المؤلفون: Shohreh Issazadeh-Navikas, Isaac Hurtado-Guerrero, José Pavia, María Jesús Pinto-Medel, Patricia Urbaneja, Natalia Mena-Vázquez, A. Alonso, Begoña Oliver-Martos, José Alcamí, Esther Calonge, Oscar Fernández, Laura Leyva, Antonio Alcami, Pablo Aliaga, Jose Luis Rodriguez-Bada, Bruno Hernáez

المساهمون: Instituto de Salud Carlos III, Unión Europea. Fondo Europeo de Desarrollo Regional (FEDER/ERDF), Regional Government of Andalusia (España), Red Española de Esclerosis Múltiple, Junta de Andalucía, European Commission

المصدر: Journal of Clinical Medicine
Volume 9
Issue 4
Digital.CSIC. Repositorio Institucional del CSIC
instname
Dipòsit Digital de la UB
Universidad de Barcelona
Journal of Clinical Medicine, Vol 9, Iss 959, p 959 (2020)
Hurtado-guerrero, I, Hernáez, B, Pinto-medel, M J, Calonge, E, Rodriguez-bada, J L, Urbaneja, P, Alonso, A, Mena-vázquez, N, Aliaga, P, Issazadeh-navikas, S, Pavia, J, Leyva, L, Alcamí, J, Alcamí, A, Fernández, Ó & Oliver-martos, B 2020, ' Antiviral, Immunomodulatory and Antiproliferative Activities of Recombinant Soluble IFNAR2 without IFN-ß Mediation ', Journal of Clinical Medicine, vol. 9, no. 4, pp. 959 . https://doi.org/10.3390/jcm9040959Test
Repisalud
Instituto de Salud Carlos III (ISCIII)

مصطلحات موضوعية: 0301 basic medicine, medicine.medical_treatment, soluble receptors, Cell, Immunology, lcsh:Medicine, Interferó, IFNAR, Pharmacology, Virus, Article, law.invention, immunology, 03 medical and health sciences, 0302 clinical medicine, Immune system, law, Interferon, medicine, Immunologia, Receptor, Soluble receptors, business.industry, lcsh:R, General Medicine, interferon, 030104 developmental biology, medicine.anatomical_structure, Cytokine, Recombinant DNA, Signal transduction, business, 030217 neurology & neurosurgery, medicine.drug

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0674109f235fe62e9bedb2c4821b8dcbTest
https://hdl.handle.net/20.500.12105/9486Test

المؤلفون: Shohreh Issazadeh-Navikas, Ava Handley, Roger Pocock, Pedro Moreira, Sandeep Gopal, Camilla Nehammer, Huikyong Lee, Leelee Ng, Patrick Ejlerskov, David C. Rubinsztein

المساهمون: Handley, Ava [0000-0003-1543-1551], Rubinsztein, David C [0000-0001-5002-5263], Pocock, Roger [0000-0002-5515-3608], Apollo - University of Cambridge Repository

المصدر: eLife, Vol 8 (2019)
eLife
Nehammer, C, Ejlerskov, P, Gopal, S, Handley, A, Ng, L, Moreira, P, Lee, H, Issazadeh-Navikas, S, Rubinsztein, D C & Pocock, R 2019, ' Interferon-beta-induced miR-1 alleviates toxic protein accumulation by controlling autophagy ', eLife, vol. 8 . https://doi.org/10.7554/eLife.49930Test

مصطلحات موضوعية: 0301 basic medicine, medicine.medical_treatment, Protein aggregation, Mice, 0302 clinical medicine, Interferon, genetics, mammalian cells, Biology (General), 3' Untranslated Regions, Caenorhabditis elegans, Huntingtin Protein, biology, microRNA, Chemistry, General Neuroscience, GTPase-Activating Proteins, General Medicine, 3. Good health, Cell biology, Cytokine, C. elegans, Medicine, Research Article, medicine.drug, autophagy, QH301-705.5, Science, Chemical biology, chemical biology, General Biochemistry, Genetics and Molecular Biology, protein aggregation, Protein Aggregates, 03 medical and health sciences, Biochemistry and Chemical Biology, genomics, medicine, biochemistry, Animals, Humans, RNA, Messenger, Caenorhabditis elegans Proteins, Base Sequence, General Immunology and Microbiology, human cells, Autophagy, rab7 GTP-Binding Proteins, Genetics and Genomics, Interferon-beta, biology.organism_classification, MicroRNAs, 030104 developmental biology, rab GTP-Binding Proteins, Mutant Proteins, Rab, Peptides, 030217 neurology & neurosurgery, HeLa Cells

وصف الملف: application/pdf; application/zip; text/xml

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::34810bf881501a7c32975e5884fc3cf7Test
https://elifesciences.org/articles/49930Test

المؤلفون: Shohreh Issazadeh-Navikas, Cord Brakebusch, Alba Manresa-Arraut, Flemming Fryd Johansen, Henrik Hasseldam

المصدر: Frontiers in Immunology, Vol 9 (2018)
Frontiers in Immunology
Manresa-Arraut, A, Johansen, F F, Brakebusch, C, Issazadeh-Navikas, S & Hasseldam, H 2018, ' RhoA Drives T-Cell Activation and Encephalitogenic Potential in an Animal Model of Multiple Sclerosis ', Frontiers in Immunology, vol. 9, 1235, pp. 1-14 . https://doi.org/10.3389/fimmu.2018.01235Test

مصطلحات موضوعية: lcsh:Immunologic diseases. Allergy, 0301 basic medicine, Encephalomyelitis, Autoimmune, Experimental, RHOA, T-Lymphocytes, medicine.medical_treatment, T cell, Immunology, experimental autoimmune encephalomyelitis, Regulator, Lymphocyte Activation, multiple sclerosis, Immunophenotyping, neuroinflammation, Mice, 03 medical and health sciences, 0302 clinical medicine, T-cell, medicine, Animals, Immunology and Allergy, Small GTPase, Antigens, Neuroinflammation, Original Research, Mice, Knockout, biology, Experimental autoimmune encephalomyelitis, Transendothelial and Transepithelial Migration, RhoA, medicine.disease, Actin cytoskeleton, Immunohistochemistry, Cell biology, Disease Models, Animal, 030104 developmental biology, Cytokine, medicine.anatomical_structure, biology.protein, Cytokines, Myelin-Oligodendrocyte Glycoprotein, Disease Susceptibility, rhoA GTP-Binding Protein, lcsh:RC581-607, Biomarkers, 030217 neurology & neurosurgery

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::005008af8cf6f2733fad7b30b4ea40deTest
https://doi.org/10.3389/fimmu.2018.01235Test