نتائج البحث - "S. Chamberlain"

1

Gene Therapy Rescues Cardiac Dysfunction in Duchenne Muscular Dystrophy Mice by Elevating Cardiomyocyte Deoxy-Adenosine Triphosphate

المؤلفون: John K. Hall, Farid Moussavi-Harami, Stephen C. Kolwicz, Jeffrey S. Chamberlain, Stephen D. Hauschka, Xiolan Chen, Michael Regnier, Guy L. Odom

المصدر: JACC: Basic to Translational Science, Vol 4, Iss 7, Pp 778-791 (2019)
JACC: Basic to Translational Science

مصطلحات موضوعية: 0301 basic medicine, musculoskeletal diseases, medicine.medical_specialty, congenital, hereditary, and neonatal diseases and abnormalities, lcsh:Diseases of the circulatory (Cardiovascular) system, Duchenne muscular dystrophy, Genetic enhancement, viruses, Cardiomyopathy, rAAV, recombinant adeno-associated viral vector, 030204 cardiovascular system & hematology, ribonucleotide reductase, dystrophin, 03 medical and health sciences, PRECLINICAL RESEARCH, 0302 clinical medicine, μDys, microdystrophin, Internal medicine, recombinant adeno-associated virus vectors, cTnT, cardiac troponin T, Medicine, Vector (molecular biology), biology, business.industry, dADP, deoxy-adenosine diphosphate, Cardiac muscle, Cardiac reserve, RNR, ribonucleotide reductase, CMV, cytomegalovirus, DMD, Duchenne muscular dystrophy, medicine.disease, 3. Good health, 030104 developmental biology, Endocrinology, medicine.anatomical_structure, Ribonucleotide reductase, lcsh:RC666-701, CK8, miniaturized murine creatine kinase regulatory cassette, biology.protein, diastolic dysfunction, dATP, deoxy-adenosine triphosphate, Cardiology and Cardiovascular Medicine, business, Dystrophin, cardiomyopathy, mdx, mouse muscular dystrophy model

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::35da563ba80c05668887e4f5b7413550Test
http://www.sciencedirect.com/science/article/pii/S2452302X19301913Test

2

A Five-Repeat Micro-Dystrophin Gene Ameliorated Dystrophic Phenotype in the Severe DBA/2J-mdx Model of Duchenne Muscular Dystrophy

المؤلفون: Xiufang Pan, Brittney Haffner, N. Nora Yang, Sean X. Duan, Jeffrey S. Chamberlain, Gang Yao, Julian Ramos, Joel S. Schneider, Keqing Zhang, Dongsheng Duan, Kasun Kodippili, Nalinda B. Wasala, Chady H. Hakim, Yongping Yue

المصدر: Molecular Therapy. Methods & Clinical Development
Molecular Therapy: Methods & Clinical Development, Vol 6, Iss C, Pp 216-230 (2017)

مصطلحات موضوعية: 0301 basic medicine, musculoskeletal diseases, Duchenne muscular dystrophy, medicine.medical_specialty, lcsh:QH426-470, Cardiomyopathy, nNOS, adeno-associated virus, medicine.disease_cause, DBA/2J, 03 medical and health sciences, 0302 clinical medicine, Glycoprotein complex, Fibrosis, Internal medicine, DMD, systemic gene therapy, Genetics, medicine, lcsh:QH573-671, Molecular Biology, Adeno-associated virus, biology, lcsh:Cytology, micro-dystrophin, Skeletal muscle, AAV, Anatomy, medicine.disease, musculoskeletal system, 3. Good health, lcsh:Genetics, 030104 developmental biology, Endocrinology, medicine.anatomical_structure, biology.protein, Molecular Medicine, Original Article, Dystrophin, mdx, cardiomyopathy, 030217 neurology & neurosurgery, Calcification

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ac3482a155f312c6eb069079d23f389Test
http://europepmc.org/articles/PMC5596503Test

3

Translation of Cardiac Myosin Activation With 2-Deoxy-ATP to Treat Heart Failure Via an Experimental Ribonucleotide Reductase-Based Gene Therapy

المؤلفون: Jeffrey S. Chamberlain, Charles E. Murry, Gregory G. Mahairas, Sam L. Teichman, Guy L. Odom, Farid Moussavi-Harami, Stephen D. Hauschka, Kassandra S. Thomson, Xiaolan Chen, Michael Regnier

المصدر: JACC: Basic to Translational Science, Vol 1, Iss 7, Pp 666-679 (2016)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::691ca22994e9dd83baec522dbf42f3edTest

4

The Impact of Perioperative Blood Glucose Levels on Pancreatic Cancer Prognosis and Surgical Outcomes

المؤلفون: Siddharth R. Raghavan, Umashankar K. Ballehaninna, Ronald S. Chamberlain

المصدر: Pancreas. 42:1210-1217

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::705eae4afc127b64cdc11ebb67eecc7aTest
https://doi.org/10.1097/mpa.0b013e3182a6db8eTest

5

Effects of aging on thyroarytenoid muscle regeneration

المؤلفون: Heidi Kletzien, Edward Schultz, Connie S. Chamberlain, Nadine P. Connor, Kyungah Lee, Diane M. Bless

المصدر: The Laryngoscope. 122:2800-2807

مصطلحات موضوعية: Pathology, medicine.medical_specialty, Endocrinology, Otorhinolaryngology, business.industry, Regeneration (biology), Internal medicine, Laryngeal Muscle, Rat model, medicine, Thyroarytenoid muscle, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::107b98db9af6379ef75ee6d100b225e0Test
https://doi.org/10.1002/lary.23589Test

6

Follistatin-mediated skeletal muscle hypertrophy is regulated by Smad3 and mTOR independently of myostatin

المؤلفون: Mark A. Febbraio, Claudia Beyer, Jeffrey S. Chamberlain, Catherine E. Winbanks, Julie R. McMullen, James M. Allen, Hongwei Qian, Patricio V. Sepulveda, Justin L. Chen, Graeme I. Lancaster, Kate L. Weeks, Rachel E. Thomson, Paul Gregorevic, Craig A. Harrison

المصدر: The Journal of Cell Biology

مصطلحات موضوعية: Follistatin, endocrine system, medicine.medical_specialty, P70-S6 Kinase 1, Myostatin, mTORC2, Article, Mice, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, medicine, Animals, Humans, Smad3 Protein, Muscle, Skeletal, Protein kinase B, Research Articles, PI3K/AKT/mTOR pathway, 030304 developmental biology, 0303 health sciences, biology, TOR Serine-Threonine Kinases, RPTOR, Skeletal muscle, Hypertrophy, Cell Biology, musculoskeletal system, Cell biology, Mice, Inbred C57BL, HEK293 Cells, medicine.anatomical_structure, Endocrinology, embryonic structures, biology.protein, hormones, hormone substitutes, and hormone antagonists, 030217 neurology & neurosurgery, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a2b447ce78fd65c4c14c4c30fa1b8ef6Test
https://doi.org/10.1083/jcb.201109091Test

7

Age-dependent Dystrophin Loss and Genetic Reconstitution Establish a Molecular Link Between Dystrophin and Heart Performance During Aging

المؤلفون: Jeffrey S. Chamberlain, Michael Daly, DeWayne Townsend, Joseph M. Metzger

المصدر: Molecular Therapy. 19(10):1821-1825

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09c588f7562b5b7d7ef7963667817517Test

8

Agenesis of the Dorsal Pancreas and Its Association With Pancreatic Tumors

المؤلفون: Ronald S. Chamberlain, Sujit Vijay Sakpal, Lucretia Sexcius, Nitin Babel

المصدر: Pancreas. 38:367-373

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcc4f665d3ea58e5fd5e04d01dadbbc9Test
https://doi.org/10.1097/mpa.0b013e318196c401Test

9

Onset of Experimental Severe Cardiac Fibrosis Is Mediated by Overexpression of Angiotensin-Converting Enzyme 2

المؤلفون: James M. Allen, Kirsten Gilday, Godfrey L. Smith, Martin W. McBride, Anna F. Dominiczak, Rachel Masson, Stuart A. Nicklin, Delyth Graham, Claudio Napoli, Margaret A. Craig, Andrew H. Baker, Jeffrey S. Chamberlain, Paul Gregorevic

المصدر: Hypertension. 53:694-700

مصطلحات موضوعية: Male, medicine.medical_specialty, Heart Diseases, Cardiac fibrosis, Angiotensin-Converting Enzyme Inhibitors, Blood Pressure, Peptidyl-Dipeptidase A, Biology, Severity of Illness Index, Gene Expression Regulation, Enzymologic, Enalapril, Polysaccharides, Transduction, Genetic, Fibrosis, Rats, Inbred SHR, Internal medicine, Renin–angiotensin system, Internal Medicine, medicine, Animals, Myocytes, Cardiac, Ultrasonography, Ejection fraction, Gene Expression Profiling, Gene Transfer Techniques, medicine.disease, Rats, Disease Models, Animal, Blood pressure, Endocrinology, Hypertension, Angiotensin-converting enzyme 2, ACE inhibitor, Angiotensin-Converting Enzyme 2, hormones, hormone substitutes, and hormone antagonists, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cbcdd208d77547aa7cd5e2063376f176Test
https://doi.org/10.1161/hypertensionaha.108.122333Test

10

Sarcolemma-localized nNOS is required to maintain activity after mild exercise

المؤلفون: Yvonne M. Kobayashi, Daniel R. Thedens, Robert M. Weiss, Jeffrey S. Chamberlain, Robert W. Crawford, Swapnesh Parikh, Steven A. Moore, Kevin P. Campbell, Erik P. Rader, Nikhil K. Iyengar, John A. Faulkner

المصدر: Nature

مصطلحات موضوعية: medicine.medical_specialty, Hemodynamics, Physiology, Physical exercise, Nitric Oxide Synthase Type I, Biology, Nitric Oxide, Article, Nitric oxide, Mice, chemistry.chemical_compound, Sarcolemma, Muscular Diseases, Internal medicine, medicine, Animals, Edema, Humans, Exercise physiology, Muscle, Skeletal, Cyclic GMP, Exercise, Fatigue, Cyclic Nucleotide Phosphodiesterases, Type 5, Multidisciplinary, Specific force, Skeletal muscle, Phosphodiesterase 5 Inhibitors, Enzyme Activation, Mice, Inbred C57BL, Nitric oxide synthase, Disease Models, Animal, Protein Transport, Endocrinology, medicine.anatomical_structure, chemistry, Mice, Inbred mdx, biology.protein, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::630803ae24186581478bb5f333dda9b4Test
https://doi.org/10.1038/nature07414Test

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