نتائج البحث - "Interleukin 4"

1

The elastin peptide VGVAPG increases CD4+ T-cell IL-4 production in patients with chronic obstructive pulmonary disease

المؤلفون: Julien Ancel, Jeanne-Marie Perotin, Pierre Gaudry, Gaëtan Deslée, Richard Le Naour, Flora Lemaire, Frank Antonicelli, Sandra Dury, Sandra Audonnet, François Lebargy

المساهمون: Pathologies Pulmonaires et Plasticité Cellulaire - UMR-S 1250 (P3CELL), Université de Reims Champagne-Ardenne (URCA)-Institut National de la Santé et de la Recherche Médicale (INSERM), Centre Hospitalier Universitaire de Reims (CHU Reims)

المصدر: Respiratory Research, Vol 22, Iss 1, Pp 1-11 (2021)
Respiratory Research
Respiratory Research, BioMed Central, 2021, 22 (1), ⟨10.1186/s12931-020-01609-4⟩

مصطلحات موضوعية: Adult, CD4-Positive T-Lymphocytes, Male, medicine.medical_specialty, [SDV]Life Sciences [q-bio], T cells, Peripheral blood mononuclear cell, Flow cytometry, Pulmonary Disease, Chronic Obstructive, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Internal medicine, medicine, Humans, COPD, Receptor, Cells, Cultured, Interleukin 4, Aged, 030304 developmental biology, lcsh:RC705-779, 0303 health sciences, biology, medicine.diagnostic_test, business.industry, Research, IL-4, lcsh:Diseases of the respiratory system, Middle Aged, medicine.disease, Desmosine, Endocrinology, 030228 respiratory system, chemistry, Leukocytes, Mononuclear, biology.protein, Cytokines, Female, Interleukin-4, Elastin peptides, business, Oligopeptides, Elastin, CD8

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::041aa9e7a2083bcd00839c25446e0b41Test
https://doaj.org/article/94d7a278c11548dbbed63fbcd45b8770Test

2

Validated and longitudinally stable asthma phenotypes based on cluster analysis of the ADEPT study

المؤلفون: Loza, M. J., Djukanovic, R., Chung, K. F., Horowitz, D., Ma, K., Branigan, P., Barnathan, E. S., Susulic, V. S., Silkoff, P. E., Sterk, P. J., Baribaud, F., Strambu, I., Laviolette, M., Singh, D., FitzGerald, J. M., Lam, S., Kelsen, S., Eich, A., Ludwig-Sengpiel, A., hupp, G.C., Backer, V., Porsbjerg, C., Girodet, P. O., Berger, P., Leigh, R., Kline, J., Dransfield, M., Calhoun, W., Hussaini, A., Khatri, S., Chanez, P., Ian, A., Fleming Louis, J., David, G., Sile, H., Scott, K., Sally, M., Andrea, M., Stelios, P., Christos, R., Kirsty, R., Kai, S., Coen, W., Xian, Y., Nora, A., Ariane, W., Kees, v.D., Marianne, v.d.P., Wim, v.A., Sterk Peter, J., Barbara, S., Lara, R., Rene, L., Paul, B., Elisabeth, B., Koos, Z., Tamara, D., Simone, H., Annemiek, D., Pieter-Paul, H., Saeeda, L. -L, Hassan, A., Betrand, D. M., Diane, L., Antonios, A., Kjell, A., Charles, A., Philipp, B., Per, B., David, B., Sven-Erik, D., Ingrid, D., Cristina, G., James Anna, J., Roelinde, M., Shama, N., Anne, P., Stacey, R., Wheelock Craig, E., Hector, G., Maciej, K., Johan, K., Marcus, S., Bansal Aruna, T., Frédéric, B., Navin, R., An, B., Inge, D. L., Martine, R., Behndig Annelie, F., Thomas, S., Jorge, B., De Jorge, A., Ann, B., Gunilla, H., Björn, Nordlund, Jon, K., Wilhelm, Z., Alix, B., Jörgen, Ö., van Marleen, G., de Maria, G. V., Lars, L., Ulf, N., Jeannette, B., Boedigheimer Michel, J., Richard, H., Xugang, H., Wen, Y., Hans, B., Klaus, B., Jonathan, T., Nadja, V., Grazyna, B., Jacek, M., Joost, B., Ben, N., Anthony, P., Doroteya, S., Armin, B., Jens, H., Norbert, K., Dominic, B., Schofield James, P. R., Skipp Paul, J., Leon, C., Bob, T., Massimo, C., Rocha João Pedro, C. P., Julaiha, G., Andrew, M. -G, Adesimbo, S., Amphun, C., Romanas, C., Caroline, M., Pascal, C., Courtney, C., Jessica, E., Val, H., Kennington Erika, J., Leanne, M., Malayka, R. -A, Leanne, R., Jessica, S., Jenny, V., Samantha, W., Breda, F., Amanda, R., David, S., Chris, C., David, M., John, R., Sousa Ana, R., Julie, C., Arnaldo, D'Amico, Giorgio, P., Marco, S., Barbro, D., Ann-Sofie, L., Pim, B., Patrick, D., Kamran, T., Clair, B., Kerry, G., Aleksandra, D., Neil, F., Trevor, G., Scott, W., Rosalia, E., Davide, C., Magnus, E., Veit, E., Damijan, E., Klaus, F., Katja, N., Corinna, S., Frans, W., Kathrin, R., Matthias, Klüglich, Fowler Stephen, J., Murray Clare, S., Jorgen, V., Ashley, W., Urs, F., Martina, G., Gabriella, G., Ildiko, H., Marton, S., Lilla, T., Zsoka, W., Thomas, G., Neil, G., Yi-ke, G., John, H., Siân, W., Elisabeth, H., Nikos, L., Karin, S., Lorraine, H., Lisa, M., Jane, M., Sandy, P., Emma, R., Caroline, S., Tim, H., Uruj, H., Cecile, H., Matthews John, G., Peter, H., Graham, R., Juliette, K., Dyson, K., Hugo, K., Anton, V., Richard, K., Alan, K., Shaw Dominick, E., Maxim, K., Linn, K., Bart, L., Sarah, M., Pippa, P., Alexander, M., Mikus, Maria, Nilsson, Peter, Montse, M., Philip, M., Paolo, M., Nadia, M., Giuseppe, S., Salvatore, V., Antonio, P., Laurie, P., Susanna, P., Ioannis, P., Anthony, R., Wolfgang, S., Kristiane, W., Florian, S., Smith Katherine, M., Päivi, S., John-Olof, T., von Christophe, G., Jonathan, W., Wilson Susan, J., Elizabeth, Y., Therapeutics), For the ADEPT (Airways Disease Endotyping for Personalized, investigators, U-BIOPRED (Unbiased Biomarkers for the Prediction of Respiratory Disease Outcome Consortium)

المصدر: Respiratory Research. 17(1)

مصطلحات موضوعية: Biological markers, Cluster analysis, Observational study, biological marker, interleukin 13, interleukin 4, autacoid, IL4 protein, human, interleukin-13, adult, airway obstruction, Article, asthma, bronchus biopsy, controlled study, disease classification, disease severity, female, gene expression, lung function, major clinical study, male, phenotype, respiratory tract disease assessment, Th2 cell, classification, cross-sectional study, forced expiratory volume, fuzzy logic, gene expression regulation, genetics, genotype, immunology, longitudinal study, lung, pathophysiology, predictive value, prognosis, reproducibility, severity of illness index, time factor, validation study, vital capacity, Cross-Sectional Studies, Humans, Inflammation Mediators, Interleukin-13, Interleukin-4, Longitudinal Studies, Predictive Value of Tests, Reproducibility of Results, Th2 Cells, Time Factors

وصف الملف: print

الوصول الحر: https://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-280494Test
https://doi.org/10.1186/s12931-016-0482-9Test

View record in SwePub

3

Loss of the adhesion G-protein coupled receptor ADGRF5 in mice induces airway inflammation and the expression of CCL2 in lung endothelial cells

المؤلفون: Fumimasa Kubo, Taku Fukuzawa, Tomoya Sato, Rahmaningsih Mara Sabirin, Ryotaro Demizu, Nobuhiro Nakamura, Shigehisa Hirose, Donna Maretta Ariestanti, Souta Oki

المصدر: Respiratory Research
Respiratory Research, Vol 20, Iss 1, Pp 1-21 (2019)

مصطلحات موضوعية: 0301 basic medicine, Gene Expression, Inflammation, Receptors, G-Protein-Coupled, Type 2 immune response, Mice, 03 medical and health sciences, Mucous cell metaplasia, 0302 clinical medicine, Endothelial cell, medicine, COPD, Animals, Humans, Bronchitis, Lung, Type 2 cytokine, Chemokine CCL2, Interleukin 4, lcsh:RC705-779, Mice, Knockout, Ig-Hepta, medicine.diagnostic_test, Chemistry, Research, Endothelial Cells, lcsh:Diseases of the respiratory system, Adhesion GPCR, respiratory system, Mast cell, Fibrosis, Molecular biology, Mucus, Asthma, Mice, Inbred C57BL, Endothelial stem cell, 030104 developmental biology, Bronchoalveolar lavage, medicine.anatomical_structure, 030228 respiratory system, GPR116, Inflammation Mediators, medicine.symptom

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6a752b0e14c309cac084503edf5d49eTest
https://doi.org/10.1186/s12931-019-0973-6Test

4

IL-4-induced caveolin-1-containing lipid rafts aggregation contributes to MUC5AC synthesis in bronchial epithelial cells

المؤلفون: Fan Yu, Xiang-Ping Yang, Peng-Cheng Cai, Wan-Li Ma, Liang Xiong, Xin-Liang He, Feng Chen, Yu Xia, Shan-Shan Rao, Hong Ye

المصدر: Respiratory Research
Respiratory Research, Vol 18, Iss 1, Pp 1-11 (2017)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::47caa3a26b99ad506f73413029600652Test
https://doi.org/10.1186/s12931-017-0657-zTest

5

The cationic amino acid transporter 2 is induced in inflammatory lung models and regulates lung fibrosis

المؤلفون: Kathryn A Niese, Marc E. Rothenberg, Nives Zimmermann, Lesley G. Ellies, Monica G. Chiaramonte

المصدر: Respiratory Research, Vol 11, Iss 1, p 87 (2010)
Respiratory Research
Niese, Kathryn A; Chiaramonte, Monica G; Ellies, Lesley G; Rothenberg, Marc E; & Zimmermann, Nives. (2010). The cationic amino acid transporter 2 is induced in inflammatory lung models and regulates lung fibrosis. Respiratory Research, 11(1), 87. doi: http://dx.doi.org/10.1186/1465-9921-11-87Test. Retrieved from: http://www.escholarship.org/uc/item/08r3r3gmTest

مصطلحات موضوعية: Pulmonary and Respiratory Medicine, Arginine, Ovalbumin, Pulmonary Fibrosis, Inflammation, Mice, Transgenic, Biology, 03 medical and health sciences, chemistry.chemical_compound, Bleomycin, Mice, 0302 clinical medicine, Pulmonary fibrosis, medicine, Animals, Amino acid transporter, RNA, Messenger, Lung, Interleukin 4, 030304 developmental biology, Mice, Knockout, lcsh:RC705-779, 0303 health sciences, Arginine transport, Arginase, Macrophages, Research, lcsh:Diseases of the respiratory system, Ornithine, respiratory system, medicine.disease, Molecular biology, Asthma, 3. Good health, respiratory tract diseases, Up-Regulation, Mice, Inbred C57BL, Disease Models, Animal, chemistry, Amino Acid Transport Systems, Basic, Cytokines, Collagen, Interleukin-4, medicine.symptom, Inflammation Mediators, STAT6 Transcription Factor, 030215 immunology