نتائج البحث - "Mark Aronovitz"

1

Endothelial Pannexin 1 Regulates Cardiac Response to Myocardial Infarction

المؤلفون: Philip J. Hall, Colleen K. Duffy, Scott R. Johnstone, Brant E. Isakson, Gregory L. Martin, Manxiu Ma, Mark Aronovitz, Robert M. Blanton, Norbert Leitinger, Matthew J. Wolf, Alex P. Young, Miranda E. Good, Abigail G. Wolpe

المصدر: Circ Res

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c9eb3728064f6d7b4c243a075b6c96aTest
https://doi.org/10.1161/circresaha.120.317272Test

2

Abstract 116: T Cells Contribute To Diastolic Dysfunction And Cardiac Hypertrophy In A Preclinical Model Of Heart Failure With Preserved Ejection Fraction

المؤلفون: Marina Anastasiou, Sasha Smolgovsky, Abraham L Bayer, Robert M. Blanton, Francisco J. Carrillo-Salinas, Kuljeet Kaur, Mark Aronovitz, Pilar Alcaide

المصدر: Circulation Research. 129

مصطلحات موضوعية: medicine.medical_specialty, Physiology, business.industry, Internal medicine, Cardiac hypertrophy, Cardiology, medicine, Diastole, Cardiology and Cardiovascular Medicine, Heart failure with preserved ejection fraction, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::b67c1d105dfa4bec8afb2cc9abb58d1fTest
https://doi.org/10.1161/res.129.suppl_1.116Test

3

Mineralocorticoid Receptor in Smooth Muscle Contributes to Pressure Overload-Induced Heart Failure

المؤلفون: Mark Aronovitz, Francisco J. Carrillo-Salinas, Ane M. Salvador, M. Elizabeth Moss, Joshua J. Man, Iris Z. Jaffe, Pilar Alcaide, Seung Kyum Kim, Gregory L. Martin, Lauren A Biwer

المصدر: Circ Heart Fail

مصطلحات موضوعية: medicine.medical_specialty, Myocytes, Smooth Muscle, Cardiomegaly, Constriction, Pathologic, 030204 cardiovascular system & hematology, Muscle, Smooth, Vascular, Article, Muscle hypertrophy, Constriction, 03 medical and health sciences, Gene Knockout Techniques, Mice, 0302 clinical medicine, Mineralocorticoid receptor, Smooth muscle, Internal medicine, medicine, Animals, Arterial Pressure, Myocytes, Cardiac, Aorta, 030304 developmental biology, Pressure overload, Heart Failure, 0303 health sciences, Ventricular Remodeling, business.industry, medicine.disease, Phenotype, Endothelial stem cell, Disease Models, Animal, Endocrinology, Receptors, Mineralocorticoid, Echocardiography, Heart failure, cardiovascular system, Cardiology and Cardiovascular Medicine, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f68c7b3b631d8b3c24123ff531069835Test
https://pubmed.ncbi.nlm.nih.gov/33517669Test

4

Isolevuglandin-Modified Cardiac Proteins Drive CD4+ T-Cell Activation in the Heart and Promote Cardiac Dysfunction

المؤلفون: Njabulo Ngwenyama, Ane M. Salvador, Francisco Velazquez, Mark Aronovitz, Pilar Alcaide, Annet Kirabo, David G. Harrison, Venkataraman Amarnath, Tania Nevers, Albert K. Tai, Robert M. Blanton, Francisco J. Carrillo-Salinas

المصدر: Circulation

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5e0815366a3235eb7d10b7159fc9f1dTest
https://pubmed.ncbi.nlm.nih.gov/33750212Test

5

Bone Morphogenetic Protein 9 Reduces Cardiac Fibrosis and Improves Cardiac Function in Heart Failure

المؤلفون: Navin K. Kapur, Richard H. Karas, Vikram Paruchuri, Mark Aronovitz, Peter Natov, Kevin J. Morine, Xiaoying Qiao, Sam York, Yali Zhang

المصدر: Circulation. 138:513-526

مصطلحات موضوعية: 0301 basic medicine, Cardiac function curve, medicine.medical_specialty, Cardiac fibrosis, GDF2, Haploinsufficiency, 030204 cardiovascular system & hematology, Article, Ventricular Function, Left, Smad1 Protein, 03 medical and health sciences, 0302 clinical medicine, Fibrosis, Physiology (medical), Internal medicine, Growth Differentiation Factor 2, medicine, Animals, Humans, Smad3 Protein, Phosphorylation, Heart Failure, Mice, Knockout, Ventricular Remodeling, biology, business.industry, Myocardium, Endoglin, Recovery of Function, Transforming growth factor beta, Fibroblasts, medicine.disease, Growth Differentiation Factors, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Heart failure, Bone Morphogenetic Protein 9, Cardiology, biology.protein, Cardiology and Cardiovascular Medicine, business, Signal Transduction

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13decba3dde67dd198b3405ed50f1754Test
https://doi.org/10.1161/circulationaha.117.031635Test

6

Abstract 113: Recombinant Tafazzin Enzyme Replacement Therapy Rescues Metabolic and Functional Defects in a Mouse Model of Barth Syndrome

المؤلفون: Robert M. Blanton, Gregory Martin, Mark Aronovitz, Lauren Richey, Wei-Ming Chien, Corinne J Thomas, Kelly Tam, Michael T. Chin, Douglas Strathdee, Junya Awata, Ana Dinca

المصدر: Circulation Research. 125

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_________::810003d8c1726858ddd487996b6cd3a0Test
https://doi.org/10.1161/res.125.suppl_1.113Test

7

Th1 effector T cells selectively orchestrate cardiac fibrosis in nonischemic heart failure

المؤلفون: Mark Aronovitz, Pilar Alcaide, Ane M. Salvador, Tania Nevers, Njabulo Ngwenyama, Francisco Velazquez, Robert M. Blanton, Francisco J. Carrillo-Salinas

المصدر: The Journal of Experimental Medicine

مصطلحات موضوعية: 0301 basic medicine, medicine.medical_specialty, Adoptive cell transfer, Cardiac fibrosis, Integrin alpha4, Receptors, Antigen, T-Cell, alpha-beta, T cell, Immunology, Context (language use), 030204 cardiovascular system & hematology, Article, Interferon-gamma, 03 medical and health sciences, 0302 clinical medicine, Antigen, Transforming Growth Factor beta, Fibrosis, Internal medicine, Cell Adhesion, medicine, Animals, Immunology and Allergy, Myofibroblasts, Research Articles, Heart Failure, Mice, Knockout, biology, business.industry, Myocardium, Transforming growth factor beta, Fibroblasts, Th1 Cells, Flow Cytometry, medicine.disease, Mice, Inbred C57BL, 030104 developmental biology, Endocrinology, medicine.anatomical_structure, Microscopy, Fluorescence, Heart failure, cardiovascular system, Cancer research, biology.protein, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba21b0510064670d912794b4f563053eTest
https://doi.org/10.1084/jem.20161791Test

8

Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model

المؤلفون: Timothy D. Calamaras, Robert M. Blanton, Jennifer J. DuPont, Mark Aronovitz, Joseph C. McCarthy, Iris Z. Jaffe

المصدر: JACC: Basic to Translational Science
JACC: Basic to Translational Science, Vol 2, Iss 3, Pp 285-296 (2017)

مصطلحات موضوعية: 0301 basic medicine, lcsh:Diseases of the circulatory (Cardiovascular) system, medicine.medical_specialty, Decreased potassium, heart failure, 030204 cardiovascular system & hematology, HF, heart failure, vascular function, Constriction, PRECLINICAL RESEARCH, 03 medical and health sciences, 0302 clinical medicine, L-NAME, L-NG-nitroarginine methyl ester, Serelaxin, Fibrosis, Internal medicine, medicine, In patient, relaxin, LV, left ventricular, Relaxin, business.industry, AngII, angiotensin II, SBP, systolic blood pressure, medicine.disease, Angiotensin II, 030104 developmental biology, lcsh:RC666-701, Heart failure, cardiovascular system, Cardiology, Cardiology and Cardiovascular Medicine, business

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4d69ca7640cf8b7063853a556a7f6a5dTest
https://doi.org/10.1016/j.jacbts.2017.03.011Test

9

Targeted disruption of glycogen synthase kinase-3β in cardiomyocytes attenuates cardiac parasympathetic dysfunction in type 1 diabetic Akita mice

المؤلفون: Marian Haburcak, Bo Wang, Jonas B. Galper, Yali Zhang, Sami F. Noujaim, Ho-Jin Park, Mark Aronovitz, Robert M. Blanton, Charles M. Welzig, Thomas Force

المصدر: PLoS ONE, Vol 14, Iss 4, p e0215213 (2019)

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3334bc8f6079c266e9f58be1fe5e8753Test
https://pubmed.ncbi.nlm.nih.gov/30978208Test

10

Gut dysbiosis induced by cardiac pressure overload enhances adverse cardiac remodeling in a T cell-dependent manner

المؤلفون: Albert K. Tai, Pilar Alcaide, David Jetton, Marina Anastasiou, Njabulo Ngwenyama, Mark Aronovitz, Francisco J. Carrillo-Salinas, Sasha Smolgovsky, Kuljeet Kaur

المصدر: Gut Microbes, Vol 12, Iss 1 (2020)
Gut Microbes
article-version (VoR) Version of Record

مصطلحات موضوعية: Male, 0301 basic medicine, Cardiac fibrosis, T-Lymphocytes, heart failure, Gut flora, Lymphocyte Activation, Mice, 0302 clinical medicine, Basic Helix-Loop-Helix Transcription Factors, t cells, Ventricular Remodeling, biology, aryl hydrocarbon receptor, Tryptophan, Gastroenterology, Endomyocardial Fibrosis, Infectious Diseases, medicine.anatomical_structure, cardiovascular system, Hypertrophy, Left Ventricular, 030211 gastroenterology & hepatology, medicine.symptom, Research Article, Research Paper, Microbiology (medical), medicine.medical_specialty, T cell, Inflammation, Microbiology, Lymphocyte Depletion, 03 medical and health sciences, Immune system, Internal medicine, Ventricular Pressure, medicine, Animals, lcsh:RC799-869, Pressure overload, Hypertrophy, Right Ventricular, gut microbiota, Fatty Acids, Volatile, medicine.disease, biology.organism_classification, Aryl hydrocarbon receptor, Gastrointestinal Microbiome, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Endocrinology, Receptors, Aryl Hydrocarbon, inflammation, Heart failure, biology.protein, Dysbiosis, lcsh:Diseases of the digestive system. Gastroenterology

الوصول الحر: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::106b679d2ae4e183c4c188987b675982Test
https://doi.org/10.1080/19490976.2020.1823801Test

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