يعرض 1 - 3 نتائج من 3 نتيجة بحث عن '"Peng, Shaohua"', وقت الاستعلام: 1.03s تنقيح النتائج
  1. 1
    دورية أكاديمية
    Kinase-Impaired BRAF Mutations in Lung Cancer Confer Sensitivity to Dasatinib

    المؤلفون: Sen, Banibrata, Peng, Shaohua, Tang, Ximing, Erickson, Heidi S., Galindo, Hector, Mazumdar, Tuhina, Stewart, David J., Wistuba, Ignacio, Johnson, Faye M.

    مصطلحات موضوعية: B-RAF, CELLS, ACTIVATION, INHIBITORS, MELANOMA, CRAF, APOPTOSIS, TUMORS, 03 Good Health and Well-being, 03 Salud y bienestar

    وصف الملف: 10 páginas

    العلاقة: MEDLINE:22649091; https://doi.org/10.1126/scitranslmed.3003513Test; https://repositorio.uc.cl/handle/11534/77249Test; WOS:000304865400005

    الإتاحة: https://doi.org/10.1126/scitranslmed.3003513Test
    https://repositorio.uc.cl/handle/11534/77249Test

    View record in BASE

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  2. 2
    دورية أكاديمية
    Matrilin‐2 prevents the TNFα‐induced apoptosis of WB‐F344 cells via suppressing JNK pathway.

    المؤلفون: Luo, Junming1,2 (AUTHOR), Peng, Shaohua3 (AUTHOR), Bai, Wenbin4 (AUTHOR), Wu, Zhaoyu5 (AUTHOR), Shan, Zhongshu6 (AUTHOR), Wu, Zetao6 (AUTHOR), Yuan, Xin6 (AUTHOR), Che, Xiaoming4 (AUTHOR), Duan, Zhili1 (AUTHOR), Peng, Jinwu7 (AUTHOR), Wang, Yichun2 (AUTHOR) wangyichun2005@sina.com, Zhang, Shukun8 (AUTHOR) zhangshukun0457@126.com

    المصدر: Biotechnology & Applied Biochemistry. May/Jun2019, Vol. 66 Issue 3, p309-315. 7p.

    مصطلحات موضوعية: *TUMOR necrosis factors, *PROGENITOR cells, *CELLS, *APOPTOSIS, *LIVER cells


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  3. 3
    دورية أكاديمية
    Mutations of the LIM protein AJUBA mediate sensitivity of head and neck squamous cell carcinoma to treatment with cell-cycle inhibitors.

    المؤلفون: Zhang, Ming1,2, Singh, Ratnakar1, Peng, Shaohua1, Mazumdar, Tuhina1, Sambandam, Vaishnavi1, Shen, Li3, Tong, Pan3, Li, Lerong3, Kalu, Nene N.1, Pickering, Curtis R.4,5, Frederick, Mitchell6, Myers, Jeffrey N.4,5, Wang, Jing3,5, Johnson, Faye M.1,5 fmjohns@mdanderson.org

    المصدر: Cancer Letters. Apr2017, p71-82. 12p.

    مصطلحات موضوعية: *HEAD & neck cancer treatment, *GENETIC mutation, *PROTEIN structure, *BIOMARKERS, *POLO-like kinases, *PROTEIN metabolism, *ANIMAL experimentation, *ANTHROPOMETRY, *ANTINEOPLASTIC agents, *APOPTOSIS, *CELL lines, *CELL physiology, *CELLULAR signal transduction, *DRUG therapy, *DOSE-effect relationship in pharmacology, *GENES, *GENETIC techniques, *HEAD tumors, *HETEROCYCLIC compounds, *MICE, *NECK tumors, *PROTEIN kinases, *PROTEIN-tyrosine kinases, *PROTEINS, *SQUAMOUS cell carcinoma, *SULFUR compounds, *TIME, *TRANSFERASES, *UREA, *PHENOTYPES, *NUCLEAR proteins, *PROTEIN kinase inhibitors, *GENOTYPES, *CELL cycle proteins, *CHEMICAL inhibitors, *PHARMACODYNAMICS


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