نتائج البحث - "Shohreh Issazadeh Navikas"

1

Effects of a single dose of psilocybin on behaviour, brain 5-HT2A receptor occupancy and gene expression in the pig

المؤلفون: Nídia Fernandez Ros, Elham Jaberi, Brice Ozenne, Kristian Linnet, Shohreh Issazadeh-Navikas, Hanne D. Hansen, Lene Lundgaard Donovan, Sys Stybe Johansen, Gitte M. Knudsen, Jens Vilstrup Johansen

المصدر: European Neuropsychopharmacology. 42:1-11

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::18c2f95bf1d689d8f0b6c949fecd73e9Test
https://doi.org/10.1016/j.euroneuro.2020.11.013Test

2

CHST6 mutations identified in Iranian MCD patients and CHST6 mutations reported worldwide identify targets for gene editing approaches including the CRISPR/Cas system

المؤلفون: Alireza Baradaran-Rafii, Elahe Elahi, Iman Safari, Shohreh Issazadeh-Navikas

المصدر: International Ophthalmology. 40:2223-2235

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0208dd9c1c96aa059dd10a1123fb827fTest
https://doi.org/10.1007/s10792-020-01401-9Test

3

Neuronal TNFα, Not α-Syn, Underlies PDD-Like Disease Progression in IFNβ-KO Mice

المؤلفون: Emilie Tresse, Joao N. Duarte, Gisela Jimenez-Duran, Shohreh Issazadeh-Navikas, Oliver Kretz, Desiree Loreth, Tobias Goldmann, Yawei Liu, Erika B Villanueva, Marco Prinz, Patrick Ejlerskov

المصدر: Villanueva, E B, Tresse, E, Liu, Y, Duarte, J N, Jimenez-Duran, G, Ejlerskov, P, Kretz, O, Loreth, D, Goldmann, T, Prinz, M & Issazadeh-Navikas, S 2021, ' Neuronal TNFα, not α-syn, underlies PDD-like disease progression in IFNβ-KO mice ', Annals of Neurology, vol. 90, no. 5, pp. 789-807 . https://doi.org/10.1002/ana.26209Test

مصطلحات موضوعية: Faculty of Health and Medical Sciences, Lewy Body Disease, Amyloid beta, alpha-synuclein, Plaque, Amyloid, Neuropathology, phosphorylated tau, Parkinson's disease dementia, Inclusion bodies, neuroinflammation, Pathogenesis, chemistry.chemical_compound, Medicine, Animals, interferon-beta, Neuroinflammation, Alpha-synuclein, Mice, Knockout, Neurons, tumor necrosis factor alpha, Lewy body, biology, business.industry, Tumor Necrosis Factor-alpha, Neurodegeneration, Parkinson Disease, medicine.disease, amyloid-beta, nervous system diseases, nervous system, Neurology, chemistry, Neuroinflammatory Diseases, Cancer research, biology.protein, Disease Progression, Lewy Bodies, Neurology (clinical), business, Neuroglia

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37b55ccc20235ff8195895b406f982c2Test
https://pubmed.ncbi.nlm.nih.gov/34476836Test

4

IFN‐β rescues neurodegeneration by regulating mitochondrial fission via STAT5, PGAM5, and Drp1

المؤلفون: Elham Jaberi, Lluís Riera-Ponsati, Wei Qi Guinevere Sew, Shohreh Issazadeh-Navikas, Karsten Ruscher, Emilie Tresse

المصدر: EMBO J

مصطلحات موضوعية: Dynamins, Programmed cell death, Formins, Mitochondrion, Biology, medicine.disease_cause, Mitochondrial Dynamics, Neuroprotection, General Biochemistry, Genetics and Molecular Biology, Cell Line, Mice, 03 medical and health sciences, 0302 clinical medicine, Cell Line, Tumor, Phosphoprotein Phosphatases, STAT5 Transcription Factor, medicine, Animals, Molecular Biology, 030304 developmental biology, Neurons, 0303 health sciences, General Immunology and Microbiology, General Neuroscience, Neurodegeneration, Parkinson Disease, Interferon-beta, Articles, medicine.disease, Mitochondria, Cell biology, INF2, Phosphorylation, Mitochondrial fission, 030217 neurology & neurosurgery, Oxidative stress

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d14b6fd4caf9e5619339c122ef47fc3Test
https://doi.org/10.15252/embj.2020106868Test

5

PIAS2-mediated blockade of IFN-β signaling:a basis for sporadic Parkinson disease dementia

المصدر: Magalhaes, J, Tresse, E, Ejlerskov, P, Hu, E, Liu, Y, Marin, A, Montalant, A, Satriano, L, Rundsten, C F, Carlsen, E M M, Rydbirk, R, Sharifi-Zarchi, A, Andersen, J B, Aznar, S, Brudek, T, Khodosevich, K, Prinz, M, Perrier, J F M, Sharma, M, Gasser, T & Issazadeh-Navikas, S 2021, ' PIAS2-mediated blockade of IFN-β signaling : a basis for sporadic Parkinson disease dementia ', Molecular Psychiatry, vol. 26, pp. 6083–6099 . https://doi.org/10.1038/s41380-021-01207-wTest
Molecular Psychiatry

مصطلحات موضوعية: 0301 basic medicine, Neurite, Diseases, medicine.disease_cause, Article, Mice, 03 medical and health sciences, Cellular and Molecular Neuroscience, 0302 clinical medicine, Mitophagy, mental disorders, medicine, Animals, Humans, Dementia, Molecular Biology, Mice, Knockout, Gene knockdown, business.industry, Dopaminergic Neurons, Parkinson Disease, Interferon-beta, medicine.disease, Protein Inhibitors of Activated STAT, Psychiatry and Mental health, 030104 developmental biology, Nerve Degeneration, alpha-Synuclein, Cancer research, Phosphorylation, Ectopic expression, Signal transduction, business, 030217 neurology & neurosurgery, Oxidative stress, Signal Transduction, Neuroscience

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b29a8e8c7d012f8dd270676bd23231b7Test
https://curis.ku.dk/portal/da/publications/pias2mediated-blockade-of-ifn-signalingTest(d386990f-e2ac-44da-a127-61b65c96e865).html

6

ADAM12 is a costimulatory molecule that determines Th1 cell fate and mediates tissue inflammation

المؤلفون: Marie Kveiborg, Mahdieh Hadi, Yawei Liu, Belinda Carrion, Shohreh Issazadeh-Navikas, Robert Bockermann, Iman Safari

المصدر: Liu, Y, Bockermann, R, Hadi, M, Safari, I, Carrion, B, Kveiborg, M & Issazadeh-Navikas, S 2021, ' ADAM12 is a costimulatory molecule that determines Th1 cell fate and mediates tissue inflammation ', Cellular & Molecular Immunology, vol. 18, pp. 1904-1919 . https://doi.org/10.1038/s41423-020-0486-8Test
Cellular and Molecular Immunology

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7a56e83362200b2478021a8b9b21fd33Test
https://curis.ku.dk/ws/files/279717262/ADAM12_is_a_costimulatory_molecule_that_determines_Th1_cell_fate_and_mediates_tissue_inflammation_publisher_version_.pdfTest

7

A mutation in

المؤلفون: Ensieh, Darbari, Davood, Zare-Abdollahi, Afagh, Alavi, Mozhgan, Rezaei Kanavi, Sepehr, Feizi, Seyed Bagher, Hosseini, Alireza, Baradaran-Rafii, Hamid, Ahmadieh, Shohreh, Issazadeh-Navikas, Elahe, Elahi

المصدر: Molecular vision. 26

مصطلحات موضوعية: Adult, Male, Base Sequence, Vesicular Transport Proteins, Genes, Recessive, Embryo, Mammalian, Pedigree, Consanguinity, Young Adult, Corneal Opacity, Gene Expression Regulation, Anterior Eye Segment, Mutation, Humans, Family, Female, Genetic Predisposition to Disease, Eye Abnormalities, Child

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=pmid________::d53ecf1b62c28d1eede7bf2ce463a22bTest
https://pubmed.ncbi.nlm.nih.gov/33273802Test

8

Antiviral, Immunomodulatory and Antiproliferative Activities of Recombinant Soluble IFNAR2 without IFN-ß Mediation

المؤلفون: Shohreh Issazadeh-Navikas, Isaac Hurtado-Guerrero, José Pavia, María Jesús Pinto-Medel, Patricia Urbaneja, Natalia Mena-Vázquez, A. Alonso, Begoña Oliver-Martos, José Alcamí, Esther Calonge, Oscar Fernández, Laura Leyva, Antonio Alcami, Pablo Aliaga, Jose Luis Rodriguez-Bada, Bruno Hernáez

المساهمون: Instituto de Salud Carlos III, Unión Europea. Fondo Europeo de Desarrollo Regional (FEDER/ERDF), Regional Government of Andalusia (España), Red Española de Esclerosis Múltiple, Junta de Andalucía, European Commission

المصدر: Journal of Clinical Medicine
Volume 9
Issue 4
Digital.CSIC. Repositorio Institucional del CSIC
instname
Dipòsit Digital de la UB
Universidad de Barcelona
Journal of Clinical Medicine, Vol 9, Iss 959, p 959 (2020)
Hurtado-guerrero, I, Hernáez, B, Pinto-medel, M J, Calonge, E, Rodriguez-bada, J L, Urbaneja, P, Alonso, A, Mena-vázquez, N, Aliaga, P, Issazadeh-navikas, S, Pavia, J, Leyva, L, Alcamí, J, Alcamí, A, Fernández, Ó & Oliver-martos, B 2020, ' Antiviral, Immunomodulatory and Antiproliferative Activities of Recombinant Soluble IFNAR2 without IFN-ß Mediation ', Journal of Clinical Medicine, vol. 9, no. 4, pp. 959 . https://doi.org/10.3390/jcm9040959Test
Repisalud
Instituto de Salud Carlos III (ISCIII)

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0674109f235fe62e9bedb2c4821b8dcbTest
https://hdl.handle.net/20.500.12105/9486Test

9

Identification of unique and shared mitochondrial DNA mutations in neurodegeneration and cancer by single-cell mitochondrial DNA structural variation sequencing (MitoSV-seq)

المؤلفون: Emilie Tresse, Joachim Weischenfeldt, Kirsten Grønbæk, Shohreh Issazadeh-Navikas, Elham Jaberi

المصدر: Jaberi, E, Tresse, E, Grønbæk, K, Weischenfeldt, J & Issazadeh-Navikas, S 2020, ' Identification of unique and shared mitochondrial DNA mutations in neurodegeneration and cancer by single-cell mitochondrial DNA structural variation sequencing (MitoSV-seq) ', EBioMedicine, vol. 57, 102868, pp. 1-17 . https://doi.org/10.1016/j.ebiom.2020.102868Test
EBioMedicine, Vol 57, Iss, Pp 102868-(2020)
EBioMedicine

مصطلحات موضوعية: Mitochondrial DNA, Research paper, lcsh:Medicine, Disease, Biology, DNA, Mitochondrial, Polymorphism, Single Nucleotide, General Biochemistry, Genetics and Molecular Biology, Structural variation, Mice, Neoplasms, medicine, Animals, Humans, media_common.cataloged_instance, Single-Cell Sequencing, European union, Neurodegeneration, media_common, Genetics, lcsh:R5-920, Point mutation, lcsh:R, High-Throughput Nucleotide Sequencing, Disease biomarker, Sequence Analysis, DNA, General Medicine, Neuron, medicine.disease, Heteroplasmy, Single cell sequencing, Genome, Mitochondrial, Genomic Structural Variation, Mutation, Nerve Degeneration, Single-Cell Analysis, lcsh:Medicine (General)

وصف الملف: application/pdf

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4dcdfb3950b32ff6118ffa396c794707Test
https://curis.ku.dk/ws/files/246674452/1_s2.0_S2352396420302437_main.pdfTest

10

Interferon-β-induced miR-1 alleviates toxic protein accumulation by controlling autophagy

المؤلفون: Shohreh Issazadeh-Navikas, Ava Handley, Roger Pocock, Pedro Moreira, Sandeep Gopal, Camilla Nehammer, Huikyong Lee, Leelee Ng, Patrick Ejlerskov, David C. Rubinsztein

المساهمون: Handley, Ava [0000-0003-1543-1551], Rubinsztein, David C [0000-0001-5002-5263], Pocock, Roger [0000-0002-5515-3608], Apollo - University of Cambridge Repository

المصدر: eLife, Vol 8 (2019)
eLife
Nehammer, C, Ejlerskov, P, Gopal, S, Handley, A, Ng, L, Moreira, P, Lee, H, Issazadeh-Navikas, S, Rubinsztein, D C & Pocock, R 2019, ' Interferon-beta-induced miR-1 alleviates toxic protein accumulation by controlling autophagy ', eLife, vol. 8 . https://doi.org/10.7554/eLife.49930Test

مصطلحات موضوعية: 0301 basic medicine, medicine.medical_treatment, Protein aggregation, Mice, 0302 clinical medicine, Interferon, genetics, mammalian cells, Biology (General), 3' Untranslated Regions, Caenorhabditis elegans, Huntingtin Protein, biology, microRNA, Chemistry, General Neuroscience, GTPase-Activating Proteins, General Medicine, 3. Good health, Cell biology, Cytokine, C. elegans, Medicine, Research Article, medicine.drug, autophagy, QH301-705.5, Science, Chemical biology, chemical biology, General Biochemistry, Genetics and Molecular Biology, protein aggregation, Protein Aggregates, 03 medical and health sciences, Biochemistry and Chemical Biology, genomics, medicine, biochemistry, Animals, Humans, RNA, Messenger, Caenorhabditis elegans Proteins, Base Sequence, General Immunology and Microbiology, human cells, Autophagy, rab7 GTP-Binding Proteins, Genetics and Genomics, Interferon-beta, biology.organism_classification, MicroRNAs, 030104 developmental biology, rab GTP-Binding Proteins, Mutant Proteins, Rab, Peptides, 030217 neurology & neurosurgery, HeLa Cells

وصف الملف: application/pdf; application/zip; text/xml

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::34810bf881501a7c32975e5884fc3cf7Test
https://elifesciences.org/articles/49930Test

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