يعرض 1 - 9 نتائج من 9 نتيجة بحث عن '"ENDOCRINE-DISRUPTING CHEMICALS"', وقت الاستعلام: 1.19s تنقيح النتائج
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    العلاقة: Perng, Wei; Cantoral, Alejandra; Soria‐contreras, Diana C.; Betanzos‐robledo, Larissa; Kordas, Katarzyna; Liu, Yun; Mora, Ana M.; Corvalan, Camila; Pereira, Anita; Cardoso, Marly Augusto; Chavarro, Jorge E.; Breton, Carrie V.; Meeker, John D.; Harley, Kim G.; Eskenazi, Brenda; Peterson, Karen E.; Tellez‐rojo, Martha Maria (2021). "Exposure to obesogenic endocrine disrupting chemicals and obesity among youth of Latino or Hispanic origin in the United States and Latin America: A lifecourse perspective." Obesity Reviews 22: n/a-n/a.; https://hdl.handle.net/2027.42/168302Test; Obesity Reviews; Valdez R, Athens MA, Thompson GH, Bradshaw BS, Stern MP. Birthweight and adult health outcomes in a biethnic population in the USA. Diabetologia. 1994; 37 ( 6 ): 624 - 631. https://doi.org/10.1007/bf00403383Test; Buck Louis GM, Zhai S, Smarr MM, et al. Endocrine disruptors and neonatal anthropometry, NICHD Fetal Growth Studies - Singletons. Environ Int. 2018; 119: 515 - 526. https://doi.org/10.1016/j.envint.2018.07.024Test; Noor N, Ferguson KK, Meeker JD, et al. Pregnancy phthalate metabolite concentrations and infant birth weight by gradations of maternal glucose tolerance. Int J Hyg Environ Health. 2019; 222 ( 3 ): 395 - 401. https://doi.org/10.1016/j.ijheh.2018.12.005Test; Deierlein AL, Wolff MS, Pajak A, et al. Longitudinal associations of phthalate exposures during childhood and body size measurements in young girls. Epidemiology. 2016; 27 ( 4 ): 492 - 499. https://doi.org/10.1097/EDE.0000000000000489Test; Perng W, Ringham BM, Glueck DH, et al. An observational cohort study of weight- and length- derived anthropometric indicators with body composition at birth and 5- mo: the Healthy Start study. Am J Clin Nutr. 2017; 106 ( 2 ): 559 - 567. https://doi.org/10.3945/ajcn.116.149617Test; Baird J, Fisher D, Lucas P, Kleijnen J, Roberts H, Law C. Being big or growing fast: Systematic review of size and growth in infancy and later obesity. Br Med J. 2005; 331 ( 7522 ): 929 - 931. https://doi.org/10.1136/bmj.38586.411273.E0Test; Lithell HO, McKeigue PM, Berglund L, Mohsen R, Lithell UB, Leon DA. Relation of size at birth to non- insulin dependent diabetes and insulin concentrations in men aged 50- 60- years. Br Med J. 1996; 312 ( 7028 ): 406 - 410. https://doi.org/10.1136/bmj.312.7028.406Test; Hales CN, Barker DJP, Clark PMS, et al. Fetal and infant growth and impaired glucose tolerance at age 64. Br Med J. 1991; 303 ( 6809 ): 1019 - 1022. https://doi.org/10.1136/bmj.303.6809.1019Test; Eriksson JG, Forsén T, Tuomilehto J, Osmond C, Barker DJP. Early growth and coronary heart disease in later life: Longitudinal study. Br Med J. 2001; 322 ( 7292 ): 949 - 953. https://doi.org/10.1136/bmj.322.7292.949Test; Haugaard LK, Baker JL, Perng W, et al. Growth in total height and its components and cardiometabolic health in childhood. PLoS One. 2016; 11 ( 9 ): 1 - 15, e0163564. https://doi.org/10.1371/journal.pone.0163564Test; Eskenazi B, Harley K, Bradman A, et al. Association of in utero organophosphate pesticide exposure and fetal growth and length of gestation in an agricultural population. Environ Health Perspect. 2004; 112 ( 10 ): 1116 - 1124. https://doi.org/10.1289/ehp.6789Test; Centers for Disease Control and Prevention. Benzophenone- 3 (BP- 3). National Biomonitoring Program. https://www.cdc.gov/biomonitoring/BenzophenoneTest- 3_FactSheet.html. Published 2017. Accessed August 15, 2020.; Corvalan C, Garmendia ML, Jones- Smith J, et al. Nutrition status of children in Latin America. Obes Rev. 2017; 18 ( suppl 2 ): 7 - 18. https://doi.org/10.1111/obr.12571Test; Grun F, Watanabe H, Zamanian Z, et al. Endocrine- disrupting organotin compounds are potent inducers of adipogenesis in vertebrates. Mol Endocrinol. 2006; 20 ( 9 ): 2141 - 2155. https://doi.org/10.1210/me.2005Test- 0367; Newbold RR, Padilla- Banks E, Snyder RJ, Jefferson WN. Developmental exposure to estrogenic compounds and obesity. Birth Defects Res a Clin Mol Teratol. 2005; 73 ( 7 ): 478 - 480. https://doi.org/10.1002/bdra.20147Test; Ridolfi AS, à lvarez GB, Rodríguez Girault M. In: Alvarez A, Polti MA, eds. Bioremediation in Latin America: current research and perspectives. Springer International Publishing; 2014 https://doi.org/10.1007/978Test- 3- 319- 05738- 5; Boeke CE, Oken E, Kleinman KP, Rifas- Shiman SL, Taveras EM, Gillman MW. Correlations among adiposity measures in school- aged children. BMC Pediatr. 2013; 13 ( 1 ): 1 - 8. https://doi.org/10.1186/1471Test- 2431- 13- 99; Biri A, Bozkurt N, Turp A, Kavutcu M, Himmetoglu à , Durak Ä°. Role of oxidative stress in intrauterine growth restriction. Gynecol Obstet Invest. 2007; 64 ( 4 ): 187 - 192. https://doi.org/10.1159/000106488Test; Eissa MA, Dai S, Mihalopoulos NL, Day RS, Harrist RB, Labarthe DR. Trajectories of fat mass index, fat free- mass index, and waist circumference in children. Project heartBeat! Am J Prev Med. 2009; 37 ( suppl 1 ): S34 - S39. https://doi.org/10.1016/j.amepre.2009.04.005Test; Guo SS, Chumlea WC, Roche AF, Siervogel RM. Age- and maturity- related changes in body composition during adolescence into adulthood: the FELS longitudinal study. Int J Obes (Lond). 1997; 21 ( 12 ): 1167 - 1175. https://doi.org/10.1038/sj.ijo.0800531Test; Faulk C, Dolinoy DC. Timing is everything: the when and how of environmentally induced changes in the epigenome of animals. Epigenetics. 2011; 6 ( 7 ): 791 - 797. https://doi.org/10.4161/epi.6.7.16209Test; Waterland RA, Michels KB. Epigenetic epidemiology of the developmental origins hypothesis. Annu Rev Nutr. 2007; 27 ( 1 ): 363 - 388. https://doi.org/10.1146/annurev.nutr.27.061406.093705Test; Laubach ZM, Perng W, Dolinoy DC, Faulk CD, Holekamp KE, Getty T. Epigenetics and the maintenance of developmental plasticity: extending the signalling theory framework. Biol Rev. 2018; 93 ( 3 ): 1323 - 1338. https://doi.org/10.1111/brv.12396Test; Wu T, Mendola P, Buck GM. Ethnic differences in the presence of secondary sex characteristics and menarche among US girls: the Third National Health and Nutrition Examination Survey, 1988- 1994. Pediatrics. 2002; 110 ( 4 ): 752 - 757. https://doi.org/10.1542/peds.110.4.752Test; United States Consumer Product Safety Commission. Phthalates business guidance & small entity compliance guide. https://www.cpsc.gov/BusinessTest- - Manufacturing/Business- Education/Business- Guidance/Phthalates- Information. Published 2019. Accessed February 21, 2020.; Centers for Disease Control and Prevention. Fourth report on human exposure to environmental chemicals, updated tables. https://www.cdc.gov/exposurereport/pdf/FourthReport_UpdatedTables_Volume1_Jan2019Test- 508.pdf. Published 2019. Accessed February 24, 2020.; Sjödin A, Wong L- Y, Jones RS, et al. Serum concentrations of polybrominated diphenyl ethers (PBDEs) and polybrominated biphenyl (PBB) in the United States population: 2003- 2004. Environ Sci Technol. 2008; 42 ( 4 ): 1377 - 1384. https://doi.org/10.1021/es702451pTest; Nguyen VK, Kahana A, Heidt J, et al. A comprehensive analysis of racial disparities in chemical biomarker concentrations in United States women, 1999- 2014. Environ Int. 2020; 137: 105496- 1 - 105496- 14. https://doi.org/10.1016/j.envint.2020.105496Test; Carter- Pokras O, Zambrana RE, Poppell CF, Logie LA, Guerrero- Preston R. The environmental health of Latino children. J Pediatr Heal Care off Publ Natl Assoc Pediatr Nurse Assoc Pract. 2007; 21 ( 5 ): 307 - 314. https://doi.org/10.1016/j.pedhc.2006.12.005Test; Lewis RC, Meeker JD, Peterson KE, et al. Predictors of urinary bisphenol A and phthalate metabolite concentrations in Mexican children. Chemosphere. 2013; 93 ( 10 ): 2390 - 2398. https://doi.org/10.1016/j.chemosphere.2013.08.038Test; Wu H, Kupsco AJ, Deierlein AL, et al. Trends and patterns of phthalates and phthalate alternatives exposure in pregnant women from Mexico City during 2007- 2010. Environ Sci Technol. 2020; 54 ( 3 ): 1740 - 1749. https://doi.org/10.1021/acs.est.9b05836Test; Fromme H, Tittlemier SA, Volkel W, Wilhelm M, Twardella D. Perfluorinated compounds- exposure assessment for the general population in Western countries. Int J Hyg Environ Health. 2009; 212 ( 3 ): 239 - 270. https://doi.org/10.1016/j.ijheh.2008.04.007Test; Buckley JP, Kim H, Wong E, Rebholz CM. Ultra- processed food consumption and exposure to phthalates and bisphenols in the US National Health and Nutrition Examination Survey, 2013- 2014. Environ Int. 2019; 131: 105057- 1 - 105057- 24. https://doi.org/10.1016/j.envint.2019.105057Test; Bleiweiss- Sande R, Goldberg J, Evans EW, Chui K, Bailey C, Sacheck J. Chemicals, cans and factories: how grade school children think about processed foods. Public Health Nutr. 2020; 23 ( 10 ): 1735 - 1744. https://doi.org/10.1017/S1368980019003859Test; Pan American Health Organization, World Health Organization. Plan of action for the prevention of obesity in children and adolescents. https://www.paho.org/hq/dmdocuments/2015/ObesityTest- Plan- Of- Action- Child- Eng- 2015.pdf. Published 2014. Accessed May 25, 2020.; World Health Organization. Overweight and obesity. https://www.who.int/newsTest- room/fact- sheets/detail/obesity- and- overweight. Published 2020. Accessed May 24, 2020.; National Academies of Sciences, Engineering, and Medicine; Health and Medicine Division; Food and Nutrition Board. Roundtable on obesity solutions. In: Callahan EA, ed. Current Status and Response to the Global Obesity Pandemic: Proceedings of a Workshop. National Academies Press (US); 2019. https://doi.org/10.17226/25273Test; Gupta N, Goel K, Shah P, Misra A. Childhood obesity in developing countries: epidemiology, determinants, and prevention. Endocr Rev. 2012; 33 ( 1 ): 48 - 70. https://doi.org/10.1210/er.2010Test- 0028; Singh AS, Mulder C, Twisk JWR, van Mechelen W, Chinapaw MJM. Tracking of childhood overweight into adulthood: a systematic review of the literature. Obes Rev. 2008; 9 ( 5 ): 474 - 488. https://doi.org/10.1111/j.1467Test- 789X.2008.00475.x; Gordon- Larsen P, The NS, Adair LS. Longitudinal trends in obesity in the United States from adolescence to the third decade of life. Obesity (Silver Spring). 2010; 18 ( 9 ): 1801 - 1804. https://doi.org/10.1038/oby.2009.451Test; Baker JL, Olsen LW, Sørensen TIA. Childhood body- mass index and the risk of coronary heart disease in adulthood. N Engl J Med. 2007; 357 ( 23 ): 2329 - 2337. https://doi.org/10.1056/NEJMoa072515Test; U.S. Department of Health and Human Services Office of Minority Health. Obesity and Hispanic Americans. https://minorityhealth.hhs.gov/omh/browse.aspx?lvl=4%26lvlid=70Test. Published 2020. Accessed May 24, 2020.; McKeown RE. The epidemiologic transition: changing patterns of mortality and population dynamics. Am J Lifestyle Med. 2009; 3 ( suppl 1 ): S19 - S26. https://doi.org/10.1177/1559827609335350Test; Zota AR, Phillips CA, Mitro SD. Recent fast food consumption and bisphenol A and phthalates exposures among the U.S. population in NHANES, 2003- 2010. Environ Health Perspect. 2016; 124 ( 10 ): 1521 - 1528. https://doi.org/10.1289/ehp.1510803Test; Rivera JA, Barquera S, González- Cossío T, Olaiz G, Sepúlveda J. Nutrition transition in Mexico and in other Latin American countries. Nutr Rev. 2004; 62 ( suppl 2 ): S149 - S157. https://doi.org/10.1111/j.1753Test- 4887.2004.tb00086.x; Popkin BM, Reardon T. Obesity and the food system transformation in Latin America. Obes Rev. 2018; 19 ( 8 ): 1028 - 1064. https://doi.org/10.1111/obr.12694Test; Comuzzie AG, Cole SA, Laston SL, et al. Novel genetic loci identified for the pathophysiology of childhood obesity in the Hispanic population. PLoS One. 2012; 7 ( 12 ): 1 - 9, e51954. https://doi.org/10.1371/journal.pone.0051954Test; Lanigan J. Prevention of overweight and obesity in early life. Proc Nutr Soc. 2018; 77 ( 3 ): 247 - 256. https://doi.org/10.1017/S0029665118000411Test; Baillie- Hamilton PF. Chemical toxins: a hypothesis to explain the global obesity epidemic. J Altern Complement Med. 2002; 8 ( 2 ): 185 - 192. https://doi.org/10.1089/107555302317371479Test; Grun F, Blumberg B. Environmental obesogens: organotins and endocrine disruption via nuclear receptor signaling. Endocrinology. 2006; 147 ( suppl 6 ): S50 - S55. https://doi.org/10.1210/en.2005Test- 1129; Muscogiuri G, Barrea L, Laudisio D, Savastano S, Colao A. Obesogenic endocrine disruptors and obesity: myths and truths. Arch Toxicol. 2017; 91 ( 11 ): 3469 - 3475. https://doi.org/10.1007/s00204Test- 017- 2071- 1; United Nations Development Programme. Energy & environment practice gender mainstreaming guidance series chemicals management. Chemicals and Gender. 2011; 1 - 28. https://www.undp.org/content/undp/en/home/librarypage/environmentTest- energy/chemicals_management/chemicals- and- gender.html. Published 2011. Accessed February 24, 2020.; Janesick A, Blumberg B. Endocrine disrupting chemicals and the developmental programming of adipogenesis and obesity. Birth Defects Res C Embryo Today. 2011; 93 ( 1 ): 34 - 50. https://doi.org/10.1002/bdrc.20197Test; Diamanti- Kandarakis E, Bourguignon JP, Giudice LC, et al. Endocrine- disrupting chemicals: an Endocrine Society scientific statement. Endocr Rev. 2009; 30 ( 4 ): 293 - 342. https://doi.org/10.1210/er.2009Test- 0002; Grun F, Blumberg B. Endocrine disrupters as obesogens. Mol Cell Endocrinol. 2009; 304 ( 1- 2 ): 19 - 29. https://doi.org/10.1016/j.mce.2009.02.018Test; Janesick A, Blumberg B. Minireview: PPARgamma as the target of obesogens. J Steroid Biochem Mol Biol. 2011; 127 ( 1- 2 ): 4 - 8. https://doi.org/10.1016/j.jsbmb.2011.01.005Test; Main KM, Mortensen GK, Kaleva MM, et al. Human breast milk contamination with phthalates and alterations of endogenous reproductive hormones in infants three months of age. Environ Health Perspect. 2006; 114 ( 2 ): 270 - 276. https://doi.org/10.1289/ehp.8075Test; Pan G, Hanaoka T, Yoshimura M, et al. Decreased serum free testosterone in workers exposed to high levels of di- n- butyl phthalate (DBP) and di- 2- ethylhexyl phthalate (DEHP): a cross- sectional study in China. Environ Health Perspect. 2006; 114 ( 11 ): 1643 - 1648. https://doi.org/10.1289/ehp.9016Test; Swan SH. Environmental phthalate exposure in relation to reproductive outcomes and other health endpoints in humans. Environ Res. 2008; 108 ( 2 ): 177 - 184. https://doi.org/10.1016/j.envres.2008.08.007Test; Talsness CE, Kuriyama SN, Sterner- Kock A, et al. In utero and lactational exposures to low doses of polybrominated diphenyl ether- 47 alter the reproductive system and thyroid gland of female rat offspring. Environ Health Perspect. 2008; 116 ( 3 ): 308 - 314. https://doi.org/10.1289/ehp.10536Test; Du G, Hu J, Huang H, et al. Perfluorooctane sulfonate (PFOS) affects hormone receptor activity, steroidogenesis, and expression of endocrine- related genes in vitro and in vivo. Environ Toxicol Chem. 2013; 32 ( 2 ): 353 - 360. https://doi.org/10.1002/etc.2034Test; Ronn M, Lind L, Orberg J, et al. Bisphenol A is related to circulating levels of adiponectin, leptin and ghrelin, but not to fat mass or fat distribution in humans. Chemosphere. 2014; 112: 42 - 48. https://doi.org/10.1016/j.chemosphere.2014.03.042Test; Darbre PD. Endocrine disruptors and obesity. Curr Obes Rep. 2017; 6 ( 1 ): 18 - 27. https://doi.org/10.1007/s13679Test- 017- 0240- 4; Nappi F, Barrea L, Di Somma C, et al. Endocrine aspects of environmental - obesogen- pollutants. Int J Environ Res Public Health. 2016; 13 ( 8 ): 765- 1 - 765- 16. https://doi.org/10.3390/ijerph13080765Test; Liu Y, Peterson KE. Maternal exposure to synthetic chemicals and obesity in the offspring: recent findings. Curr Environ Heal Reports. 2015; 2 ( 4 ): 339 - 347. https://doi.org/10.1007/s40572Test- 015- 0068- 6; Mudipalli A. Lead hepatotoxicity & potential health effects. Indian J Med Res. 2007; 126 ( 6 ): 518 - 527.; Cupul- Uicab LA, Hernandez- Avila M, Terrazas- Medina EA, Pennell ML, Longnecker MP. Prenatal exposure to the major DDT metabolite 1,1- dichloro- 2,2- bis(p- chlorophenyl)ethylene (DDE) and growth in boys from Mexico. Environ Res. 2010; 110 ( 6 ): 595 - 603. https://doi.org/10.1016/j.envres.2010.06.001Test; Garced S, Torres- Sanchez L, Cebrian ME, Claudio L, Lopez- Carrillo L. Prenatal dichlorodiphenyldichloroethylene (DDE) exposure and child growth during the first year of life. Environ Res. 2012; 113: 58 - 62. https://doi.org/10.1016/j.envres.2011.12.002Test; Arrebola JP, Cuellar M, Bonde JP, Gonzalez- Alzaga B, Mercado LA. Associations of maternal o,p- ²- DDT and p,p- ²- DDE levels with birth outcomes in a Bolivian cohort. Environ Res. 2016; 151: 469 - 477. https://doi.org/10.1016/j.envres.2016.08.008Test; Harley KG, Huen K, Aguilar Schall R, et al. Association of organophosphate pesticide exposure and paraoxonase with birth outcome in Mexican- American women. PLoS One. 2011; 6 ( 8 ): 1 - 10, e23923. https://doi.org/10.1371/journal.pone.0023923Test; Harley KG, Chevrier J, Aguilar Schall R, Sjodin A, Bradman A, Eskenazi B. Association of prenatal exposure to polybrominated diphenyl ethers and infant birth weight. Am J Epidemiol. 2011; 174 ( 8 ): 885 - 892. https://doi.org/10.1093/aje/kwr212Test; Warner M, Aguilar Schall R, Harley KG, Bradman A, Barr D, Eskenazi B. In utero DDT and DDE exposure and obesity status of 7- year- old Mexican- American children in the CHAMACOS cohort. Environ Health Perspect. 2013; 121 ( 5 ): 631 - 636. https://doi.org/10.1289/ehp.1205656Test; Warner M, Wesselink A, Harley KG, Bradman A, Kogut K, Eskenazi B. Prenatal exposure to dichlorodiphenyltrichloroethane and obesity at 9- years of age in the CHAMACOS study cohort. Am J Epidemiol. 2014; 179 ( 11 ): 1312 - 1322. https://doi.org/10.1093/aje/kwu046Test; Heggeseth B, Harley K, Warner M, Jewell N, Eskenazi B. Detecting associations between early- life DDT exposures and childhood growth patterns: a novel statistical approach. PLoS One. 2015; 10 ( 6 ): 1 - 13, e0131443. https://doi.org/10.1371/journal.pone.0131443Test; Erkin- Cakmak A, Harley KG, Chevrier J, et al. In utero and childhood polybrominated diphenyl ether exposures and body mass at age 7- years: the CHAMACOS study. Environ Health Perspect. 2015; 123 ( 6 ): 636 - 642. https://doi.org/10.1289/ehp.1408417Test; Warner M, Ye M, Harley K, Kogut K, Bradman A, Eskenazi B. Prenatal DDT exposure and child adiposity at age 12: The CHAMACOS study. Environ Res. 2017; 159: 606 - 612. https://doi.org/10.1016/j.envres.2017.08.050Test; Harley KG, Berger K, Rauch S, et al. Association of prenatal urinary phthalate metabolite concentrations and childhood BMI and obesity. Pediatr Res. 2017; 82 ( 3 ): 405 - 415. https://doi.org/10.1038/pr.2017.112Test; Heggeseth BC, Holland N, Eskenazi B, Kogut K, Harley KG. Heterogeneity in childhood body mass trajectories in relation to prenatal phthalate exposure. Environ Res. 2019; 175: 22 - 33. https://doi.org/10.1016/j.envres.2019.04.036Test; Harley KG, Aguilar Schall R, Chevrier J, et al. Prenatal and postnatal bisphenol A exposure and body mass index in childhood in the CHAMACOS cohort. Environ Health Perspect. 2013; 121 ( 4 ): 514 - 520. https://doi.org/10.1289/ehp.1205548Test; Yang TC, Peterson KE, Meeker JD, et al. Exposure to bisphenol A and phthalates metabolites in the third trimester of pregnancy and BMI trajectories. Pediatr Obes. 2018; 13 ( 9 ): 550 - 557. https://doi.org/10.1111/ijpo.12279Test; Yang TC, Peterson KE, Meeker JD, et al. Bisphenol A and phthalates in utero and in childhood: association with child BMI z- score and adiposity. Environ Res. 2017; 156: 326 - 333. https://doi.org/10.1016/j.envres.2017.03.038Test; Bowman A, Peterson KE, Dolinoy DC, et al. Phthalate exposures, DNA methylation and adiposity in Mexican children through adolescence. Front Public Health. 2019; 7: 162- 1 - 162- 17. https://doi.org/10.3389/fpubh.2019.00162Test; Maresca MM, Hoepner LA, Hassoun A, et al. Prenatal exposure to phthalates and childhood body size in an urban cohort. Environ Health Perspect. 2016; 124 ( 4 ): 514 - 520. https://doi.org/10.1289/ehp.1408750Test; Buckley JP, Engel SM, Mendez MA, et al. Prenatal phthalate exposures and childhood fat mass in a New York City cohort. Environ Health Perspect. 2016; 124 ( 4 ): 507 - 513. https://doi.org/10.1289/ehp.1509788Test; Buckley JP, Herring AH, Wolff MS, Calafat AM, Engel SM. Prenatal exposure to environmental phenols and childhood fat mass in the Mount Sinai Children’s Environmental Health Study. Environ Int. 2016; 91: 350 - 356. https://doi.org/10.1016/j.envint.2016.03.019Test; Starling AP, Adgate JL, Hamman RF, et al. Perfluoroalkyl substances during pregnancy and offspring weight and adiposity at birth: examining mediation by maternal fasting glucose in the Healthy Start Study. Environ Health Perspect. 2017; 125 ( 6 ):067016- 1- 067016- 9. https://doi.org/10.1289/EHP641Test; Starling AP, Adgate JL, Hamman RF, Kechris K, Calafat AM, Dabelea D. Prenatal exposure to per- and polyfluoroalkyl substances and infant growth and adiposity: the Healthy Start Study. Environ Int. 2019; 131: 104983- 1 - 104983- 16. https://doi.org/10.1016/j.envint.2019.104983Test

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    العلاقة: Padmanabhan, V.; Sarma, H. N.; Savabieasfahani, M.; Steckler, T. L.; Veiga-Lopez, A.; (2010). "Developmental reprogramming of reproductive and metabolic dysfunction in sheep: native steroids vs. environmental steroid receptor modulators." International Journal of Andrology 33(2 Proceedings of the 5th Copenhagen Workshop on Endocrine Disrupters, 20-22 May 2009 ): 394-404.; https://hdl.handle.net/2027.42/78725Test; International Journal of Andrology

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