نتائج البحث - "Mark Aronovitz"

1

Endothelial Pannexin 1 Regulates Cardiac Response to Myocardial Infarction

المؤلفون: Philip J. Hall, Colleen K. Duffy, Scott R. Johnstone, Brant E. Isakson, Gregory L. Martin, Manxiu Ma, Mark Aronovitz, Robert M. Blanton, Norbert Leitinger, Matthew J. Wolf, Alex P. Young, Miranda E. Good, Abigail G. Wolpe

المصدر: Circ Res

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c9eb3728064f6d7b4c243a075b6c96aTest
https://doi.org/10.1161/circresaha.120.317272Test

2

Distinct Phenotypes Induced by Three Degrees of Transverse Aortic Constriction in Mice

المؤلفون: Phyllis Zhang, Timothy D. Calamaras, Gordon S. Huggins, Mark Aronovitz, Daniel A. Richards, Robert M. Blanton, Kelly Tam, Gregory L. Martin, Peiwen Liu, Heather K. Bowditch

المصدر: Scientific Reports, Vol 9, Iss 1, Pp 1-15 (2019)
Scientific Reports

مصطلحات موضوعية: 0301 basic medicine, Male, medicine.medical_specialty, Cardiac fibrosis, Diastole, lcsh:Medicine, Hemodynamics, Aorta, Thoracic, Constriction, Pathologic, 030204 cardiovascular system & hematology, Left ventricular hypertrophy, Article, Constriction, Mice, Random Allocation, Ventricular Dysfunction, Left, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, medicine, Animals, Cardiac structure, Decompensation, 030212 general & internal medicine, lcsh:Science, Heart Failure, Multidisciplinary, Ejection fraction, business.industry, Aortic constriction, Myocardium, lcsh:R, medicine.disease, Phenotype, Fibrosis, Hypertensive heart disease, 3. Good health, Mice, Inbred C57BL, Disease Models, Animal, stomatognathic diseases, 030104 developmental biology, Heart failure, Cardiology, Hypertrophy, Left Ventricular, lcsh:Q, Cardiology and Cardiovascular Medicine, business, 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b05c37157db5f436e893b04224d9d6e0Test
https://doi.org/10.1016/j.cardfail.2019.07.096Test

3

Targeted disruption of glycogen synthase kinase-3β in cardiomyocytes attenuates cardiac parasympathetic dysfunction in type 1 diabetic Akita mice

المؤلفون: Marian Haburcak, Bo Wang, Jonas B. Galper, Yali Zhang, Sami F. Noujaim, Ho-Jin Park, Mark Aronovitz, Robert M. Blanton, Charles M. Welzig, Thomas Force

المصدر: PLoS ONE, Vol 14, Iss 4, p e0215213 (2019)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3334bc8f6079c266e9f58be1fe5e8753Test
https://pubmed.ncbi.nlm.nih.gov/30978208Test

4

Gut dysbiosis induced by cardiac pressure overload enhances adverse cardiac remodeling in a T cell-dependent manner

المؤلفون: Albert K. Tai, Pilar Alcaide, David Jetton, Marina Anastasiou, Njabulo Ngwenyama, Mark Aronovitz, Francisco J. Carrillo-Salinas, Sasha Smolgovsky, Kuljeet Kaur

المصدر: Gut Microbes, Vol 12, Iss 1 (2020)
Gut Microbes
article-version (VoR) Version of Record

مصطلحات موضوعية: Male, 0301 basic medicine, Cardiac fibrosis, T-Lymphocytes, heart failure, Gut flora, Lymphocyte Activation, Mice, 0302 clinical medicine, Basic Helix-Loop-Helix Transcription Factors, t cells, Ventricular Remodeling, biology, aryl hydrocarbon receptor, Tryptophan, Gastroenterology, Endomyocardial Fibrosis, Infectious Diseases, medicine.anatomical_structure, cardiovascular system, Hypertrophy, Left Ventricular, 030211 gastroenterology & hepatology, medicine.symptom, Research Article, Research Paper, Microbiology (medical), medicine.medical_specialty, T cell, Inflammation, Microbiology, Lymphocyte Depletion, 03 medical and health sciences, Immune system, Internal medicine, Ventricular Pressure, medicine, Animals, lcsh:RC799-869, Pressure overload, Hypertrophy, Right Ventricular, gut microbiota, Fatty Acids, Volatile, medicine.disease, biology.organism_classification, Aryl hydrocarbon receptor, Gastrointestinal Microbiome, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Endocrinology, Receptors, Aryl Hydrocarbon, inflammation, Heart failure, biology.protein, Dysbiosis, lcsh:Diseases of the digestive system. Gastroenterology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::106b679d2ae4e183c4c188987b675982Test
https://doi.org/10.1080/19490976.2020.1823801Test

5

SMOOTH MUSCLE CELL MINERALOCORTICOID RECEPTOR AS A MEDIATOR OF CARDIOVASCULAR STIFFNESS WITH AGING

المؤلفون: Amy McCurley, Isaac E. Stillman, Jennifer J. DuPont, M. Elizabeth Moss, Mark Aronovitz, Seung Kyum Kim, S. Ananth Karumanchi, Demetra D. Christou, Iris Z. Jaffe

مصطلحات موضوعية: 0301 basic medicine, Male, medicine.medical_specialty, medicine.drug_class, Cell, Myocytes, Smooth Muscle, Gene Expression, 030204 cardiovascular system & hematology, Spironolactone, Article, Muscle, Smooth, Vascular, 03 medical and health sciences, Mice, 0302 clinical medicine, Mediator, Mineralocorticoid receptor, Vascular Stiffness, Fibrosis, Internal medicine, Internal Medicine, medicine, Animals, Humans, Receptor, Cellular Senescence, Aged, Mineralocorticoid Receptor Antagonists, Exercise Tolerance, business.industry, Gene Expression Profiling, musculoskeletal system, medicine.disease, Gene expression profiling, 030104 developmental biology, medicine.anatomical_structure, Endocrinology, Blood pressure, Receptors, Mineralocorticoid, Treatment Outcome, Mineralocorticoid, cardiovascular system, Disease Progression, business, hormones, hormone substitutes, and hormone antagonists

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fdd7e29358615b48a4c66365fd6f1f12Test
https://europepmc.org/articles/PMC5843545Test/

6

Left Ventricular T-Cell Recruitment Contributes to the Pathogenesis of Heart Failure

المؤلفون: Ane M. Salvador, Pilar Alcaide, Mark Aronovitz, Navin K. Kapur, Robert M. Blanton, Richard H. Karas, Francisco Velazquez, Tania Nevers, Andrew Knapp, Anna Grodecki-Pena

المصدر: Circulation: Heart Failure. 8:776-787

مصطلحات موضوعية: Adult, Male, medicine.medical_specialty, Pathology, Endothelium, Cardiac fibrosis, Heart Ventricles, T-Lymphocytes, medicine.medical_treatment, T cell, Inflammation, Lymphocyte Activation, Ventricular Function, Left, Article, Muscle hypertrophy, Ventricular Dysfunction, Left, Internal medicine, Cell Adhesion, medicine, Animals, Humans, Ventricular remodeling, Cells, Cultured, Heart Failure, Mice, Knockout, Microscopy, Video, Ventricular Remodeling, business.industry, Endothelial Cells, Middle Aged, medicine.disease, Fibrosis, Myocardial Contraction, Coculture Techniques, Chemotaxis, Leukocyte, Disease Models, Animal, Kinetics, Cytokine, medicine.anatomical_structure, Case-Control Studies, Heart failure, Disease Progression, Cardiology, Hypertrophy, Left Ventricular, medicine.symptom, Cardiology and Cardiovascular Medicine, business, Genes, T-Cell Receptor alpha

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::19f4b2b43fde9572aca5927886080de0Test
https://doi.org/10.1161/circheartfailure.115.002225Test

7

Endothelial mineralocorticoid receptor contributes to systolic dysfunction induced by pressure overload without modulating cardiac hypertrophy or inflammation

المؤلفون: Ane M. Salvador, Mark Aronovitz, M. Elizabeth Moss, Pilar Alcaide, Iris Z. Jaffe, Kathleen B. Mueller, Robert M. Blanton

المصدر: Physiological Reports

مصطلحات موضوعية: 0301 basic medicine, Cardiac function curve, Cardiovascular Conditions, Disorders and Treatments, Male, medicine.medical_specialty, Physiology, Cardiac fibrosis, Systole, Immunology, heart failure, Inflammation, Blood Pressure, Cardiomegaly, 030204 cardiovascular system & hematology, Biology, Proinflammatory cytokine, Signalling Pathways, 03 medical and health sciences, chemistry.chemical_compound, Mice, 0302 clinical medicine, Mineralocorticoid receptor, Physiology (medical), Internal medicine, medicine, Ventricular Dysfunction, Animals, Cells, Cultured, Original Research, mineralocorticoid receptor, Pressure overload, Aldosterone, Ventricular Remodeling, Tumor Necrosis Factor-alpha, medicine.disease, Intercellular Adhesion Molecule-1, Fibrosis, Mice, Inbred C57BL, 030104 developmental biology, Endocrinology, Receptors, Mineralocorticoid, chemistry, Heart failure, Cardiology, endothelial cell, Endothelium, Vascular, medicine.symptom, Adhesion molecules

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18e89ab9b16f31c4e365eb26f13c91c6Test
http://europepmc.org/articles/PMC5492203Test

8

Reduced Activin Receptor Like-Kinase 1 Activity Promotes Cardiac Fibrosis in Heart Failure

المؤلفون: Lyanne Buiten, Kevin J. Morine, Emily E. Mackey, Prerna Nepali, Robert M. Blanton, Richard H. Karas, Keshan Ughreja, S. Paul Oh, Mark Aronovitz, Jonathan Levine, Navin K. Kapur, Xiaoying Qiao, Vikram Paruchuri

مصطلحات موضوعية: 0301 basic medicine, Adult, Male, medicine.medical_specialty, Cardiac fibrosis, medicine.medical_treatment, Activin Receptors, Type II, Article, Pathology and Forensic Medicine, 03 medical and health sciences, Mice, Fibrosis, Internal medicine, TGF beta signaling pathway, medicine, Animals, Humans, Heart Failure, biology, Ventricular Remodeling, business.industry, Myocardium, General Medicine, Transforming growth factor beta, Middle Aged, medicine.disease, Brain natriuretic peptide, 030104 developmental biology, Cytokine, Endocrinology, Heart failure, biology.protein, Female, Cardiology and Cardiovascular Medicine, business, Activin Receptors, Type I, Transforming growth factor

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99e7d3078e647d8f759c1fbb78bca7abTest
https://europepmc.org/articles/PMC5863230Test/

9

Aldosterone Promotes Vascular Remodeling by Direct Effects on Smooth Muscle Cell Mineralocorticoid Receptors

المؤلفون: S. Ananth Karumanchi, Iris Z. Jaffe, Mark Aronovitz, Dafina Pruthi, Amy McCurley, Carol Galayda

المصدر: Arteriosclerosis, Thrombosis, and Vascular Biology. 34:355-364

مصطلحات موضوعية: Male, Placental growth factor, medicine.medical_specialty, Time Factors, medicine.drug_class, Myocytes, Smooth Muscle, Pregnancy Proteins, Biology, Antibodies, Muscle, Smooth, Vascular, Article, Mice, chemistry.chemical_compound, Fibrosis, Internal medicine, medicine, Animals, RNA, Messenger, Receptor, Aldosterone, Cell Proliferation, Placenta Growth Factor, Mice, Knockout, Vascular Endothelial Growth Factor Receptor-1, Cell growth, Endothelial Cells, medicine.disease, Mice, Inbred C57BL, Vascular endothelial growth factor B, Disease Models, Animal, Carotid Arteries, Receptors, Mineralocorticoid, Endocrinology, chemistry, Mineralocorticoid, Knockout mouse, cardiovascular system, Carotid Artery Injuries, Cardiology and Cardiovascular Medicine

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd6abf6f52188708c765f294546f8ad4Test
https://doi.org/10.1161/atvbaha.113.302854Test

10

Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Infiltration, Cardiac Remodeling, and Function in Pressure Overload–Induced Heart Failure

المؤلفون: Mark Aronovitz, Ane M. Salvador, Robert M. Blanton, Richard H. Karas, Francisco Velazquez, Tania Nevers, Bonnie Wang, Ana Molina, Iris Z. Jaffe, Pilar Alcaide

المصدر: Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease

مصطلحات موضوعية: 0301 basic medicine, Male, Time Factors, Cardiac fibrosis, T-Lymphocytes, Interleukin-1beta, Aorta, Thoracic, 030204 cardiovascular system & hematology, Lymphocyte Activation, Ventricular Function, Left, Ventricular Dysfunction, Left, 0302 clinical medicine, Medicine, Thoracic aorta, adhesion molecules, Myofibroblasts, Cells, Cultured, Original Research, remodeling, Mice, Knockout, Ventricular Remodeling, Intercellular Adhesion Molecule-1, Constriction, Chemotaxis, Leukocyte, medicine.anatomical_structure, Cardiology, cardiovascular system, Disease Progression, Hypertrophy, Left Ventricular, Cardiology and Cardiovascular Medicine, Signal Transduction, medicine.medical_specialty, Heart Ventricles, Diastole, Bone Marrow Cells, Proinflammatory cytokine, 03 medical and health sciences, medicine.artery, Internal medicine, Animals, Arterial Pressure, Ventricular remodeling, Pressure overload, Heart Failure, business.industry, Interleukin-6, Macrophages, medicine.disease, Fibrosis, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Ventricle, inflammation, Heart failure, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::39de25ade0b349e7cbf51b679bea4648Test
http://europepmc.org/articles/PMC4943280Test

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