نتائج البحث - "A. Faggioni"

1

Could autophagy dysregulation link neurotropic viruses to Alzheimer’s disease?

المؤلفون: Mara Cirone, Maria Anele Romeo, Alberto Faggioni

المصدر: Neural Regeneration Research, Vol 14, Iss 9, Pp 1503-1506 (2019)
Neural Regeneration Research

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::475b8b3fd2378f624cd288c5ba302831Test
http://www.nrronline.org/article.asp?issn=1673-5374;year=2019;volume=14;issue=9;spage=1503;epage=1506;aulast=RomeoTest

2

BFRF1 protein is involved in EBV-mediated autophagy manipulation

المؤلفون: Marzia Dimarco, Roberta Santarelli, Antonella Farina, Giuseppina Farina, Mara Cirone, Antonio Angeloni, Roberta Gonnella, Alberto Faggioni, Sandro Valia

المصدر: Microbes and infection. 22(10)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3f8a8243a84bd1a0c7ba1803341545eTest
https://pubmed.ncbi.nlm.nih.gov/32882412Test

3

HHV-6A infection dysregulates autophagy/UPR interplay increasing beta amyloid production and tau phosphorylation in astrocytoma cells as well as in primary neurons, possible molecular mechanisms linking viral infection to Alzheimer's disease

المؤلفون: Aurelia Gaeta, Maria Anele Romeo, Alberto Faggioni, Gabriella D'Orazi, Maria Saveria Gilardini Montani, Mara Cirone

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5b8d9a05435a471e8f26267643142fd8Test
http://hdl.handle.net/11573/1374144Test

4

Quercetin Interrupts the Positive Feedback Loop Between STAT3 and IL-6, Promotes Autophagy, and Reduces ROS, Preventing EBV-Driven B Cell Immortalization

المؤلفون: Roberta Gonnella, Maria Saveria Gilardini Montani, Roberta Santarelli, Gabriella D'Orazi, Maria Anele Romeo, Marisa Granato, C. Zompetta, Mara Cirone, Alberto Faggioni

المصدر: Biomolecules, Vol 9, Iss 9, p 482 (2019)
Biomolecules
Volume 9
Issue 9

مصطلحات موضوعية: 0301 basic medicine, autophagy, Epstein-Barr virus (EBV), IL-6, LCLs, quercetin, ROS, SQSTM1/p62, STAT3, Epstein-Barr Virus Infections, viruses, lcsh:QR1-502, medicine.disease_cause, Biochemistry, lcsh:Microbiology, chemistry.chemical_compound, 0302 clinical medicine, hemic and lymphatic diseases, Sequestosome-1 Protein, heterocyclic compounds, Cells, Cultured, Feedback, Physiological, chemistry.chemical_classification, B-Lymphocytes, biology, Chemistry, virus diseases, respiratory system, Cell biology, Gene Expression Regulation, Neoplastic, medicine.anatomical_structure, 030220 oncology & carcinogenesis, Quercetin, STAT3 Transcription Factor, Cell Survival, Article, 03 medical and health sciences, medicine, Animals, Humans, Molecular Biology, B cell, Reactive oxygen species, Interleukin-6, Autophagy, Epstein–Barr virus (EBV), STAT3, biochemical phenomena, metabolism, and nutrition, Cell Transformation, Viral, 030104 developmental biology, Cell culture, STAT protein, biology.protein, Reactive Oxygen Species, Carcinogenesis

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3caa4a7edb646dbbc0c444d705ba95f0Test
https://www.mdpi.com/2218-273X/9/9/482Test

5

EBV reduces autophagy, intracellular ROS and mitochondria to impair monocyte survival and differentiation

المؤلفون: Maria Rosaria Torrisi, Alberto Faggioni, Marisa Granato, Mara Cirone, Roberta Santarelli, M. S. Gilardini Montani, Roberta Gonnella

المصدر: Autophagy. 15(4)

مصطلحات موضوعية: 0301 basic medicine, Herpesvirus 4, Human, SQSTM1/p62, Apoptosis, Monocytes, Autophagy-Related Protein 5, STAT3, Sequestosome-1 Protein, RNA, Small Interfering, NFE2L2, education.field_of_study, Kelch-Like ECH-Associated Protein 1, ATG5, EBV, ROS, autophagy, dendritic cells, monocytes, Cell Differentiation, BECN1, Cell biology, Mitochondria, DNA-Binding Proteins, medicine.anatomical_structure, Monocyte differentiation, Signal Transduction, Research Paper, STAT3 Transcription Factor, Cell Survival, NF-E2-Related Factor 2, Biology, Mitochondrial Proteins, 03 medical and health sciences, Sequestosome 1, medicine, Autophagy, Humans, education, Molecular Biology, 030102 biochemistry & molecular biology, Monocyte, Autophagosomes, Granulocyte-Macrophage Colony-Stimulating Factor, rab7 GTP-Binding Proteins, Cell Biology, Dendritic Cells, TFAM, 030104 developmental biology, Mitochondrial biogenesis, rab GTP-Binding Proteins, Interleukin-4, Reactive Oxygen Species, Transcription Factors

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e297bfa7c4288ddb3e938956aa88d05dTest
https://pubmed.ncbi.nlm.nih.gov/30324853Test

6

Epstein-Barr virus encoded EBNA2 alters immune checkpoint PD-L1 expression by downregulating miR-34a in B cell lymphomas

المصدر: Leukemia
Leukemia 33, 132–147 (2018)

مصطلحات موضوعية: 0301 basic medicine, PD-L1, Cancer Research, Epstein-Barr Virus Infections, Herpesvirus 4, Human, medicine.medical_treatment, Lymphocyte, T-Lymphocytes, lymphoma, Biology, medicine.disease_cause, B7-H1 Antigen, Article, Small hairpin RNA, 03 medical and health sciences, Viral Proteins, 0302 clinical medicine, immune system diseases, EBV, hemic and lymphatic diseases, medicine, Biomarkers, Tumor, Tumor Cells, Cultured, Humans, B cell, EBNA2, microRNA, Hematology, Immunotherapy, medicine.disease, Prognosis, Epstein–Barr virus, Immune checkpoint, Lymphoma, Gene Expression Regulation, Neoplastic, MicroRNAs, 030104 developmental biology, medicine.anatomical_structure, Oncology, Epstein-Barr Virus Nuclear Antigens, 030220 oncology & carcinogenesis, Cancer cell, Cancer research, Lymphoma, Large B-Cell, Diffuse

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9be03e7582e0c541ff2cd7c1469d6adTest
http://hdl.handle.net/11573/1118305Test

7

EBV up-regulates PD-L1 on the surface of primary monocytes by increasing ROS and activating TLR signaling and STAT3

المؤلفون: Luca Falcinelli, Maria Saveria Gilardini Montani, Livia Di Renzo, Alberto Faggioni, Marisa Granato, Roberta Santarelli, Mara Cirone, Laura Cuomo, Marina Vitillo, Roberta Gonnella

مصطلحات موضوعية: STAT3 Transcription Factor, 0301 basic medicine, MAPK/ERK pathway, PD-L1, Epstein-Barr Virus Infections, medicine.medical_treatment, Immunology, EBV, MAPK, STAT3, TLR, monocytes, Biology, Virus Replication, B7-H1 Antigen, 03 medical and health sciences, Immune system, medicine, Humans, Immunology and Allergy, Phosphorylation, RNA, Small Interfering, Cell Death, Monocyte, Toll-Like Receptors, Free Radical Scavengers, HLA-DR Antigens, Cell Biology, Up-Regulation, Cell biology, 030104 developmental biology, medicine.anatomical_structure, Cytokine, Myeloid Differentiation Factor 88, biology.protein, RNA Interference, Reactive Oxygen Species, Protein Processing, Post-Translational, Intracellular, Oncovirus, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::752b55d578dcc9776e81daf0535ed913Test
http://hdl.handle.net/11573/1137175Test

8

Chloroquine supplementation increases the cytotoxic effect of curcumin against Her2/neu overexpressing breast cancer cells in vitro and in vivo in nude mice while counteracts it in immune competent mice

المؤلفون: Mara Cirone, Roberto Bei, Giulia d'Amati, Alberto Faggioni, Gabriella D'Orazi, Rosanna Mattera, Roberta Bernardini, Marisa Granato, Laura Masuelli, Monica Benvenuto, Maurizio Mattei

المصدر: OncoImmunology, Vol 6, Iss 11 (2017)

مصطلحات موضوعية: 0301 basic medicine, lcsh:Immunologic diseases. Allergy, Programmed cell death, autophagy, T cell, Immunology, athymic nude mice, Balb/c mice, cancer, chloroquine, curcumin, Her2/neu, immunology and allergy, immunology, oncology, Biology, lcsh:RC254-282, HER2/neu, 03 medical and health sciences, 0302 clinical medicine, Immune system, medicine, Immunology and Allergy, Cytotoxic T cell, Original Research, balb/c mice, Settore MED/04 - Patologia Generale, Autophagy, FOXP3, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Molecular biology, 030104 developmental biology, medicine.anatomical_structure, Oncology, 030220 oncology & carcinogenesis, her2/neu, Cancer cell, Cancer research, biology.protein, lcsh:RC581-607

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab2bbb1ea38d69f8df8fc8b0596140d4Test
https://doaj.org/article/f394136392ee4755867c3d9621fa4ed7Test

9

EBV early lytic protein BFRF1 alters emerin distribution and post-translational modification

المؤلفون: F. Libotte, Antonella Farina, E. Buono, Giuseppina Farina, Shivangi Yadav, Alberto Faggioni, Sandro Valia

المصدر: Virus research. 232

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Herpesvirus 4, Human, Viral egress, Nuclear Envelope, viruses, Cell, Emerin, Active Transport, Cell Nucleus, Hyperphosphorylation, Biology, Virus Replication, Virus, 03 medical and health sciences, Viral Proteins, EBV, Virology, Cell Line, Tumor, medicine, Distribution (pharmacology), Animals, Humans, Nuclear membrane, Pathogen, Virus Release, BFRF1, B-Lymphocytes, Nuclear envelope, Virus Assembly, Virion, Membrane Proteins, Nuclear Proteins, Callithrix, Cell biology, 030104 developmental biology, Infectious Diseases, medicine.anatomical_structure, Lytic cycle, Host-Pathogen Interactions, Protein Multimerization, Protein Processing, Post-Translational

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bab1e7aa82397739a89b88874c85c772Test
https://pubmed.ncbi.nlm.nih.gov/28238874Test

10

Epstein-Barr virus lytic infection promotes activation of Toll-like receptor 8 innate immune response in systemic sclerosis monocytes

المؤلفون: Michael York, Silvia Quarta, Stefania Lenna, G. Alessandra Farina, Maria Trojanowska, Annarita Vestri, Antonella Farina, Stefania Morrone, Edoardo Rosato, Valentina Lacconi, Alberto Faggioni, Giovanna Peruzzi, David H. Dreyfus

المصدر: Arthritis Research & Therapy

مصطلحات موضوعية: 0301 basic medicine, Male, Innate immune response, Chemokine, Epstein-Barr Virus Infections, Herpesvirus 4, Human, Gene Expression, Virus Replication, Monocytes, 0302 clinical medicine, Interferon, hemic and lymphatic diseases, Immunology and Allergy, skin and connective tissue diseases, Cells, Cultured, Toll-like receptor, EBV reactivation, biology, integumentary system, Reverse Transcriptase Polymerase Chain Reaction, Middle Aged, 3. Good health, medicine.anatomical_structure, Host-Pathogen Interactions, Quinolines, Systemic sclerosis, IFN inducible genes, Toll-like receptor 8, Rheumatology, Immunology, Female, medicine.drug, Research Article, Adult, CD14, Blotting, Western, CD16, Peripheral blood mononuclear cell, 03 medical and health sciences, Young Adult, Cell Line, Tumor, medicine, Humans, Aged, Innate immune system, Scleroderma, Systemic, Monocyte, Gene Expression Profiling, Immunity, Innate, Thiazoles, 030104 developmental biology, biology.protein, 030215 immunology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::035daf7a37730bf5716c9cc3a69c6a00Test
https://pubmed.ncbi.nlm.nih.gov/28245863Test

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