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1دورية أكاديمية
المساهمون: BIOLOGY (NU), MECHANOBIOLOGY INSTITUTE
المصدر: Unpaywall 20201031
مصطلحات موضوعية: caveolin 1, dexamethasone, genomic DNA, guanine nucleotide exchange factor, guanosine triphosphatase, protein Cdc42, Rho like small guanosine triphosphatase, RhoA guanine nucleotide binding protein, unclassified drug, glucocorticoid, small interfering RNA, disease treatment, enzyme activity, gene expression, genetic engineering, mutation, pathogen, animal cell, animal experiment, animal model, animal tissue, Article, CDK20 gene, cell culture, cell migration, circular dichroism, controlled study, DLC1 gene, enzyme activation, female
العلاقة: Ashraf, S, Kudo, H, Rao, J (2018). Mutations in six nephrosis genes delineate a pathogenic pathway amenable to treatment. Nature Communications 9 (1) : 1960. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-018-04193-wTest; https://scholarbank.nus.edu.sg/handle/10635/178411Test
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2دورية أكاديمية
المساهمون: DEPT OF BIOCHEMISTRY
المصدر: Unpaywall 20201031
مصطلحات موضوعية: guanosine triphosphatase activating protein, protein binding, Rho guanine nucleotide binding protein, amino acid sequence, Candida albicans, candidiasis, chemistry, diploidy, gene expression regulation, genetics, haploidy, metabolism, microbiology, mutation, phenotype, physiology, ultrastructure, Fungal, GTPase-Activating Proteins, rho GTP-Binding Proteins
العلاقة: Yang, S.L, Zeng, G, Chan, F.Y, Wang, Y.-M, Yang, D, Wang, Y (2018). Sac7 and Rho1 regulate the white-to-opaque switching in Candida albicans. Scientific Reports 8 (1) : 875. ScholarBank@NUS Repository. https://doi.org/10.1038/s41598-018-19246-9Test; https://scholarbank.nus.edu.sg/handle/10635/178524Test
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3دورية أكاديمية
المؤلفون: Bi, M, Gladbach, A, Van Eersel, J, Ittner, A, Przybyla, M, Van Hummel, A, Chua, S.W, Hoven, J.V.D, Lee, W.S, Müller, J, Parmar, J, Jonquieres, G.V, Stefen, H, Guccione, E, Fath, T, Housley, G.D, Klugmann, M, Ke, Y.D, Ittner, L.M
المساهمون: DEPT OF BIOCHEMISTRY
المصدر: Unpaywall 20201031
مصطلحات موضوعية: glutamic acid, guanosine triphosphatase activating protein, mitogen activated protein kinase, Ras protein, SynGAP1 protein, tau protein, unclassified drug, mouse, animal, brain, compartmentalization, experimental study, gene expression, inhibition, protein, stroke, toxicity, animal experiment, animal model, Article, brain damage, cerebrovascular accident, controlled study, disease exacerbation, enzyme activation, excitotoxicity, gene overexpression, middle cerebral artery occlusion, nerve cell, nonhuman
العلاقة: Bi, M, Gladbach, A, Van Eersel, J, Ittner, A, Przybyla, M, Van Hummel, A, Chua, S.W, Hoven, J.V.D, Lee, W.S, Müller, J, Parmar, J, Jonquieres, G.V, Stefen, H, Guccione, E, Fath, T, Housley, G.D, Klugmann, M, Ke, Y.D, Ittner, L.M (2017). Tau exacerbates excitotoxic brain damage in an animal model of stroke. Nature Communications 8 (1) : 473. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-017-00618-0Test; https://scholarbank.nus.edu.sg/handle/10635/178581Test
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4دورية أكاديمية
المؤلفون: Hall, A.R, Burke, N, Dongworth, R.K, Kalkhoran, S.B, Dyson, A, Vicencio, J.M, Dorn, G.W, Yellon, D.M, Hausenloy, D.J
المساهمون: DUKE-NUS MEDICAL SCHOOL
المصدر: Unpaywall 20201031
مصطلحات موضوعية: antimycin A1, calcium ion, cytochrome c oxidase, mitofusin 1, mitofusin 2, reactive oxygen metabolite, reduced nicotinamide adenine dinucleotide dehydrogenase (ubiquinone), succinate dehydrogenase (ubiquinone), ubiquinol cytochrome c reductase, calcium, carrier protein, guanosine triphosphatase, Mfn1 protein, mouse, Mfn2 protein, mitochondrial permeability transition pore, acute heart infarction, animal experiment, animal model, Article, cell organelle, confocal microscopy, controlled study, disorders of mitochondrial functions, echocardiography, fractional shortening, heart infarction size, heart left ventricle hypertrophy, heart mitochondrion, heart output
العلاقة: Hall, A.R, Burke, N, Dongworth, R.K, Kalkhoran, S.B, Dyson, A, Vicencio, J.M, Dorn, G.W, Yellon, D.M, Hausenloy, D.J (2016). Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction. Cell Death and Disease 7 (5) : e2238. ScholarBank@NUS Repository. https://doi.org/10.1038/cddis.2016.139Test; https://scholarbank.nus.edu.sg/handle/10635/180381Test
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5دورية أكاديمية
المؤلفون: Laviolette L.A., Mermoud J., Calvo I.A., Olson N., Boukhali M., Steinlein O.K., Roider E., Sattler E.C., Huang D., Teh B.T., Motamedi M., Haas W., Iliopoulos O.
المساهمون: DEAN'S OFFICE (DUKE-NUS MEDICAL SCHOOL), DUKE-NUS MEDICAL SCHOOL
المصدر: Unpaywall 20200831
مصطلحات موضوعية: afatinib, early endosome antigen 1, epidermal growth factor receptor, estrone, guanosine triphosphatase activating protein, immunoglobulin G antibody, lysosome associated membrane protein 1, protein kinase B, rab7a protein, scatter factor, scatter factor receptor, STAT3 protein, tumor suppressor protein, unclassified drug, EGFR protein, human, FLCN protein, oncoprotein, Rab protein, Rab7 protein, anatomy, cancer, cells and cell components, enzyme, enzyme activity, gene, gene expression, growth, inhibitor, ligand
العلاقة: Laviolette L.A., Mermoud J., Calvo I.A., Olson N., Boukhali M., Steinlein O.K., Roider E., Sattler E.C., Huang D., Teh B.T., Motamedi M., Haas W., Iliopoulos O. (2017). Negative regulation of EGFR signalling by the human folliculin tumour suppressor protein. Nature Communications 8 : 15866. ScholarBank@NUS Repository. https://doi.org/10.1038/ncomms15866Test; https://scholarbank.nus.edu.sg/handle/10635/174490Test
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6دورية أكاديمية
المؤلفون: Kopecki, Zlatko, O'Neill, Geraldine M., Arkell, Ruth, Cowin, Allison J
المصدر: The Journal of Investigative Dermatology
مصطلحات موضوعية: Keywords: actin, alpha actinin, fibronectin, flightless i, guanosine triphosphatase, paxillin, protein, protein p30, Rac1 protein, Rho factor, unclassified drug, animal cell, animal experiment, animal model, animal tissue, article, cell adhesion, cell level, cell m
العلاقة: http://hdl.handle.net/1885/68799Test; https://openresearch-repository.anu.edu.au/bitstream/1885/68799/7/01_Kopecki_Regulation_of_Focal_Adhesions_2011.pdf.jpgTest; https://openresearch-repository.anu.edu.au/bitstream/1885/68799/9/02_Kopecki_Regulation_of_Focal_Adhesions_2011.pdf.jpgTest
الإتاحة: https://doi.org/10.1038/jid.2011.69Test
http://hdl.handle.net/1885/68799Test
https://openresearch-repository.anu.edu.au/bitstream/1885/68799/7/01_Kopecki_Regulation_of_Focal_Adhesions_2011.pdf.jpgTest
https://openresearch-repository.anu.edu.au/bitstream/1885/68799/9/02_Kopecki_Regulation_of_Focal_Adhesions_2011.pdf.jpgTest -
7مورد إلكتروني
المصدر: The Journal of Investigative Dermatology