نتائج البحث - "Type I interferon production"

1

Host Genetic Risk Factors for Chlamydia trachomatis-Related Infertility in Women

المؤلفون: Yutong Liu, Catherine L. Haggerty, William M. Geisler, Wujuan Zhong, Robert D. Kirkcaldy, Xiaojing Zheng, Harold C. Wiesenfeld, Yun Li, Gloria E Anyalechi, Catherine M. O'Connell, Karen R. Hammond, Jason P. Fine, Toni Darville, Sharon L. Hillier, Michael P. Steinkampf

المصدر: J Infect Dis

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d56f61335d0a5accc1a4a0356b85611Test
https://doi.org/10.1093/infdis/jiab149Test

2

A paucigranulocytic asthma host environment promotes the emergence of virulent influenza viral variants

المؤلفون: Monique I. J. Spronken, Anjana C. Karawita, Alexander A. Khromykh, Keng Yih Chew, Cassandra L. Pegg, Lauren E. Steele, Kirsty R. Short, Myrna J. M. Bunte, Simon Phipps, Yin Xiang Setoh, Nathalie A. J. Verzele, Katina D. Hulme, Kyle R. Upton, Helle Bielefeldt-Ohmann, Conor J. Bloxham, Silvie Van den Hoecke, Bram Vrancken, Maria B. Sukkar

المساهمون: Virology

المصدر: eLife, Vol 10 (2021)
eLife
eLife, 10:e61803, 1-21. eLife Sciences Publications

مصطلحات موضوعية: 0301 basic medicine, Male, Mouse, viruses, Receptor for Advanced Glycation End Products, 0601 Biochemistry and Cell Biology, medicine.disease_cause, influenza virus, immunology, Mice, Immunology and Inflammation, Influenza A Virus, H1N1 Subtype, Interferon, Biology (General), Coronavirus, Mutation, Microbiology and Infectious Disease, General Neuroscience, General Medicine, 3. Good health, interferons, host-pathogen evolution, Host-Pathogen Interactions, Medicine, Female, medicine.drug, Research Article, Human, QH301-705.5, infectious disease, Science, 030106 microbiology, Virulence, Biology, General Biochemistry, Genetics and Molecular Biology, Virus, 03 medical and health sciences, Orthomyxoviridae Infections, medicine, Animals, human, mouse, Asthma, General Immunology and Microbiology, microbiology, COVID-19, asthma, Type I interferon production, medicine.disease, Mice, Inbred C57BL, 030104 developmental biology, inflammation, Infectious disease (medical specialty), Immunology, mutation

وصف الملف: application/pdf; Electronic

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::341c42c431fa162d9b26f731140706d5Test
https://doi.org/10.7554/elife.61803Test

3

DDX58 and Classic Singleton-Merten Syndrome

المؤلفون: Carlos Ferreira, Michele Nehrebecky, Yanick J. Crow, Ji Woo Park, Adriana Almeida de Jesus, William A. Gahl, Tracy A Briggs, Sun Hur, Pamela J. Gardner, Raphaela Goldbach-Mansky

المصدر: Journal of Clinical Immunology
Ferreira, C R, Crow, Y J, Gahl, W A, Gardner, P J, Goldbach-Mansky, R, Hur, S, de Jesús, A A, Nehrebecky, M, Park, J W & Briggs, T A 2019, ' DDX58 and Classic Singleton-Merten Syndrome ', Journal of clinical immunology, vol. 39, no. 1, pp. 75-80 . https://doi.org/10.1007/s10875-018-0572-1Test

مصطلحات موضوعية: 0301 basic medicine, Male, Pathology, Interferonopathy, Lydia Becker Institute, medicine.disease_cause, 0302 clinical medicine, Interferon, Odontodysplasia, Immunology and Allergy, Receptors, Immunologic, Promoter Regions, Genetic, retinoic acid-inducible gene I, Mutation, Middle Aged, Phenotype, Singleton-Merten syndrome, Gain of Function Mutation, Interferon Type I, type I interferon, DEAD Box Protein 58, Original Article, Dental Enamel Hypoplasia, Female, Metacarpus, medicine.drug, Adult, medicine.medical_specialty, Singleton Merten syndrome, Immunology, Aortic Diseases, Cell Line, 03 medical and health sciences, Muscular Diseases, Psoriasis, ResearchInstitutes_Networks_Beacons/lydia_becker_institute_of_immunology_and_inflammation, Arthropathy, medicine, Humans, Vascular Calcification, business.industry, Sequela, medicine.disease, Type I interferon production, 030104 developmental biology, HEK293 Cells, Osteoporosis, business, 030215 immunology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb649cc4efdfd038ec4ae95dc3cc2dc6Test
http://europepmc.org/articles/PMC6394545Test

4

The Nucleoprotein of H7N9 Influenza Virus Positively Regulates TRAF3-Mediated Innate Signaling and Attenuates Viral Virulence in Mice

المؤلفون: Qiyun Zhu, Bo Zhang, Yanli Wei, Yan Zeng, Shuai Xu, Cao-Qi Lei, Zhengxiang Wang, Yingying Du, Xuegang Zhang

المصدر: J Virol

مصطلحات موضوعية: Viral protein, viruses, Immunology, Gene Expression, Cellular Response to Infection, Apoptosis, Biology, Influenza A Virus, H7N9 Subtype, Virus Replication, medicine.disease_cause, Microbiology, Virus, Mice, Influenza A Virus, H1N1 Subtype, Immune system, Interferon, Virology, Influenza A virus, medicine, Animals, Humans, Lung, Mice, Inbred BALB C, Innate immune system, TNF Receptor-Associated Factor 3, Virulence, Ubiquitination, Type I interferon production, Baculoviral IAP Repeat-Containing 3 Protein, Immunity, Innate, Cell biology, Disease Models, Animal, Nucleoproteins, Viral replication, A549 Cells, Insect Science, Interferon Type I, DEAD Box Protein 58, Female, Signal Transduction, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45da1edecf37b76c707b0953c396420fTest
https://doi.org/10.1128/jvi.01640-20Test

5

Decreased Type I Interferon Production by Plasmacytoid Dendritic Cells Contributes to Severe Dengue

المؤلفون: Denis R. St. Laurent, Darragh Duffy, Veasna Duong, Tineke Cantaert, Axelle Vanderlinden, Carolina Scagnolari, Nikaïa Smith, Vinit Upasani, Federica Frasca, Philippe Dussart, Vincent Bondet, Sokchea Lay, Guido Antonelli, Sothy Heng, Heidi Auerswald, Sowath Ly

المساهمون: Immunologie [Phnom Penh], Institut Pasteur du Cambodge, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), University of Groningen [Groningen], Department of Molecular Medicine, Institut Pasteur, Fondation Cenci Bolognetti - Istituto Pasteur Italia, Fondazione Cenci Bolognetti, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome] (UNIROMA), Réseau International des Instituts Pasteur (RIIP), Immunologie Translationnelle - Translational Immunology lab, Institut Pasteur [Paris] (IP), Kantha Bopha Hospitals Foundation, This study was supported by a grant from Institut Pasteur (ACIP n°221 -2019, DeSiReS: Dengue Severe Immune Response Studies). TC was funded by the Institute Pasteur International Network and is an HHMI-Wellcome Trust International Research Scholar (208710/Z/17/Z). VU was funded by the Institute Pasteur International Network Calmette and Yersin Ph.D. scholarship. DD acknowledges support from the Agence Nationale de la Recherche (ANR grant number CE17001002)., ANR-16-CE17-0005,GENMSMD,Dissection génétique de la Susceptibilité Mendélienne aux infections mycobactériennes chez l'homme(2016), ANR-16-CE17-0010,IFNX,Investigation des interferonopathies type I humaine(2016), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome], Institut Pasteur [Paris]

المصدر: Frontiers in Immunology, Vol 11 (2020)
Frontiers in Immunology
Frontiers in Immunology, 2020, 11, pp.605087. ⟨10.3389/fimmu.2020.605087⟩
Frontiers in Immunology, Frontiers, 2020, 11, pp.605087. ⟨10.3389/fimmu.2020.605087⟩
Frontiers in Immunology, 11:605087. Frontiers Media SA

مصطلحات موضوعية: Male, Time Factors, viruses, Plasmacytoid dendritic cell, Dengue virus, medicine.disease_cause, Antibodies, Viral, Severity of Illness Index, DISEASE, Dengue, 0302 clinical medicine, Immunology and Allergy, Platelet, Child, Original Research, 0303 health sciences, interferon stimulated genes, Antibody titer, virus diseases, simoa immunoassay, Viral Load, Acquired immune system, Prognosis, myloid dendritic cells, Single Molecule Imaging, 3. Good health, B-CELLS, Child, Preschool, Host-Pathogen Interactions, Interferon Type I, POSTTRANSCRIPTIONAL REGULATION, [SDV.IMM]Life Sciences [q-bio]/Immunology, type I interferon, Female, Cambodia, Viral load, lcsh:Immunologic diseases. Allergy, Adolescent, Immunology, Enzyme-Linked Immunosorbent Assay, RECEPTOR IFNAR-2, 03 medical and health sciences, Immune system, ENHANCEMENT, medicine, plasmacytoid dendritic cell, Humans, 030304 developmental biology, dengue virus, business.industry, Platelet Count, Dendritic Cells, biochemical phenomena, metabolism, and nutrition, Type I interferon production, Antibodies, Neutralizing, ALPHA, VIRUS-INFECTION REQUIRES, HEMORRHAGIC-FEVER, Case-Control Studies, IFN-BETA, business, lcsh:RC581-607, 030215 immunology, RESPONSES

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab42c88242b43c752954a404e305f0d5Test
http://hdl.handle.net/11573/1473176Test

6

TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis

المؤلفون: Alexander A. Navarini, Curdin Conrad, Alessio Mylonas, Anne-Karine Lapointe, Jeremy Di Domizio, Olivier Demaria, Michel Gilliet, Cyrine Belkhodja, Lars E. French, Maxime Vernez

المساهمون: University of Zurich, Conrad, Curdin

المصدر: Nature Communications, Vol 9, Iss 1, Pp 1-11 (2018)
Nature Communications
Nature communications, vol. 9, no. 1, pp. 25

مصطلحات موضوعية: 0301 basic medicine, Male, T-Lymphocytes, General Physics and Astronomy, Autoimmunity, medicine.disease_cause, 030207 dermatology & venereal diseases, 0302 clinical medicine, Crohn Disease, Interferon, lcsh:Science, Cells, Cultured, Mice, Inbred BALB C, Multidisciplinary, 10177 Dermatology Clinic, Antibodies, Monoclonal, Middle Aged, 3100 General Physics and Astronomy, Adalimumab/adverse effects, Adalimumab/immunology, Adalimumab/therapeutic use, Adolescent, Adult, Aged, Animals, Antibodies, Monoclonal/adverse effects, Antibodies, Monoclonal/immunology, Antibodies, Monoclonal/therapeutic use, Autoimmunity/drug effects, Autoimmunity/immunology, Crohn Disease/drug therapy, Crohn Disease/immunology, Crohn Disease/metabolism, Cytokines/genetics, Cytokines/immunology, Cytokines/metabolism, Dendritic Cells/drug effects, Dendritic Cells/immunology, Dendritic Cells/metabolism, Female, Humans, Infliximab/adverse effects, Infliximab/immunology, Infliximab/therapeutic use, Interferon Type I/genetics, Interferon Type I/immunology, Interferon Type I/metabolism, Psoriasis/chemically induced, Psoriasis/immunology, T-Lymphocytes/drug effects, T-Lymphocytes/immunology, T-Lymphocytes/metabolism, Tumor Necrosis Factor-alpha/antagonists & inhibitors, Tumor Necrosis Factor-alpha/immunology, Tumor Necrosis Factor-alpha/metabolism, Young Adult, Interferon Type I, Cytokines, Tumor necrosis factor alpha, medicine.drug, Science, 610 Medicine & health, 1600 General Chemistry, General Biochemistry, Genetics and Molecular Biology, Article, 03 medical and health sciences, 1300 General Biochemistry, Genetics and Molecular Biology, Psoriasis, medicine, Adalimumab, business.industry, Tumor Necrosis Factor-alpha, General Chemistry, Dendritic Cells, medicine.disease, Type I interferon production, Infliximab, 030104 developmental biology, Immunology, lcsh:Q, business, Interferon type I

وصف الملف: application/pdf; 2018_LF_TNF_blockade_induces_a_dysregulated_type_I_interferon_response_without_autoimmunity_in_paradoxical_psoriasis.pdf - application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbbc7decb248c10970a313c075620f0fTest
https://doaj.org/article/180fc992d72e4fcbbf1d606055e86015Test