نتائج البحث - "Type I interferon production"

المساهمون: Immunologie [Phnom Penh], Institut Pasteur du Cambodge, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), University of Groningen [Groningen], Department of Molecular Medicine, Institut Pasteur, Fondation Cenci Bolognetti - Istituto Pasteur Italia, Fondazione Cenci Bolognetti, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome] (UNIROMA), Réseau International des Instituts Pasteur (RIIP), Immunologie Translationnelle - Translational Immunology lab, Institut Pasteur [Paris] (IP), Kantha Bopha Hospitals Foundation, This study was supported by a grant from Institut Pasteur (ACIP n°221 -2019, DeSiReS: Dengue Severe Immune Response Studies). TC was funded by the Institute Pasteur International Network and is an HHMI-Wellcome Trust International Research Scholar (208710/Z/17/Z). VU was funded by the Institute Pasteur International Network Calmette and Yersin Ph.D. scholarship. DD acknowledges support from the Agence Nationale de la Recherche (ANR grant number CE17001002)., ANR-16-CE17-0005,GENMSMD,Dissection génétique de la Susceptibilité Mendélienne aux infections mycobactériennes chez l'homme(2016), ANR-16-CE17-0010,IFNX,Investigation des interferonopathies type I humaine(2016), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome], Institut Pasteur [Paris]

المصدر: Frontiers in Immunology, Vol 11 (2020)
Frontiers in Immunology
Frontiers in Immunology, 2020, 11, pp.605087. ⟨10.3389/fimmu.2020.605087⟩
Frontiers in Immunology, Frontiers, 2020, 11, pp.605087. ⟨10.3389/fimmu.2020.605087⟩
Frontiers in Immunology, 11:605087. Frontiers Media SA

مصطلحات موضوعية: Male, Time Factors, viruses, Plasmacytoid dendritic cell, Dengue virus, medicine.disease_cause, Antibodies, Viral, Severity of Illness Index, DISEASE, Dengue, 0302 clinical medicine, Immunology and Allergy, Platelet, Child, Original Research, 0303 health sciences, interferon stimulated genes, Antibody titer, virus diseases, simoa immunoassay, Viral Load, Acquired immune system, Prognosis, myloid dendritic cells, Single Molecule Imaging, 3. Good health, B-CELLS, Child, Preschool, Host-Pathogen Interactions, Interferon Type I, POSTTRANSCRIPTIONAL REGULATION, [SDV.IMM]Life Sciences [q-bio]/Immunology, type I interferon, Female, Cambodia, Viral load, lcsh:Immunologic diseases. Allergy, Adolescent, Immunology, Enzyme-Linked Immunosorbent Assay, RECEPTOR IFNAR-2, 03 medical and health sciences, Immune system, ENHANCEMENT, medicine, plasmacytoid dendritic cell, Humans, 030304 developmental biology, dengue virus, business.industry, Platelet Count, Dendritic Cells, biochemical phenomena, metabolism, and nutrition, Type I interferon production, Antibodies, Neutralizing, ALPHA, VIRUS-INFECTION REQUIRES, HEMORRHAGIC-FEVER, Case-Control Studies, IFN-BETA, business, lcsh:RC581-607, 030215 immunology, RESPONSES

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab42c88242b43c752954a404e305f0d5Test
http://hdl.handle.net/11573/1473176Test

5

TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis

المؤلفون: Alexander A. Navarini, Curdin Conrad, Alessio Mylonas, Anne-Karine Lapointe, Jeremy Di Domizio, Olivier Demaria, Michel Gilliet, Cyrine Belkhodja, Lars E. French, Maxime Vernez

المساهمون: University of Zurich, Conrad, Curdin

المصدر: Nature Communications, Vol 9, Iss 1, Pp 1-11 (2018)
Nature Communications
Nature communications, vol. 9, no. 1, pp. 25

مصطلحات موضوعية: 0301 basic medicine, Male, T-Lymphocytes, General Physics and Astronomy, Autoimmunity, medicine.disease_cause, 030207 dermatology & venereal diseases, 0302 clinical medicine, Crohn Disease, Interferon, lcsh:Science, Cells, Cultured, Mice, Inbred BALB C, Multidisciplinary, 10177 Dermatology Clinic, Antibodies, Monoclonal, Middle Aged, 3100 General Physics and Astronomy, Adalimumab/adverse effects, Adalimumab/immunology, Adalimumab/therapeutic use, Adolescent, Adult, Aged, Animals, Antibodies, Monoclonal/adverse effects, Antibodies, Monoclonal/immunology, Antibodies, Monoclonal/therapeutic use, Autoimmunity/drug effects, Autoimmunity/immunology, Crohn Disease/drug therapy, Crohn Disease/immunology, Crohn Disease/metabolism, Cytokines/genetics, Cytokines/immunology, Cytokines/metabolism, Dendritic Cells/drug effects, Dendritic Cells/immunology, Dendritic Cells/metabolism, Female, Humans, Infliximab/adverse effects, Infliximab/immunology, Infliximab/therapeutic use, Interferon Type I/genetics, Interferon Type I/immunology, Interferon Type I/metabolism, Psoriasis/chemically induced, Psoriasis/immunology, T-Lymphocytes/drug effects, T-Lymphocytes/immunology, T-Lymphocytes/metabolism, Tumor Necrosis Factor-alpha/antagonists & inhibitors, Tumor Necrosis Factor-alpha/immunology, Tumor Necrosis Factor-alpha/metabolism, Young Adult, Interferon Type I, Cytokines, Tumor necrosis factor alpha, medicine.drug, Science, 610 Medicine & health, 1600 General Chemistry, General Biochemistry, Genetics and Molecular Biology, Article, 03 medical and health sciences, 1300 General Biochemistry, Genetics and Molecular Biology, Psoriasis, medicine, Adalimumab, business.industry, Tumor Necrosis Factor-alpha, General Chemistry, Dendritic Cells, medicine.disease, Type I interferon production, Infliximab, 030104 developmental biology, Immunology, lcsh:Q, business, Interferon type I

وصف الملف: application/pdf; 2018_LF_TNF_blockade_induces_a_dysregulated_type_I_interferon_response_without_autoimmunity_in_paradoxical_psoriasis.pdf - application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbbc7decb248c10970a313c075620f0fTest
https://doaj.org/article/180fc992d72e4fcbbf1d606055e86015Test

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Neutrophils drive type I interferon production and autoantibodies in patients with Wiskott-Aldrich syndrome

المؤلفون: Giulia Maria Piperno, Federica Benvenuti, Genni Enza Marcovecchio, Asma Naseem, Maria Carmina Castiello, Maria Pia Cicalese, Anna Villa, Elena Fontana, Luigi D. Notarangelo, Karla E Cervantes-Luevano, Marita Bosticardo, Alessandro Aiuti, Nicoletta Caronni, Paolo Uva

المصدر: Journal of Allergy and Clinical Immunology. 142:1605-1617.e4

مصطلحات موضوعية: Adult, Male, 0301 basic medicine, Adolescent, Neutrophils, Immunology, Gene Expression, Plasmacytoid dendritic cell, Granulocyte, medicine.disease_cause, Extracellular Traps, Article, Autoimmunity, Young Adult, 03 medical and health sciences, 0302 clinical medicine, medicine, Animals, Humans, Immunology and Allergy, B-cell activating factor, Autoantibodies, Mice, Knockout, B-Lymphocytes, biology, business.industry, Infant, Dendritic Cells, Neutrophil extracellular traps, Type I interferon production, Wiskott-Aldrich Syndrome, Mice, Inbred C57BL, 030104 developmental biology, medicine.anatomical_structure, Child, Preschool, Neutrophil elastase, Myeloperoxidase, Interferon Type I, biology.protein, Female, business, 030215 immunology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::52ac470a8912ba2e936b8dc41e826de0Test
https://doi.org/10.1016/j.jaci.2017.11.063Test

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Wiskott-Aldrich syndrome protein-mediated actin dynamics control type-I interferon production in plasmacytoid dendritic cells

المؤلفون: Elisa Bonomi, Ulrich Kalinke, Stefania Gobessi, Anna Villa, Marco Catucci, Ayse Metin, Luigi D. Notarangelo, Francesca Prete, Sophie Hambleton, Maria Carmina Castiello, William Vermi, Robbert G. M. Bredius, Mayrel Labrada, Mirjam van der Burg, Federica Benvenuti, Caterina Cancrini, Alessandro Aiuti

المساهمون: Immunology, International Centre for Genetic Engineering and Biotechnology, Padriciano 99, 34149 Trieste, Italy., Prete, Francesca, Catucci, Marco, Labrada, Mayrel, Gobessi, Stefania, Castiello Maria, Carmina, Bonomi, Elisa, Aiuti, Alessandro, Vermi, William, Cancrini, Caterina, Metin, Ayse, Hambleton, Sophie, Bredius, Robbert, Notarangelo Luigi, Daniele, van der Burg, Mirjam, Kalinke, Ulrich, Villa, Anna, Benvenuti, Federica

المصدر: Journal of Experimental Medicine, 210(2), 355-374. Rockefeller University Press
The Journal of Experimental Medicine

مصطلحات موضوعية: Male, Wiskott–Aldrich syndrome, Autoimmunity, Plasmacytoid dendritic cell, Receptor, Interferon alpha-beta, Mice, 0302 clinical medicine, immune system diseases, Interferon, hemic and lymphatic diseases, Immunology and Allergy, Mice, Knockout, 0303 health sciences, biology, Wiskott–Aldrich syndrome protein, hemic and immune systems, Endocytosis, Cell biology, Wiskott-Aldrich Syndrome, 3. Good health, Interferon Type I, Female, Wiskott-Aldrich Syndrome Protein, medicine.drug, Signal Transduction, congenital, hereditary, and neonatal diseases and abnormalities, Immunology, macromolecular substances, Article, 03 medical and health sciences, medicine, Animals, Humans, RNA, Messenger, 030304 developmental biology, Innate immune system, Base Sequence, TLR9, Interferon-alpha, Dendritic Cells, Cell Biology, medicine.disease, Type I interferon production, Actins, Immunity, Innate, Mice, Inbred C57BL, Disease Models, Animal, Toll-Like Receptor 9, biology.protein, Interferon type I, 030215 immunology