نتائج البحث - "Gwi Ran Kim"

1

Histone deacetylase inhibitor LMK235 attenuates vascular constriction and aortic remodelling in hypertension

المؤلفون: Seung-Jung Kee, Marc Pflieger, Thomas Kurz, Hae Jin Kee, Gwi Ran Kim, Simei Sun, Matthias U. Kassack, Sin Young Choi, Young Mi Seok, Yuhee Ryu, Myung Ho Jeong

المصدر: Journal of Cellular and Molecular Medicine

مصطلحات موضوعية: 0301 basic medicine, Male, Angiotensin receptor, medicine.medical_specialty, Vascular smooth muscle, hypertension, medicine.drug_class, Aortic Diseases, Muscle, Smooth, Vascular, Nitric oxide, Rats, Sprague-Dawley, 03 medical and health sciences, chemistry.chemical_compound, Mice, 0302 clinical medicine, calcium calmodulin‐dependent protein kinase II, Internal medicine, Rats, Inbred SHR, Renin–angiotensin system, medicine, Animals, Antihypertensive Agents, Histone deacetylase 5, vascular hyperplasia, Angiotensin II, Histone deacetylase inhibitor, Cell Biology, Original Articles, HDAC5, HDAC4, Rats, Histone Deacetylase Inhibitors, 030104 developmental biology, Endocrinology, chemistry, Gene Expression Regulation, Vasoconstriction, 030220 oncology & carcinogenesis, Benzamides, Molecular Medicine, Original Article, medicine.symptom

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::864b3a40fdb5d328dc25c76d3bf1cb31Test
http://europepmc.org/articles/PMC6433685Test

2

Gentisic acid attenuates pressure overload-induced cardiac hypertrophy and fibrosis in mice through inhibition of the ERK1/2 pathway

المؤلفون: Myung Ho Jeong, Gwi Ran Kim, Hae Jin Kee, Li Jin, Yuhee Ryu, Simei Sun, Sin Young Choi

المصدر: Journal of Cellular and Molecular Medicine

مصطلحات موضوعية: Male, 0301 basic medicine, Cardiac fibrosis, Gentisates, cardiac fibrosis, Constriction, Pathologic, 030204 cardiovascular system & hematology, Pharmacology, Electrocardiography, Mice, chemistry.chemical_compound, 0302 clinical medicine, gentisic acid, Fibrosis, Diltiazem, Phosphorylation, Gentisic acid, Aorta, cardiac hypertrophy, Brain natriuretic peptide, GATA4/Sp1, Bisoprolol, cardiovascular system, Molecular Medicine, Original Article, Hypertrophy, Left Ventricular, medicine.drug, MAP Kinase Signaling System, Sp1 Transcription Factor, medicine.drug_class, Cardiomegaly, 03 medical and health sciences, transverse aortic constriction, Pressure, medicine, Animals, Beta blocker, Pressure overload, ERK1/2 MAPK signalling pathway, business.industry, Myocardium, Original Articles, Cell Biology, medicine.disease, GATA4 Transcription Factor, 030104 developmental biology, Gene Expression Regulation, chemistry, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f18040d5769ef9bb2fa8b1a356471c6Test
https://doi.org/10.1111/jcmm.13869Test

3

Gallic acid attenuates calcium calmodulin-dependent kinase II-induced apoptosis in spontaneously hypertensive rats

المؤلفون: Hae Jin Kee, Myung Ho Jeong, Zhe Hao Piao, Sin Young Choi, Chun Ping Liu, Bin Liu, Li Jin, Gwi Ran Kim, Yuhee Ryu, Simei Sun

المصدر: Journal of Cellular and Molecular Medicine

مصطلحات موضوعية: Male, 0301 basic medicine, Apoptosis, Blood Pressure, Essential hypertension, Rats, Inbred WKY, chemistry.chemical_compound, Atrial natriuretic peptide, Rats, Inbred SHR, Natriuretic Peptide, Brain, Myocytes, Cardiac, p300-CBP Transcription Factors, Gallic acid, bcl-2-Associated X Protein, biology, Caspase 3, cardiac hypertrophy, Angiotensin II, spontaneously hypertensive rats (SHR), Brain natriuretic peptide, Isoenzymes, Nitric oxide synthase, Hypertension, cardiovascular system, Molecular Medicine, Original Article, Hypertrophy, Left Ventricular, Atrial Natriuretic Factor, Signal Transduction, medicine.medical_specialty, Cardiotonic Agents, Cell Line, 03 medical and health sciences, Gallic Acid, Internal medicine, Ca2+/calmodulin-dependent protein kinase, medicine, Animals, Antihypertensive Agents, Ca2+/calmodulin‐dependent protein kinase II, Original Articles, Cell Biology, medicine.disease, Rats, 030104 developmental biology, Endocrinology, Gene Expression Regulation, chemistry, biology.protein, Nitric Oxide Synthase, Tumor Suppressor Protein p53, Calcium-Calmodulin-Dependent Protein Kinase Type 2

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fd0c03e67ab3a1e36bf6670923376bdTest
https://doi.org/10.1111/jcmm.13419Test

4

Gallic acid attenuates hypertension, cardiac remodeling, and fibrosis in mice with N G-nitro-L-arginine methyl ester-induced hypertension via regulation of histone deacetylase 1 or histone deacetylase 2

المؤلفون: Sin Young Choi, Gwi Ran Kim, Yuhee Ryu, Simei Sun, Jae Yeong Cho, Hae Jin Kee, Ming Quan Lin, Myung Ho Jeong, Li Jin, Zhe Hao Piao

المصدر: Journal of Hypertension. 35:1502-1512

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a55b615687603aee17f6d3b645aee114Test
https://doi.org/10.1097/hjh.0000000000001327Test

5

Histone deacetylase and GATA-binding factor 6 regulate arterial remodeling in angiotensin II-induced hypertension

المؤلفون: Ming Quan Lin, Yuhee Ryu, Seon Young Yu, Hae Jin Kee, Sin Young Choi, Myung Ho Jeong, Hyung-Seok Kim, Li Jin, Soo-Na Cho, Gwi Ran Kim

المصدر: Journal of Hypertension. 34:2206-2219

مصطلحات موضوعية: Male, 0301 basic medicine, Physiology, Upstream and downstream (transduction), Gene Expression, Secondary hypertension, Aorta, Thoracic, Blood Pressure, Cell Cycle Proteins, Vascular Remodeling, 030204 cardiovascular system & hematology, Hydroxamic Acids, Kidney, Histone Deacetylases, Muscle, Smooth, Vascular, Muscle hypertrophy, Mice, 03 medical and health sciences, 0302 clinical medicine, GATA6 Transcription Factor, Internal Medicine, medicine, Animals, Pyrroles, Phosphorylation, Cells, Cultured, Cell Proliferation, Cell Nucleus, Cell growth, business.industry, Angiotensin II, DNA, Hypertrophy, medicine.disease, Histone Deacetylase Inhibitors, Repressor Proteins, 030104 developmental biology, Hypertension, Cancer research, Histone deacetylase, Vascular pathology, Calcium-Calmodulin-Dependent Protein Kinase Type 2, Cardiology and Cardiovascular Medicine, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::119ebb757c92d5bfaff641c9cc1f7441Test
https://doi.org/10.1097/hjh.0000000000001081Test

6

Gentisic acid prevents the transition from pressure overload-induced cardiac hypertrophy to heart failure

المؤلفون: Hyung-Seok Kim, Gwi Ran Kim, Seung-Jung Kee, Sin Young Choi, Myung Ho Jeong, Simei Sun, Hae Jin Kee, Yuhee Ryu

المصدر: Scientific Reports, Vol 9, Iss 1, Pp 1-14 (2019)
Scientific Reports

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4a850dd2a423b0d5834c7b852538decTest
https://doi.org/10.1038/s41598-019-39423-8Test

7

Tubastatin A suppresses renal fibrosis via regulation of epigenetic histone modification and Smad3-dependent fibrotic genes

المؤلفون: Sin Young Choi, Myung Ho Jeong, Yuhee Ryu, Hae Jin Kee, Gwi Ran Kim, Inkyeom Kim, Jae Yeong Cho, Soo-Na Cho, Hyung-Seok Kim

المصدر: Vascular Pharmacology. 72:130-140

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::110bf3544a9646dac4348020ad19afdcTest
https://doi.org/10.1016/j.vph.2015.04.006Test

8

Gallic acid improves cardiac dysfunction and fibrosis in pressure overload-induced heart failure

المؤلفون: Hae Jin Kee, Hyung-Seok Kim, Bin Liu, Gwi Ran Kim, Zhe Hao Piao, Li Jin, Sin Young Choi, Myung Ho Jeong, Simei Sun, Yuhee Ryu

المصدر: Scientific Reports, Vol 8, Iss 1, Pp 1-11 (2018)
Scientific Reports

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6b2251ee0ab594a8f200feca735efe3cTest
https://doi.org/10.1038/s41598-018-27599-4Test

9

Gallic Acid Reduces Blood Pressure and Attenuates Oxidative Stress andCardiac Hypertrophy in Spontaneously Hypertensive Rats

المؤلفون: Hae Jin Kee, Myung Ho Jeong, Gwi Ran Kim, Simei Sun, Zhe Hao Piao, Young Mi Seok, Sin Young Choi, Li Jin, Bin Liu, Ming Quan Lin, Yuhee Ryu

المصدر: SCIENTIFIC REPORTS(7)
Scientific Reports, Vol 7, Iss 1, Pp 1-14 (2017)
Scientific Reports

مصطلحات موضوعية: Male, 0301 basic medicine, medicine.medical_specialty, lcsh:Medicine, Blood Pressure, Cardiomegaly, 030204 cardiovascular system & hematology, Left ventricular hypertrophy, Essential hypertension, medicine.disease_cause, Article, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Gallic Acid, Rats, Inbred SHR, Internal medicine, Renin–angiotensin system, medicine, Animals, Humans, cardiovascular diseases, lcsh:Science, Multidisciplinary, NADPH oxidase, biology, Chemistry, GATA4, Angiotensin II, lcsh:R, Blood Pressure Determination, Heart, medicine.disease, Malondialdehyde, GATA4 Transcription Factor, Rats, Oxidative Stress, 030104 developmental biology, Endocrinology, Blood pressure, Gene Expression Regulation, Hypertension, NADPH Oxidase 2, Homeobox Protein Nkx-2.5, biology.protein, cardiovascular system, lcsh:Q, Oxidative stress

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23585899b4d6652c5681d93ca50df413Test
http://open-repository.kisti.re.kr/cube/handle/open_repository/479323.doTest

10

Gallic acid prevents isoproterenol-induced cardiac hypertrophy and fibrosis through regulation of JNK2 signaling and Smad3 binding activity

المؤلفون: Sin Young Choi, Hae Jin Kee, Jae Yeong Cho, Ming Quan Lin, Yuhee Ryu, Gwi Ran Kim, Myung Ho Jeong, Zhe Hao Piao, Li Jin

المصدر: Scientific Reports
SCIENTIFIC REPORTS(6)

مصطلحات موضوعية: 0301 basic medicine, MAPK/ERK pathway, Male, medicine.medical_specialty, Heart Ventricles, Cardiomegaly, Ventricular Function, Left, Article, 03 medical and health sciences, chemistry.chemical_compound, Mice, Downregulation and upregulation, Atrial natriuretic peptide, Fibrosis, Internal medicine, Gallic Acid, medicine, Animals, Mitogen-Activated Protein Kinase 9, Gallic acid, Smad3 Protein, Cells, Cultured, Multidisciplinary, Chemistry, Kinase, Isoproterenol, Brain natriuretic peptide, medicine.disease, Disease Models, Animal, 030104 developmental biology, Endocrinology, Gene Expression Regulation, cardiovascular system, Signal transduction, Protein Binding, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::180d3c4658f1920ea095284ad012eb65Test
https://pubmed.ncbi.nlm.nih.gov/27703224Test

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