يعرض 1 - 3 نتائج من 3 نتيجة بحث عن '"Axonal Transport"', وقت الاستعلام: 0.72s تنقيح النتائج
  1. 1
    دورية أكاديمية
    Normal levels of KIF5 but reduced KLC1 levels in both Alzheimer disease and Alzheimer disease in Down syndrome: evidence suggesting defects in anterograde transport.

    المؤلفون: Chen, Xu-Qiao, Das, Utpal, Park, Gooho, Mobley, William C

    المصدر: Alzheimer's research & therapy. 13(1)

    مصطلحات موضوعية: APP, Alzheimer’s disease, Axonal transport, Down syndrome, Dp16, KIF5, KLC1, Alzheimer’, s disease, Medical and Health Sciences

    وصف الملف: application/pdf

    الوصول الحر: https://escholarship.org/uc/item/6s75n7kkTest

    View record in eScholarship

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  2. 2
    دورية أكاديمية
    Normal levels of KIF5 but reduced KLC1 levels in both Alzheimer disease and Alzheimer disease in Down syndrome: evidence suggesting defects in anterograde transport

    المؤلفون: Xu-Qiao Chen, Utpal Das, Gooho Park, William C. Mobley

    المصدر: Alzheimer’s Research & Therapy, Vol 13, Iss 1, Pp 1-20 (2021)

    مصطلحات موضوعية: Alzheimer’s disease, Down syndrome, Axonal transport, KIF5, APP, Dp16, Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571, Neurology. Diseases of the nervous system, RC346-429

    وصف الملف: electronic resource

    العلاقة: https://doaj.org/toc/1758-9193Test

    الوصول الحر: https://doaj.org/article/b1d6b1a81e6c469a96beb036522311a7Test

    View record in DOAJ النص الكامل

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  3. 3
    Normal levels of KIF5 but reduced KLC1 levels in both Alzheimer disease and Alzheimer disease in Down syndrome: evidence suggesting defects in anterograde transport

    المؤلفون: Utpal Das, Xu-Qiao Chen, Gooho Park, William C. Mobley

    المصدر: Alzheimer’s Research & Therapy, Vol 13, Iss 1, Pp 1-20 (2021)
    Alzheimer's research & therapy, vol 13, iss 1
    Alzheimer's Research & Therapy

    مصطلحات موضوعية: 0301 basic medicine, Cerebellum, Aging, Down syndrome, Kinesins, Neurodegenerative, Alzheimer's Disease, Medical and Health Sciences, lcsh:RC346-429, Mice, Amyloid beta-Protein Precursor, 0302 clinical medicine, Amyloid precursor protein, 2.1 Biological and endogenous factors, Aetiology, Axonal transport, Temporal cortex, education.field_of_study, biology, Dp16, medicine.anatomical_structure, Neurology, Kinesin light chain 1, Neurological, Alzheimer's disease, Microtubule-Associated Proteins, Alzheimer’s disease, medicine.medical_specialty, Cognitive Neuroscience, Intellectual and Developmental Disabilities (IDD), 1.1 Normal biological development and functioning, KLC1, lcsh:RC321-571, s disease, 03 medical and health sciences, Alzheimer Disease, Underpinning research, Internal medicine, medicine, Acquired Cognitive Impairment, Dementia, Animals, education, Alzheimer’, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, lcsh:Neurology. Diseases of the nervous system, KIF5, Research, Neurosciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), medicine.disease, Brain Disorders, 030104 developmental biology, Endocrinology, biology.protein, Axoplasmic transport, Neurology (clinical), APP, 030217 neurology & neurosurgery

    وصف الملف: application/pdf

    الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7f725823a490cb09dea95e5d2c2eff33Test
    https://doaj.org/article/b1d6b1a81e6c469a96beb036522311a7Test


    أضف إلى المفضلة
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