رسالة جامعية
From age to aging: Biological age and its role in the criminal career.
| العنوان: | From age to aging: Biological age and its role in the criminal career. |
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| المؤلفون: | Tanksley, Peter T. |
| بيانات النشر: | University of Cincinnati / OhioLINK, 2020. |
| سنة النشر: | 2020 |
| المجموعة: | Ohiolink ETDs |
| Original Material: | http://rave.ohiolink.edu/etdc/view?acc_num=ucin1601995918198417Test |
| مصطلحات موضوعية: | Criminology, Crime, Criminal Career, Aging, Biological Age, Pace of Aging, Stress |
| الوصف: | The age-crime relationship is one of the most canonical and law-like relationships that criminology has observed; however, the age-crime relationship is more complicated than criminological theories have yet appreciated. Criminologists generally view the influence of age in one of two ways: (1) invariant, inexplicable, and non-interactive—and thus scientifically uninteresting—or (2) as signifying nothing so much as the timing of certain developmental and societal events that are actually responsible for explaining the variation in crime. Both approaches view the age-crime relationship in a unidirectional manner (i.e., age impacts crime), never considering the opposite possibility. This dissertation reconsiders the age-crime relationship by applying a new conceptualization of age that is rooted in the physiological integrity of the human organism: biological age. By viewing age as a biological construct that indicates one’s progress in the process of birth, maturation, senescence, and ultimately death, I am able to reverse the causal arrow and ask the question: how does crime influence (biological) age?In order to explore the relationship between biological age and crime, I conduct three studies in which I leverage two recently developed methods for quantifying biological age and use data from two longitudinal cohort studies—the Health and Retirement Study (HRS; United States) and the Dunedin Longitudinal Study (DLS; New Zealand). In Chapter 1, I provide a brief introduction to three relationships that make up a tripartite structure: (1) age and crime, (2) age and health, and (3) crime and health. These interrelated literatures provide the theoretical and empirical premises for the rest of the dissertation. In Chapter 2, I examine the impact of lifetime incarceration on the biological age in the HRS and find that individuals with a history of incarceration tend to experience faster biological aging, but only among non-Black respondents. In Chapter 3, I use the DLS to explore how early offending and criminal justice contact impacts biological aging throughout middle adulthood and find that criminal justice involvement (criminal conviction) predicts accelerated biological aging even after accounting for prior offending behavior. In Chapter 4, I again use the DLS to examine whether individuals who persist in offending longer than others also have a more accelerated pace of biological aging and find that the evidence is, at most, only suggestive. In Chapter 5, I summarize the overall findings of the study and describe how concepts like biological age may be situated in the current theories of age and crime in the criminological literature. |
| Original Identifier: | oai:etd.ohiolink.edu:ucin1601995918198417 |
| نوع الوثيقة: | text |
| اللغة: | English |
| الإتاحة: | http://rave.ohiolink.edu/etdc/view?acc_num=ucin1601995918198417Test |
| حقوق: | unrestricted This thesis or dissertation is protected by copyright: some rights reserved. It is licensed for use under a Creative Commons license. Specific terms and permissions are available from this document's record in the OhioLINK ETD Center. |
| رقم الانضمام: | edsndl.OhioLink.oai.etd.ohiolink.edu.ucin1601995918198417 |
| قاعدة البيانات: | Networked Digital Library of Theses & Dissertations |
| الوصف غير متاح. |