المؤلفون: Thomas Jespersen, Jau-Nian Chen, Katarzyna Kulej, Raffaela Torggler, Martin R. Larsen, Jonas M. la Cour, Triantafyllos Zacharias, Kevin Wang, Martin W. Berchtold

المصدر: Berchtold, M W, Zacharias, T, Kulej, K, Wang, K, Torggler, R, Jespersen, T, Chen, J-N, Larsen, M R & la Cour, J M 2016, ' The arrhythmogenic calmodulin mutation D129G dysregulates cell growth, calmodulin-dependent kinase II activity, and cardiac function in zebrafish ', Journal of Biological Chemistry, vol. 291, no. 52, pp. 26636-26646 . https://doi.org/10.1074/jbc.M116.758680Test
The Journal of biological chemistry, vol 291, iss 52

مصطلحات موضوعية: 0301 basic medicine, calmodulin, Protein Conformation, Mutant, heart failure, Arrhythmias, Cardiovascular, medicine.disease_cause, Medical and Health Sciences, Biochemistry, Tachycardia, Myocytes, Cardiac, Phosphorylation, Zebrafish, Cells, Cultured, Mutation, Cultured, catecholaminergic polymorphic ventricular tachycardia, Kinase, Autophosphorylation, Biological Sciences, Cell biology, Long QT Syndrome, Heart Disease, Cardiac, Biochemistry & Molecular Biology, medicine.medical_specialty, animal structures, Calmodulin, Cells, Biology, 03 medical and health sciences, Internal medicine, medicine, cell signaling, Animals, Humans, Viability assay, Molecular Biology, Cell Proliferation, Myocytes, calcium, Cell growth, Ventricular, DT40, Arrhythmias, Cardiac, Cell Biology, biology.organism_classification, 030104 developmental biology, Endocrinology, Chemical Sciences, biology.protein, Tachycardia, Ventricular, Calcium, Ca2+/calmodulin-dependent protein kinase II, Calcium-Calmodulin-Dependent Protein Kinase Type 2

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4450c2b0ac980a35a6b31621121d1a37Test
https://pubmed.ncbi.nlm.nih.gov/27815504Test

المؤلفون: Paola Vagnarelli, William C. Earnshaw, Hiromi Ogawa, Zhenjie Xu, Kumiko Samejima

المصدر: The Journal of Biological Chemistry
Xu, Z, Vagnarelli, P, Ogawa, H, Samejima, K & Earnshaw, W C 2010, ' Gradient of Increasing Aurora B Kinase Activity Is Required for Cells to Execute Mitosis ', Journal of Biological Chemistry, vol. 285, no. 51, pp. 40163-40170 . https://doi.org/10.1074/jbc.M110.181545Test

مصطلحات موضوعية: INCENP, Chromosomal Proteins, Non-Histone, Amino Acid Motifs, Aurora inhibitor, Aurora B kinase, Mitosis, Cell Cycle Proteins, Spindle Apparatus, Biology, Protein Serine-Threonine Kinases, Biochemistry, Microtubules, Spindle elongation, Cell Line, Avian Proteins, Gene Knockout, 03 medical and health sciences, 0302 clinical medicine, Aurora Kinases, Animals, Molecular Biology, 030304 developmental biology, Cytokinesis, 0303 health sciences, Chromosome Passenger Complex, Cell Cycle, DT40, Cell Biology, Molecular biology, Cell biology, Protein Structure, Tertiary, Spindle checkpoint, Chromosome passenger complex, Mutation, Chickens, Microtubule-Associated Proteins, 030217 neurology & neurosurgery, Cell Division, Aurora B Kinase

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7a73a9c8625685fd5b658656aae7cb3Test
https://pubmed.ncbi.nlm.nih.gov/20956539Test