دورية أكاديمية

HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia

التفاصيل البيبلوغرافية
العنوان: HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia
المؤلفون: de Barrios O., Galaras A., Trincado J.L., Azagra A., Collazo O., Meler A., Agraz-Doblas A., Bueno C., Ballerini P., Cazzaniga G., Stam R.W., Varela I., De Lorenzo P., Valsecchi M.G., Hatzis P., Menéndez P., Parra M.
المصدر: Leukemia ; https://www.scopus.com/inward/record.uri?eid=2-s2.0-85096982699&doi=10.1038%2fs41375-020-01097-x&partnerID=40&md5=51e1818bf18e73dded40f6215305b372Test
سنة النشر: 2021
المجموعة: University of Thessaly Institutional Repository / Ιδρυματικό Αποθετήριο Πανεπιστημίου Θεσσαλίας
مصطلحات موضوعية: doxycycline, histone deacetylase 7, HDAC7 protein, human, histone deacetylase, mixed lineage leukemia protein, acute lymphoblastic leukemia, Article, B lymphocyte differentiation, bioinformatics, cancer prognosis, cancer survival, CCL5 gene, CD86 gene, chromosome translocation 11, chromosome translocation 4, clinical article, clinical outcome, cohort analysis, controlled study, disease association, gene, gene expression, gene expression level, gene ontology, gene overexpression, genetic analysis, HDAC7 gene, human cell, infant
الوصف: [No abstract available]
نوع الوثيقة: article in journal/newspaper
اللغة: English
تدمد: 08876924
العلاقة: http://hdl.handle.net/11615/73126Test
DOI: 10.1038/s41375-020-01097-x
الإتاحة: https://doi.org/10.1038/s41375-020-01097-xTest
http://hdl.handle.net/11615/73126Test
رقم الانضمام: edsbas.FB1AA029
قاعدة البيانات: BASE
الوصف
تدمد:08876924
DOI:10.1038/s41375-020-01097-x