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1دورية أكاديمية
المؤلفون: Bruno Cesar Costa Soares, Hnin Ei Ei Khine, Boonchoo Sritularak, Pithi Chanvorachote, Rosa Alduina, Rungroch Sungthong, Chatchai Chaotham
المصدر: Frontiers in Pharmacology, Vol 15 (2024)
مصطلحات موضوعية: lung cancer, ROS, apoptosis, mitochondrial outer membrane permeabilization, DNA damage, Cymensifin A, Therapeutics. Pharmacology, RM1-950
وصف الملف: electronic resource
العلاقة: https://www.frontiersin.org/articles/10.3389/fphar.2024.1361085/fullTest; https://doaj.org/toc/1663-9812Test
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2دورية أكاديمية
المصدر: BMC Research Notes, Vol 14, Iss 1, Pp 1-7 (2021)
مصطلحات موضوعية: BAX, Intrinsic apoptosis, Mitochondrial dysfunction, Mitochondrial outer membrane permeabilization, Mitochondrial fragmentation, Elamipretide (SS31), Medicine, Biology (General), QH301-705.5, Science (General), Q1-390
وصف الملف: electronic resource
العلاقة: https://doaj.org/toc/1756-0500Test
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3دورية أكاديمية
المؤلفون: Xiaoke Chi, Dang Nguyen, James M Pemberton, Elizabeth J Osterlund, Qian Liu, Hetal Brahmbhatt, Zhi Zhang, Jialing Lin, Brian Leber, David W Andrews
المصدر: eLife, Vol 9 (2020)
مصطلحات موضوعية: primary murine neurons, MEF cells, BMK cells, apoptosis, Bcl-2 family proteins, mitochondrial outer membrane permeabilization, Medicine, Science, Biology (General), QH301-705.5
وصف الملف: electronic resource
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4دورية أكاديمية
المؤلفون: Belizário, J.E., Alves, J., Occhiucci, J.M., Garay-Malpartida, M., Sesso, A.
المصدر: Brazilian Journal of Medical and Biological Research. August 2007 40(8)
مصطلحات موضوعية: Mitochondrial outer membrane permeabilization, Permeability transition pore, Cytochrome c, Reactive oxygen species, Caspases, BCL-2
وصف الملف: text/html
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5دورية أكاديمية
المؤلفون: Deng, Jing
المصدر: Deng, Jing. 2017. “How to unleash mitochondrial apoptotic blockades to kill cancers?” Acta Pharmaceutica Sinica. B 7 (1): 18-26. doi:10.1016/j.apsb.2016.08.005. http://dx.doi.org/10.1016/j.apsb.2016.08.005Test.
مصطلحات موضوعية: ASH, American Society of Hematology, ATAP, amphipathic tail-anchoring peptide, BAD, BCL-2-associated death promoter protein, BAK, BCL-2 homologous antagonist killer, BAX, BCL-2-associated X protein, BCL-2, B-cell lymphoma 2, BCL-w (BCL2L2), BCL-2-like protein 2, BCL-xL, B-cell lymphoma X long, BFL-1 (BCL2A1), BCL-2-related protein A1, BCR, B-cell receptor, BH3, BCL-2 homology 3, BID, BH3 interacting domain death agonist, BIK, BCL-2-interacting killer, BIM, BCL-2-interacting mediator of cell death, BOK, BCL-2 related ovarian killer, BTK, Bruton׳s tyrosine kinase, CDK, cyclin-dependent kinase, CLL, chronic lymphocytic leukemia, CHOP, cyclophosphamide, hydroxydaunorubicin, oncovin-vincristine and prednisone, CML, chronic myelogenous leukemia, CR, complete response, EGFR, epidermal growth factor receptor, ER, endoplasmic reticulum, ERK, extracellular signal-regulated kinase, FDA, U. S. Food and Drug Administration, GSK-3, glycogen synthase kinase-3, ITK, interleukin-2-inducible T-cell kinase, MCL, myeloid cell leukemia, MOMP, mitochondrial outer membrane permeabilization, NHL, non-Hodgkin lymphoma, NIH, National Institutes of Health, NSCLC, non-small cell lung cancer, PI3K, phosphatidylinositol-3-kinase, PUMA, p53 up-regulated modulator of apoptosis, SLL, small lymphocytic lymphoma, T-ALL, T-acute lymphocytic leukemia, Apoptosis, BCL-2 family, Mitochondrial priming, BH3 profiling, Targeted therapy, Combination therapy
العلاقة: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5237704/pdfTest/; Acta Pharmaceutica Sinica. B