نتائج البحث - "Qinhong Xu"

1

RETRACTED ARTICLE: LncRNA KCNQ1OT1 inhibits the radiosensitivity and promotes the tumorigenesis of hepatocellular carcinoma via the miR-146a-5p/ACER3 axis

المؤلفون: Lin Wang, Lijing Zhou, Ganghua Yang, Lei Zhang, Yong Wan, Fandi Meng, Xiankui Meng, Qinhong Xu

المصدر: Cell Cycle. 19:2519-2529

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::22771a7da1df74db46014a7f06da1134Test
https://doi.org/10.1080/15384101.2020.1809259Test

2

Overexpression of circ_0001445 decelerates hepatocellular carcinoma progression by regulating miR-942-5p/ALX4 axis

المؤلفون: Fandi Meng, Yong Wan, Lijing Zhou, Lin Wang, Lei Zhang, Ganghua Yang, Qinhong Xu

المصدر: Biotechnology Letters. 42:2735-2747

مصطلحات موضوعية: Male, 0106 biological sciences, 0301 basic medicine, Carcinoma, Hepatocellular, Carcinogenesis, Cell, Bioengineering, Biology, medicine.disease_cause, 01 natural sciences, Applied Microbiology and Biotechnology, Mice, 03 medical and health sciences, In vivo, Cell Line, Tumor, 010608 biotechnology, medicine, Animals, Humans, Glycolysis, neoplasms, Cell Proliferation, Transition (genetics), Cell growth, Liver Neoplasms, Cell cycle process, RNA, Circular, General Medicine, medicine.disease, digestive system diseases, DNA-Binding Proteins, Gene Expression Regulation, Neoplastic, MicroRNAs, 030104 developmental biology, medicine.anatomical_structure, Hepatocellular carcinoma, Disease Progression, Cancer research, Heterografts, Female, Transcription Factors, Biotechnology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1cc7f2ff6fddf31ac541083371cb74cTest
https://doi.org/10.1007/s10529-020-02985-zTest

3

Curcumin Suppresses Hepatic Stellate Cell-Induced Hepatocarcinoma Angiogenesis and Invasion through Downregulating CTGF

المؤلفون: Jianjun Lei, Wanxing Duan, Dong Zhang, Shan Shao, Zheng Wang, Liang Han, Qinhong Xu, Xuqi Li, Wei Li

المصدر: Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity, Vol 2019 (2019)

مصطلحات موضوعية: Vascular Endothelial Growth Factor A, 0301 basic medicine, Aging, Carcinoma, Hepatocellular, Curcumin, Epithelial-Mesenchymal Transition, Article Subject, NF-E2-Related Factor 2, Angiogenesis, medicine.medical_treatment, Down-Regulation, Biochemistry, Neovascularization, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Hepatic Stellate Cells, Human Umbilical Vein Endothelial Cells, medicine, Humans, Neoplasm Invasiveness, Epithelial–mesenchymal transition, lcsh:QH573-671, Neovascularization, Pathologic, lcsh:Cytology, Interleukin-6, Growth factor, Liver Neoplasms, Connective Tissue Growth Factor, Hep G2 Cells, Cell Biology, General Medicine, Hypoxia-Inducible Factor 1, alpha Subunit, Glutathione, Chemokine CXCL12, digestive system diseases, CTGF, 030104 developmental biology, Matrix Metalloproteinase 9, chemistry, Tumor progression, 030220 oncology & carcinogenesis, Hepatic stellate cell, Cancer research, medicine.symptom, Research Article

وصف الملف: text/xhtml

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64e25c5d49a0a8c86e0a4f5b7b82f5d7Test
https://doi.org/10.1155/2019/8148510Test

4

Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of YAP/HIF-1α

المؤلفون: Jianjun Lei, Weikun Qian, Qinhong Xu, Jie Li, Junyu Cao, Zhengdong Jiang, Liang Cheng, Bin Yan, Cancan Zhou, Liankang Sun, Ke Chen, Qingyong Ma

المصدر: Experimental Cell Research. 371:63-71

مصطلحات موضوعية: 0301 basic medicine, Homeobox protein NANOG, C-Met, Primary Cell Culture, Biology, 03 medical and health sciences, Paracrine signalling, chemistry.chemical_compound, 0302 clinical medicine, Cancer stem cell, Cell Line, Tumor, Hexokinase, Spheroids, Cellular, Pancreatic cancer, Paracrine Communication, Tumor Microenvironment, medicine, Humans, Pancreas, Adaptor Proteins, Signal Transducing, Tumor microenvironment, Hepatocyte Growth Factor, SOXB1 Transcription Factors, Pancreatic Stellate Cells, YAP-Signaling Proteins, Nanog Homeobox Protein, Cell Biology, Proto-Oncogene Proteins c-met, Hypoxia-Inducible Factor 1, alpha Subunit, Phosphoproteins, medicine.disease, Gene Expression Regulation, Neoplastic, Pancreatic Neoplasms, Protein Transport, 030104 developmental biology, chemistry, Culture Media, Conditioned, 030220 oncology & carcinogenesis, Cancer research, Hepatocyte growth factor, Stem cell, Glycolysis, Octamer Transcription Factor-3, Signal Transduction, Transcription Factors, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4d858a9b7ce3dd45f62ccd7864d06d9Test
https://doi.org/10.1016/j.yexcr.2018.07.041Test

5

NAF-1 Inhibition by Resveratrol Suppresses Cancer Stem Cell-Like Properties and the Invasion of Pancreatic Cancer

المؤلفون: Tao Qin, Liang Cheng, Ying Xiao, Weikun Qian, Jie Li, Zheng Wu, Zheng Wang, Qinhong Xu, Wanxing Duan, Lucas Wong, Erxi Wu, Qingyong Ma, Jiguang Ma

المصدر: Frontiers in Oncology
Frontiers in Oncology, Vol 10 (2020)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b8cbabc314ae9f0751f31ce68a973d1bTest
https://doi.org/10.3389/fonc.2020.01038Test

6

Resveratrol Ameliorates the Malignant Progression of Pancreatic Cancer by Inhibiting Hypoxia-induced Pancreatic Stellate Cell Activation

المؤلفون: Tao Qin, Qinhong Xu, Jie Li, Jianjun Lei, Ying Xiao, Xuqi Li, Zheng Wang, Wanxing Duan, Liankang Sun, Qingyong Ma, Jiguang Ma, Weikun Qian

المصدر: Cell Transplantation
Cell Transplant
Cell Transplantation, Vol 29 (2020)

مصطلحات موضوعية: 0301 basic medicine, Stromal cell, endocrine system diseases, pancreatic cancer, Biomedical Engineering, Pancreatic stellate cell, lcsh:Medicine, Resveratrol, Adenocarcinoma, resveratrol, Antioxidants, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Pancreatic cancer, medicine, Humans, Interleukin 6, Transplantation, biology, Chemistry, hypoxia, lcsh:R, Pancreatic Stellate Cells, Cell Biology, PSCs, medicine.disease, Cell Hypoxia, Desmoplasia, Vascular endothelial growth factor A, 030104 developmental biology, medicine.anatomical_structure, stromal desmoplasia, 030220 oncology & carcinogenesis, Hepatic stellate cell, biology.protein, Cancer research, Disease Progression, Original Article, medicine.symptom, Corrigendum, Carcinoma, Pancreatic Ductal

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8c69248eb24c1ab384e219ecbe8de2baTest
http://europepmc.org/articles/PMC7563930Test

7

Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions

المؤلفون: Jiguang Ma, Zhengdong Jiang, Liang Zong, Xin Chen, Liang Cheng, Bin Yan, Qinhong Xu, Xuqi Li, Jianjun Lei, Wanxing Duan, Ke Chen, Zheng Wang, Cancan Zhou, Qingyong Ma

المصدر: Journal of Experimental & Clinical Cancer Research, Vol 37, Iss 1, Pp 1-14 (2018)
Journal of Experimental & Clinical Cancer Research : CR

مصطلحات موضوعية: Male, 0301 basic medicine, Cancer Research, endocrine system diseases, Gene Expression, Cell Cycle Proteins, Cell Communication, Smad2 Protein, Mice, Invasion, Tumor Microenvironment, Pancreatic stellate cells, Chemistry, Nuclear Proteins, Middle Aged, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Gene Expression Regulation, Neoplastic, Oncology, Gene Knockdown Techniques, Disease Progression, Female, YAP, Adult, Epithelial-Mesenchymal Transition, Stromal cell, Cell Survival, Mice, Transgenic, lcsh:RC254-282, Transforming Growth Factor beta1, 03 medical and health sciences, Cell Line, Tumor, Pancreatic cancer, medicine, Animals, Humans, Epithelial–mesenchymal transition, Aged, Neoplasm Staging, Tumor microenvironment, Cell growth, Research, medicine.disease, Coculture Techniques, digestive system diseases, Pancreatic Neoplasms, CTGF, Disease Models, Animal, 030104 developmental biology, Tumor progression, Hepatic stellate cell, Cancer research, Neoplasm Grading, Stromal Cells, Transcription Factors

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fed4d7268751332e64a4617fe50e8e99Test
http://link.springer.com/article/10.1186/s13046-018-0740-4Test

8

Hyperglycemia aggravates microenvironment hypoxia and promotes the metastatic ability of pancreatic cancer

المؤلفون: Jiguang Ma, Qingyong Ma, Qinhong Xu, Weikun Qian, Liang Cheng, Bin Yan, Liang Han, Han Liu, Wei Li

المصدر: Computational and Structural Biotechnology Journal, Vol 16, Iss, Pp 479-487 (2018)
Computational and Structural Biotechnology Journal

مصطلحات موضوعية: 0301 basic medicine, lcsh:Biotechnology, Biophysics, HIF-1α, PSC, pancreatic stellate cells, STZ, streptozotocin, Biochemistry, Metastasis, 03 medical and health sciences, 0302 clinical medicine, Structural Biology, In vivo, SOD, superoxide dismutase, lcsh:TP248.13-248.65, Pancreatic cancer, Diabetes mellitus, Genetics, medicine, Hypoxia, TEM, transmission electron microscopy, EGF, epidermal growth factor, business.industry, GDNF, glial cell line-derived neurotrophic factor, CCL2, chemical chemokine 2, Hypoxia (medical), medicine.disease, Streptozotocin, VEGF, vascular endothelial growth factor, Computer Science Applications, CoCl2, cobalt chloride, ECM, endothelial cells, extracellular matrix, 030104 developmental biology, medicine.anatomical_structure, Hyperglycemia, H2O2, hydrogen peroxide, 030220 oncology & carcinogenesis, HIF-1α, hypoxia-inducible factor 1α, Cancer research, Immunohistochemistry, PNI, perineural invasion, medicine.symptom, EMT, epithelial-mesenchymal transition, Pancreas, business, Research Article, Biotechnology, medicine.drug

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::92efc4506fb8f4b87b9567b9a58e1dc1Test
https://doi.org/10.1016/j.csbj.2018.10.006Test

9

Circ-CSPP1 knockdown suppresses hepatocellular carcinoma progression through miR-493-5p releasing-mediated HMGB1 downregulation

المؤلفون: Qinhong Xu, Ganghua Yang, Lei Zhang, Fandi Meng, Yong Wan, Lin Wang

المصدر: Cellular Signalling. 86:110065

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6e228501ff0d3a50c92ccd4fedee1f4Test
https://doi.org/10.1016/j.cellsig.2021.110065Test

10

Insulin Receptor and GPCR Crosstalk Stimulates YAP via PI3K and PKD in Pancreatic Cancer Cells

المؤلفون: Steven H. Young, Fang Hao, Enrique Rozengurt, Yinglan Zhao, Jan V. Stevens, James Sinnett-Smith, Qinhong Xu

المصدر: Molecular cancer research : MCR, vol 15, iss 7

مصطلحات موضوعية: 0301 basic medicine, Chemokine CXCL5, Cancer Research, medicine.medical_treatment, Receptors, G-Protein-Coupled, chemistry.chemical_compound, Insulin receptor substrate, Receptors, Insulin, Phosphorylation, Protein Kinase C, Neurotensin, Cancer, Tumor, Adaptor Proteins, CD, Gene Expression Regulation, Neoplastic, Oncology, Pancreatic Ductal, CYR61, Signal transduction, Carcinoma, Pancreatic Ductal, Receptor, Class I Phosphatidylinositol 3-Kinases, Oncology and Carcinogenesis, Biology, Article, Cell Line, G-Protein-Coupled, Pancreatic Cancer, 03 medical and health sciences, Rare Diseases, Antigens, CD, Cell Line, Tumor, medicine, Humans, Oncology & Carcinogenesis, Antigens, Molecular Biology, PI3K/AKT/mTOR pathway, Adaptor Proteins, Signal Transducing, Neoplastic, Carcinoma, Signal Transducing, Connective Tissue Growth Factor, YAP-Signaling Proteins, Phosphoproteins, Receptor, Insulin, Pancreatic Neoplasms, CTGF, Insulin receptor, Pyrimidines, 030104 developmental biology, Gene Expression Regulation, chemistry, Cancer research, biology.protein, Digestive Diseases, Transcription Factors, Cysteine-Rich Protein 61, Developmental Biology

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::057cef87736dba67ab8747ef526205c9Test
https://doi.org/10.1158/1541-7786.mcr-17-0023Test

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