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المؤلفون: Ji Miao, Sudha B. Biddinger, Ivana Semova, Anthony N. Hollenberg, Hong Kang, Matthew D. Hirschey, David Masson, Yue Hu, Satyapal Chahar, Xiaowei Sun
المساهمون: Division of Endocrinology, Children's Hospital Boston, Harvard Medical School, Harvard Medical School [Boston] ( HMS ), Beth Israel Deaconess Medical Center, Lipides - Nutrition - Cancer [Dijon - U1231] ( LNC ), Université de Bourgogne ( UB ) -AgroSup Dijon - Institut National Supérieur des Sciences Agronomiques, de l'Alimentation et de l'Environnement-Institut National de la Santé et de la Recherche Médicale ( INSERM ), Duke University Medical Center
المصدر: Journal of Biological Chemistry
Journal of Biological Chemistry, American Society for Biochemistry and Molecular Biology, 2018, 293 (6), pp.2006-2014. 〈http://www.jbc.org/content/293/6/2006.longTest〉. 〈10.1074/jbc.M117.782557〉مصطلحات موضوعية: 0301 basic medicine, Male, medicine.medical_specialty, mTORC1, Mechanistic Target of Rapamycin Complex 1, Biochemistry, metabolic syndrome, fructose, 03 medical and health sciences, chemistry.chemical_compound, Mice, 0302 clinical medicine, Internal medicine, Gene expression, medicine, Animals, glucose, Molecular Biology, [ SDV.BBM ] Life Sciences [q-bio]/Biochemistry, Molecular Biology, Triglycerides, lipogenesis, Mice, Knockout, Fatty liver, dyslipidemia, mammalian target of rapamycin (mTOR), Lipid metabolism, Fructose, Cell Biology, medicine.disease, Mice, Inbred C57BL, 030104 developmental biology, Endocrinology, Metabolism, chemistry, Gene Expression Regulation, Liver, Lipogenesis, Metabolic syndrome, Signal transduction, biological phenomena, cell phenomena, and immunity, 030217 neurology & neurosurgery, Signal Transduction
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e0151552b633fb1a9d27448adf7dd3bTest
https://hal-univ-bourgogne.archives-ouvertes.fr/hal-01730773Test -
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المؤلفون: Ming‑Xu Hu, Li Liu, Rui‑Hong Zhang, Ping Zhang, Ke‑Li Yun, Hong‑Cai Zhang, Li Sui
المصدر: Bioscience Reports, Vol 35, Iss 2, p e00186 (2015)
Bioscience Reportsمصطلحات موضوعية: Programmed cell death, autophagy, Biophysics, lcsh:Life, lcsh:QR1-502, endoplasmic reticulum (ER) stress, Metal toxicity, mTOR, mammalian target of rapamycin, mTORC1, Mechanistic Target of Rapamycin Complex 1, Pharmacology, Biology, Biochemistry, lcsh:Microbiology, ER, endoplasmic reticulum, Mice, ROS, reactive oxygen species, UPR, unfolded protein response, mammalian target of rapamycin (mTOR) pathway, S6K1, S6 kinase 1, Animals, Humans, Molecular Biology, Cells, Cultured, DMEM, Dulbecco's Modified Essential Medium, Original Paper, Myocardium, TOR Serine-Threonine Kinases, Endoplasmic reticulum, Autophagy, mTORC1, mammalian target of rapamycin complex 1, Cell Biology, Fibroblasts, lead toxicity, LC3, light chain 3, Cell biology, lcsh:QH501-531, Lead, Apoptosis, Multiprotein Complexes, Toxicity, Unfolded protein response, 3-MA, 3-methyladenine, cardiofibroblasts
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8b99c3e54419619f587d1db7268a04f5Test
http://www.bioscirep.org/bsr/035/e186/bsr035e186.htmTest