يعرض 1 - 10 نتائج من 16 نتيجة بحث عن '"Extracellular Matrix Proteins--metabolism"', وقت الاستعلام: 0.90s تنقيح النتائج
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    المساهمون: Cardiovascular and Metabolic Disorders [Singapor] (cvmd), Duke-NUS Medical School [Singapore], Università degli studi di Bari Aldo Moro = University of Bari Aldo Moro (UNIBA), Ospedale San Giovanni di Dio, Firenze, National Heart and Lung Institute [London] (NHLI), Imperial College London-Royal Brompton and Harefield NHS Foundation Trust, Génétique Evolutive Humaine - Human Evolutionary Genetics, Institut Pasteur [Paris] (IP)-Centre National de la Recherche Scientifique (CNRS), Faculty of Medicine [London, UK], Imperial College London, Agency for science, technology and research [Singapore] (A*STAR), Institute of Molecular and Cell Biology, National University of Singapore (NUS)-Agency for science, technology and research [Singapore] (A*STAR), MRC London Institute of Medical Sciences (LMC), National Heart Centre Singapore (NHCS), Royal Brompton and Harefield NHS Foundation Trust, Cardiovascular Research Centre [London], Centro Nacional de Investigaciones Cardiovasculares Carlos III [Madrid, Spain] (CNIC), Instituto de Salud Carlos III [Madrid] (ISC), University of Bristol [Bristol], Institute of Physiology [Prague], Czech Academy of Sciences [Prague] (CAS), University of Cambridge [UK] (CAM), The Alan Turing Institute, MRC Biostatistics Unit [Cambridge, UK], Max Delbrück Center for Molecular Medicine [Berlin] (MDC), Helmholtz-Gemeinschaft = Helmholtz Association, German Center for Cardiovascular Research (DZHK), Berlin Institute of Health (BIH), Charité - UniversitätsMedizin = Charité - University Hospital [Berlin], The research was primarily supported by National Medical Research Council (NMRC) Singapore grant NMRC/CBRG/0106/2016 (to E.P.) and the British Heart Foundation (BHF) Ph.D. Studentship grant FS/11/25/28740 (to E.P). We acknowledge additional funding support from European Union FP7 CardioNeT-ITN-289600 (to E.L.-P., S.A.C., and P.J.R.B.), Heart Research UK (to P.J.R.B.), NIHR CV BRU of Royal Brompton and Harefield, NHS Foundation Trust (to S.A.C. and P.J.R.B.), BHF (to S.A.C.), Leducq Foundation (to S.A.C.), MRC UK (to S.A.C.), BHF Program Grant no. RG/15/5/31446 (to C.E. and E.P.). M.P. was supported by Praemium Academiae award of the Czech Academy of Sciences and grant 14-36804G from the Czech Science Foundation., We wish to thank Dr. Jacques Behmoaras for contributing critical and constructive comments to the manuscript., European Project: 289600,EC:FP7:PEOPLE,FP7-PEOPLE-2011-ITN,CARDIONET(2012), Moreno-Moral, Aida [0000-0002-8155-3146], Barton, Paul J. R. [0000-0002-1165-7767], Schafer, Sebastian [0000-0002-6909-8275], Bottolo, Leonardo [0000-0002-6381-2327], Cook, Stuart A. [0000-0001-6628-194X], Petretto, Enrico [0000-0003-2163-5921], Apollo - University of Cambridge Repository, National Medical Research Council (Singapur), British Heart Foundation, Unión Europea. Comisión Europea, National Institutes of Health (Estados Unidos), Czech Science Foundation, Fondation Leducq, NHS Foundation Trusth, University of Bari Aldo Moro (UNIBA), Institut Pasteur [Paris]-Centre National de la Recherche Scientifique (CNRS), Leducq Foundation for Cardiovascular Research, Commission of the European Communities, Heart Research UK, Medical Research Council (MRC), Barton, Paul JR [0000-0002-1165-7767], Cook, Stuart A [0000-0001-6628-194X]

    المصدر: Nature Communications
    Nature Communications, 2019, 10 (1), pp.3616. ⟨10.1038/s41467-019-11551-9⟩
    Nature Communications, Vol 10, Iss 1, Pp 1-19 (2019)
    Chen, H, Moreno-Moral, A, Pesce, F, Devapragash, N, Mancini, M, Heng, E L, Rotival, M, Srivastava, P K, Harmston, N, Shkura, K, Rackham, O J L, Yu, W P, Sun, X M, Tee, N G Z, Tan, E L S, Barton, P J R, Felkin, L E, Lara-Pezzi, E, Angelini, G, Beltrami, C, Pravenec, M, Schafer, S, Bottolo, L, Hubner, N, Emanueli, C, Cook, S A & Petretto, E 2019, ' WWP2 regulates pathological cardiac fibrosis by modulating SMAD2 signalling ', Nature Communications, vol. 10, 3616 . https://doi.org/10.1038/s41467-019-11551-9Test
    Nature Communications, Nature Publishing Group, 2019, 10 (1), pp.3616. ⟨10.1038/s41467-019-11551-9⟩
    Repisalud
    Instituto de Salud Carlos III (ISCIII)

    مصطلحات موضوعية: Male, 45/43, BLOOD-PRESSURE, 02 engineering and technology, MESH: Protein Isoforms, MESH: Heart Diseases / genetics, Mice, Transforming Growth Factor beta, 42/44, Fibrosis, Protein Isoforms, Medicine, MESH: Animals, lcsh:Science, MESH: Ubiquitin-Protein Ligases / genetics, MESH: Fibrosis / metabolism, MESH: Genetic Predisposition to Disease* / genetics, GENE-EXPRESSION, Regulation of gene expression, Extracellular Matrix Proteins, MESH: Middle Aged, MESH: Fibrosis / genetics, TGF-BETA, MESH: Heart Diseases / metabolism, MESH: Ubiquitin-Protein Ligases / metabolism, 3. Good health, Cell biology, Ubiquitin ligase, FIBROBLAST, MESH: Young Adult, Science & Technology - Other Topics, 64/60, Cardiomyopathies, 0210 nano-technology, Cardiac function curve, Science, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, REVEALS, Humans, Aged, MESH: Adolescent, Science & Technology, MESH: Humans, [SDV.GEN.GPO]Life Sciences [q-bio]/Genetics/Populations and Evolution [q-bio.PE], IDENTIFICATION, MESH: Smad2 Protein / metabolism, MESH: Extracellular Matrix Proteins / metabolism, MESH: Adult, MESH: Cardiomyopathies / metabolism, medicine.disease, 030104 developmental biology, Cardiovascular and Metabolic Diseases, E3 UBIQUITIN LIGASE, RAT, DETERMINANT, lcsh:Q, Myocardial fibrosis, MESH: Female, 0301 basic medicine, Cardiac fibrosis, General Physics and Astronomy, Smad2 Protein, Gene Regulatory Networks, 13/89, MESH: Aged, MESH: Transforming Growth Factor beta / metabolism, Multidisciplinary, biology, MESH: Smad2 Protein / genetics, article, Middle Aged, MESH: Cardiomyopathies / genetics, 021001 nanoscience & nanotechnology, 692/4019/592/75/74, MESH: Gene Expression Regulation, Multidisciplinary Sciences, Centre for Surgical Research, HEART-FAILURE, Female, 38/39, Adult, Adolescent, Heart Diseases, MESH: Mice, Transgenic, Ubiquitin-Protein Ligases, Mice, Transgenic, 14/32, 82/80, Young Adult, Animals, Genetic Predisposition to Disease, 14/35, MESH: Mice, Pressure overload, business.industry, General Chemistry, MESH: Male, Gene Expression Regulation, [SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human genetics, Ubiquitin ligases, Heart failure, 13/51, biology.protein, 631/45/474/2073, MESH: Gene Regulatory Networks, business

    وصف الملف: application/pdf; text; application/zip; text/xml

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    المساهمون: University of Hawaii, Universität zu Lübeck [Lübeck], Biologie Neurovasculaire et Mitochondriale Intégrée (BNMI), Université d'Angers (UA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Netherlands Institute for Neuroscience (NIN), Royal Netherlands Academy of Arts and Sciences (KNAW), Munich Heart Alliance, German Centre for Cardiovascular Research, partner site Munich Heart Alliance, Munich, Hungarian Academy of Sciences (MTA), MUMC+: *AB Onderzoekers (9), RS: MHeNs - R3 - Neuroscience, Oogheelkunde, Centre National de la Recherche Scientifique (CNRS)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université d'Angers (UA), ANS - Amsterdam Neuroscience, Human Genetics, Other departments

    المصدر: The American journal of pathology
    The American journal of pathology, 2014, 184 (1), pp.159-70. ⟨10.1016/j.ajpath.2013.09.015⟩
    American Journal of Pathology, 184(1), 159-70. Elsevier Science
    The American journal of pathology, 184(1), 159-70. Elsevier B.V.
    American journal of pathology, 184(1), 159-170. Elsevier Inc.

    وصف الملف: application/pdf

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    المساهمون: ACS - Pulmonary hypertension & thrombosis, Graduate School, ACS - Diabetes & metabolism, RS: CARIM - R1.02 - Vascular aspects thrombosis and haemostasis, RS: CARIM - R3.02 - Hypertension and target organ damage, Pediatric Surgery

    المصدر: PloS one, vol. 13, no. 3, pp. e0193967
    PLoS ONE, 13(3):e0193967. Public Library of Science
    PLoS ONE
    PLOS ONE
    PLOS ONE, 13(3):e0193967. Public Library of Science
    PLoS One (online), 13(3):e0193967. Public Library of Science
    PLOS ONE, Vol. 13, No 3 (2018) P. e0193967
    PLoS ONE, Vol 13, Iss 3, p e0193967 (2018)

    مصطلحات موضوعية: Male, Myocardium/metabolism, medicine.medical_treatment, Organic chemistry, Blood Pressure, Vascular Medicine, Biochemistry, Electrocardiography, Ventricular Dysfunction, Left, 0302 clinical medicine, 80 and over, lcsh:Science, ddc:616, Aged, 80 and over, Extracellular Matrix Proteins, Ventricular Dysfunction, Left/diagnostic imaging/epidemiology/metabolism/pathology, Heart, Vitamins, Physical sciences, Myocardium/metabolism/pathology, Blood, PRESERVED EJECTION FRACTION, ARTERIAL STIFFNESS, Switzerland, MENAQUINONE-7 SUPPLEMENTATION, medicine.medical_specialty, Endothelium, B vitamins, Extracellular Matrix Proteins/metabolism, 03 medical and health sciences, Calcium-Binding Proteins/metabolism, Humans, Aged, Myocardium, lcsh:R, Calcium-Binding Proteins, Biology and Life Sciences, Proteins, nutritional and metabolic diseases, medicine.disease, 030104 developmental biology, Blood pressure, lcsh:Q, Myocardial Ischemia/diagnostic imaging/metabolism/pathology/surgery, K-DEPENDENT PROTECTION, BONE MORPHOGENETIC PROTEIN-4, Adult, Cardiomyopathy, Dilated/diagnostic imaging, Cardiomyopathy, Dilated/metabolism, Cardiomyopathy, Dilated/pathology, Cardiomyopathy, Dilated/surgery, Cohort Studies, Female, Heart/diagnostic imaging, Heart Transplantation, Middle Aged, Multivariate Analysis, Myocardial Ischemia/diagnostic imaging, Myocardial Ischemia/metabolism, Myocardial Ischemia/pathology, Myocardial Ischemia/surgery, Myocardium/pathology, Netherlands, Ventricular Dysfunction, Left/diagnostic imaging, Ventricular Dysfunction, Left/epidemiology, Ventricular Dysfunction, Left/metabolism, Ventricular Dysfunction, Left/pathology, Young Adult, 0301 basic medicine, POWERFUL PREDICTOR, Vitamin K, Physiology, Myocardial Ischemia, lcsh:Medicine, 030204 cardiovascular system & hematology, Matrix gla protein, Ventricular Dysfunction, Medicine and Health Sciences, VITAMIN-K, Post-Translational Modification, Phosphorylation, Dilated/diagnostic imaging, GENERAL-POPULATION, Heart transplantation, Multidisciplinary, biology, Dilated cardiomyopathy, Body Fluids, Chemistry, medicine.anatomical_structure, Cardiology, HEART-FAILURE, Anatomy, Left/diagnostic imaging, Research Article, Cardiomyopathy, Dilated, Histology, Cardiomyopathy, Diastole, Blood Plasma, Chemical compounds, Cardiomyopathy, Dilated/diagnostic imaging/metabolism/pathology/surgery, Internal medicine, Organic compounds, medicine, business.industry, FILLING PRESSURE, Heart failure, Cardiovascular Anatomy, Arterial stiffness, biology.protein, business

    وصف الملف: application/pdf; Electronic-eCollection

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    المساهمون: School of Molecular and Microbial Sciences, University of Queensland [Brisbane], Génétique des Biofilms, Institut Pasteur [Paris]-Centre National de la Recherche Scientifique (CNRS), Griffith University [Brisbane], This work was supported by grants from the Australian National Health and Medical Research Council (631654), the Australian Research Council (DP1097032), the University of Queensland, the Institut Pasteur, the CNRS URA 2172, the Network of Excellence EuroPathoGenomics, and the European Community (LSHB-CT-2005-512061).M.A.S. is supported by an ARC Future Fellowship, and J.V. was a Marie-Curie Fellow., We thank Laura Zanichelli, Tim J. Wells, Chelsea Stewart, and Barbara Arnts for expert assistance, as well as Sylvie Rimsky for providing the purified native H-NS protein and Per Klemm for providing strain CFT073amp., European Project: 512061,Network of Excellence EuroPathoGenomics, Institut Pasteur [Paris] (IP)-Centre National de la Recherche Scientifique (CNRS)

    المصدر: Digital.CSIC. Repositorio Institucional del CSIC
    instname
    Infection and Immunity
    Infection and Immunity, American Society for Microbiology, 2012, 80 (1), pp.321-332. ⟨10.1128/IAI.05322-11⟩
    Infection and Immunity, 2012, 80 (1), pp.321-332. ⟨10.1128/IAI.05322-11⟩

    مصطلحات موضوعية: MESH: Escherichia coli Infections/microbiology/pathology, medicine.disease_cause, urologic and male genital diseases, Polymerase Chain Reaction, MESH: Bacterial/genetics/*metabolism, Fimbriae Proteins, Mice, MESH: Extracellular Matrix Proteins/metabolism, Uropathogenic Escherichia coli, MESH: Animals, MESH: Urinary Tract Infections/microbiology/pathology, Cloning, Molecular, Escherichia coli Infections, MESH: Biofilms/growth & development, Extracellular Matrix Proteins, 0303 health sciences, biology, Membrane transport protein, Escherichia coli Proteins, MESH: DNA, MESH: Fimbriae Proteins/metabolism, Phenotype, female genital diseases and pregnancy complications, 3. Good health, Infectious Diseases, Urinary Tract Infections, Female, MESH: Escherichia coli K12/genetics/pathogenicity, MESH: Bacterial/genetics, DNA, Bacterial, Virulence Factors, MESH: Cloning, Immunology, MESH: Escherichia coli Proteins/genetics/*metabolism, Virulence, MESH: Virulence Factors/genetics/*metabolism, Microbiology, 03 medical and health sciences, MESH: Gene Expression Profiling, MESH: Uropathogenic Escherichia coli/*genetics/*pathogenicity, medicine, Animals, Adhesins, Bacterial, Gene, Escherichia coli, MESH: Mice, 030304 developmental biology, Escherichia coli K12, 030306 microbiology, Gene Expression Profiling, Membrane Transport Proteins, MESH: Polymerase Chain Reaction, MESH: Inbred C57BL, bacterial infections and mycoses, Molecular Pathogenesis, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, Mice, Inbred C57BL, Gene expression profiling, Bacterial adhesin, Biofilms, MESH: Molecular, biology.protein, Parasitology, MESH: Membrane Transport Proteins/genetics/*metabolism, MESH: Adhesins, MESH: Female

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    المساهمون: University of Zurich, Fuentealba, Pablo

    المصدر: Europe PubMed Central
    Fuentealba, P, Klausberger, T, Karayannis, T, Suen, W Y, Huck, J, Tomioka, R, Rockland, K, Capogna, M, Studer, M, Morales, M & Somogyi, P 2010, ' Expression of COUP-TFII nuclear receptor in restricted GABAergic neuronal populations in the adult rat hippocampus ', The Journal of neuroscience : the official journal of the Society for Neuroscience, vol. 30, no. 5, pp. 1595-609 . https://doi.org/10.1523/JNEUROSCI.4199-09.2010Test

    وصف الملف: 1595.full.pdf - application/pdf

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    المصدر: Journal of Cellular Physiology

    مصطلحات موضوعية: Enzymologic, Time Factors, Physiology, Decorin, Clinical Biochemistry, Bone Morphogenetic Protein 2, Messenger/metabolism, Core Binding Factor Alpha 1 Subunit, Cell Differentiation/genetics, chemistry.chemical_compound, Mice, Versicans, Osteogenesis, Bone Morphogenetic Protein 2/metabolism, Receptors, Biglycan, Recombinant Proteins/metabolism, Regulation of gene expression, Mice, Knockout, Extracellular Matrix Proteins, Cell Differentiation, Heparan sulfate, 3T3 Cells, Fibroblast Growth Factor 2/metabolism, Recombinant Proteins, Cell biology, Protein Processing, Biochemistry, Fibroblast growth factor receptor, Versican, Cell Lineage/genetics, Fibroblast Growth Factor 2, Proteoglycans, RNA Interference, Sulfotransferases, Heparin Lyase/genetics/metabolism, 060103 Cell Development Proliferation and Death, Knockout, Biology, Transfection, Fibroblast Growth Factor/metabolism, Dermatan sulfate, Gene Expression Regulation, Enzymologic, Extracellular Matrix Proteins/metabolism, Glypicans/genetics/*metabolism, Osteogenesis/genetics, Glypicans, Animals, Humans, Heparanase, Cell Lineage, RNA, Messenger, Core Binding Factor Alpha 1 Subunit/genetics/metabolism, Cell Proliferation, Sulfotransferases/genetics/metabolism, Osteoblasts, Osteoblasts/enzymology/*metabolism, Post-Translational, Cell Biology, Proteoglycans/metabolism, Receptors, Fibroblast Growth Factor, carbohydrates (lipids), Gene Expression Regulation, chemistry, Heparin Lyase, biology.protein, RNA, Protein Processing, Post-Translational, Versicans/metabolism

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    المساهمون: Groningen Kidney Center (GKC), Groningen Institute for Organ Transplantation (GIOT)

    المصدر: The British journal of dermatology, 124(1), 13-20. Wiley

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    المصدر: BMC Cancer
    BMC Cancer, Vol 6, Iss 1, p 140 (2006)