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المؤلفون: Francesco Bertoni, Steven A. Carr, Manuela Cavalli, Holger Moch, Anna Rinaldi, Arianna Vallerga, Azzurra Mutti, Tanya Svinkina, Ze Dong, Hana Janouskova, Geniver El Tekle, Tiziano Bernasocchi, Simon Linder, Giuseppina M. Carbone, Daniela Bossi, Marianna Kruithof-de Julio, Roger Geiger, Laura P. Brandt, Wilbert Zwart, Andrea Rinaldi, Simone Mosole, Marita Zoma, Marco Bolis, Jean-Philippe Theurillat, Filippo Spriano, Andrea Alimonti, Peter Schraml, Cai-Guang Yang, Domenico Albino, Valentina Ceserani, Namrata D. Udeshi, Mark A. Rubin
المصدر: Nature Communications
Nature Communications, Vol 12, Iss 1, Pp 1-18 (2021)
Bernasocchi, Tiziano; El Tekle, Geniver; Bolis, Marco; Mutti, Azzurra; Vallerga, Arianna; Brandt, Laura P.; Spriano, Filippo; Svinkina, Tanya; Zoma, Marita; Ceserani, Valentina; Rinaldi, Anna; Janouskova, Hana; Bossi, Daniela; Cavalli, Manuela; Mosole, Simone; Geiger, Roger; Dong, Ze; Yang, Cai-Guang; Albino, Domenico; Rinaldi, Andrea; ... (2021). Dual functions of SPOP and ERG dictate androgen therapy responses in prostate cancer. Nature communications, 12(1), p. 734. Nature Publishing Group 10.1038/s41467-020-20820-x <http://dx.doi.org/10.1038/s41467-020-20820-xTest>
Nature Communications, 12مصطلحات موضوعية: Proteomics, Male, Cancer therapy, medicine.drug_class, Science, Mice, Nude, 610 Medicine & health, Cell Cycle Proteins, SPOP, medicine.disease_cause, Article, Tumour biomarkers, Prostate cancer, Mice, Transcriptional Regulator ERG, Cell Line, Tumor, medicine, Biomarkers, Tumor, Animals, Humans, Immunoprecipitation, Transcription factor, Cancer genetics, Oncogene Proteins, biology, Nuclear Proteins, Prostatic Neoplasms, Ubiquitin-Protein Ligase Complexes, medicine.disease, Androgen, Immunohistochemistry, Ubiquitin ligase, Androgen receptor, DNA-Binding Proteins, Repressor Proteins, HEK293 Cells, Androgen Therapy, Receptors, Androgen, Mutation, biology.protein, Cancer research, Carcinogenesis, 610 Medizin und Gesundheit, Co-Repressor Proteins, Protein Binding, Signal Transduction
وصف الملف: application/pdf; application/application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4fe8919843ab06744fd7a44c9ed8c87aTest
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المؤلفون: Carlo V. Catapano, Giuseppina M. Carbone, Andrea Zitella, Fausto Sessa, Jessica Merulla, Gianluca Civenni, George N. Thalmann, Simona Rossi, Giovanna Chiorino, Gioacchino D'Ambrosio, Marco Losa, Domenico Albino, Paola Ostano, Maurizia Mello-Grand, Ramón García-Escudero, Cecilia Dallavalle
المصدر: Dallavalle, Cecilia; Albino, Domenico; Civenni, Gianluca; Merulla, Jessica; Ostano, Paola; Mello-Grand, Maurizia; Rossi, Simona; Losa, Marco; D'Ambrosio, Gioacchino; Sessa, Fausto; Thalmann, George; Garcia-Escudero, Ramon; Zitella, Andrea; Chiorino, Giovanna; Catapano, Carlo V; Carbone, Giuseppina M (2016). MicroRNA-424 impairs ubiquitination to activate STAT3 and promote prostate tumor progression. Journal of clinical investigation, 126(12), pp. 4585-4602. American Society for Clinical Investigation 10.1172/JCI86505 <http://dx.doi.org/10.1172/JCI86505Test>
مصطلحات موضوعية: Male, STAT3 Transcription Factor, 0301 basic medicine, Ubiquitin-Protein Ligases, Mice, Nude, 610 Medicine & health, medicine.disease_cause, Mice, 03 medical and health sciences, 0302 clinical medicine, Ubiquitin, Cell Line, Tumor, medicine, Animals, Humans, Gene silencing, RNA, Neoplasm, STAT3, Transcription factor, Mice, Inbred BALB C, biology, Medicine (all), fungi, Protein turnover, Prostatic Neoplasms, General Medicine, Neoplasm Proteins, Ubiquitin ligase, MicroRNAs, 030104 developmental biology, Tumor progression, 030220 oncology & carcinogenesis, Neoplastic Stem Cells, biology.protein, Cancer research, Carcinogenesis, Transcription Factors, Research Article
وصف الملف: application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9850d2ba51cd08aeabc7246dd9860c1cTest
https://boris.unibe.ch/92727/1/render.pdfTest -
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المؤلفون: Fausto Sessa, Nicole Longoni, George N. Thalmann, Carlo V. Catapano, Manuela Sarti, Giuseppina M. Carbone, Laura Curti, Gianluca Civenni, Sandra Pinton, Domenico Albino, Giovanna Chiorino, Maurizia Mello-Grand, Gioacchino D'Ambrosio
المصدر: Cancer research
مصطلحات موضوعية: Male, Oncology, Cancer Research, medicine.medical_specialty, Epithelial-Mesenchymal Transition, Biology, medicine.disease_cause, Mice, 03 medical and health sciences, Prostate cancer, 0302 clinical medicine, Transcriptional Regulator ERG, Cancer stem cell, Internal medicine, medicine, Animals, Humans, Epithelial–mesenchymal transition, 030304 developmental biology, Epithelial cell differentiation, 0303 health sciences, ETS transcription factor family, ETS Homologous Factor, Prostatic Neoplasms, Cell Differentiation, Epithelial Cells, medicine.disease, 3. Good health, Gene Expression Regulation, Tumor progression, 030220 oncology & carcinogenesis, Neoplastic Stem Cells, Trans-Activators, Carcinogenesis, Transcription Factors
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5bb074bdd0236894a1176ba18af17111Test
http://hdl.handle.net/11383/1758077Test