نتائج البحث - "Paloma Martín Sanz"

1

Hepatic COX-2 expression protects mice from an alcohol-high fat diet-induced metabolic disorder by involving protein acetylation related energy metabolism

المؤلفون: Marta Casado, Liya Pi, Wei Wei, Minjie Chen, Xicui Sun, Qi Cao, Paloma Martín-Sanz, Carme Cucarella, Bin Ren

المساهمون: National Institutes of Health (US), Ministerio de Economía y Competitividad (España)

المصدر: Alcohol (Fayetteville, N.Y.)
Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: Male, medicine.medical_specialty, Health (social science), Liquid diet, White adipose tissue, Protein acetylation, Toxicology, Diet, High-Fat, Biochemistry, Article, 03 medical and health sciences, Behavioral Neuroscience, chemistry.chemical_compound, Mice, 0302 clinical medicine, Fatty liver disease, Internal medicine, medicine, Animals, Protein kinase B, Liver injury, Triglyceride, Chemistry, Cholesterol, Metabolic disorder, protein acetylation, Acetylation, General Medicine, COX-2, medicine.disease, Lipid Metabolism, 030227 psychiatry, Mice, Inbred C57BL, Endocrinology, Metabolism, Neurology, Liver, Cyclooxygenase 2, fatty liver disease, Steatosis, Energy Metabolism, metabolism, 030217 neurology & neurosurgery

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::89ddf36ac5ef4491264f586cb5c1bc7fTest
http://europepmc.org/articles/PMC8095085Test

2

COX-2 Expression in Hepatocytes Improves Mitochondrial Function after Hepatic Ischemia-Reperfusion Injury

المؤلفون: Marina Fuertes-Agudo, María Luque-Tévar, Carme Cucarella, Rocío Brea, Lisardo Boscá, Rubén Quintana-Cabrera, Paloma Martín-Sanz, Marta Casado

المساهمون: Ministerio de Ciencia e Innovación (España), Centro de Investigación Biomédica en Red Enfermedades Hepáticas y Digestivas (España), Centro de Investigación Biomédica en Red Enfermedades Cardiovaculares (España), Comunidad de Madrid, European Commission, Ministerio de Economía y Competitividad (España), Casado, Marta [0000-0001-6457-4650], Fuertes-Agudo, Marina [0000-0002-8146-6165], Boscá, Lisardo [0000-0002-0253-5469], Quintana-Cabrera, Ruben [0000-0002-0601-349X], Casado, Marta, Fuertes-Agudo, Marina, Boscá, Lisardo, Quintana-Cabrera, Ruben

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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Antioxidants; Volume 11; Issue 9; Pages: 1724

مصطلحات موضوعية: Liver, Physiology, Ischemia-reperfusion, Clinical Biochemistry, Mitochondrial dynamics, Prostaglandins, Cell Biology, COX-2, High-resolution respirometry, prostaglandins, liver, ischemia-reperfusion, high-resolution respirometry, mitochondrial dynamics, Molecular Biology, Biochemistry

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e977ba3525e74af3162976806880b9dTest
http://hdl.handle.net/10261/280010Test

3

Beyond classic concepts in thyroid homeostasis: Immune system and microbiota

المؤلفون: José Moisés Laparra, Silvia González-Ramos, Lisardo Boscá, Victoria Fernández-García, Paloma Martín-Sanz

المساهمون: Ministerio de Ciencia, Innovación y Universidades (España), Agencia Estatal de Investigación (España), Centro de Investigación Biomédica en Red Enfermedades Cardiovaculares (España), Comunidad de Madrid

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9529dda4ceac52a30d52f700ceff8e52Test
http://hdl.handle.net/10261/264834Test

4

NOD1 deficiency promotes an imbalance of thyroid hormones and microbiota homeostasis in mice fed high fat diet

المؤلفون: Yolanda Sanz, Lisardo Boscá, Kevin J. Portune, María Jesús Obregón, Marta Paz-García, Paloma Martín-Sanz, Silvia González-Ramos, Emilio F. Acosta-Medina, Victoria Fernández-García, Antonio Castrillo

المساهمون: Ministerio de Economía, Industria y Competitividad (España), European Commission, Comunidad de Madrid, Fundación Ramón Areces, Instituto de Salud Carlos III

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-17 (2020)

مصطلحات موضوعية: 0301 basic medicine, Physiology, Thyroid Gland, Adipose tissue, lcsh:Medicine, White adipose tissue, Gut flora, Biochemistry, 0302 clinical medicine, Nod1 Signaling Adaptor Protein, Brown adipose tissue, Adipocytes, Homeostasis, lcsh:Science, Mice, Knockout, Multidisciplinary, High fat diet, Biodiversity, Lipids, Intestines, medicine.anatomical_structure, Liver, medicine.medical_specialty, Thyroid Hormones, Adipose Tissue, White, Immunology, 030209 endocrinology & metabolism, Biology, Diet, High-Fat, Article, Proinflammatory cytokine, 03 medical and health sciences, Internal medicine, Nucleotide-binding oligomerization domain protein, medicine, Animals, Metabolomics, Resting energy expenditure, Obesity, Inflammation, lcsh:R, Body Weight, Feeding Behavior, Glucose Tolerance Test, biology.organism_classification, Gastrointestinal Microbiome, Fatty Liver, Mice, Inbred C57BL, body regions, 030104 developmental biology, Endocrinology, lcsh:Q, Hormone

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e9526fde07d5bb70a2b7cfdb147d7c3Test
http://hdl.handle.net/10261/217922Test

5

NOD1 activation in cardiac fibroblasts induces myocardial fibrosis in a murine model of type 2 diabetes

المؤلفون: Pilar González-Peramato, Verónica Terrón, Noelia Agra, Silvia González-Ramos, Almudena Val-Blasco, María Teresa Vallejo-Cremades, Gemma Benito, Patricia Prieto, Carmen Delgado, Ivette Pacheco, María Fernández-Velasco, Lisardo Boscá, Paloma Martín-Sanz

المساهمون: Red de Investigación Cardiovascular (España), Instituto de Salud Carlos III, Ministerio de Economía y Competitividad (España), European Commission

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: Blood Glucose, 0301 basic medicine, Agonist, medicine.medical_specialty, Cardiac fibrosis, medicine.drug_class, Mice, Transgenic, Inflammation, 030204 cardiovascular system & hematology, Diaminopimelic Acid, Biochemistry, Diabetes Mellitus, Experimental, Pathogenesis, Mice, 03 medical and health sciences, 0302 clinical medicine, Insulin resistance, Transforming Growth Factor beta, Fibrosis, Nod1 Signaling Adaptor Protein, Internal medicine, Nitriles, medicine, Animals, Humans, Insulin, Sulfones, Receptor, Molecular Biology, business.industry, Myocardium, NF-kappa B, Cell Biology, Endomyocardial Fibrosis, medicine.disease, body regions, 030104 developmental biology, Endocrinology, Diabetes Mellitus, Type 2, Gene Expression Regulation, NIH 3T3 Cells, Myocardial fibrosis, Insulin Resistance, medicine.symptom, business, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21ae27f84acad49739b0c03b005dcd10Test
https://doi.org/10.1042/bcj20160556Test

6

Endothelial NOD1 directs myeloid cell recruitment in atherosclerosis through VCAM-1

المؤلفون: Alberto del Monte-Monge, Miguel A. Lasunción, Marta Paz-García, Cristina Rodríguez, Cristina Rius, Silvia González-Ramos, Lisardo Boscá, Paloma Martín-Sanz, Vicente Andrés, José Martínez-González, Victoria Fernández-García, Oliver Soehnlein

المساهمون: Ministerio de Economía y Competitividad (España), Ministerio de Ciencia, Innovación y Universidades (España), Agencia Estatal de Investigación (España), Centro de Investigación Biomédica en Red Enfermedades Cardiovaculares (España), Comunidad de Madrid, German Research Foundation, Comunidad de Madrid (España), Deutsche Forschungsgemeinschaft (Alemania), Netherlands Organization for Scientific Research, Unión Europea. Fondo Europeo de Desarrollo Regional (FEDER/ERDF)

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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Repisalud
Instituto de Salud Carlos III (ISCIII)
FASEB JOURNAL
r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau

مصطلحات موضوعية: 0301 basic medicine, Male, Myeloid, Vascular Cell Adhesion Molecule-1, Inflammation, Biochemistry, 03 medical and health sciences, chemistry.chemical_compound, Mice, 0302 clinical medicine, Apolipoproteins E, Cell Movement, Receptor-Interacting Protein Serine-Threonine Kinase 2, Nod1 Signaling Adaptor Protein, NOD1, Genetics, medicine, Animals, Humans, Myeloid Cells, VCAM-1, Autocrine signalling, Molecular Biology, Cells, Cultured, Innate immunity, Innate immune system, business.industry, Monocyte, Pattern recognition receptor, NF-kappa B, Leukocyte, Atherosclerosis, Cardiovascular disease, Lipoproteins, LDL, Mice, Inbred C57BL, body regions, Autocrine Communication, 030104 developmental biology, medicine.anatomical_structure, chemistry, Cancer research, Endothelium, Vascular, medicine.symptom, business, 030217 neurology & neurosurgery, Biotechnology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2a57c134651acb8a640af7880f0c3477Test
http://hdl.handle.net/10261/208024Test

7

Caveolin-1-dependent activation of the metalloprotease TACE/ADAM17 by TGF-β in hepatocytes requires activation of Src and the NADPH oxidase NOX1

المؤلفون: Isabel Fabregat, Rafael Mayoral, Paloma Martín-Sanz, Jèssica Mainez, Gustavo Egea, Joaquim Moreno-Càceres

المساهمون: Ministerio de Educación, Cultura y Deporte (España), European Commission, Ministerio de Economía y Competitividad (España), Instituto de Salud Carlos III, Generalitat de Catalunya

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: 0301 basic medicine, TACE/ADAM17, TGF- β, Cell Survival, Blotting, Western, Caveolin 1, Apoptosis, ADAM17 Protein, Biology, Biochemistry, 03 medical and health sciences, 0302 clinical medicine, Transforming Growth Factor beta, NOX1, Animals, NADH, NADPH Oxidoreductases, Enzyme Inhibitors, Phosphorylation, Molecular Biology, Cells, Cultured, Mice, Knockout, NADPH oxidase, Kinase, NADPH Oxidase 1, Cell Biology, Transforming growth factor beta, Molecular biology, 3. Good health, Enzyme Activation, ErbB Receptors, ADAM Proteins, Pyrimidines, src-Family Kinases, 030104 developmental biology, CAV1, 030220 oncology & carcinogenesis, Hepatocytes, biology.protein, RNA Interference, Reactive Oxygen Species, Src, Proto-oncogene tyrosine-protein kinase Src

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5b9b645b96ab7a54e6e73b6dd953a2aTest
https://doi.org/10.1111/febs.13669Test

8

Metabolic signatures linked to macrophage polarization: from glucose metabolism to oxidative phosphorylation

المؤلفون: Paloma Martín-Sanz, Susana Alemany, Marina Mojena, Silvia González-Ramos, Patricia Prieto, María Fernández-Velasco, Lisardo Boscá

المساهمون: Comunidad de Madrid, Red de Investigación Cardiovascular (España), Ministerio de Economía y Competitividad (España)

المصدر: Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: Anabolism, Macrophages, Macrophage polarization, Inflammation, Metabolism, Oxidative phosphorylation, Macrophage Activation, Biology, Biochemistry, Glucose, medicine, Humans, Phosphorylation, Macrophage, Glycolysis, medicine.symptom, Energy Metabolism, Oxidation-Reduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2858f0b33b95bf24f219c6c88c007611Test
https://doi.org/10.1042/bst20150107Test

9

Protection against gamma-radiation injury by protein tyrosine phosphatase 1B

المؤلفون: Silvia González-Ramos, Marina Mojena, Adrián Povo-Retana, Daniel Rico, Ángela M. Valverde, Victoria Fernández-García, Paloma Martín-Sanz, Alberto Tejedor, María Pimentel-Santillana, Lisardo Boscá, Patricia Rada

المساهمون: Agencia Estatal de Investigación (España), Ministerio de Economía y Competitividad (España), Fundación Ramón Areces, European Commission, Instituto de Salud Carlos III, Ministerio de Economía, Industria y Competitividad (España), Comunidad de Madrid

المصدر: Redox Biology, Vol 17, Iss, Pp 213-223 (2018)
Repositorio Institucional de la Consejería de Sanidad de la Comunidad de Madrid
Consejería de Sanidad de la Comunidad de Madrid
Redox Biology
Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: 0301 basic medicine, Irradiation sensitivity, p53, Cell viability, DNA damage, Clinical Biochemistry, Cell, Protein tyrosine phosphatase, Biochemistry, 03 medical and health sciences, Mice, Immune system, Downregulation and upregulation, medicine, Animals, Fragmentation (cell biology), Phosphorylation, Radiation Injuries, lcsh:QH301-705.5, Cell Proliferation, Mice, Knockout, Protein Tyrosine Phosphatase, Non-Receptor Type 1, lcsh:R5-920, Lethality, Chemistry, Macrophages, Organic Chemistry, Wild type, Fibroblasts, beta-Galactosidase, Glutathione, Hedgehog signaling pathway, Cell biology, 030104 developmental biology, medicine.anatomical_structure, lcsh:Biology (General), Gene Expression Regulation, Gamma Rays, RNA Interference, lcsh:Medicine (General), Reactive Oxygen Species, hormones, hormone substitutes, and hormone antagonists, DNA Damage

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f51547af9ade10c42d006efd81520ca8Test
https://pubmed.ncbi.nlm.nih.gov/29705509Test

10

Disruption of tumor necrosis factor alpha receptor 1 signaling accelerates NAFLD progression in mice upon a high-fat diet

المؤلفون: A.S. Arboatti, Cristina E. Carnovale, Juan A. Monti, María Paula Ceballos, Ariel D. Quiroga, Paloma Martín-Sanz, M.G. Sedlmeier, María Teresa Ronco, Daniel E. Francés, Eduardo Roggero, Omar Motiño, Flavia Lambertucci, Gerardo B. Pisani, Silvina R. Villar, María de Luján Alvarez

المساهمون: Consejo Nacional de Investigaciones Científicas y Técnicas (Argentina), Ministerio de Economía y Competitividad (España)

المصدر: CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Digital.CSIC. Repositorio Institucional del CSIC
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مصطلحات موضوعية: 0301 basic medicine, Male, HIGH-FAT DIET, IRS1, Endocrinology, Diabetes and Metabolism, Clinical Biochemistry, Interleukin-1beta, IL-1 BETA, Apoptosis, White adipose tissue, Biochemistry, Non-alcoholic Fatty Liver Disease, Nonalcoholic fatty liver disease, IL-1 beta, Insulin, Mice, Knockout, Nutrition and Dietetics, Hepatocyte apoptotic process, biology, purl.org/becyt/ford/3.1 [https], Medicina Básica, High-fat diet, Liver, Receptors, Tumor Necrosis Factor, Type I, purl.org/becyt/ford/3 [https], Signal Transduction, INSULIN RESISTANCE, medicine.medical_specialty, CIENCIAS MÉDICAS Y DE LA SALUD, Diet, High-Fat, Fisiología, Proinflammatory cytokine, 03 medical and health sciences, Insulin resistance, INFLAMMATION, Internal medicine, Nonalcoholic liver disease, medicine, Animals, Molecular Biology, Protein kinase B, Inflammation, NONALCOHOLIC LIVER DISEASE, business.industry, Macrophages, medicine.disease, Mice, Inbred C57BL, TNFR1, Insulin receptor, 030104 developmental biology, Endocrinology, biology.protein, business

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcdcc388e2877340d74d646f05705bb6Test
http://hdl.handle.net/10261/188143Test

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