المؤلفون: Filippo de Braud, Claudio Vernieri, Alessandra Gambacurta, Silvia D'Amico, Silvia Baldari, Gabriele Toietta, Francesca Bernassola, Flavio Di Giacinto, Ewa K. Krasnowska, Isabella Manni, Marco Ranalli, Maria Lucibello, Giulia Piaggio

المصدر: Cells 9 (2020). doi:10.3390/cells9051260
info:cnr-pdr/source/autori:Silvia D'Amico .; Ewa Krystyna Krasnowska .; Isabella Manni I.; Gabriela Toietta .; Silvia Baldari .; Giulia Piaggio G.; Marco Ranalli ; Alesandra Gambacurta ; Claudio Vernieri ; Flavio Di Giacinto .; Francesca Bernassola .; Filippo de Braud ; Maria Lucibello ./titolo:DHA Affects Microtubule Dynamics Through Reduction of Phospho-TCTP Levels and Enhances the Antiproliferative Effect of T-DM1 in Trastuzumab-Resistant HER2-Positive Breast Cancer Cell Lines/doi:10.3390%2Fcells9051260/rivista:Cells/anno:2020/pagina_da:/pagina_a:/intervallo_pagine:/volume:9
Cells
Volume 9
Issue 5

مصطلحات موضوعية: Receptor, ErbB-2, T-DM1, medicine.medical_treatment, Apoptosis, Mice, SCID, Microtubules, Phosphatidylinositol 3-Kinases, chemistry.chemical_compound, Trastuzumab, Cytotoxic T cell, Epidermal growth factor receptor, Phosphorylation, skin and connective tissue diseases, biology, Settore BIO/11, Tumor Protein, Translationally-Controlled 1, General Medicine, Artemisinins, DHA, Female, Signal Transduction, medicine.drug, musculoskeletal diseases, congenital, hereditary, and neonatal diseases and abnormalities, phospho-TCTP, Mitosis, Breast Neoplasms, Article, Breast cancer, Cell Line, Tumor, Biomarkers, Tumor, medicine, Animals, Humans, HER2-positive breast cancer, Cell Proliferation, Chemotherapy, business.industry, medicine.disease, Oxidative Stress, chemistry, Drug Resistance, Neoplasm, Trastuzumab emtansine, biology.protein, Cancer research, Translationally controlled tumour protein, Reactive Oxygen Species, business, Proto-Oncogene Proteins c-akt, DNA Damage

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50d87d8b3dbe15279ffc19762694738eTest
https://doi.org/10.3390/cells9051260Test

المؤلفون: Gareth J. Browne, Loredana Zocchi, Martina Malatesta, Aaron Ciechanover, Francesca Bernassola, Angelo Peschiaroli, Ewen Gallagher, Richard A. Knight, Mario Rossi, Rami I. Aqeilan, Flavia Scialpi, Gerry Melino

المصدر: Cell death and differentiation. 15(7)

مصطلحات موضوعية: adaptor protein, cytokine, mutant protein, Notch receptor, protein c jun, protein Itch, regulator protein, transcription factor, transforming growth factor beta, ubiquitin protein ligase E3, apoptosis, autoimmune disease, autoimmunity, binding affinity, carboxy terminal sequence, cell differentiation, cell fate, cell growth, cell proliferation, cell renewal, cell stress, clonal anergy, disease course, DNA damage, enzyme activation, enzyme activity, enzyme analysis, enzyme localization, enzyme specificity, enzyme substrate, epidermis cell, gene expression regulation, gene mutation, growth regulation, human, immune response, immunological tolerance, immunopathogenesis, immunopathology, immunoregulation, inflammation, keratinocyte, lung disease, lymphocyte differentiation, malignant neoplastic disease, malignant transformation, mutational analysis, nonhuman, pathophysiology, priority journal, protein binding, protein degradation, protein expression, protein family, protein phosphorylation, protein processing, protein protein interaction, protein stability, regulatory mechanism, review, signal transduction, skin disease, T lymphocyte activation, transcription regulation, ubiquitination, Animals, Cell Death, Immune System, Keratinocytes, Mice, Mice, Mutant Strains, Neoplasms, Phosphorylation, Protein Transport, Receptor, Epidermal Growth Factor, Receptors, Chemokine, Repressor Proteins, Signal Transduction, Skin, Substrate Specificity, Transforming Growth Factor beta, TRPC Cation Channels, Ubiquitin, Ubiquitin-Protein Ligases, Agouti, Mus, skin and connective tissue diseases, Settore BIO/11, Ubiquitin ligase, ErbB Receptors, Molecular Biology, Epidermal Growth Factor, Immunology, Chemokine, Receptors, biology, Mutant Strains, medicine.symptom, Signal transduction, Receptor, Inflammation, Immune system, Immunity, medicine, Cell Biology, Transforming growth factor beta, biology.protein

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d75d8a9d69263a342ecb749494c14652Test
https://pubmed.ncbi.nlm.nih.gov/18552861Test