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1
المؤلفون: Saburo Ito, Jun Araya, Katsutoshi Nakayama, Kenji Kobayashi, Hiromichi Hara, Naoki Takasaka, Kazuyoshi Kuwano, Shunsuke Minagawa
المصدر: Respiratory Investigation. 54:397-406
مصطلحات موضوعية: 0301 basic medicine, Pulmonary and Respiratory Medicine, Senescence, Aging, Pathogenesis, Pulmonary Disease, Chronic Obstructive, 03 medical and health sciences, Idiopathic pulmonary fibrosis, 0302 clinical medicine, Autophagy, Leukocytes, Homeostasis, Humans, Medicine, Cellular Senescence, Telomere Shortening, Lung, business.industry, Epithelial Cells, medicine.disease, Idiopathic Pulmonary Fibrosis, respiratory tract diseases, Telomere, Pulmonary Alveoli, 030104 developmental biology, medicine.anatomical_structure, 030220 oncology & carcinogenesis, Immunology, Cytokines, Cytokine secretion, business, Myofibroblast
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5d28632d51dc147b78a10810fafb3b3bTest
https://doi.org/10.1016/j.resinv.2016.03.010Test -
2Cigarette smoke exposure worsens acute lung injury in antibiotic-treated bacterial pneumonia in mice
المؤلفون: Xiaohui Fang, Suzaynn F. Schick, Jason Abbott, Carolyn S. Calfee, Matthew L. Springer, Jeffrey E. Gotts, Naoki Takasaka, Lauren Chun, Michael A. Matthay, Stephen L. Nishimura
المصدر: American journal of physiology. Lung cellular and molecular physiology, vol 315, iss 1
Gotts, JE; Chun, L; Abbott, J; Fang, X; Takasaka, N; Nishimura, SL; et al.(2018). Cigarette smoke exposure worsens acute lung injury in antibiotic-treated bacterial pneumonia in mice. American Journal of Physiology-Lung Cellular and Molecular Physiology, 315(1), L25-L40. doi: 10.1152/ajplung.00405.2017. UCSF: Retrieved from: http://www.escholarship.org/uc/item/26t407hcTestمصطلحات موضوعية: 0301 basic medicine, ARDS, Physiology, Medical Physiology, Respiratory System, medicine.disease_cause, 2.2 Factors relating to physical environment, Mice, 0302 clinical medicine, 2.2 Factors relating to the physical environment, 2.1 Biological and endogenous factors, Aetiology, Lung, Acute Respiratory Distress Syndrome, medicine.diagnostic_test, cigarette smoke, Bacterial, Anti-Bacterial Agents, medicine.anatomical_structure, Streptococcus pneumoniae, Infectious Diseases, Smoking and Health, 5.1 Pharmaceuticals, Pneumococcal pneumonia, Pneumonia & Influenza, Respiratory, Pneumococcal, Female, Development of treatments and therapeutic interventions, Infection, pneumococcus, Research Article, Pulmonary and Respiratory Medicine, Acute Lung Injury, Pulmonary Edema, Lung injury, 03 medical and health sciences, Rare Diseases, Physiology (medical), Tobacco, Pneumonia, Bacterial, medicine, Animals, pneumonia, Tobacco Smoke and Health, business.industry, Prevention, Bacterial pneumonia, Cell Biology, Pneumonia, Pneumococcal, medicine.disease, respiratory tract diseases, Pneumonia, 030104 developmental biology, Bronchoalveolar lavage, Good Health and Well Being, 030228 respiratory system, Immunology, Tobacco Smoke Pollution, business
وصف الملف: application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3126195881829f5218fd6fc42769e51Test
https://escholarship.org/uc/item/26t407hcTest -
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المؤلفون: Haru Yanagisawa, Naoki Takasaka, Jeffrey E. Gotts, Carolyn S. Calfee, Xiaohui Fang, Michael A. Matthay, Jason Abbott, Stephen L. Nishimura
المصدر: Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco, vol 19, iss 9
Gotts, JE; Abbott, J; Fang, X; Yanagisawa, H; Takasaka, N; Nishimura, SL; et al.(2017). Cigarette smoke exposure worsens endotoxin-induced lung injury and pulmonary edema in mice. Nicotine and Tobacco Research, 19(9), 1033-1039. doi: 10.1093/ntr/ntx062. UCSF: Retrieved from: http://www.escholarship.org/uc/item/9nw9b9pzTestمصطلحات موضوعية: 0301 basic medicine, Adult, Lipopolysaccharides, ARDS, Pulmonary toxicity, medicine.medical_treatment, Clinical Sciences, Original Investigations, Pulmonary Edema, Lung injury, 03 medical and health sciences, Mice, 0302 clinical medicine, medicine, Animals, Lung, Marketing, Respiratory Distress Syndrome, medicine.diagnostic_test, business.industry, Public Health, Environmental and Occupational Health, Lung Injury, respiratory system, Pulmonary edema, medicine.disease, Neutrophilia, respiratory tract diseases, 030104 developmental biology, medicine.anatomical_structure, Bronchoalveolar lavage, Cytokine, 030228 respiratory system, Immunology, Public Health and Health Services, Tobacco Smoke Pollution, Public Health, medicine.symptom, business
وصف الملف: application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c959d84694ae6b28f1f55323cf9462aTest
https://escholarship.org/uc/item/9nw9b9pzTest -
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المؤلفون: Saburo Ito, Amanda Goodsell, Naoki Takasaka, Mitsuo Hashimoto, Jody L. Baron, Haruhiko Yanagisawa, Stephen L. Nishimura, Shunsuke Minagawa, Catherine Moermans, Royce Ma, Alison L. Budelsky, Jun Araya
المصدر: American journal of physiology. Lung cellular and molecular physiology, vol 312, iss 1
Yanagisawa, H; Hashimoto, M; Minagawa, S; Takasaka, N; Ma, R; Moermans, C; et al.(2016). Role of Il-17A in murine models of COPD airway disease. American Journal of Physiology-Lung Cellular and Molecular Physiology, 312(1), L122-L130. doi: 10.1152/ajplung.00301.2016. UCSF: Retrieved from: http://www.escholarship.org/uc/item/5c63d8ttTestمصطلحات موضوعية: 0301 basic medicine, Physiology, Pulmonary Fibrosis, Respiratory System, Medical Physiology, Interleukin-1beta, Inbred C57BL, interleukin-17, Mice, Pulmonary Disease, Chronic Obstructive, 0302 clinical medicine, Fibrosis, Receptors, Pulmonary fibrosis, 2.1 Biological and endogenous factors, Lung, COPD, Receptors, Interleukin-17, cigarette smoke, Innate lymphoid cell, Interleukin-17, Smoking, respiratory system, Respiratory, Interleukin 17, medicine.symptom, Research Article, Pulmonary and Respiratory Medicine, Chronic Obstructive, Chronic Obstructive Pulmonary Disease, Inflammation, Adenoviridae, Pulmonary Disease, 03 medical and health sciences, Neutralization Tests, Physiology (medical), medicine, Animals, Animal, business.industry, fibrosis, Cell Biology, Pneumonia, medicine.disease, respiratory tract diseases, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Poly I-C, 030228 respiratory system, airway, inflammation, Disease Models, Immunology, Airway, business, CD8
وصف الملف: application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::037918cdf8d43f9f1b498320a3a7539aTest
https://europepmc.org/articles/PMC5283930Test/ -
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المؤلفون: Toshiaki Morikawa, Jun Kojima, Kenichiro Shimizu, Saburo Ito, Naoki Takasaka, Yusuke Kurita, Takanori Numata, Jun Araya, Makoto Kawaishi, Masahiro Yoshida, Kazuyoshi Kuwano, Hiroshi Wakui, Satoko Fujii, Yumi Kaneko, Kenji Kobayashi, Stephen L. Nishimura, Makoto Odaka, Katsutoshi Nakayama, Hiromichi Hara, Shunsuke Minagawa, Toru Harada
المصدر: Autophagy
مصطلحات موضوعية: Male, Baf A1, bafilomycin A1, Mitochondrial ROS, senescence, PINK1, PTEN-induced putative kinase 1, Mitochondrial Degradation, NAC, N-acetylcysteine, CS, cigarette smoke, Mitochondrion, Pulmonary Disease, Chronic Obstructive, HBEC, human bronchial epithelial cell, Mitophagy, PCD, programmed cell death, Lung, Cellular Senescence, Smoking, ROS, Tobacco Products, Middle Aged, Immunohistochemistry, Mitochondria, Cell biology, Biochemistry, Female, Cell aging, Adult, Senescence, Ubiquitin-Protein Ligases, Bronchi, PINK1, Biology, ROS, reactive oxygen species, MAP1LC3/LC3, microtubule-associated protein 1 light chain 3, Autophagy, Humans, COPD, TLR, toll-like receptor, Molecular Biology, Aged, EM, electron microscopy, FEV1, forced expiratory volume in one second, Epithelial Cells, Cell Biology, PARK2, WB, western blotting, respiratory tract diseases, Microscopy, Electron, COPD, chronic obstructive pulmonary disease, FVC, forced vital capacity, SA-β-Gal, senescence-associated β-galactosidase, Clinical Research Paper, Reactive Oxygen Species, Protein Kinases, CSE, cigarette smoke extract
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f12d72e8c6593d2486e43cd47706f378Test
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المؤلفون: Toshiaki Morikawa, Yoko Yumino, Saburo Ito, Makoto Odaka, Makoto Kawaishi, Kazuyoshi Kuwano, Naoki Takasaka, Katsutoshi Nakayama, Stephen L. Nishimura, Shunsuke Minagawa, Takanori Numata, Jun Hirano, Jun Araya, Hiromichi Hara, Satoko Fujii, Jun-ichi Kojima, Takeo Ishikawa
المصدر: Oncoimmunology, vol 1, iss 5
Oncoimmunologyمصطلحات موضوعية: Senescence, autophagy, senescence, Chronic Obstructive Pulmonary Disease, Cell, Immunology, Oncology and Carcinogenesis, Biology, Pathogenesis, Ubiquitin, Tobacco, ubiquitin, medicine, Immunology and Allergy, 2.1 Biological and endogenous factors, COPD, Secretion, Aetiology, Lung, Tobacco Smoke and Health, Autophagy, p62, Interleukin, medicine.disease, Cell biology, respiratory tract diseases, medicine.anatomical_structure, Good Health and Well Being, Oncology, biology.protein, Respiratory, Research Paper
وصف الملف: application/pdf
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b0af84350e99bc0ca92919b853189104Test
https://escholarship.org/uc/item/0775b5knTest