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Inhibited autophagy may contribute to heme toxicity in cardiomyoblast cells

التفاصيل البيبلوغرافية
العنوان: Inhibited autophagy may contribute to heme toxicity in cardiomyoblast cells
المؤلفون: Alexandra, Gyongyosi, Kitti, Szoke, Ferenc, Fenyvesi, Zsolt, Fejes, Ildiko Beke, Debreceni, Bela, Nagy, Arpad, Tosaki, Istvan, Lekli
المصدر: Biochemical and biophysical research communications. 511(4)
سنة النشر: 2019
مصطلحات موضوعية: Oxidative Stress, Cell Survival, Autophagy, Animals, Hemin, Protoporphyrins, Reactive Oxygen Species, Heme Oxygenase-1, Myoblasts, Cardiac, Rats
الوصف: Several groups have demonstrated that induction of heme-oxygenase-1 (HO-1) could protect the myocardium against ischemic events; however, heme accumulation could lead to toxicity. The aim of the present study was to investigate the role of autophagy in heme toxicity. H9c2 cardiomyoblast cells were treated with different dose of hemin or cobalt-protoporphyrin IX (CoPP
تدمد: 1090-2104
الوصول الحر: https://explore.openaire.eu/search/publication?articleId=pmid________::1179d4442a491b3706064a9ef58bf91dTest
https://pubmed.ncbi.nlm.nih.gov/30833080Test
رقم الانضمام: edsair.pmid..........1179d4442a491b3706064a9ef58bf91d
قاعدة البيانات: OpenAIRE
الوصف
تدمد:10902104