7267017487277678e38bacTest
    https://doi.org/10.1016/j.molmet.2022.101579Test


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  2. 2
    Sertoli cell-derived exosome-mediated transfer of miR-145-5p inhibits Leydig cell steroidogenesis by targeting steroidogenic factor 1

    المؤلفون: Yadong Huang, Jiaxin Mei, Yan Tang, Jinlian Liang, Qihao Zhang, Hanhao Li, Qi Xiang, Yan Yang, Zhen Cao, Rufei Huang, Huan Xia

    المصدر: FASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES. 35(6)

    مصطلحات موضوعية: 0301 basic medicine, Steroidogenic factor 1, Male, endocrine system, Biology, Exosomes, Steroidogenic Factor 1, Biochemistry, Exosome, 03 medical and health sciences, Mice, 0302 clinical medicine, Lipid droplet, Gene expression, Testis, Genetics, medicine, Animals, Molecular Biology, Testosterone, Sertoli Cells, Leydig cell, Leydig Cells, Sertoli cell, Microvesicles, Cell biology, Mice, Inbred C57BL, MicroRNAs, 030104 developmental biology, medicine.anatomical_structure, Steroids, 030217 neurology & neurosurgery, Biotechnology

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60fd94469b4f5fd328747595815e55a1Test
    https://pubmed.ncbi.nlm.nih.gov/34010469Test


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  3. 3
    Expression of progesterone receptor, estrogen receptors α and β, and kisspeptin in the hypothalamus during perinatal development of gonad-lacking steroidogenic factor-1 knockout mice

    المؤلفون: Yayoi Ikeda, Ayako Tagami, Mamiko Maekawa, Tomoko Kato-Inui

    المصدر: Brain Research. 1712:167-179

    مصطلحات موضوعية: Male, 0301 basic medicine, Steroidogenic factor 1, endocrine system, medicine.medical_specialty, Gonad, Hypothalamus, Estrogen receptor, Biology, Steroidogenic Factor 1, Mice, 03 medical and health sciences, 0302 clinical medicine, Kisspeptin, Pregnancy, Internal medicine, Progesterone receptor, medicine, Animals, Estrogen Receptor beta, Gonads, Molecular Biology, Mice, Knockout, Neurons, Kisspeptins, Sex Characteristics, Sexual differentiation, Estradiol, General Neuroscience, Arcuate Nucleus of Hypothalamus, Estrogen Receptor alpha, Preoptic Area, Mice, Inbred C57BL, Preoptic area, 030104 developmental biology, Endocrinology, medicine.anatomical_structure, Receptors, Estrogen, Female, Neurology (clinical), Receptors, Progesterone, 030217 neurology & neurosurgery, Developmental Biology

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23b32030a9fa4f6aef7e4382f66dfd6fTest
    https://doi.org/10.1016/j.brainres.2019.02.016Test


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  4. 4
    Nr5a1-Cre-mediated Tspo conditional knockout mice with low growth rate and prediabetes symptoms – A mouse model of stress diabetes

    المؤلفون: Vassilios Papadopoulos, Jinjiang Fan, Enrico Campioli

    المصدر: Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865:56-62

    مصطلحات موضوعية: Blood Glucose, Male, 0301 basic medicine, Steroidogenic factor 1, endocrine system, medicine.medical_specialty, medicine.medical_treatment, 030209 endocrinology & metabolism, Adrenocorticotropic hormone, Biology, Steroidogenic Factor 1, Mitochondrial Proteins, Prediabetic State, Mice, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Receptors, GABA, Downregulation and upregulation, Stress, Physiological, Corticosterone, Internal medicine, Conditional gene knockout, medicine, Translocator protein, Animals, Humans, Calgranulin A, Prediabetes, Molecular Biology, Mice, Knockout, Integrases, Insulin, medicine.disease, 3. Good health, Mice, Inbred C57BL, Disease Models, Animal, Cholesterol, Glucose, 030104 developmental biology, Endocrinology, Diabetes Mellitus, Type 2, Gene Expression Regulation, Liver, chemistry, Hyperglycemia, biology.protein, Molecular Medicine, Female

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f788d08f996ec0c9bcbe6051f457457eTest
    https://doi.org/10.1016/j.bbadis.2018.10.022Test


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  5. 5
    Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats

    المؤلفون: Yun-Fei Zou, Linguo Pei, Hui Wang, Min Liu, Guanghui Chen, Qi Zhang

    المصدر: Epigenetics. 14:245-259

    مصطلحات موضوعية: Male, 0301 basic medicine, Steroidogenic factor 1, endocrine system, Cancer Research, medicine.medical_specialty, 3-Hydroxysteroid Dehydrogenases, Offspring, Histone Deacetylase 2, Biology, Testicular Diseases, Epigenesis, Genetic, Mice, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Glucocorticoid receptor, Pregnancy, Corticosterone, Internal medicine, Testis, medicine, Animals, Testosterone, Rats, Wistar, Molecular Biology, Gene knockdown, Fetus, Ethanol, Sperm Count, Histone deacetylase 2, Lysine, Leydig Cells, Acetylation, 030104 developmental biology, Endocrinology, chemistry, In utero, Prenatal Exposure Delayed Effects, 030220 oncology & carcinogenesis, Female, Research Paper

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3dec92f8f1d18ba3dd041df470760c02Test
    https://doi.org/10.1080/15592294.2019.1581595Test


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  6. 6
    Transcription factor 21 regulates expression of ERβ and SF-1 via upstream stimulatory factor-2 in endometriotic tissues

    المؤلفون: Qing Xue, Pei Li Wu, Zhao Tong Dong, Xin Li, Yan Zhou, Serdar E. Bulun, Cheng Zeng, Ying Fang Zhou

    المصدر: Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1861:706-717

    مصطلحات موضوعية: Adult, 0301 basic medicine, USF2, Biophysics, Mice, Nude, Steroidogenic Factor 1, Biochemistry, Upstream Stimulatory Factor, E-Box Elements, Mice, Young Adult, 03 medical and health sciences, 0302 clinical medicine, Structural Biology, Gene expression, Basic Helix-Loop-Helix Transcription Factors, Genetics, Transcriptional regulation, Animals, Estrogen Receptor beta, Humans, Promoter Regions, Genetic, Molecular Biology, Transcription factor, Cells, Cultured, Estrogen receptor beta, Cell Proliferation, Gene knockdown, Chemistry, Promoter, Cell biology, 030104 developmental biology, Gene Expression Regulation, 030220 oncology & carcinogenesis, Upstream Stimulatory Factors, Female, Stromal Cells, Endometritis

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50e2b512893092db71cbe8f6c7a94cd4Test
    https://doi.org/10.1016/j.bbagrm.2018.06.008Test


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  7. 7
    Heterozygous deletion of Sox9 in mouse mimics the gonadal sex reversal phenotype associated with campomelic dysplasia in humans

    المؤلفون: Stefan Bagheri-Fam, Alexander N. Combes, Cheuk Kwan Ling, Dagmar Wilhelm

    المصدر: Human molecular genetics. 29(23)

    مصطلحات موضوعية: Male, endocrine system, Heterozygote, Sex Differentiation, Disorders of Sex Development, SOX9, Biology, Steroidogenic Factor 1, Andrology, 03 medical and health sciences, Mice, Genetics, medicine, Animals, Humans, Disorders of sex development, Allele, Gonads, Molecular Biology, Genetics (clinical), 030304 developmental biology, Mice, Knockout, 0303 health sciences, Sexual differentiation, 030305 genetics & heredity, Campomelic Dysplasia, Gene Expression Regulation, Developmental, SOX9 Transcription Factor, General Medicine, Sex reversal, medicine.disease, Campomelic dysplasia, Mice, Inbred C57BL, Disease Models, Animal, Phenotype, embryonic structures, Knockout mouse, Female, Haploinsufficiency

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f3eee5d9e37919f1ade556d38285fcaTest
    https://pubmed.ncbi.nlm.nih.gov/33305798Test


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  8. 8
    Withdrawal: Orphan nuclear receptor Nur77 induces zinc finger protein GIOT-1 gene expression, and GIOT-1 acts as a novel corepressor of orphan nuclear receptor SF-1 via recruitment of HDAC2

    المؤلفون: Kwang-Hoon, Song, Yun-Yong, Park, Hae Jin, Kee, Cheol Yi, Hong, Yong-Soo, Lee, Seung-Won, Ahn, Hye-Jin, Kim, Keesook, Lee, Hyun, Kook, In-Kyu, Lee, Heung-Sik, Choi

    مصطلحات موضوعية: Male, Chromatin Immunoprecipitation, Receptors, Steroid, Molecular Sequence Data, Histone Deacetylase 2, Receptors, Cytoplasmic and Nuclear, Electrophoretic Mobility Shift Assay, Steroidogenic Factor 1, Biochemistry, Histone Deacetylases, Mice, Testis, Nuclear Receptor Subfamily 4, Group A, Member 1, Animals, Humans, Withdrawals/Retractions, RNA, Small Interfering, Promoter Regions, Genetic, Molecular Biology, Homeodomain Proteins, Base Sequence, Cell Biology, DNA, DNA-Binding Proteins, Repressor Proteins, Gene Expression Regulation, Transcription Factors

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d21a33556823ff3a07fb5865c14d4448Test
    https://europepmc.org/articles/PMC6916490Test/


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  9. 9
    Identification of a pituitary ERα-activated enhancer triggering the expression of Nr5a1, the earliest gonadotrope lineage-specific transcription factor

    المؤلفون: Florence Petit, Joëlle Cohen-Tannoudji, David L'Hôte, Vincent Pacini, Jean-Noël Laverrière, Bruno Quérat

    المساهمون: Unité de Biologie Fonctionnelle et Adaptative (BFA (UMR_8251 / U1133)), Université Paris Diderot - Paris 7 (UPD7)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

    المصدر: Epigenetics & Chromatin, Vol 12, Iss 1, Pp 1-17 (2019)
    Epigenetics & Chromatin
    Epigenetics & Chromatin, BioMed Central, 2019, 12 (1), ⟨10.1186/s13072-019-0291-8⟩

    مصطلحات موضوعية: Transcription, Genetic, lcsh:QH426-470, Gonadotrope specification, Gonadotrophs, Biology, Steroidogenic Factor 1, Chromatin remodeling, Cell Line, [SDV.BDLR.RS]Life Sciences [q-bio]/Reproductive Biology/Sexual reproduction, Histones, Mice, 03 medical and health sciences, 0302 clinical medicine, Transcription (biology), Gene expression, Genetics, Animals, Humans, Estrogen receptor, Epigenetics, Promoter Regions, Genetic, Enhancer, Molecular Biology, Transcription factor, 030304 developmental biology, 0303 health sciences, Base Sequence, Research, Estrogen Receptor alpha, Epigenetic, Cell Differentiation, DNA-Directed RNA Polymerases, Chromatin Assembly and Disassembly, Chromatin, Cell biology, lcsh:Genetics, Enhancer Elements, Genetic, [SDV.BDD.EO]Life Sciences [q-bio]/Development Biology/Embryology and Organogenesis, Regulatory sequence, Pituitary Gland, Nr5a1, CRISPR-Cas Systems, Sequence Alignment, 030217 neurology & neurosurgery

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4bc1e26afb0d608f46ef2df57d6f6caTest
    https://doi.org/10.1186/s13072-019-0291-8Test


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  10. 10
    Fetal Leydig Cells Persist as an Androgen-Independent Subpopulation in the Postnatal Testis

    المؤلفون: Yuichi Shima, Sawako Matsuzaki, Ilpo Huhtaniemi, Takashi Baba, Ken Ichirou Morohashi, Kanako Miyabayashi, Shigeaki Kato, Hiroyuki Otake

    المصدر: Molecular Endocrinology. 29:1581-1593

    مصطلحات موضوعية: Male, Steroidogenic factor 1, medicine.medical_specialty, medicine.drug_class, Cellular differentiation, Green Fluorescent Proteins, Biology, Steroidogenic Factor 1, Mice, Endocrinology, Internal medicine, Testis, medicine, Animals, Cell Lineage, Testosterone, Molecular Biology, Original Research, Mice, Knockout, Fetus, Sexual differentiation, luteinizing hormone/choriogonadotropin receptor, Gene Expression Regulation, Developmental, Leydig Cells, Cell Differentiation, General Medicine, Receptors, LH, Embryo, Mammalian, Androgen, Mice, Inbred C57BL, Androgen receptor, Receptors, Androgen, Androgens, Signal Transduction

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f55d0a32c77cfdd8b8dcdcbbf63f17dcTest
    https://doi.org/10.1210/me.2015-1200Test


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